Does seeing an infarct in the medulla cause your heart to skip a beat?
Does medullary anatomy send you into respiratory arrest?
Never fear, here is a thread on the major medullary syndromes!
2/The medulla is like a toll booth.
Everything going down into the cord must pass through the medulla & everything from the cord going back up to the brain must too.
That’s a lot of tracts for a very small territory. Luckily you don’t need to know every tract
3/Medulla has 4 main vascular territories, spread out like a fan:
Anteromedial, anterolateral, lateral, and posterior.
You don’t need to remember their names, just the territory they cover—and I’ll show you how
4/The anterior territory is fed by the anterior spinal artery.
As a result, you can commonly get bilateral anteromedial infarcts as a result of the fact that the anterior spinal artery is unpaired.
5/This results in a classic appearance for a medial medullary infarct.
Some say it looks like ear buds, while others say it looks like a heart.
But if you see either—that’s a medial medullary infarct
6/Anterolateral territory has more variable anatomy—being fed by a combination of feeders from the anterior spinal artery, vertebral perforators, and branches of the PICA.
Different articles will say different vessels are dominant—which means NONE of them really are.
7/Because it is fed by combinations of different vessels, it is relatively uncommon to see isolated infarcts in this territory because the different vessels can collateralize for each other
8/Finally, both the lateral and posterior territories are both fed by branches of the PICA.
So if there is a PICA infarct, they go out together
9/Infarcts of both of these territories give us what I call the classic “bruised cheek” appearance of the medulla
It looks like the little chubby cheek of the medulla has been in hit with lateral/posterior PICA medullary infarcts. (often just called lateral medullary infarcts)
10/I think the vascular territories of the medulla look like a praying mantis face
The antennae is hypoglossal nerve that comes out it the “head” here.
This face will help you remember the medullary syndromes associated with medial & lateral medullary infarcts.
11/So what are the main deficits associated with medial medullary infarcts?
Well, it will hit the pyramids (weakness)
It will affect the medial lemniscus (sensory)
And the exiting hypoglossal nerves
12/So let’s look at what happens if we take out the medial part of our praying mantis:
You take out the antennae which are medial (hypoglossal)
And you take out the little arms that are always medial under his chin (so motor) as well as their little feelers (sensory).
13/Now let’s talk lateral medullary syndrome. It takes out both the lateral & posterior territories supplied by the PICA
It is complex, but the main tracts involved are the spinothalamic, sympathetic tracts, spinal trigeminal, and CN 9 & 10.
How to remember these?
14/I remember the tracts bc lateral medullary syndrome is a result of an injury to the lateral aspect or SIDE of the medulla—a Side Trauma
And the tracts involved start w/S & T just like Side Trauma
15/While the spinal trigeminal & sympathetic tracts are ipsilateral, the spinothalamic tract is contralateral.
I remember that it crosses b/c it has an O in the middle for OPPOSITE side
I call it the SPINE to OPPOSITE THALAMUS tract
16/To remember what is involved in lateral medullary syndrome—look what is involved on our praying mantis face:
his far lateral eyes = horner’s from sympathetics
his face = spinal trigeminal
his jowls/chewing mechanisms = dysphagia from CN 9 & 10 dysfunction
17/And then, remember, the lateral/posterior medullary infarct looks like a punch to the face—so that burns!
That can help you to remember spinothalamic pain & temperature sensation loss
Other tracts are involved as well, but these main ones will help you get to the diagnosis!
18/So now you know the arterial territories of the medulla & how to remember the associated syndromes
So next time you have a medullary infarct, just remember the praying mantis face & you won’t have to bug anyone to help you!
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2/Vessel injury can be from direct penetration, but most commonly is from head/neck movement during trauma causing vessel stretching/twisting while the rest of the vessel is fixed.
Just like how your arm breaks if it’s twisted or pulled while the rest of you is fixed!
3/As a result, the most common sites of injury occur at a junction between where the vessel is mobile & where it is fixed (skullbase, transverse foramen).
At C2-3, the carotid runs over the transverse foramen of C2, essentially fixing it at this point as well.
1/Hate it when one radiologist called the stenosis mild, the next one said moderate--but it was unchanged?!
How do you grade it? Do you estimate? Measure? Guess???
Let this thread take the load off your back w/a lumbar grading system that’s easy, reproducible & evidence-based!
2/Lumbar stenosis has always been controversial.
In 2012, they tried to survey spine experts to come to a consensus as to what are the most important criteria for canal & foraminal stenosis.
And the consensus was…that there was no consensus.
So what should you use to call it?
3/Well, you don’t want just gestalt it—that is a recipe for inconsistency & disagreement.
But you don’t want to measure everything either—measurements are not only cumbersome, they introduce reader variability & absolute measurements don’t mean the same thing in every patient.
1/Asking “How old are you?” can be dicey—both in real life & on MRI!
Do you know how to tell the age of blood on MRI?
Here’s a thread on how to date blood on MRI!
Read this so next time you see a hemorrhage, your guess on when it happened will always be in the right vein!
2/If you ask someone how to date blood on MRI, they’ll spit out a crazy mnemonic about babies that tells you what signal blood should be on T1 & T2 imaging by age.
But mnemonics are crutch—they help you memorize, but not understand
If you understand, you don’t need to memorize
3/If you look at the mnemonic, you will notice one thing—the T1 signal is all you need to tell if blood is acute, subacute or chronic.
T2 signal will tell if it is early or late in each of those time periods—but that type of detail isn’t needed in real life
Has measuring for Chiari 1 malformations become a real headache?
Can’t quite peg when to call a Chiari? Looking for a better way?
Let the new SCANtastic decompress it for you w/new @theAJNR Chiari research: ajnr.org/content/early/…
@TheAJNR 2/Most know that a Chiari 1 malformation (CM) is when the tonsils extend below the foramen magnum (FM).
Classically, it’s bc the posterior fossa is underdeveloped & small, while the cerebellum is normal sized, so it doesn’t fit in the posterior fossa & herniates down.
@TheAJNR 3/Most call tonsils 5mm below the FM as a CM.
This is from a paper looking at normal & symptomatic CMs.
Tonsils are like people—they come in all different shapes & sizes.
CMs were always >2 mm below the FM & normals were never >5mm below.