Ten cognitive check points (general rules or heuristics to help guide my practice) I use in the ICU to help make sure I don't get too far off track. A thread🧵👇
1/10: Shock with a narrow pulse pressure (<30mmHg) needs an urgent #POCUS (or echo) to identify occult RV/LV failure, obstructive shock, or severe hypovolemia.
2/10: When you see a pt. who is "persistently hypovolemic" that transiently responds to fluid loading. First, ensure not bleeding.🩸 Second, instead of assuming they are just really dry, think about dynamic LVOTo, intracavitary gradients, or diastolic dysfunction. Giving them 15L of fluid to treat persistent fluid responsiveness is likely not the right move.
3/10: If a nurse (or anyone!) asks you the same question twice (e.g. should we consider getting a CT scan) don't take it that your judgement is being question - this is often a great indicator they have a different mental model about your pt. Explore the why and challenge your own assumptions.
4/10: The diagnosis of "septic shock" does mean the patient has distributive shock. 🫀❌ Even if it is pure septic shock, patients may have any phenotype of shock depending on their chronic comorbidities, acute cardiomyopathy, or secondary processes (core pulmonale, pericardial effusions etc.)
5/10: A persistently elevated lactate in a septic pt does NOT mean they are under-resuscitated or "dry" Step 1) do you have source control Step 2) Are you treating the right phenotype of shock? Step 3) Can we optimize real-time markers of perfusion (e.g. cap refill time) through volume, inotropes, MAP challenge, inodilators etc.
6/10: Be wary of admitting a patient to the ICU with a diagnosis of sepsis NYD (not yet diagnosed)- in the ICU, we should be able to figure out the D (diagnosis). Repeat the primary history, re-examine, consider pan-CT, echo, focused TEE etc. Think about where infection can hide! (valves, belly, heart etc.)
7/10: There is no such thing as a 'stable' post cardiac arrest patient. They just died. They are by definition the patient closest to death in your department/hospital. The disease that caused them to die is still at play, time matters! 🫀⌛️Prioritize diagnosis, ensuring adequate access (Art + venous), and definitive treatment.
8/10: Post cardiac arrest pts. are simultaneously a 1) cardiac patient (their heart stopped) 2) neuro patient (we care about their brain) 3) and trauma patient (someone just crushed their chest for like 30 min).
If you are CT scanning post arrest, low threshold to PAN scan (head, chest, abdo)
Also, if you are going to order a CT chest, consider a PE protocol so you never have to discuss on rounds whether the patient could have an occult PE as the etiology arrest!!! 🫀🧠🦴
9/10: If you are using POCUS to justify giving more fluids, you are probably doing it wrong.
10/10: If a pt or family says they want everything done - clarify this!!!
When you dive into it, it often means not giving up on them. They want you to try to diagnosis them, correct treatable causes, and if they are dying, provide them a comfortable and dignified death.
Rarely is it life at all cost or indefinite prolongation of dying with life support.
These are just a few observations from a very early career intensivist! Would love to hear everyone else's heuristics they use in their practice. @emily_fri @nickmmark @emcrit @PulmCrit @iceman_ex @IM_Crit_ @ThinkingCC @AndromedaShock @ChrisCarrollMD @precordialthump @Wilkinsonjonny @jon_silversides @Bram_Rochwerg @KimLewisMD
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(1/9) Here’s how to become the best doctor you can in 2025…
Some advice (e.g. learn from your pts) is timeless but some thing are different than when Osler trained.
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(2/9) Learn from your patients
Learning ~= cases seen × learning extracted per case
Maximizing both is key.
Volume exposes you to varied presentations, and reflection deepens your understanding.
There’s no substitute for either. Perhaps in the coming years AI simulated presentations may assist in pattern recognition (e.g. high exposure to simulated pathology) but not quite there yet.
(3/9) Develop skills beyond knowledge
Knowledge matters, but communication, listening, problem-solving, studying, and teamwork matter more in practice.
When trainees struggle, it’s often these skills, not medical knowledge, that hold them back.
(1/x) In fellowship, I managed a peri-arrest patient in the middle of the night who changed my understanding and appreciation for hemodynamics, ultrasound, and TEE.
I've seen similar cases dozens of times now, yet this commonly gets missed, even at top institutions worldwide.
A 🧵
(2/x) When I met this pt. they had a HR of 170 and a blood pressure on arterial line of 50/30. They were mottled head to toe, ashen, and looked like they were seconds away from arresting.
They had a recent NSTEMI and my first thought was cardiogenic shock or a mechanical complication of their MI --> VSD, tamponade, free wall rupture etc.
No transthoracic views on echo.
(3/x) I started the usually therapy, phenylephrine pushes, levophed, vaso pushes, and epi infusion.
No response.
I started bolusing aliquots of 20-50mcg of epinephrine ... no response. We intubated while this was going on.
