If you’re a parent and you aren’t screaming #AirborneAIDS here on Twitter, you are letting those three fuckers get away with their misinformation propaganda!
Cleavage of a Neuroinvasive Human Respiratory HCoV-OC43 Spike Glycoprotein by Proprotein Convertases Modulates Neurovirulence and Virus Spread within the Central Nervous System (CNS) - 2015
#AirborneMadCow
#WearN95 ncbi.nlm.nih.gov/pmc/articles/P…
We knew beta-CoVs (SARS/MERS/OC43) had neurovirulence causing neurological damage and sometimes even fatal encephalitis for DECADES prior to the pandemic.
Jan - JN.1
Feb - JN.2
Mar - H.1.V
Apr - BQ.3 "Gismonda" - TRG refuses naming & gets replaced
May - JN.8.8
Jun - JN.7
Jul - XCC
Aug - JN.2.78 - FDA approves JN.2 updated vaccine
Sep - XBJ
Oct - JN.9.99
Nov - BQ.7 "Klymene" - US elects Trump
Dec - BQ.7.777
We cannot pharmaceutically keep pace with an airborne virus capable of genetic recombination where GI persistence is causing lymphocytopenia regardless of immunity.
The eradication of SARS is the only viable solution.
But the political climate is not there yet.
#ZeroCOVID
We cannot continue to play "whack a mole" with a virus whose Quasi-Species Swarm abilities will evade human immunity challenges.
SARS itself is not the problem.
Every other virus/bacteria we declared "endemic" is going to be the problem.
I have no qualms calling out the #AirborneAIDS “Tone Police” as what they are - Misinformation Minimizers.
#MisinformationMinimizer
#MedTwitter is against acknowledging there’s SARS persistence in a majority of SARS infections and SARS destroys T Cells leading to lymphocytopenia enabling Opportunistic Infections (OIs) from latent bacteria that are common everywhere.
“It’s actually immune debt syndrome” Jasnah
Jasnah the expert minimizer didn’t even bother to Google Azvudine and look up to see how Azvudine has evolved well beyond “mostly in vitro”!
#DontLookUP
Let's set the record straight on ORF8!
There's virologists venturing beyond their expertise, spewing wild nonsense about ORF8 in SARS-CoV w/o having a grasp of bacteria or ribosomes. Hold tight as we debunk JN.1 chaos and uncover SARS-CoV ribosome frameshifting.
#TEXTBOOKSARS
1/ The ribosomal frameshift mechanism in SARS-CoV is crucial for optimizing viral protein synthesis. 🦠🧬A flawed reproduction process allows SARS to produce mRNA viral proteins in specific ratios, enhancing its ability to replicate and propagate.
#BoxOfNails
2/ One consequence of ribosomal frameshifting in coronaviruses is the generation of mRNAs with long 3' untranslated regions (3'UTRs). These extended 3'UTRs can potentially trigger the host NMD (Nonsense-Mediated mRNA Decay) pathway. ⚙️🧬 en.wikipedia.org/wiki/Ribosomal…
🔬ELI5: Let's dive into why knowledge about bacteria🦠is super important for understanding how SARS-CoV behaves and grasping the mysteries of #LongCOVID.
#BoxOfNails
#HijackedChickenCoop en.wikipedia.org/wiki/Bacterial…
1/10 🔬 Scientific Advancement: Our understanding of bacterial evolution has recently evolved. Previously attributed to genetic drift, recent research emphasizes the pivotal role of genetic recombination in shaping bacterial genomes. 🧬
@ejustin46
@ejustin46 2/10 🔄 Genetic Recombination: It's not just mutations! Transformation, transduction, and conjugation are key players in bacterial evolution. These processes drive the exchange of genetic material, influencing adaptability.
#GeneticExchange en.wikipedia.org/wiki/Genetic_r…