What do phospholipids do in mammalian cell membranes? They turn sunlight into a DC electric current to electrify the membranes. What does electrification of the membranes mean? Look at the top line of the slide below. With out this......nothing below it on the slide can happen. If you have this condition you cannot because it is an auto immune conditon related to poor sun light and too much nnEMF day and night. This thread contains that answer because it is linked to to the etiology of so many disease centralized MDs are impotent to repiar. ----> forum.jackkruse.com/threads/why-do…
2. Phospholipids help by preventing the accumulation of fats in the liver. It plays a major role in the transportation and removal of cholesterol from the cells. Fatty liver is not what many of the shills on the internet tell you. @MaxGulhaneMD
3. Properties Of Human Phospholipids
They are photoelectric signal mediators.
They are amphipathic molecules.
They anchor proteins within the cell membranes that are all voltage regulated.
They are the major constituents of mammalian cell membranes.
They are the components of bile and lipoproteins that tells us information if the surface and interiors of the cells are operating within electric and magnetic signals of the sun properly.
4. Phospholipids are compound lipids, consisting of phosphoric acids, nitrogen base, alcohol and fatty acids. These compound lipids are major components of the cell membrane and also provide a fluid character to the membranes. In cell membranes, these phospholipids have a hydrophilic head and a hydrophobic tail, which forms the inside of the bilayer.
There are two types of phospholipids
Glycerophospholipids
They are the major types of phospholipids, which occur in the biological membrane. It consists of glycerol-based phospholipids. Fatty liver disease is a disease that tells us about a light mismatch on the skin and eye.
Sphingophospholipids
They are the important constituents of myelin and are abundantly found in the brain and nervous tissues. It consists of sphingosine as alcohol. MS is a disease that tells us there is a photoelectric mismatch.
5. The fatty acid tails of phospholipids face inward towards the cytoplasm where other electric and magnetochemical biomolecules await instructions from the sun, away from water, whereas the phosphate heads face the outward aqueous side where water is acting like an electromagnetic capicitor. Since the heads face outward, one layer is exposed to the interior of the cell and one layer is exposed to the exterior.
6. Centralized medicine believes that the underlying cause of heart attack, stroke, and peripheral vascular disease, atherosclerosis is the major cause of death and morbidity in the United States and the industrial world. The discovery by Virchow more than 100 years ago that atheroma contained a yellow fatty substance, later identified as cholesterol by Windaus, suggested a role for lipids in the pathogenesis of atherosclerosis. Centralized medicine has not moved one iota from this idea.
I have moved light years past this idea in decentralized medicine. Why? I understand light. Phospholipids are photo electric structures. Lipids are not the problem.........LIGHT is. The light you choose to live under and imbibe is the cause of PAD.
7. I believe people must understand history to understand how to embrace decentralized thinking and make it their lexicon. Centralized fiat incentives determine outcomes in all aspects of human existence.
Here is a lesson for you on academia, and I hope you take it well.
1.
2. My response to 2 captured scientists of centralization:
3. Tom, a student of mine, said, "Robert Maxwell poisoned the well for profit in a true centralized fashion. It’s incredible what peeling back the onion reveals." My response:
Distill the decentralized lessons: I believe people must understand history of medicine to understand how to embrace decentralized thinking and make it their lexicon. Centralized fiat incentives determine outcomes in all aspects of human existence. Thius is especially true in HEALTHCARE. The scientific method isn't worth the paper it's printed on these days because none of is true. It is a digital ledger of PhD nonsense of what corporations have bought and paid for and has been allowed in by experts hired by Federal agencies captured by the government to print what they see fit. To print the narrative, they want the sheep to follow.
Taking a sabbatical from lying is the only thing that may save your life from disease & death, but it may also save your life in the end.
What has my time in medicine taught me?
That my pen is mightier than my scalpel because my pen is filled with ink that writes decentralized ideas down that make others realize that they have been lied to for decades.
8. I'll give you a simple example of how detailed & decreeing you have to be to be in my tribe. Look at the abstract title below. Normally you'd expect I'd love a paper with this title right?
But there is a key detail that is WRONG. The clue is highlighted in blue by me in the abstract pic.
Do you know what was wrong?
9. What is wrong with the abstract?
What does quantum thermodynamics says about the flow of energy based on Arrhenius 4th law of thermodynamics and Pirogene's dissipative state ideas in physics?
In physical chemistry, the Arrhenius equation is often now referred to the fourth law of thermodynamics and it is a formula for the temperature dependence of reaction rates.
Arrhenius provided a physical justification and interpretation for the formula. It can be used to model the temperature variation of diffusion coefficients, population of crystal vacancies, creep rates, and many other thermally induced processes and reactions. Isn't temperature something else the circadian clock genes pay attention too? Yep. This tells you light and temperature are both important and if they are then this implies timing is critical as temperature and light vary. The Eyring equation, developed in 1935, also expresses the relationship between rate and energy. Rate is a term that implies timing, doesn't it?
Go listen to Pirogene's Nobel speech. Notice what he says about time? Two for two.
It is not all about energy even though it appears to be. Same thing why it is not all about food either. Food is not medicine without consioderation of timing. It is about how the system experiences time. The paper is not quite right. This is the decentralized truth. Details matter. They demand your focus.
Timing controls how energy flows in the matter in cells because that is NATURE's decentralized truth.
10. My work in quantum biology is a very difficult read for many. This science has patterns & references to physics and physical chemistry, which usually repel centralized humanists/transhumanists. But do not be afraid to enter my realm. Even if you do not understand everything, my work will sharpen your appetite for knowledge. This is why I will never dumb my work down for the centralizd dumbed down masses. They need what I know and I am going to force it down their throats. I want you to remain hungry for the wisdom Nature provides to cut your umbilical cord to centralized healthcare.