(0/x) How do I approach the first 2 minutes of meeting a new critically ill pt?
My approach has been forged from my learnings as a paramedic.
Here's my approach as an ICU doc 🧵
(1/x) I start with a scene survey.
I want to understand who's in the room and what are their roles. Is someone currently leading the resuscitation? What monitors are on the patient? What drugs are hooked up to the patient?
In EMS, one of the first things we are taught is to not rush to the patient.
This is for your own safety in case there's a hazard, but also because you can learn so much about what's going on by observing your environment as you approach the patient.
This gets missed in the hospitals.
Take some time and even by understanding what pumps they're on, you can often learn a lot about what might be going on in this very moment. You can also learn what help you might have if 💩🪭
(2/x) Next, I start perform a very quick (<15 seconds) scan of the patient
Essentially quickly checking the ABCs
This involves:
Reviewing the monitor for current vitals
Looking at the patient's skin colour
Looking for mottling
Looking at the Work of Breathing
Looking for LOC
Approaching the patient and briefly feeling their feet or hands for temperature and pulse characteristics
My goal here is to determine whether we need to go straight into resuscitation within seconds (cardiac arrest) or peri-arrest, or whether I can take a bit more detailed handover from the team that's already caring for the patient.
(0/x) For the past 2 years I have worked in our chronic ICU helping wean difficult to wean patients with neurological weakness.
Here are some of the things I’ve learned from my vastly expert multi-disciplinary team 🧵
Really these are just great practices for weaning ALL ICU pts. off ventilators...
(1/x) Deeming a patient impossible to wean should only be done by groups of clinicians with extensive experience in this.
In fact, evidence suggests that physicians are often poor at predicting weaning failure.
We've had patients with neuromuscular weakness who have been ventilated for close to a year or longer who have successfully weaned entirely from mechanical ventilation with thoughtful, deliberate weaning strategies.
(2/x) Optimizing volume status is crucial to weaning patients.
For neuromuscular weakness patients, even having 500 mL of pleural effusion adds a pound of weight onto each diaphragm.
When multiplied over the thousands of breaths per day, this significantly contributes to weaning failure.
Optimize your patient's volume status to help them wean!
(1/9) Its July and medical trainees are starting their medical school residency or fellowship.
Here is my advice for any ICU fellows starting out to avoid making some of the same mistakes that I've made in the past.
A 🧵
Tip 1: Just go see the patient.
When somebody is calling the intensive care unit in the hospital, they are calling for help. We are the 911 for hospitalized patients.
Even if there may not be a clear ICU indication based on your initial phone call with the referring physician or nurse, just go see the patient.
Often I find that we can still improve patient care for those patients even if they don't need vasopressors, ventilator, or inotropes. Often they are quite sick, and our expertise managing sick patients can be very useful.
In 2025, we need to move away from the restricted notion that the intensive care unit expertise only pertains to ventilators, vasopressors, and inotropes.
We are experts in managing acutely unwell patients even before they require ICU, so we should lend our expertise to help patients anywhere in the hospital.
Tip 2: When you first meet a patient or family, listen more than you speak.
Ask the patient and family lots of questions about their values, functions, and what's important to them in their life.
Try to get to know them as a person before they became into the ICU.
Taking time to ask the patient and family these questions up front will actually answer a lot of questions for you down the road and save time in conversations.
It also builds rapport, shows that you are committed to their loved one, and will often get you useful clinical information that helps solve any diagnostic puzzles you have
Avoid launching into a medical update within the first 5 minutes of meeting a family.
Often they are not yet primed to hear this information, and spending a bit of time building rapport ahead of time is invaluable.
(0/x) Do you want to be the Obi-Wan of diuresis? (or Darth Vader if you tilt to darkness)
Are you still starting diuresis with 40mg of 80mg of IV lasix?
Below are the lessons I learned from @ArgaizR and @FH_Verbrugge on our most recent HCProunds as they show us how to wield the ‘force’ to make sodium disappear from the body 👇
(1/x) If you missed the webinar, check it out here 👇- it is one of our best.
First, we need to separate a patient's volume status from their electrolytes.
Contraction alkalosis is a misnomer because, yes, when you diuresis patients with Lasix monotherapy, they become alkalotic and their volume status contracts, but this is not a cause-and-effect relationship.
The contraction of the volume status is not what's driving the alkalosis.
If you use multi-modal diuresis, e.g., integrating acetazolamide early on, you will find that your patient's bicarb will not increase, and then you can assess your volume status clinically based on physical exam and potentially ultrasound to assess for congestion on the right or left side.
(2/x) Second: we should start acetazolamide early when we diurese patients.
This prevents alkalosis that may limit diuresis and acetazolamide is CHLORIDE SPARING.
Why does this matter?
Hypochloremia is a potent activator of diuretic resistance. Acetazolamide might attenuate this.