Thread by @DrJackKruse

1. Question asked to me this AM via DM by a concerned parent will be today's biophysics lesson: "Why is using sunblock on my kids skin afflicted with roscea bad advice. I was told this this week at the peditrician's office. They also tried to cousel us in taking the jab."

Answer: The answer is here in a Patreon blog I wrote years ago about that condition and how sunblock affects it. Also there are implications of this peditricians advice that is harmful to the future development of your kids CNS and PNS. That is a bit more complicated but here is the genesis of how that problem can manifest. Deuterium in humans is kept in the circulatory system and out of the cytosol and mitochondrial organelles by design because it acts as a photosensitizer for the endogenous production of light that assists RBCs in their task of oxygen and CO2 deliver to tissues.

Deuterium is essentially a photosensitizer for the creation of UVA, UVB, and UVC light for RBCs in our circulatory system. It requires surface level UVA light to begin to work. So when you block sunlight with clothing or sunblock you are blocking endogenous light production in the circulatory system for the needs of your RBCs. Moreover, this has collateral affects. It affects oxygen deliver to you colony of mtDNA but it also affects the circadian cycle of your RBCs and can lead to anemia and other blood dyscrasias. This is very easy to assess with an O2 Hb sat % monitor you can buy for 25 bucks on Amazon and a tubr of toxic sunblock or UV blocking shirt above the hand you put the monitir on.

The UV blocking effect in RBCs is registered in peroxiredoxins of the RBCs since adult human RBCs have no mitochondria. Peroxiredoxin-2 (PRDX2) in preventing hydrogen peroxide-induced oxidative stress in the red blood cell. patreon.com/posts/21334201

2. What few clinicians and researcher know about human blood is how circadian cycles are controlled by the RBCs. One of the most abundant proteins in erythrocytes after hemoglobin is peroxiredoxin. They are a ubiquitous family of antioxidant enzymes that control cytokine-induced peroxide levels and thereby mediate solar signal transduction in mammalian cells.

I hope you recall that cytochrome 2, 3, and 4 make hydrogen peroxide free radicals that are critically related to peroxiredoxins. Recall H202 is quenched by catalase in the blood. Do you know catalase is the fastest enzyme in the human body? There is a reason for that. Moreover, Catalase is another heme related protein subject to retinal destruction when you abuse the wrong light. Most skin diseases are linked to this biophysical reality. Your pediatrician and dermatologist are impotent in helping you because they are undereducated how light operates in your skin and blood.

Peroxiredoxins are tightly regulated by phosphorylation, and light changes phosphoryaltion big time. More? Redox status such as sulfonation, acetylation, nitration, truncation and oligomerization states are all changed by light stress in cells. All of these are affected by the light environment of the mammal in question.

3. Mitochondrial experts do not have much to say when we talk about RBCs. They believe that you need photosensitizers to change blood signaling. They are idiots too, for having this opinion. Why?

These enzymes in RBCs share the same basic catalytic mechanism, in which a redox-active cysteine (the peroxidatic cysteine) in the active site is oxidized to a sulfenic acid by the peroxide substrate. Cysteine becomes afunctional when it is oxidized by light and it turns out this is a big issue in the methionine cycle = because it ruins PER circadian gene that controls diurnal function being made in the AM in the cytosol by sunlight and then entering the nucleus to affect its function.

See my comments in Quantum Thermodynamics #14 about methionine as a TIME CRYSTAL for your blood. I guarrantee you that your experts have zero clue about this pathway. patreon.com/posts/19082581

4. The recycling of the sulfenic acid back to a thiol is what distinguishes the three enzyme classes. 2-Cys peroxiredoxins are reduced by thiols such as thioredoxins, thioredoxin-like proteins, and glutathione, while the 1-Cys enzymes are reduced by ascorbic acid and/or glutathione in the presence of GST-π. This can be augmented with the coherent domains sunlight creates in water of the blood (93% by volume FYI) by clinicians who understand the mechanisms I mentioned in detail the November 2018 webinar for my members.

