1. Question asked to me this AM via DM by a concerned parent will be today's biophysics lesson: "Why is using sunblock on my kids skin afflicted with roscea bad advice. I was told this this week at the peditrician's office. They also tried to cousel us in taking the jab."
Answer: The answer is here in a Patreon blog I wrote years ago about that condition and how sunblock affects it. Also there are implications of this peditricians advice that is harmful to the future development of your kids CNS and PNS. That is a bit more complicated but here is the genesis of how that problem can manifest. Deuterium in humans is kept in the circulatory system and out of the cytosol and mitochondrial organelles by design because it acts as a photosensitizer for the endogenous production of light that assists RBCs in their task of oxygen and CO2 deliver to tissues.
Deuterium is essentially a photosensitizer for the creation of UVA, UVB, and UVC light for RBCs in our circulatory system. It requires surface level UVA light to begin to work. So when you block sunlight with clothing or sunblock you are blocking endogenous light production in the circulatory system for the needs of your RBCs. Moreover, this has collateral affects. It affects oxygen deliver to you colony of mtDNA but it also affects the circadian cycle of your RBCs and can lead to anemia and other blood dyscrasias. This is very easy to assess with an O2 Hb sat % monitor you can buy for 25 bucks on Amazon and a tubr of toxic sunblock or UV blocking shirt above the hand you put the monitir on.
The UV blocking effect in RBCs is registered in peroxiredoxins of the RBCs since adult human RBCs have no mitochondria. Peroxiredoxin-2 (PRDX2) in preventing hydrogen peroxide-induced oxidative stress in the red blood cell. patreon.com/posts/21334201
2. What few clinicians and researcher know about human blood is how circadian cycles are controlled by the RBCs. One of the most abundant proteins in erythrocytes after hemoglobin is peroxiredoxin. They are a ubiquitous family of antioxidant enzymes that control cytokine-induced peroxide levels and thereby mediate solar signal transduction in mammalian cells.
I hope you recall that cytochrome 2, 3, and 4 make hydrogen peroxide free radicals that are critically related to peroxiredoxins. Recall H202 is quenched by catalase in the blood. Do you know catalase is the fastest enzyme in the human body? There is a reason for that. Moreover, Catalase is another heme related protein subject to retinal destruction when you abuse the wrong light. Most skin diseases are linked to this biophysical reality. Your pediatrician and dermatologist are impotent in helping you because they are undereducated how light operates in your skin and blood.
Peroxiredoxins are tightly regulated by phosphorylation, and light changes phosphoryaltion big time. More? Redox status such as sulfonation, acetylation, nitration, truncation and oligomerization states are all changed by light stress in cells. All of these are affected by the light environment of the mammal in question.
3. Mitochondrial experts do not have much to say when we talk about RBCs. They believe that you need photosensitizers to change blood signaling. They are idiots too, for having this opinion. Why?
These enzymes in RBCs share the same basic catalytic mechanism, in which a redox-active cysteine (the peroxidatic cysteine) in the active site is oxidized to a sulfenic acid by the peroxide substrate. Cysteine becomes afunctional when it is oxidized by light and it turns out this is a big issue in the methionine cycle = because it ruins PER circadian gene that controls diurnal function being made in the AM in the cytosol by sunlight and then entering the nucleus to affect its function.
See my comments in Quantum Thermodynamics #14 about methionine as a TIME CRYSTAL for your blood. I guarrantee you that your experts have zero clue about this pathway. patreon.com/posts/19082581
4. The recycling of the sulfenic acid back to a thiol is what distinguishes the three enzyme classes. 2-Cys peroxiredoxins are reduced by thiols such as thioredoxins, thioredoxin-like proteins, and glutathione, while the 1-Cys enzymes are reduced by ascorbic acid and/or glutathione in the presence of GST-π. This can be augmented with the coherent domains sunlight creates in water of the blood (93% by volume FYI) by clinicians who understand the mechanisms I mentioned in detail the November 2018 webinar for my members.
5. If you use sunblock or UV blocking clothing what happens in this system? When the AM UV light signal is defective, for any reason, from the skin to RBCs you set up the brain for disease because you are altering oxygen and NO deliver in the arterial bed that feds the brain. Recall 20% of cardiac output is sent to the brain. In diseases like AD/PD/ALS this changes lipid and gas permeability of the BBB. I covered this in my Vermont 2017 talk on YT via when I showed some slides of how light waves resonate with RBC's to make toroids sound waves in the blood vessels that release NO and activate the peroxiredoxin system to action from the arterial wall to change the microcirculation of the brain and BBB.