There is a well-known theorem of minimum entropy production derived by Prigogine, which states that entropy exported from a system reaches a minimum, or becomes zero, at thermodynamic equilibrium and at steady states close to thermodynamic equilibrium. Prigogine's theorem is a direct consequence of Onsager's reciprocity relationship which holds at steady states close to thermodynamic equilibrium. The principle of internal entropy compensation, is in addition to, and implies the principle of minimum entropy production, and may even be valid in regimes far from thermodynamic equilibrium.
He had some very interesting things to say about TIME in his work and Nobel Prize speech. I think they are key to understanding circadian biology and timing in dencentralized networks in cells. All of our time reversible equations in physics created by Newton, Einstein, and Schrödinger describe a simplification of what actually occurs in nature. We live our lives with eyes blinkered, dismissing reality as the exception to our neatly formed approximations of what time really is. nobelprize.org/prizes/chemist…
11. Prigogine defined dissipative structures and their role in thermodynamic systems far from equilibrium, a discovery that won him the Nobel Prize in Chemistry in 1977. In summary, Ilya Prigogine discovered that the importation and dissipation of energy into chemical systems could result in the emergence of new structures (hence dissipative structures) due to internal self-reorganization. In his 1955 text, Prigogine drew connections between dissipative structures and the Rayleigh-Bénard instability, and the Turing mechanism. Prigogine's theorem is germane to these ideas.
Mitochondria are dissipative structures in cells, but not the only ones inside of cells. They transform energy and create order from the disorder in light energy they use to operate. The water mitochondria create is probably the single most important dissipative structure that life is based upon in cells. According to Prigogine, determinism loses its explanatory power in the face of irreversibility and instability in dissipative systems. This is a major departure from the approach of Newton, Einstein and Schrödinger, all of whom expressed their theories in terms of deterministic equations.
Indeterminism is the opposite of determinism and is related to chance. Chance is related to probability. In science, most specifically quantum theory in physics links directly to probability and not cause and effect. Now go listen to what I said two weeks ago in Prague.......ABOUT TIME. @BTCPrague
@MaxGulhaneMD 12. I'm re writing the biology books using the physics of time. That is where the truth lies. Few see where I am going. It is time they do.
13. You dont need diverse research.......that is corporate cronyism DEI BS.
They want to dilute science just like they diluted PEER reivew to keep us all from the truth.
You have to make the acurate diagnosis when the public is sick.
BigHarma wants to censor real speech in science because it goes to the hear tof their monopoly and they sit next to the money printer = Cantillon effect 101.
@MaxGulhaneMD 14. We need concentration on science that focus specificially on Time.
15. Just how ignorant are the centralized experts.......unfathomable.
Are they any other reactions like Vitamin A in mammals? Can I introduce you to Vitamin D?
Production of Vitamin D in the Skin is electromagnetic like a ripe fruit falling from a tree.
During exposure to sunlight, the ultraviolet B photons with energies between 290 and 315 nm are absorbed by provitamin D3 (7-dehydrocholesterol) in the skin. This absorption results in a photolysis of the B-ring of provitamin D3 resulting in the formation of previtamin D3.
However, because previtamin D3 is thermodynamically unstable, it quickly undergoes an isomerization (rearrangement) of its triple bond system to form vitamin D3. Do you see right here in the sentence that the words unstable & quickly are used. These words imply timing is involved, don't they? Has centralized science accounted for this?
This photo-isomerization process is enhanced in skin cells because the previtamin D3 is synthesized in the cell membrane of mammals when it is fully electrified. When this happens it restricts its movement making sure UVB photons interact with the cholesterol molecule thereby accelerating the transformation of previtamin D3 to vitamin D3. Once vitamin D3 is formed in the skin cell membrane, it is photoelectrically release like when an apple falls from a tree to the ground. This is the fractal aspect of nature from the nano to the macro levels no one visualizes. When the chemical is photo readied by UVB light conversion it is no longer restricted in its movement and freely translocates into the extracellular space to find its way into the dermal capillary bloodstream where it is bound to a specific vitamin D-binding protein
16. A picture above is a schematic representation of the photochemical and thermal events that result in the synthesis of vitamin D3 in the skin, and the photodegradation of previtamin D3 and vitamin D3 to biologically inert photoproducts.
7-Dehydrocholesterol (7-DHC) a seasonal non visual photoreceptor in the skin is converted to previtamin D3 by the action of solar ultraviolet B radiation. Once formed, previtamin D3 is transformed into vitamin D3 by a heat-dependent (ΔH) process. When heat is involved it implies so is timing.
Vitamin D3 exits the skin into the dermal capillary blood system and is bound to a specific vitamin D-binding protein (DBP). When previtamin D3 and vitamin D3 are exposed to solar ultraviolet B radiation, they are converted to a variety & myriad of photoproducts that have little or no activity on calcium metabolism. This picture was found in Holick MF (1995) Vitamin D: Photobiology, metabolism, and clinical applications. In: DeGroot LJ, et al. (eds.) Endocrinology, 3rd edn, pp. 990–1013. Philadelphia: W.B. Saunders.
When human skin is exposed to sunlight, it is the solar ultraviolet B photons with energies between 290 and 315 nm that are responsible for causing the photolysis of 7-dehydrocholesterol to previtamin D3. This photochemical process occurs in the plasma membrane of keratocytes; as a result, the thermodynamically unstable cis, cis isomer of previtamin D3 is rapidly transformed by a rearrangement of double bonds to form vitamin D3.