5. If you use sunblock or UV blocking clothing what happens in this system? When the AM UV light signal is defective, for any reason, from the skin to RBCs you set up the brain for disease because you are altering oxygen and NO deliver in the arterial bed that feds the brain. Recall 20% of cardiac output is sent to the brain. In diseases like AD/PD/ALS this changes lipid and gas permeability of the BBB. I covered this in my Vermont 2017 talk on YT via when I showed some slides of how light waves resonate with RBC's to make toroids sound waves in the blood vessels that release NO and activate the peroxiredoxin system to action from the arterial wall to change the microcirculation of the brain and BBB.

How does this change the circadian mechanism in the brain? It alters the SCN and molecular clock linkage. For the timing gear shift in RBC peroxiredoxins, the amino acid cysteine is the key time crystal that is likely defective and leads to an oxidized state which does not work. This will affect phosphorylation of PER protein and it won't operate in the brain and leads to eventual apoptosis in the frontal and temporal lobes of man where glucose metabolism is defective where the BBB BECOMES more permeable from nnEMF around us in our environment. This means you are sensitized to blue light/nnEMF light stress. You think those new Apple VR glasses are risk free. Think again. They will buy me lots of Bitcoin in the future in my clinics.

Mechanical trauma of concussions acts the same way (hear that NFL/NHL) & can also induce this in my opinion and I think this mechanism is shared with ALS with head and neck trauma. Once the phosphorylation of PER gene is altered, all circadian hell breaks loose in the CNA. It leads to collateral destruction in neurons over time by altering the chaos/self-ordering effects found in how entropy is used in cells to self organize.

6. Why is understanding methionine important as a time crystal? It is one of the key rare sulfur-containing amino acids that help us tell time by passing phosphorus to the PER one gene product every AM. Sulfur also cools the temperature of these proteins for environmental sensing and adjusting of the PER gene product.

How?

7. Throughout 4.5 billion years of molecular evolution, proteins have evolved in order to maintain the spatial proximity between aromatic residues (Trp, Tyr and Phe) and disulfide bridges (SS) (Petersen et al, 1999).

We might call this mechanism of how light interacts with certain aromatic amino acids and sulfur-containing amino acids how the fractal geometry of life begins. It is a key molecular quantum connection you must understand because it is BASIC to most photoelectric diseases in humans.

There is a very special spatial geometric relationship that exists because the process is quantized to light frequencies that our star releases to us on Earth wirelessly. This has not been well appreciated by modern healthcare. This is also why the sun reduces all-cause mortality and why it can never be replaced in healthcare. I also believe this is why solar redox is critical to having a child with great morphogenesis and the normal adult form. It can also take a normal organ like the brain and turn it into mush over a life time spent in blue light and nnEMF.

Doyou understand now why your experts are impotent to fix chronic disease built around modern lighting? They do not shit about light or why light controls matters at all.

HOW DOES THIS OPERATE BELOW THE CELL LEVEL UNCLE JACK?

To suggest this is lunacy is not wise when you really understand the biophysics of aromatic amino acids in our cells well. This was the topic of the Vermont 2018 talk.

The interaction of the most powerful part of the solar spectrum of light (UVA/B/C) measures the collisions in the aromatic amino acids and in the disulfide bridges. THIS IS why the 2017 Nobel Prize in medicine is important to understand and fully understand to understand what UNCLE Jack is teaching you. PER is a protein made every AM by SUNLIGHT stimulus as the picture below shows.

You are letting dumbass tell you stupid things to do. Blocking the sun with clothing and sunscreen is suicide for your brain, arteries, RBCs and your circadian mechanism.

8. Now for the goods why clinicians and most mitochondrial experts in centralized science are dumbasses. Why is this picture above such a big deal in understanding neurodegeneration photoelectric origins from blockade of the sun from the skin?
When PER1 cycling is off chronically because of aberrant light or nnEMF so is NAD+ at cytochrome 1 in the mitochondrial matrix and this causes advanced aging in man = David Sinclair's paper in Dec 2013. This is about the only thing Sinclair has done in his career that was worth it. Aging links directly to low NAD+ levels. So does every chronic disease. When NAD+ is low so is hypoxia. Guess what else is destroyed inside of you? MELANIN.