How does this change the circadian mechanism in the brain? It alters the SCN and molecular clock linkage. For the timing gear shift in RBC peroxiredoxins, the amino acid cysteine is the key time crystal that is likely defective and leads to an oxidized state which does not work. This will affect phosphorylation of PER protein and it won't operate in the brain and leads to eventual apoptosis in the frontal and temporal lobes of man where glucose metabolism is defective where the BBB BECOMES more permeable from nnEMF around us in our environment. This means you are sensitized to blue light/nnEMF light stress. You think those new Apple VR glasses are risk free. Think again. They will buy me lots of Bitcoin in the future in my clinics.
Mechanical trauma of concussions acts the same way (hear that NFL/NHL) & can also induce this in my opinion and I think this mechanism is shared with ALS with head and neck trauma. Once the phosphorylation of PER gene is altered, all circadian hell breaks loose in the CNA. It leads to collateral destruction in neurons over time by altering the chaos/self-ordering effects found in how entropy is used in cells to self organize.
6. Why is understanding methionine important as a time crystal? It is one of the key rare sulfur-containing amino acids that help us tell time by passing phosphorus to the PER one gene product every AM. Sulfur also cools the temperature of these proteins for environmental sensing and adjusting of the PER gene product.
How?
7. Throughout 4.5 billion years of molecular evolution, proteins have evolved in order to maintain the spatial proximity between aromatic residues (Trp, Tyr and Phe) and disulfide bridges (SS) (Petersen et al, 1999).
We might call this mechanism of how light interacts with certain aromatic amino acids and sulfur-containing amino acids how the fractal geometry of life begins. It is a key molecular quantum connection you must understand because it is BASIC to most photoelectric diseases in humans.
There is a very special spatial geometric relationship that exists because the process is quantized to light frequencies that our star releases to us on Earth wirelessly. This has not been well appreciated by modern healthcare. This is also why the sun reduces all-cause mortality and why it can never be replaced in healthcare. I also believe this is why solar redox is critical to having a child with great morphogenesis and the normal adult form. It can also take a normal organ like the brain and turn it into mush over a life time spent in blue light and nnEMF.
Doyou understand now why your experts are impotent to fix chronic disease built around modern lighting? They do not shit about light or why light controls matters at all.
HOW DOES THIS OPERATE BELOW THE CELL LEVEL UNCLE JACK?
To suggest this is lunacy is not wise when you really understand the biophysics of aromatic amino acids in our cells well. This was the topic of the Vermont 2018 talk.
The interaction of the most powerful part of the solar spectrum of light (UVA/B/C) measures the collisions in the aromatic amino acids and in the disulfide bridges. THIS IS why the 2017 Nobel Prize in medicine is important to understand and fully understand to understand what UNCLE Jack is teaching you. PER is a protein made every AM by SUNLIGHT stimulus as the picture below shows.
You are letting dumbass tell you stupid things to do. Blocking the sun with clothing and sunscreen is suicide for your brain, arteries, RBCs and your circadian mechanism.
8. Now for the goods why clinicians and most mitochondrial experts in centralized science are dumbasses. Why is this picture above such a big deal in understanding neurodegeneration photoelectric origins from blockade of the sun from the skin?
When PER1 cycling is off chronically because of aberrant light or nnEMF so is NAD+ at cytochrome 1 in the mitochondrial matrix and this causes advanced aging in man = David Sinclair's paper in Dec 2013. This is about the only thing Sinclair has done in his career that was worth it. Aging links directly to low NAD+ levels. So does every chronic disease. When NAD+ is low so is hypoxia. Guess what else is destroyed inside of you? MELANIN.
See how it all fits. See why your experts are ignornant yet? Understand my disdain for food gurus, supplement pushers, mitochondrial/longevity experts, and exercise gurus yet? They all have no idea what key data they are LACKING. Now you do.
9. NAD+ is one of the more immediate players in cytochrome 1 that is a huge driver of circadian biology in humans. It concerns itself with electrons and proton motions in mitochondria. IMAGINE THAT.
I told you NAD+ is made from the other time crystal in biology called Tryptophan. Tryptophan also makes melatonin and melatonin is what controls autophagy and apoptosis in humans cells. NAD+ is the coenzyme called nicotinamide adenine dinucleotide (NAD+). It participates in a variety of redox reactions in the matrix that help generate coherent domains in CSF that surrounds the brain and that comes from the water insider your blood vessels. See how it all links yet, dumbasses?