Approximately 50% of previtamin D3 is converted to vitamin D3 within 2 hours. See time plays another role in biology of mammals. As vitamin D3 is formed in the membrane, its open flexible structure is likely jettisoned from the plasma membrane into the extracellular space. Once vitamin D3 enters the extracellular fluid space, it is attracted to the vitamin D binding protein (DBP) in the circulation, and thus enters the dermal capillary bed.
Is there another layer of complexity linked to time?
Why does Vitamin D receptor vary in thermal reactions? It turns out evolution uses realtivity and quantum mechanics in the Vitamin d receptor types. This is why there is existence of different forms of the 1,25-dihydroxyvitamin D3(1,25-(OH)2D3) receptor in mammals. The classic nuclear receptor for 1,25-(OH)2D3 is associated with nuclear genomic activity and their is another uncharacterized membrane receptors for both 1,25-(OH)2D3and 1,25-dihydroxyprevitamin D3(1, 25-(OH)2preD3) associated with non-genomic activity in mammals. One can get through the nuclear membrane and the others cannot. Timing yet again is why this occurs.
Look at the picture below. Stringing the lessons together yet?
TIME IS EVERYWHERE WHEN YOUR PERSPECTIVE GETS DOWN TO THE NANO LEVEL.
@MaxGulhaneMD 17. I hate ignorant people who think they are wise and I challenge them like a great white shark challenges their prey. I do it for the public's health.
Centralized healthcare is the biggest predator on the Planet for longevity.
18. How to get past the nuclear membrane once you make Vitamin D in the skin electromagnetically?
Sunlight energy is also used to sulfate cholesterol FIRST to get the Vitamin D train rolling in the skin to the blood to get to nDNA. When it is sulfated it becomes water soluble in blood. When it isn't sulfated, your liver responses to protect the nucleus in cells and it upregulates LDL production to act like a sponge to protect the AMO physics inside of cells. So, when your centralized dermatologist tells you to stay out of the sun and take the secosteroid Vitamin D from a plant or animal it is also packaged in a seed oil because it is normaly a fat soluable compound. Few realize that the simple act of taking D3 pills and living indoors behinds glass walls is raising your LDL cholesterol constantly. bit Harma knew it and this is why Big Pharma taught every doctor since they found the evidence in the 1950s military documents that they could profit massively by training doctors ignorant of light's effects to put their patients on statins. They knew to blame it on biochemistry gone awry and they baited the PhDs with the papers on the melavoant pathway and Q cycle. When you review the history of the evidence you know they are playing you like a fiddle. You are their mouse and they are the cat. Few see the game plan, and less of you get it.
Cholesterol and Vitamin D3 are nearly identical in chemical structure. Most people do not know this. Sunlight naturally sulfates these things (pic below). The only difference in both bio-molecules is a single double bond in the second ring of the cholesterol backbone. Remember light is BURIED at the electronic level in all things including cholesterol and Vitamin D. Your pills are sold to you soaked in seed oil do not. this is done by design because BigHarma knows what deuterium vs hydrogen can do. They've know about the effects of deuterium in drug delivery because of the the physical chemistry trials they have done. They never disclosed this to their curriculums to medical students or pharmacy students by design. This is why they want centralized pharmacy like WalMart, CVS, and Walgreens versus compounding pharmacies. When the drugs are ready made and all one needs to do is count your chemistry skills atrophy. A compounding pharmacist is an alchemist of physical chemistry.. They are more apt to realize what Pfizer and Moderna are really up to in New Jersey factories. I told @nayibbukele this and I put that in my law for El Salvador.
19. The AMO physics of these two biomolecules are nearly identical with one exception. HYDROGEN. This gives Vitamin D3 one less hydrogen atom than the closed ring of cholesterol has. If you spent anytime reading my Tensegrity 6 blog about my original hacks of the periodic table I did 20 years ago it should make you think a lot more carefully about atomic details.
One hydrogen is the only difference at the atomic scale between them, but from the quantum thermodynamic perspective you learn that there is a shit ton of energy buried in hydrogen's bond at the electronic and vibrational level. These are the bonds that food gurus and biochemists do not see, much less understand, so they ignore it at your peril. Here is why the public health goes awry because of a lack of focus on really what does matter in a cell. Here you will see the wisdom of my lessons in the Tensegrity 6 blog at play. Look it all the charge density sunlight adds to the system that a pill cannot. See, never divorce biophysics from biochemistry. If you do, you lose and it is through this crack that chronic disease epidemics begin.
20. The photonic energy in sunlight is used to oxidize hydrogen sulfide to make sulfate. This process sulfates everything at our surfaces and just below the surface and this makes many things water soluable and lowers charge density. Sunscreen was innovated by BigHarma to block sulfation by design. When you block sulfation you create a need for more drugs because you lower the tissues in energy and this exacerbates the timing mismatch in mtDNA. This fuels more mitochondrial mutations and creates disease phenotypes which fuels their centralized business models A cursory review of the what sulfation does to photovoltaics tells you how Nature uses sulfation to get past a highly charged membrane. It discharges the charge density to ruin the capacitance of the Vitamin D. This is how you enter the nDNA environment like a ghost. Nature has more magics it uses in sunlight. Melanin, Vitamin D, cholesterol, heparin, and nitric oxide all respond to the UV part of the spectrum when it is in sunlight. Few people know that the UV, and blue green part of the spectrum work in unison to very locally vasodilate blood vessels in the skin. I showed people the the melanopsin 2014 paper 1000's of times that showed blue light is capable of dialating blood vessels but not one person ask me about the AMO physics of the interaction.
