Ten cognitive check points (general rules or heuristics to help guide my practice) I use in the ICU to help make sure I don't get too far off track. A thread🧵👇
1/10: Shock with a narrow pulse pressure (<30mmHg) needs an urgent #POCUS (or echo) to identify occult RV/LV failure, obstructive shock, or severe hypovolemia.
2/10: When you see a pt. who is "persistently hypovolemic" that transiently responds to fluid loading. First, ensure not bleeding.🩸 Second, instead of assuming they are just really dry, think about dynamic LVOTo, intracavitary gradients, or diastolic dysfunction. Giving them 15L of fluid to treat persistent fluid responsiveness is likely not the right move.
3/10: If a nurse (or anyone!) asks you the same question twice (e.g. should we consider getting a CT scan) don't take it that your judgement is being question - this is often a great indicator they have a different mental model about your pt. Explore the why and challenge your own assumptions.
4/10: The diagnosis of "septic shock" does mean the patient has distributive shock. 🫀❌ Even if it is pure septic shock, patients may have any phenotype of shock depending on their chronic comorbidities, acute cardiomyopathy, or secondary processes (core pulmonale, pericardial effusions etc.)
5/10: A persistently elevated lactate in a septic pt does NOT mean they are under-resuscitated or "dry" Step 1) do you have source control Step 2) Are you treating the right phenotype of shock? Step 3) Can we optimize real-time markers of perfusion (e.g. cap refill time) through volume, inotropes, MAP challenge, inodilators etc.
6/10: Be wary of admitting a patient to the ICU with a diagnosis of sepsis NYD (not yet diagnosed)- in the ICU, we should be able to figure out the D (diagnosis). Repeat the primary history, re-examine, consider pan-CT, echo, focused TEE etc. Think about where infection can hide! (valves, belly, heart etc.)
7/10: There is no such thing as a 'stable' post cardiac arrest patient. They just died. They are by definition the patient closest to death in your department/hospital. The disease that caused them to die is still at play, time matters! 🫀⌛️Prioritize diagnosis, ensuring adequate access (Art + venous), and definitive treatment.
8/10: Post cardiac arrest pts. are simultaneously a 1) cardiac patient (their heart stopped) 2) neuro patient (we care about their brain) 3) and trauma patient (someone just crushed their chest for like 30 min).
If you are CT scanning post arrest, low threshold to PAN scan (head, chest, abdo)
Also, if you are going to order a CT chest, consider a PE protocol so you never have to discuss on rounds whether the patient could have an occult PE as the etiology arrest!!! 🫀🧠🦴
9/10: If you are using POCUS to justify giving more fluids, you are probably doing it wrong.
10/10: If a pt or family says they want everything done - clarify this!!!
When you dive into it, it often means not giving up on them. They want you to try to diagnosis them, correct treatable causes, and if they are dying, provide them a comfortable and dignified death.
Rarely is it life at all cost or indefinite prolongation of dying with life support.
These are just a few observations from a very early career intensivist! Would love to hear everyone else's heuristics they use in their practice. @emily_fri @nickmmark @emcrit @PulmCrit @iceman_ex @IM_Crit_ @ThinkingCC @AndromedaShock @ChrisCarrollMD @precordialthump @Wilkinsonjonny @jon_silversides @Bram_Rochwerg @KimLewisMD
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(1/10) Pericardial tamponade is a clinical diagnosis supported by #echo (2D + quantitative). A 🧵of 10 confirmed tamponade cases (by drainage!) starting with obvious and ending with subtle ones that you could easily miss.. some PEARLS along the way 👇#medtwitter #foamed
(2/10) The swinging heart sign is a strong indicator the a pericardial effusion is hemodynamically important. It also gives the ECG correlate of electrical alternans. #cardiotwitter #echofirst #ecg
(3/10) In this parasternal long axis, there is collapse of the RV free wall. Sometimes described as a person jumping on a trampoline. Remember, tamponade is ultimately a clinical diagnosis supported by echo findings, but its hard to argue with findings like this. #pocus #foamcc
Many of us use #POCUS or #resusTEE during cardiac arrest to guide management: identify reversible causes, improve CPR quality (mainly TEE), but do you ever use it to identify occult VF or VT? If not, you could be missing VF in 12% of cases. A brief 🧵🫀
#MedTwitter #foamed
We primarily rely on ECG during cardiac arrest to identify VF BUT @FTeranMD et al. identified 12% of cardiac arrest cases where asystole on ECG was actually fine VF on TEE. Why? ⬆️ BMI, ❌lead position, or ⬇️sensitivity on ECG all can lead to false neg pubmed.ncbi.nlm.nih.gov/30779977/
In this case, interestingly, the patient starts in ventricular tachycardia (this clip here). You can see there is some almost coordinated movement of the LV/RV, however, too fast to allow any ejection of blood which is why they have no pulse...
In the ICU, we often consider shunt as an etiology for hypoxia. Bubble studies can diagnosis right to left shunt and distinguish intracardiac or intrapulmonary shunt. A brief 🧵with some PEARLS to get the most out of a bubble study #echofirst #pocus #CardioTwitter #Cardiology
What to use? A 3-way stopcock with agitated saline works well. Not getting good visualization? Consider bacteriostatic saline... the preservative makes way better bubbles. Alternatively, mixing with a tiny bit of blood can help if visualization poor.
If bubbles appear on the left side within 3-5 cardiac cycles, this is suggestive of intracardiac shunt. If they appear later like beats 5+ (often more), intrapulmonary shunt. On #TEE, you can often visualize the bubbles crossing a PFO.. exact #s are a guide with some exceptions
#POCUS transcranial Doppler can be hard to integrate into practice - a 🧵on how it can change management. Trauma post OR for laparotomy (no CTs yet) - still unstable, TCDs done given low GCS.. diastolic reversal suggesting severely elevated ICP 🧠🤯😢#medtwitter #echofirst
Hypertonic saline + sedation and urgent CT scan showed evidence of subdural with midline shift - taken to the OR by NSx. Now Day 2 - EVD not working well and pt. not waking. Repeat TCD shows PI of ~2.4 which correlates to ICP = (10.93 × PI) - 1.28 = 25. Elevated!
Some 3% saline and deep sedation later, repeat TCD shows a PI of 1.02 which ~ ICP of 10-12.
When you insert a transvenous pacemaker for bradycardic shock, do you routinely titrate HR with serial VTI to maximize cardiac output? A 🧵
Patient's with bradycardic shock have low CO due to inadequate HR, but often have concomitant LV/RV failure. Restoring a HR via pacing to an arbitrary number (60bpm, 80bpm), may not optmizie their CO or resolve their shock. Using LVOT VTI you can monitor this to titrate HR
Once you set your pacemaker to a HR (say 60bpm), check a LVOT VTI. Now increase the HR by 10bpm measuring serial LVOT-VTI. The multiplication product of HR x VTI can be monitored to see when HR increases and SV starts decreasing. Anecdotally, this is often between 80-100bpm.