I'm sick and tired of dealing with people who do not do a thing to help the public get well but sell them bullshit stories of treatments that only enrich themselves.
Time for the AGE OF LIGHT to begin. @twocitizenships
10. Solar exposure and fasting work with light frequencies to slow ECT flow and this can increase the intracellular NAD/NADH ratio if the light environment is dominated by sunlight. It won't do this with artificial light, because it lowers NAD+. This is what sets off a cascade of circadian events that can destroy tissues because they involve epigenetics and the regulation of growth and metabolism of man. LIGHT DOES THIS, not food or anything else the dumbasses tell you.
NOT FOOD OR FUELS.
SUN + fasting induced by sun exposure (NO from UVA and IR from sun) ---> increased PER1 creation and cycling is controlled by AM sunlight ---> raised NAD+ -> SIRT1 -> BMAL1/CLOCK -> NAMPT -> NAD+ is increased at cytochrome 1. NAD+ major effect is to activate the sirtuins and keep PER on the schedule being made in the cytosol to penetrate the nucleus at night as the reaction above shows in the picture. SIRT 1 is another gear in the light lattice clock that keeps PER on time and SIRT is a family of deacetylase enzymes. When you understand what UV and IR light are doing to a matrix and cytosol at the same time, you can see why fasting and AM light exposure is potentially the best circadian hack one could do for a reset. It won't work in fake light (nnEMF =BBB leaky) when ALAN is present. Eating protein stimulates the thermogenic gears of the clock = why the Leptin Rx uses it fuckers.
11. So how does sunblock clothing or tech screens get you from roscea to Alzheimer's disease in 4 decades?
BACK TO THE AMINO ACIDS
The aromatic amino acids location becomes the first step in determining where the position and geometry of residues to act as nanosized antennas in the protein world that can capture UV light (from ~250-298nm). This could be another "yes or no binary code" that cells use to build tissues. You can see why the idea of a time crystal is paramount now when you are building a life form. Think about what I said in my Vermont 2017 and 2018 video now in this light!!!
The first two protein bends are always determined by nuclear DNA coding and this would save us a lot of time in morphogenesis. The last two bends are tied to the redox state which is linked to this quantum photoelectric process I am describing here now. This is what is defective in PD/AD patients. If the timing of PER is off for any reason at all the binary code of " yes and no's" buried in light frequencies in RBCs (peroxiredoxins) ferried to neurons are off and as a result the protein folding is off, and you'll make glitches in the folding of proteins and organelles in cells and tissues.
The amount of misfolding affects the ELF-UV light signal the cell can make. Bad folding = more ELF-UV release = more neural tissue destruction.
Got it yet? Do you see yet why I am hot with my profession.
The answers are all in the literature but they are too bound to the diet and exercise paradigm of bullshit to see it.
12. The amount of misfolding affects the ELF-UV light signal the cell can make. Bad folding = more ELF-UV release = more neural tissue destruction.
Once excited by the incident ELF-UV light these amino acids can enter photochemical pathways likely to have harmful or beneficial effects on protein structures (yes and no binary code plays a role here again) by affecting specific bonds like disulfide bonds in cysteine/cystine on the PER protein made every AM via RBCs peroxiredoxins. They are the key photosenstizer in blood.
In the circadian mechanism, their effect on the PER protein that is made every AM by sunlight exposure on our body is critical in the right amount of phosphorylation in our cells to tell proper quantum time in neurons and glial cells. This is why proteins cannot be clear during sleep. This is why elevating your bed when you sleep helps. It is because your addicted to light that is killing you.
These two sulfur amino acids are the rarest amino acids in our proteins, and as such can acts as the ideal photo-optical switch or gate for signaling of phosphorylation of PER because of they a relatively rare in humans.