21. This area interested me as a neurosurgeon because I was looking for a mechanism to explain the queer things that happen in brain bleeds called subarachnoid hemorrhages. We see them in trauma and in aneurysm ruptures. I have always felt that a lack of light operating in unison is behind most of the cerebrovascular pathologies that neurosurgeons treat. It took me ten years to figure it out, but altered light signaling is why AVMs and aneurysms rupture. It is also why some people develop SAH with minor trauma. I know I have been up all night taking care of these patient now and I am tired. I sacrificed my time for money to pay off legal bills to keep this info flowing to you clowns.
22. They are all symptoms of low redox power in the brain. Nitric oxide (NO) is a well-known “gasotransmitter” in the literature because a Nobel Prize was given for its discovery in 1992. It is a gas that acts focally and locally when it is released. It does not have global effects. This tells you something about TIMING too. It acts RAPIDLY, not CHRONICALLY. Vitamin D actions act CHRONICALLY.
This tells you that NO signaling is likely behind many light induced chronic diseases. Electriified membranes absorb gas and turn it into many other biomolecules. Life figured this out 3.8 billion years ago. NO is a gaseous signaling molecule that has a unique ability to induce a relaxation of the artery wall and a resulting drop in blood pressure. Recalll from my "Kruse for Dummies" lecture what I said about unique signals. They are how information is transferred in biological systems by Claude Shannon's theory of information.
Every one else is playing checkers looking at biochemical pathways while I am playing 4D chess because I understand how light operates in physical chemistry. Without full spectum sunlight, endothelial dysfunction and peripheral arterial damage should be EXPECTED by the decentralized clinician. It is not. No one in medical curriculums looks at the data in BigHarma literature to see this data much less understand the clinical significance.
UV light increases NO which lowers ATP and raises ROS/RNS. Both are incredibly important. It is the key photoswitch of mammals.
23. A lack of NO production at our integument and eye surfaces link PAD directly to cardiovascular dysfunction. The local effect become generalized in the entire organ as the lack of NO production gets worse under ALAN or nnEMF influence.
Peter Attia still does know this and he is printing money selling you statins and jabs.
This is why PAD is always linked to cardiovascular disease. The link is the aberrant use fo the electromagentic spectrum to communicate to create NO. Modern light and RF and cell radiation impairs production of NO from arginine by eNOS.
This, in turn, induces high blood pressure by causing endothelial dysfunction. Mitochondria are intimately involved in importing nitorgen into tissues to create the substrates that eventually become NO when sunlight is present. Nitrogen substrates are not created from the direct synthesis by eNOS.
Nature provided the clue to me why humans got rid of Vitamin C for glutathione in this AMO physics dance. When Vitamin C is missing in subcutaneous tissues, glutathione become more reactive with locally produced NO. This mimics a radical pair/triad effect we see in avian compass navigation.
Here is more evidence of magnetochemistry in humans being used. When glutathione and nitric oxide are powered by terrestrial sunlight this allowed humans to produce S-nitrosoglutathione (GSNO).
I think this is why human primates lost the majority of their integumentary hair and absorbed more melanin from the hair follicle to the interior. This is why Peter is bald as he works out in his blue lit gym with airpods in his ears and wants you to believe a 100 push ups some how saves you from all this? That is lunatic level thinking, in my opinion based on the science of NO.
This allowed the skin to become a better charge capacitor for the brain and heart by allowing the skin to become a depot station to store massive amounts of nitric oxide. This is why human immune T cells are so common in the skin and why leptin was placed in SQ fat. This is where anti-phospholipid sysndrome and all other auto-immune conditions come from.
Other primates do not have these phenotypes even thought their genomes are close to identical. This tells me magnetochemistry timing induced this evolutionary change. We never need genes to change this. We used timing to change the metabolic pathways in the skin using hair loss and removal of Vitamin C from the radical triad mechanism to do it.
As a consequence of this dance using more light on the skin, keratinocytes were able to sense more visible light combinations with purple, blue, and green light to easily photocatalyze the release of NO from glutathione. Not only does this cause a relaxation of the blood vessels, but it also frees up glutathione to react with hydrogen sulfide gas to produce sulfate to make every other chemical in the skin water soluable to get access to body parts to have global effects in distal organs. This is why PAD is linked to so many diseases.
This is how light develops its abscopal effects. No one has figured out how this all works in humans but this is how I have seen it for 20 plus years. To date no one has published a thing using my ideas. But I can explain why subtraction of Vitamin C in humans links to hair loss. This explains most of the integumentary and ocular diseases we see today. It also explains obesity, too. It explained to me why humans get aneuysms and AVMs.
People need to wake the fuck up and stop making stupid people famous in healthcare. We need the decentralized truth spread like a virus.forum.jackkruse.com/threads/why-do…
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Deuterium's Quantum Dual Nature: Bosons vs. Fermions
At the core is deuterium (D or ²H), an isotope of hydrogen with an extra neutron. My blog explains its particle statistics: Nucleus: A boson (spin 1, even parity), following Bose-Einstein statistics. Bosons can occupy the same quantum state, enabling phenomena like superfluidity or Bose-Einstein condensates at low temperatures.
Atom: A fermion (half-integer spin, like 1/2 or 3/2 ħ when combined with an electron), obeying Fermi-Dirac statistics and the Pauli exclusion principle. This makes deuterium atoms behave like "individualists" that can't pile up in the same state. This is why biology is racist against their use in mitochondria. The raise energy resistance.
