Lifelong Learner. Award-winning Objective Thinker. Certified outer spectrum enthusiast. Social mediaholic. Yes, my profile pic is middle finger of Galileo.
Dec 1 • 12 tweets • 4 min read
This is the part where I say, “I told you so”.
There’s bacterial involvement from human gut micro biome that amplifies SARS mitochondrial dysfunction causing increased ER stress and production of prions.
China has no issues calling SARS out as an autoimmune disease like AIDS nor do they bicker about SARS viral persistence similar to HIV so they can administer repurposed HIV medication for treatment. Political leaders elsewhere don’t have the balls to care.
The genera #mycoplasma and #spiroplasma belong to the mollicutes, a group of gram-positive firmicutes that lack a cell wall. These bacteria live in close association with their eukaryotic hosts and are characterized by an extreme reduction of the genome. journals.asm.org/doi/10.1128/mb…
If you search my timeline for #spiroplasma you will notice a great deal of research already exists on the association of this genome-reduced, wall-less, and fastidious bacteria in relation to White-Tailed Deer (WTD) and prion diseases.
The greatest obstacle to creating an effective SARS/MERS vaccine was always the limitations of the Human Immune System (HIS) against Human Coronaviruses (HCoV) where infection has persistent GI epithelium cells "re-teach" the HIS t-cells to accept HCoVs. onlinelibrary.wiley.com/doi/full/10.10…
ELI5 Summary of the abstract:
•If you got vaccinated before ever getting SARS, you end up with a better immune response than
•if you got SARS 1st, & then got the same amount of vaccination—in fact vaccination didn't seem to do much at all at that point.
Nov 13 • 14 tweets • 5 min read
SARS persistence: CD4-mediated SARS infection of T helper cells contributes to a poor immune response in patients.
SARS-infected T helper cells express higher levels of IL-10, which we could use to identify viral persistence but we don’t.
That thread by Dr. Bagai is a quality high level explanation for the way SARS impacts CD4 T helper cells. Same as HIV, T helper cell infection seems to occur in certain cases where there is seroconversion (SARS virus entering the blood).
Oct 23 • 6 tweets • 2 min read
Nivolumab is a human IgG4 monoclonal antibody that blocks PD-1. I wonder why removing the cell surface inhibitors would help prevent cancer cells from spreading - unless they presented themselves as harmless due to persistent viral spike proteins?
When the @WHO created a clinical case definition for #LongCOVID in October 2021 that was dependent on a positive PCR test, your nation canceled availability for PCR testing and refused to adopt the new clinical definition.
Instead, US CDC, UK NHS & EU dispute the definition and create their own definition.
Lack of consensus over the #LongCOVID clinical definition means your nation can refuse to report cases.
Oct 16 • 5 tweets • 2 min read
"brain fog" is minimization the loss of cognitive-linguistic abilities. Loss of language and communication abilities often arises for those who have a loss of sense of smell - except you can recover the sense of smell.
Part 2 of the "cheat sheet" for coping with "brain fog".
Oct 13 • 17 tweets • 6 min read
Theses are "cousins" waves.
People having multiple SARS infections in less than two months is no longer going to be "rare".
There’s still influential minimizer media pundits have no clue about human anatomy versus beta-CoVs promoting misinformation and lies:
- there’s no “HCoV natural immunity”
- there’s no “one and done” like influenza
Oct 9 • 10 tweets • 4 min read
#LongCOViD annihilation of t-cells from SARS infection occurs much faster than HIV because SARS produces more non-viable viral protein particles.
This is a very important chapter in the evolution of our understanding of persistent viral infections, such as HIV, made possible by our ongoing SARS pandemic. sciencenews.org/article/dorman…
Oct 7 • 15 tweets • 5 min read
Baby Boomers entering retirement are a burden to society.
Culling Boomers from the herd will save us trillions in healthcare and pensions costs.
It’s not worth saving the Boomers.
Retweet if you disagree, OK Boomer?
Do you remember that Veterans study?
Minimizing media was saying “those old Veterans don’t represent average Americans”.
Which is true.
They were once much healthier than average Americans and younger than you Boomer.
SARS Survivors often presented with all the physical symptoms of PTSD, without the trauma. That’s how we treated them, as PTSD sufferers for the first few years. We failed them. We failed to treat the root cause of SARS persistence triggered inflammation.
Fortunately, there were far fewer patients dealing with our struggle to understand how neurocognitive decline could be caused by a respiratory virus back in 2003. doi.org/10.1017/S13556…
Sep 17 • 6 tweets • 2 min read
“"In addition to induce DNA damage, SARS-CoV inhibits its repair"
No longer a preprint.
“An unresolved issue of SARS-CoV-2 disease is that patients often remain positive for viral RNA as detected by PCR many weeks after the initial infection in the absence of evidence for viral replication”
The “Nipah isn’t airborne” is reminiscent of every virologist being wrong about the ability of Nipah to mutate and adapt to human hosts.
Sep 13 • 12 tweets • 2 min read
PHOs around the world have been the target of bribery from American interests to influence WHO guidance since the Ebola PHEIC in 2019. If the Select Subcommittee wants a list of corrupt PHOs who’ve accepted American bribes, we can provide it.
Be aware, that if you follow Tom Peacock (or follow those who followed the misinformation provided by Tom Peacock) then you probably think:
-“SARS recombinant is RARE” and/or
- “SARS recombination with MonkeyPox was impossible”.
@fitterhappierAJ @FrontImmunol Infiltrating T cells are a hallmark of ongoing immune surveillance in cancer and are often referred to a Tumor-infiltrating T lymphocytes (TILs) and are generally positive for patients towards better better clinical outcomes in recurring cancer or viral infection.
@fitterhappierAJ @FrontImmunol These TILs are activated and actively searching for problems. There’s nothing good about these cells being around when there’s no problem because our immune system can damage healthy tissue.