It turns out cysteine/cystine disulfide bridges in proteins are known to be excellent quenchers of the excited state of aromatic residues by UV light in the literature. I don't believe any AD researchers realize this today. This means these disulfide residues naturally decrease the power present in UV light created in the nearby excited aromatic amino acids of the cells in a developing life form
13. Could this study be possibly hinting that Alzheimer's disease is also linked to indoor living in the blue light as Uncle Jack has been saying for 19 years? YEP. The study offers a hint of hope for staving off dementia in some people - if you treat high blood pressure aggressively, it might just help both your heart and your brain. the reason is simple. Staying indoors block most UV and 3--60% of red light allowing the blue light easy passage to and fro because it is not affected by the glass in the window and this causes a relative BLUE LIGHT HAZARD for humans over time. Indoor living is the big link to the photoelectric defect in my opinion. nytimes.com/2019/01/28/hea…
14. In this way, the cysteine/cystine disulfide bridges contribute to protein stability of PER and their circadian activity in tissues, thereby, stabilizing ubiquitin rates and helping drive proper morphogenesis via the blueprint in DNA/RNA. I believe the fidelity of this copying mechanism is tied to the OAM number of light that interacts with DNA and I assume that ELF-UV is critical in that dance. When it is off this fosters protein misfolding and I think it is linked to many brain diseases associated with misfolding like ALS, prions, and GBM. I assume each species has its own molecular resonance OAM they operate with = many diseases can happen that share many features = AD/PD/ALS and GBMs (grade 4 gliomas is associated with LACK OF UV light)
Why do I think the amount of ELF-UV light is the key in this dance of disease? Because all the pieces fit.
15. Sick neurons make too much ELF-UV light and we know UVA light affects CCO.
Chronic inhibition of cytochrome c oxidase leads to chronic neurodegenerative disease and that link is found in many papers.
UV light excitation of the aromatic residues is known to trigger electron ejection from their side chains (Bent & Hayon, 1975a; Bent & Hayon, 1975b; Bent & Hayon, 1975c; Creed, 1984a; Creed, 1984b; Kerwin & Rammele, 2007, Neves-Petersen et al., 2009a).
These electrons can be captured by disulfide bridges in simple proteins with tons of disulfide bridges like glutathione, leading to the formation of a transient disulfide electron adduct radicals, which will dissociate photoelectrically, leading to the formation of free thiol groups in the protein. What you did not know until my Patreon blogs last year is that coherent water created by the matrix can increase endogenous glutathione for matter creation and organ building and proper maintenance in organs like the brain. In AD this process is broken by RBC changes in the eye and skin to blue light and nnEMF.
This photochemical change then leads to non-optical signaling at deeper levels in the embryo as development continues using the butterfly effect = non-linear effect that goes back to Turing's 1952 paper on how morphogenesis proceeds.
The irony in all these details is that UV frequencies foster the creation of matter naturally, and do not cause cancer, by these quantum mechanisms.
Why do I say this?
16. More irony for healthcare myopic paradigm that the sun and UV light is bad: This quantum mechanism using UV light is now being used big pharma to develop drugs using nanotechnology and the ability of cells to make ELF-UV light in a process called LUMI.
Almost 75% of drugs prescribed affect the human circadian mechanism. None of the fix the problem, only light and dark can.
That is how roscea can lead to a destroyed human brain and why your doctors can't do a damn thing about with their training. Got it Elizabeth?
END OF LESSON.
• • •
Missing some Tweet in this thread? You can try to
force a refresh
What do you know about the Stiles-Crawford effect in a healthy eye? What if I told you this effect is how we sharpen central vision and narrow the periphery of the retina from too much blue light. Would you believe it? Did you know melanopsin has a specific topographic map on a healthy retina? A lesson no has taught you is incoming.
2. All opsins are topologic insulators. You might want to read that threadroll above now to understand topology well.
Topology changes in a cell = geometry change of cristae = UPE change from mitochondria = the optical signal changes in the same tissue altering physiology.
So what happens if you sustain mitochondrial damage in your retina's colony of mitochondria to the Stiles Crawford effect?
What are the implications?
3. The Stiles–Crawford effect (SCE) is the human eye's phenomenon of reduced light sensitivity when light enters the pupil from its periphery compared to the pupil's center. This effect is due to the optical properties of photoreceptors, which act as waveguides and are aligned to channel light towards the fovea, the central point of vision. The SCE makes vision less sensitive to light entering the periphery, thus reducing glare and improving visual clarity. It also keeps a lid on the amount of blue light the periphery the retina gets.
This sharpens vision, makes myopia, glaucoma, cataracts, hyperopia, and AMD almost impossible to get. Makes one resistant to mental illness too. Makes one impervious to diabetic transformations. Makes neurodegeneration rare.
Another new podcast from me with Smuggling Hope. It is good one on mental health because it explains how biomolecules absorption and emission spectra's determine reality or determine which mental illness one gets.