Molecule (D₂): A boson again, due to symmetric nuclear spins in the ground state. It forms antisymmetric (para-deuterium, spin 0 or 2 ħ, even L) and symmetric (ortho-deuterium, spin 1 ħ, odd L) states.
This contrasts with ordinary hydrogen (H₂), a fermion in its para form (spin 0) and boson in ortho (spin 1), leading to different behaviors at low temperatures. Cooling deuterium near absolute zero produces pure ortho-deuterium with residual spin-2 fractions that can't be fully removed, unlike hydrogen's para form with zero angular momentum.
The takeaway: Deuterium's bosonic tendencies make cold D₂ act like a collective fluid (Bose-Einstein-like), while H₂ follows fermionic exclusion. This affects heat capacities, neutron interactions, and low-energy systems—relevant for fusion, cryogenics, and, as we'll see, biology------> All covered here. optimalklubs.com/kruse-for-dumm…
2. Biochemical Implications: Hydrogen Isotopes in Melanin and Metabolic Pathways
The diagram links this to biology, showing a pathway from acetyl-CoA (a ketone precursor) through melanin dissociation, involving clock genes, sunlight, and electromagnetic spectra. Key points:Oxygen (O₂) from NADP⁺/NADPH cycles feeds into tryptophan hydroxylation, producing intermediates like 5-hydroxytryptophan (5-HTP) and 5-hydroxytryptamine (5-HT, serotonin).
Hydrogen ions (H⁺) are central, explicitly noted as "THIS HYDROGEN CAN BE H+ or D." Protons or deuterons from the pentose phosphate pathway (PPP) influence steps like aromatic L-amino acid decarboxylase, leading to glucogenic amino acids (e.g., alanine) or neurotransmitters.
Deuterium's heavier mass (twice hydrogen's) slows kinetic reactions (kinetic isotope effect), disrupting enzyme kinetics, and the dielectric constant of the metabolic pathways altering UPE function, mitochondrial function, and water structuring in cells. The diagram implies sunlight-driven melanin creates a "plasma" for harvesting electromagnetic energy, with H/D isotopes affecting unique codes in water and biomolecules.
3. Connection to deuterium's physics: In biological "condensates" (e.g., exclusion zone water around proteins), bosonic deuterium has to enable collective behaviors, like enhanced proton tunneling or spin-dependent reactions.
This fully explains why deuterium depletion (e.g., via diet) is UNDERexplored in health contexts—high deuterium foods must hinder fermionic electron flows in mitochondria, while low levels promote bosonic fluidity. This is the basis of low energy resistance and high energy resistance.
Here's how this idea flows:
Low éR = Efficiency and Flow: In healthy states, like during deep sleep or a good workout done in sunlight with proper recovery, éR is dialed down. Energy transforms smoothly, think laminar flow in a river, minimal turbulence. Your cells rebuild, adapt, and thrive. Physics analogy: It's like a superconductor with near-zero resistance, letting current (or bioenergy) zip through without loss.
High éR = Stress and Stagnation: Push too hard in a blue lit gym with nnEMF in your ears, via light stress or chronic stress of any type, poor diet, use of supplements, or disease states, and éR builds up like traffic jams in a circuit. Energy dissipates as heat, damage, or inflammation, accelerating entropy at the molecular level. This links to aging (those "hallmarks" like genomic instability) and diseases (e.g., mitochondrial disorders causing brain fog and exhaustion).
Biology tie-in: It's why exercise feels good in sunlight in moderation but wrecks you if overdone under blue light in agym it is because éR rises, signaling "back off and recover." Blue light allows deuterium to flow into the mitochondria and the KIE takes over.
The retina thins from the effect of man-made light and a lack of sunlight, and then your risk of Frontotemporal dementia rises. The OCT test is the best screening test for FTD. We have familial and mtDNA FTD.
The retina acts as a ‘window to the brain," and that window changes the photolithography of the retinal cells. When the light is not optimal, the retina thins and the brain degenerates. Retinal degeneration has been detectable in mutation carriers prior to the onset of cognitive symptoms, establishing retinal thinning as one of the earliest observable signs of familial (frontotemporal dementia).
nnEMF and a lack of sun lead to neural degeneration, which is primarily driven by the abnormal accumulation of misfolded proteins within neurons. Light controls the confirmation bending of proteins. The DNA code controls the AA sequence and secondary bending, but quantum processing determines tertiary and quaternary bending by UPE transformation in cells. The most common protein abnormality in FTD is the tau protein, also known as TDP-43. These protein aggregates damage and kill nerve cells, leading to brain atrophy (shrinkage) and the resulting behavioral, personality, language, and movement problems characteristic of FTD. I believe this protein also thins the retina. Light is the offender.
Blue light increases RBC turnover in the retina and changes the oxidation state of iron, and this affects the synthesis of new heme proteins in the retina and brain. Oxidative stress is a key trigger in this disease because it increases peroxide production (ROS), which alters the UPE signature of cells in the construction of the heme proteins, which changes the photolithography inside the retina and brain. Changing the photolithography = altered protein folding via the Golgi apparatus and RER. TDP-43 is a biomarker of redox imbalance in FTD-related UPEs.
The path of energy in life, based on the evidence we have as a species now, is that Optical photonics began 13.8 billion years ago. The photoelectric effect was born from the rudimentary physics present in the early universe. Photonics precedes all chemistry of all types. Functional medicine does not realize this or teach it. Molecules were and are innovated by redox chemistry, and each one has an electromagnetic barcode to program its possible states around the star it evolved in.