See how it all fits. See why your experts are ignornant yet? Understand my disdain for food gurus, supplement pushers, mitochondrial/longevity experts, and exercise gurus yet? They all have no idea what key data they are LACKING. Now you do.

9. NAD+ is one of the more immediate players in cytochrome 1 that is a huge driver of circadian biology in humans. It concerns itself with electrons and proton motions in mitochondria. IMAGINE THAT.

I told you NAD+ is made from the other time crystal in biology called Tryptophan. Tryptophan also makes melatonin and melatonin is what controls autophagy and apoptosis in humans cells. NAD+ is the coenzyme called nicotinamide adenine dinucleotide (NAD+). It participates in a variety of redox reactions in the matrix that help generate coherent domains in CSF that surrounds the brain and that comes from the water insider your blood vessels. See how it all links yet, dumbasses?

I'm sick and tired of dealing with people who do not do a thing to help the public get well but sell them bullshit stories of treatments that only enrich themselves.

Time for the AGE OF LIGHT to begin. @twocitizenships

10. Solar exposure and fasting work with light frequencies to slow ECT flow and this can increase the intracellular NAD/NADH ratio if the light environment is dominated by sunlight. It won't do this with artificial light, because it lowers NAD+. This is what sets off a cascade of circadian events that can destroy tissues because they involve epigenetics and the regulation of growth and metabolism of man. LIGHT DOES THIS, not food or anything else the dumbasses tell you.

NOT FOOD OR FUELS.

SUN + fasting induced by sun exposure (NO from UVA and IR from sun) ---> increased PER1 creation and cycling is controlled by AM sunlight ---> raised NAD+ -> SIRT1 -> BMAL1/CLOCK -> NAMPT -> NAD+ is increased at cytochrome 1. NAD+ major effect is to activate the sirtuins and keep PER on the schedule being made in the cytosol to penetrate the nucleus at night as the reaction above shows in the picture. SIRT 1 is another gear in the light lattice clock that keeps PER on time and SIRT is a family of deacetylase enzymes. When you understand what UV and IR light are doing to a matrix and cytosol at the same time, you can see why fasting and AM light exposure is potentially the best circadian hack one could do for a reset. It won't work in fake light (nnEMF =BBB leaky) when ALAN is present. Eating protein stimulates the thermogenic gears of the clock = why the Leptin Rx uses it fuckers.

11. So how does sunblock clothing or tech screens get you from roscea to Alzheimer's disease in 4 decades?

BACK TO THE AMINO ACIDS

The aromatic amino acids location becomes the first step in determining where the position and geometry of residues to act as nanosized antennas in the protein world that can capture UV light (from ~250-298nm). This could be another "yes or no binary code" that cells use to build tissues. You can see why the idea of a time crystal is paramount now when you are building a life form. Think about what I said in my Vermont 2017 and 2018 video now in this light!!!

The first two protein bends are always determined by nuclear DNA coding and this would save us a lot of time in morphogenesis. The last two bends are tied to the redox state which is linked to this quantum photoelectric process I am describing here now. This is what is defective in PD/AD patients. If the timing of PER is off for any reason at all the binary code of " yes and no's" buried in light frequencies in RBCs (peroxiredoxins) ferried to neurons are off and as a result the protein folding is off, and you'll make glitches in the folding of proteins and organelles in cells and tissues.

The amount of misfolding affects the ELF-UV light signal the cell can make. Bad folding = more ELF-UV release = more neural tissue destruction.

Got it yet? Do you see yet why I am hot with my profession.

The answers are all in the literature but they are too bound to the diet and exercise paradigm of bullshit to see it.

12. The amount of misfolding affects the ELF-UV light signal the cell can make. Bad folding = more ELF-UV release = more neural tissue destruction.

Once excited by the incident ELF-UV light these amino acids can enter photochemical pathways likely to have harmful or beneficial effects on protein structures (yes and no binary code plays a role here again) by affecting specific bonds like disulfide bonds in cysteine/cystine on the PER protein made every AM via RBCs peroxiredoxins. They are the key photosenstizer in blood.