This is the information why Jordan Peterson is sick and why he and his daughter have stumbled multiple times in getting him well. ivoox.com/.../exposing-t…...
Biophotons are ultra-weak light emissions produced by living organisms, thought to arise from metabolic processes like oxidative reactions in mitochondria. Centralized scientists and AI bots hypothesized them to play a role in cellular communication, potentially influencing gene expression or signaling pathways. FIAF, also known as angiopoietin-like protein 4 (ANGPTL4), is a protein produced by tissues like fat and the gut, and it’s a key player in lipid metabolism and microbiome construction. Neither understand how UPEs control FIAF. If the UPE is coherent then FIAF inhibits lipoprotein lipase, affecting how fats are stored or broken down, and its expression ramps up during fasting. The microbiome, meanwhile, is the community of gut microbes that can shift in response to diet, fasting, or host metabolism, influencing energy balance and health. If the UPEs in mitochondria is not coherent, the UPE changes and mental illness becomes more probable. You cannot burn fat so it changes neural signaling.
Centralized scientists and technocrats who build AI systems will say no direct study says “biophotons control FIAF to affect the microbiome,” but we can hypothesize based on related mechanisms. This is patently false. Why?
FIAF has known absorption and emission spectra to light frequencies. Because of that, a study is superfluous because each chemical in the world has an optical fingerprint. Just because a scientist has not published the work is immaterial to this BASIC biophysical fact in spectroscopy. This was what I brought Ray Peat over ten years ago, and he was impotent enough to answer my critiques of his work. This podcast covers these details.
2. Is nerve pain caused by a Lyrica deficiency?
Is Lyme disease linked to a lack of doxycycline?
Is depression due to Prozac deficiency.
Are heart attacks are due to Lipitor deficiency.
Is obesity due to Ozempic deficiency.
Are Headaches due to Tylenol deficiency?
Is Bipolar disorder due to lithium deficiency?
Any questions?
You've been conditioned by centralized medicine to ask the wrong question.
You have a solar deficiency combined with a nnEMF toxicity problem.
This alters the UPEs your mitochondria make and it is this light that changes the neural tracks that make you mentally ill. This is why your Bipolar Disorder exists. The defect is not in you; it is in your environment.
3. Light exposure,say, sunlight or specific wavelengths impacts circadian rhythms via the suprachiasmatic nucleus in the brain, which regulates hormones like dopamine, GABA,melatonin and cortisol.
These hormones influence metabolism, including fat tissue activity where FIAF is expressed. Metabolism is what makes the key UPEs.
Fasting, which boosts FIAF, is also tied to light cycles, think of how daylight affects feeding patterns. If biophotons reflect or amplify these light-driven processes at a cellular level, they might indirectly tweak FIAF production by signaling energy states or oxidative stress in cells. If you cannot burn fat you are more likely to be mentally ill.
My decentralized ideas have always been spot-on that this topic doesn’t need a scientist to spell it out in a paper, absorption/emission spectra are measurable, and biophoton emissions are detectable.
The gap isn’t in the physics; it’s in tying the specific wavelengths of biophotons (which vary by cell type and state) to FIAF’s exact optical profile in vivo. Still, if gut cells emit biophotons in, say, the 300-400 nm range, and FIAF absorbs there, the optical photonics interaction’s real, study or not. It is first principle thinking. It is obvious what they problem. It’s like saying water absorbs infrared; we don’t need a new experiment to prove it gets hot under sunlight. Biology acts like it does, but in physics they use theoretical physics and first principle thinking to make predictions when the experiments are not done or cannot be done.
When matter experiences this topologic change do you know it become capable of emitting photons? In biology we call this UPEs. That is what does all the information transferring in life to keep you alive and kicking.
2. In astronomy and cosmology Spectroscopy is a form of remote sensing, meaning it allows scientists to determine the composition of an object without physically interacting with it. This is how we examine remote atoms in deep space.
How do we know what other worlds are made of? Planets we’ve never touched, stars we’ll never reach? By reading their light. Why can't quantum biologists realize the same opportunity exists in cells?
3. Quantum biology cannot yet apply the same spectroscopic "reading of light" as astronomy because cellular components are too small to be analyzed by light in the same way, and the inherent quantum effects within cells are not easily distinguishable from background noise in typical biological systems. They do not have photomultipliers small enough to sample UPEs yet.