The TDP-43 molecule is codified by light in its absorption and emission spectra. This links it to aberrant UPE production in the central retinal pathways, as blue light increases heme turnover to alter its biophotonic signal. Look at all the places a UPE can be made from in a cell to cause this disease on the slide. You'll also see that light carries spin information that can create this disease.
Where there is a change in matter (TDP-43), there must also be a change in atomic molecular geometry in that tissue. Where normal cellular geometry ceases to exist, we will find light as the key agent of change. When both exist in simultaneity, you'd better understand Fermat's law and the photoelectric effect to understand what a disease really is. Right now, in centralized ophthalmology, none of them do. See Bruce Willis.
THE BIOPHYSICAL CODA of FTD: When Turing realized morphology in all living things had a photonic timeline in Nature, he wrote a key paper on morphogenesis. Light signaling predates biochemistry and molecular creation from redox chemistry. This paper suggested that a system of chemical substances, called morphogens, could react together as a symphony does and subsequently diffuse their electromagnetic, electrochemical, and photobiolelectric information through a tissue to sculpt it, changing its morphology without using the nuclear genome.
Turing stated in 1951 that this set of circumstances is adequate to account for the main phenomena of morphogenesis. In such a dissipative system, although the cells may originally be quite homogeneous at the embryo stage, this information upload may later develop a pattern or structure in the embryo due to the instability of the homogeneous equilibrium caused by light pollution in the parents' germ line, which is triggered by random disturbances in the tissue. Those random disturbances are UPEs. The random disturbance he hypothesized about turned out not to be random at all. UPEs are created in cells by light AMO interactions via redox chemistry that directs their own sculpting from the nuclear genome. The UPE energy transformation is driven by the mitochondrial genome, which is subject to light in the environment to create UPEs and eventually by reactive chemicals like ROS/RNS inside all cells. Any disease linked to aberrant UPEs will always present as having a spectrum of maladies. FTD has that optical signature. ncbi.nlm.nih.gov/books/NBK55928…
2. The GLP-1 Drug Connection: Weight Loss at a Cost to Your Eyes and Ears
GLP-1 drugs like semaglutide (Ozempic, Wegovy) and liraglutide (Victoza) are prescribed for type 2 diabetes and weight loss, helping millions shed pounds by curbing hunger and stabilizing blood sugar. However, literature suggests that they can affect the eyes and even the inner ear (cochlea), which ties into the light biology disruptions we just discussed.
Effects on the Eyes
These drugs cause rapid weight loss, which triggers fluid shifts and reduced blood volume in the body.📷This can stress the delicate blood vessels in your retina, leading to:
Worsening diabetic retinopathy: In people with diabetes, GLP-1s can make existing eye damage worse, causing blurred vision or macular issues.
Increased risk of vision loss diseases: Studies link them to a doubled risk of neovascular age-related macular degeneration (nAMD), a leading cause of blindness.
There's also a higher chance of non-arteritic anterior ischemic optic neuropathy (NAION), where blood flow to the optic nerve drops, potentially causing sudden vision loss.
Blurred vision and other issues: Common side effects include visual impairment, which can start as early as 10 days after use. While not everyone experiences this (risks are low but real), regular eye exams are recommended for users.
How does this relate to light biology? The fluid shifts and oxidative stress from GLP-1s can amplify ROS production, disrupting UPEs and protein folding in the retina, much like exposure to bad light. This thins the retina, mimicking the early FTD signs, and could raise dementia risk indirectly through disrupted circadian rhythms.
Interestingly, while some research suggests GLP-1s might protect against general dementia by reducing inflammation, their eye effects could counteract that, especially in FTD-prone folks. My TED talk was banned because BigHarma was afraid I was going to obliterate their GLP 1 train by showing the world why they banned stopped the leptin trials early on weight loss.
3. Since the ear also processes sensory info tied to your body's clock, this could feed into broader brain degeneration.
Effects on the Ears (Cochlea)The cochlea, your inner ear's hearing hub, isn't immune. GLP-1s are linked to hearing problems like hearing loss, vertigo, tinnitus (ringing), and eustachian tube dysfunction (feeling of ear fullness).
Rapid weight loss can disrupt fluid balance in the ear, similar to the eyes. Diabetes itself raises hearing loss risk via blood vessel damage, and some GLP-1s (like exendin-4 types) heighten it further. The GLP target in the ear is the stria vascularis.
I tried to pull the veil back on the layers of the Deep State and where they are linked and why they are linked. It is a nasty story of deceit and ideological fascism where science was stolen to control modern humans with Light, drugs, and jabs.
The historical links you must know. Why is Israel so misunderstood? Propaganda is its shield, so you can never know who is really behind it.
It is British Imperialism dressed in drag to protect its ideology of fascism, which is what a Fabian really is.
The billionaires of today are not the architects of Zionism
They’re the actors. Elon. Thiel. Gates.
They run infrastructure, but they don’t own the world.
They were groomed by systems older than corporations.
The real owners?
• BlackRock, Vanguard, BIS — own the assets
• Rockefeller, Rothschild, DuPont — own the timeline
• WEF, CFR, Chatham House, Fabians — write the script
• Think tanks + foundations — enforce ideology
Occult orders + Straussian elites — justify it all as “destiny”
This is not capitalism.
This is a technocratic theocracy—where power is hidden in algorithms, law, debt, and narrative.
They don’t fear elections.
They fear recognition.
They fear you finding out what their plan really is.
Why must we decentralize medicine?
Because it eliminates centralized Rockefeller Medicine from the World.