In the circadian mechanism, their effect on the PER protein that is made every AM by sunlight exposure on our body is critical in the right amount of phosphorylation in our cells to tell proper quantum time in neurons and glial cells. This is why proteins cannot be clear during sleep. This is why elevating your bed when you sleep helps. It is because your addicted to light that is killing you.

These two sulfur amino acids are the rarest amino acids in our proteins, and as such can acts as the ideal photo-optical switch or gate for signaling of phosphorylation of PER because of they a relatively rare in humans.

It turns out cysteine/cystine disulfide bridges in proteins are known to be excellent quenchers of the excited state of aromatic residues by UV light in the literature. I don't believe any AD researchers realize this today. This means these disulfide residues naturally decrease the power present in UV light created in the nearby excited aromatic amino acids of the cells in a developing life form

13. Could this study be possibly hinting that Alzheimer's disease is also linked to indoor living in the blue light as Uncle Jack has been saying for 19 years? YEP. The study offers a hint of hope for staving off dementia in some people - if you treat high blood pressure aggressively, it might just help both your heart and your brain. the reason is simple. Staying indoors block most UV and 3--60% of red light allowing the blue light easy passage to and fro because it is not affected by the glass in the window and this causes a relative BLUE LIGHT HAZARD for humans over time. Indoor living is the big link to the photoelectric defect in my opinion. nytimes.com/2019/01/28/hea…

14. In this way, the cysteine/cystine disulfide bridges contribute to protein stability of PER and their circadian activity in tissues, thereby, stabilizing ubiquitin rates and helping drive proper morphogenesis via the blueprint in DNA/RNA. I believe the fidelity of this copying mechanism is tied to the OAM number of light that interacts with DNA and I assume that ELF-UV is critical in that dance. When it is off this fosters protein misfolding and I think it is linked to many brain diseases associated with misfolding like ALS, prions, and GBM. I assume each species has its own molecular resonance OAM they operate with = many diseases can happen that share many features = AD/PD/ALS and GBMs (grade 4 gliomas is associated with LACK OF UV light)

Why do I think the amount of ELF-UV light is the key in this dance of disease? Because all the pieces fit.

15. Sick neurons make too much ELF-UV light and we know UVA light affects CCO.
Chronic inhibition of cytochrome c oxidase leads to chronic neurodegenerative disease and that link is found in many papers.

UV light excitation of the aromatic residues is known to trigger electron ejection from their side chains (Bent & Hayon, 1975a; Bent & Hayon, 1975b; Bent & Hayon, 1975c; Creed, 1984a; Creed, 1984b; Kerwin & Rammele, 2007, Neves-Petersen et al., 2009a).

These electrons can be captured by disulfide bridges in simple proteins with tons of disulfide bridges like glutathione, leading to the formation of a transient disulfide electron adduct radicals, which will dissociate photoelectrically, leading to the formation of free thiol groups in the protein. What you did not know until my Patreon blogs last year is that coherent water created by the matrix can increase endogenous glutathione for matter creation and organ building and proper maintenance in organs like the brain. In AD this process is broken by RBC changes in the eye and skin to blue light and nnEMF.

This photochemical change then leads to non-optical signaling at deeper levels in the embryo as development continues using the butterfly effect = non-linear effect that goes back to Turing's 1952 paper on how morphogenesis proceeds.

The irony in all these details is that UV frequencies foster the creation of matter naturally, and do not cause cancer, by these quantum mechanisms.

Why do I say this?

16. More irony for healthcare myopic paradigm that the sun and UV light is bad: This quantum mechanism using UV light is now being used big pharma to develop drugs using nanotechnology and the ability of cells to make ELF-UV light in a process called LUMI.

Almost 75% of drugs prescribed affect the human circadian mechanism. None of the fix the problem, only light and dark can.

That is how roscea can lead to a destroyed human brain and why your doctors can't do a damn thing about with their training. Got it Elizabeth?

END OF LESSON.