Just because the technology is not available or studied means we should ignore the idea. Absense of evidence is not absence of effect. This is first principle thinking that is missing from most scientists today. In physics theoretical physicists provide a first principle lens to the Standard Model to innovate. We need to do the same in quantum biology. This is what I do.
Astronomers use spectroscopy to analyze the wavelengths of light absorbed or emitted by large celestial bodies, which reveal their chemical composition. However, cellular molecules are often too small to generate a detectable light signature, and the complex, noisy environment of a living cell makes it difficult to isolate and interpret the faint quantum signals associated with specific biological processes. there is no doubt today UPEs are real and carry information. We've know that AXIOMATICALLY since the Onion root experiment in 1922.
Cutaneous antimicrobial effects of sunlight on cholesterol conversion to Vitamin D components are today's PSA boys and girls.
1,25(OH)2D made in the liver and kidneys from 25 D(OH) from the skin by the sun and cholesterol and its receptor regulate the processing of the long-chain glycosylceramides that are critical for the skin barrier formation which is crucial in defending the skin.
Do you know how the heme protein enzymes CYP control this process?
The two Vitamin D biomolecules induce toll-like receptor 2 (TLR2) and its coreceptor CD14, which initiate the innate immune response in the skin. Activation of these receptors leads to the induction of CYP27B1 (heme protein), which in turn induces cathelicidin resulting in the killing of invasive organisms.
What happens when blue light and nnEMF destroy heme proteins when you know this connection? Innate immunity is destroyed. This is why Fauci wanted you indoors during COVID he and Baric made in Ukraine and China. ncbi.nlm.nih.gov/pmc/articles/P…
2. See how the heme photoreceptors are blown away?
3. Spine tumors are not common but most of them are associated with a poorly functioning immune arm and associated with low Vitamin D levels from poor solar exposure. That is something you can prevent to avoid this outcome.
Fire your centralized doctor by hiring nature for your reversal! Nature quantizes the precise amount of melatonin from the mitochondria needed to optimize autophagy and apoptosis. 95% of melatonin is made in human mitochondria. It is not your pineal or your gut. Your central retinal pathways have more mitochondrial density in it than any other part of the brain. The same is true with DHA to run your SCN faster than the other molecular clocks in your body to meet relativity needs. Want more info on exogenous melatonin? Use Yandex search with my name and mitohack #722. Your welcome in advance.
2. Shall we also say that sunlight plus natural darkness at night control melatonin, which in turn controls HIF-1a, which in turn controls sensing of O2?
Guess what happens to your mitochondria when oxygen tensions change? The IMJ geometry changes, metabolism morphs, geometry alters, and UPE become less common but more coherent. Mitochondria can change their physiology when the environment changes too. This is a remnant of the GOE when we had chronic hypoxia. this is why we innovated HIF1 and linked it to the PER clock genes.
Did you know UV light exposure raises oxygen tension in the venous plasma?
Guess what that all implies?
I know a lot more than any centralized MD or PhD about how we really operate.
If you have a T1D child you have a light problem. That light problem has manifested in your germ line.
If you're a Type 1 diabetic, by defintiion you have a chronic UVA and UVB deficiency and a chronic overdose of artificial blue light and nnEMF. It is also axiomatic.
Look at the chart below. T1D is almost nonexistent near the equator
2. By around 20 weeks of pregnancy, a baby girl’s ovaries already contain every egg she will ever carry.
Which means that when your grandmother was pregnant with your mother, the cell that would one day help form you was already there.
Three generations, held in one body.
This isn’t folklore. It’s embryology.
Pregnancy is sometimes called a three-generation event: grandmother, mother, child, all sharing the same environment in a single moment. Scientists call it multigenerational exposure. I call transgeneration biology.
3. But biology makes it hard to imagine these things don’t matter. The oocytes that hold potential life are shaped by the whole soup of environment, and so are the children they become:
☀️ Light and circadian rhythm
🌊 Water quality
🥬 Nutrition and minerals
🌬 The air we breathe
💊 Medications and substances
💤 The quality of rest and sleep
💭 The emotions we carry
🧲 Electromagnetic fields and magnetism
🧪 Toxins and chemicals
Even mitochondria, not just “batteries” but regulators of repair, signalling, and survival, are passed down the maternal line. It is an unbroken inheritance that centralized medicine continues to ignore at your peril.
Three generations are intertwined in every pregnancy in humans.
Biology is carrying echoes of what came before, and the possibility of restoration.
Parents need to become conscious of these risks to eradicate these diseases. The transhumanists seem to know, why don't the normies?