That is a world where drugs and jabs are used as weapons of control.
Time to wake up and do your diagnosing better today than you did in the past.
2. True Evil with assorted atrocities most often occurs when the most respectable and intelligent of people fall for it. The wise never do. They sniff out and diagnose the problem of centralization to avoid collateral damage. Be careful who packs your parachutes.
3. What is modern gaslighting of the jab injured really? It is cloaked in pardons and Nobel Prizes given to the architects of the crimes.
I started a huge Barista FIRE on the boat it appears with my Bitcoin talks with him every AM. He apparently was quite influential with the young staff and told them all they were working communist hours for communist pay, and when his manager heard it spill over to our morning conversations and how many of the krewe I am Orange pilling, they axed him. He was escorted off the book quickly in Peru, and I never knew it until this AM. Some of his barista folks confirmed it got hot quickly.
What Is the FIRE Movement, and Where Does Barista FIRE Fit In?
FIRE stands for “financial independence, retire early.” The FIRE movement puts forth the idea that becoming work-optional isn’t about reaching a certain retirement age; it’s about having enough money invested that the compound interest gains can sufficiently cover your annual expenses. Bitcoin really changes the mix for young people locked into communist like employee environments.
This is achieved by reaching what is called your FIRE number. A (very) rough calculation of FIRE number is to multiply expected annual expenses when no longer working by 25. This is the amount of retirement savings you’ll want to have ready to tap, but know that performance on investment accounts can vary widely from year to year. A traditional FIRE number is typically well north of $1 million.
There are many people who don’t actually want to stop working and enter full retirement. Instead, they want to downshift to doing more fulfilling work, or they want to be able to work part-time so that they can spend more time pursuing hobbies or passions. In jobs like medicine and cruiseship workers the math works rapidly but for different reasons. So when I explained this to Christopher he seemed to catch fire like I had poured diesel fuel on him. Never thought he'd get canned for it that fast. I guess capitalism is a bad antidote for the communist boat life.
2. The Origins of the FIRE Movement
In 1998, three researchers at Trinity University published the results of a study on retirement savings.
Their projections found that, if an investor had a certain multiple of their income saved up and withdrew 4% or less of their nest egg each year, their chances of depleting their savings in a 30-year period were zero. I re-did this calculation in 2013 and added Bitcoin CAGR to the mix, and the results were more stunning. That is when I realized I could bail on centralized medicine and began teaching this method to my MD clients. Sadly, most of them crumbled because they could not fathom walking away from their jobs because of their programming. The Trinity research group was based on rates of return since the invention of the 401(k) and other tax-advantaged retirement accounts, and later became known as the "Trinity study."
3. The results of the Trinity study were first published in the American Association of Individual Investors Journal in 1998.
This research led to my reframe: If you could grow your nest egg large enough that the interest alone covered annual expenses and other medical expenses, most or even all of your principal would be protecting, preserving your wealth. I tweaked this big time. I realized that the decentralized mitochondrial life could bring medical expenses to zero and then I could power up my savings with the Bitcoin CAGR i could make money grow on trees for decades. IT was how I figured out that the one thing killing me, my job, could be eliminated rapidly. I got a huge settlement from a cruise incident related to a paleo meathead, and I yolo'd it into the program, and within a year, I found out this was going to be life-changing.
The Centralized Fiat Retarded Say Money Doesn’t Grow on Trees......but decentralized geniuses know it happens.
If your tree is a large enough nest egg due to the Bitcoin CAGR, it actually does. I began to realize that the traditional retirement age wasn’t dictated by age, but by whether you reach financial independence. Thus, the ORANGE FIRED MD lifestyle was born.
FIRE stands for Financial Independence, Retire Early. Over the years, however, many ORANGE FIRED MDs Savages began to break from core FIRE principles and define FIRE variations that better suit their lifestyle goals. I helped many of my tribe do just that. Membership matters in this tribe.
1. ANSWER: In ALAN contexts, blue light suppresses melatonin, which normally inhibits AVP and cortisol, leading to unchecked vasopressin release during "light stress." This causes a reactive hyponatremia in the posterior pituitary, activating brain osmoreceptors and RAAS, raising blood pressure and stress hormones, which fragment sleep and impair daytime recovery. They also chronically degrade the mitochondria in these neural tracts.
2. Now for the dirty details.
Precise Mechanisms of Sodium Regulation in the Brain and Circulatory SystemSodium (Na+) homeostasis is tightly regulated to maintain osmotic balance, blood pressure, and neuronal function, with disruptions like hyponatremia (serum Na+ <135 mEq/L) directly impacting sleep via arousal, cognitive fog, and fragmented rest. The brain acts as the central sensor and regulator, while the circulatory system executes adjustments through hormonal and renal pathways.
Brain's Role in Sodium Sensing and Control: The hypothalamus, particularly osmoreceptor neurons in the supraoptic and paraventricular nuclei, continuously monitors plasma osmolality and Na+ levels via stretch-sensitive ion channels. When hyponatremia occurs (e.g., from ALAN-induced vasopressin overrelease diluting blood Na+), these neurons trigger arginine vasopressin (AVP, or antidiuretic hormone) secretion from the posterior pituitary.
AVP promotes renal water reabsorption via aquaporin-2 channels in the collecting ducts, conserving water but exacerbating dilutional hyponatremia if unchecked. In the brain, low Na+ also activates the subfornical organ and organum vasculosum of the lamina terminalis (circumventricular organs lacking a blood-brain barrier), which integrate signals from baroreceptors and chemoreceptors to modulate thirst and salt appetite. Chronic hyponatremia impairs neuronal excitability, leading to symptoms like headaches and insomnia, as it alters membrane potentials and synaptic transmission.
Circulatory System Integration with Hormones: In the periphery, low Na+ (hyponatremia) is sensed by renal juxtaglomerular cells, triggering renin release, initiating RAAS: renin converts angiotensinogen to angiotensin I, then II, which stimulates aldosterone from the adrenal cortex to enhance Na+ reabsorption in the distal nephron and potassium excretion. This raises blood pressure to restore volume but can cause nocturnal hypertension, disrupting sleep architecture (e.g., reduced REM). Cortisol, released from the adrenal cortex in response to stress signals (including ALAN and hyponatremia), amplifies this by promoting Na+ retention via mineralocorticoid receptors and enhancing sympathetic activity, increasing heart rate and arousal. Vasopressin interacts with cortisol and RAAS; for instance, AVP potentiates cortisol's effects on water balance, while cortisol feedback inhibits AVP in healthy states, but ALAN disrupts this, leading to unchecked stress responses. Atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP) counterbalance by promoting natriuresis (Na+ excretion) when blood volume rises, but in hyponatremia from SIAD (syndrome of inappropriate antidiuresis), this balance fails, perpetuating low Na+ and sleep disturbances like frequent awakenings.
3. So Emily, what you should do is see the AM sunrise every AM. Drink DDW water and add high-quality salt to the water, and within 2-4 weeks, you will sleep much better. These slides support the tweets, and the last one with the D20 water spectrum really hits the mark.
A couple of points: Not all human cells have mtDNA. See adult blood cells. This is a big deal when you consider their absorption spectra. 200-600 nm light with a sharp cut-off. Also, blood still transforms energy into UPEs. And since blood fills 20% of our CO to the CNS, this also has implications. The retina and gut increase their blood flow with light use and feeding. Feeding is a light-based phenomenon since all food webs link back to photosynthesis.
In humans, light stress does not force mitochondria to react exactly the same way as infection (no ATF4 surge, different folate handling), but there are partial overlaps in ISR activation, 1C remodeling, and mtDNA signaling.
This suggests UV light acts more as an external folate depleter for DNA protection/photorepair, while infection is an internal ATF4-orchestrated defense. If environments lack natural light controls (as in modern human life), it should exacerbate mismatches in folate biology. For deeper dives, I'd recommend reviewing the ATF4-UV papers for experimental details.
Folate as a Light-Responsive Switch: My patreon has made this point over and over again, natural folate (not folic acid) is photosensitive, degraded by UVB, and shows seasonal lows in summer (higher at low latitudes, with gender differences showing men have lower levels). This would act as a "switch" for mitochondrial/genome control, linking to melatonin/tryptophan pathways and neurotransmitter synthesis (e.g., serotonin, dopamine). In low-light/artificial blue light environments (e.g., indoors), folate stability increases unnaturally, disrupting methylation/DNA synthesis and contributing to diseases like Alzheimer's, Parkinson's, or diabetes (via melanin quenching loss and superoxide buildup). People with MTHFR and COMT defects are supersensitive to ALAN and a LACK OF SUNLIGHT. This is echoed all throughout my decentralized photo-bioelectric thesis: light refines folate via photosynthesis/food webs, but artificial setups bypass this, leading to transgenerational effects.
No Seasonal/Light Controls: absent natural light cycles (e.g., constant artificial light), retinal/CNS signaling should be expected to dysregulate—e.g., excess folic acid fortification (since 1996 in North America), which overloads 1C pathways, causing cognitive haze, sleep issues, or neural migration problems in lit environments. This photonic effect is completely missed in centralized medicine and is really missed in functional medicine, which pushes for excessive folate use with SNPs. These people need more sun and less ALAN, not drugs.
Black Swan Mitochondriac View: Melanin (from UV-absorbing aromatic amino acids) quenches mitochondrial superoxide, protecting against neurodegeneration. Blue light destroys melanin and heme proteins, amplifying risks from ALAN and a lack of sunlight which aligns perfectly with UV's mitochondrial stress but without the adaptive folate restriction seen in infection.
2. Why is this tweet important to the jabbed? If you follow the work of @Kevin_McKernan you'll find in his blogs many mentions of pseudouracil and jab injuries. Then you look at my blogs on jab repair, and you'll notice I recommend Tropical relocation as the best risk reduction for the compliant. WHY?
Did you know that in humans, UVR depletes folate in skin and blood, inducing S-phase arrest, affecting uracil misincorporation (frame-shift mutations), and sensitizing apoptosis in keratinocytes. This isn't "damage" but a permissive environment for seasonal phenotypic changes, e.g., increased melanin production to protect folate stores. When you understand the photorepair mechanism, you will see this is how humans can block the DoD and BigHarma death mechanism from uracil replacement induced by the jabs. @MdBreathe None of the COVID experts have this sophistication. They are still pushing detox answers when it is crystal clear that redox biology is the path for the Savage trying to stay alive and away from disease.
3. Individuals with MTHFR C677T (thermolabile variant) are indeed supersensitive to light/folate fluctuations, as the mutation impairs folic acid conversion to 5-MTHF. High folic acid may select for this allele evolutionarily, acting as a "genetic time bomb" by promoting unmetabolized buildup and altering DNA/RNA biology.
People who have this SNP and took the jab have no choice but to move. If they do not, they will be taken out to Taper the Ponzi. This variant requires higher folate for stability, making carriers vulnerable in low-UVR (winter/indoor) settings, where blue light further degrades melanin (a superoxide quencher), exacerbating mitochondrial stress.