What do phospholipids do in mammalian cell membranes? They turn sunlight into a DC electric current to electrify the membranes. What does electrification of the membranes mean? Look at the top line of the slide below. With out this......nothing below it on the slide can happen. If you have this condition you cannot because it is an auto immune conditon related to poor sun light and too much nnEMF day and night. This thread contains that answer because it is linked to to the etiology of so many disease centralized MDs are impotent to repiar. ----> forum.jackkruse.com/threads/why-do…
2. Phospholipids help by preventing the accumulation of fats in the liver. It plays a major role in the transportation and removal of cholesterol from the cells. Fatty liver is not what many of the shills on the internet tell you. @MaxGulhaneMD
3. Properties Of Human Phospholipids
They are photoelectric signal mediators.
They are amphipathic molecules.
They anchor proteins within the cell membranes that are all voltage regulated.
They are the major constituents of mammalian cell membranes.
They are the components of bile and lipoproteins that tells us information if the surface and interiors of the cells are operating within electric and magnetic signals of the sun properly.
4. Phospholipids are compound lipids, consisting of phosphoric acids, nitrogen base, alcohol and fatty acids. These compound lipids are major components of the cell membrane and also provide a fluid character to the membranes. In cell membranes, these phospholipids have a hydrophilic head and a hydrophobic tail, which forms the inside of the bilayer.
There are two types of phospholipids
Glycerophospholipids
They are the major types of phospholipids, which occur in the biological membrane. It consists of glycerol-based phospholipids. Fatty liver disease is a disease that tells us about a light mismatch on the skin and eye.
Sphingophospholipids
They are the important constituents of myelin and are abundantly found in the brain and nervous tissues. It consists of sphingosine as alcohol. MS is a disease that tells us there is a photoelectric mismatch.
5. The fatty acid tails of phospholipids face inward towards the cytoplasm where other electric and magnetochemical biomolecules await instructions from the sun, away from water, whereas the phosphate heads face the outward aqueous side where water is acting like an electromagnetic capicitor. Since the heads face outward, one layer is exposed to the interior of the cell and one layer is exposed to the exterior.
6. Centralized medicine believes that the underlying cause of heart attack, stroke, and peripheral vascular disease, atherosclerosis is the major cause of death and morbidity in the United States and the industrial world. The discovery by Virchow more than 100 years ago that atheroma contained a yellow fatty substance, later identified as cholesterol by Windaus, suggested a role for lipids in the pathogenesis of atherosclerosis. Centralized medicine has not moved one iota from this idea.
I have moved light years past this idea in decentralized medicine. Why? I understand light. Phospholipids are photo electric structures. Lipids are not the problem.........LIGHT is. The light you choose to live under and imbibe is the cause of PAD.
7. I believe people must understand history to understand how to embrace decentralized thinking and make it their lexicon. Centralized fiat incentives determine outcomes in all aspects of human existence.
Here is a lesson for you on academia, and I hope you take it well.
1.
2. My response to 2 captured scientists of centralization:
3. Tom, a student of mine, said, "Robert Maxwell poisoned the well for profit in a true centralized fashion. It’s incredible what peeling back the onion reveals." My response:
Distill the decentralized lessons: I believe people must understand history of medicine to understand how to embrace decentralized thinking and make it their lexicon. Centralized fiat incentives determine outcomes in all aspects of human existence. Thius is especially true in HEALTHCARE. The scientific method isn't worth the paper it's printed on these days because none of is true. It is a digital ledger of PhD nonsense of what corporations have bought and paid for and has been allowed in by experts hired by Federal agencies captured by the government to print what they see fit. To print the narrative, they want the sheep to follow.
Taking a sabbatical from lying is the only thing that may save your life from disease & death, but it may also save your life in the end.
What has my time in medicine taught me?
That my pen is mightier than my scalpel because my pen is filled with ink that writes decentralized ideas down that make others realize that they have been lied to for decades.
8. I'll give you a simple example of how detailed & decreeing you have to be to be in my tribe. Look at the abstract title below. Normally you'd expect I'd love a paper with this title right?
But there is a key detail that is WRONG. The clue is highlighted in blue by me in the abstract pic.
Do you know what was wrong?
9. What is wrong with the abstract?
What does quantum thermodynamics says about the flow of energy based on Arrhenius 4th law of thermodynamics and Pirogene's dissipative state ideas in physics?
In physical chemistry, the Arrhenius equation is often now referred to the fourth law of thermodynamics and it is a formula for the temperature dependence of reaction rates.
Arrhenius provided a physical justification and interpretation for the formula. It can be used to model the temperature variation of diffusion coefficients, population of crystal vacancies, creep rates, and many other thermally induced processes and reactions. Isn't temperature something else the circadian clock genes pay attention too? Yep. This tells you light and temperature are both important and if they are then this implies timing is critical as temperature and light vary. The Eyring equation, developed in 1935, also expresses the relationship between rate and energy. Rate is a term that implies timing, doesn't it?
Go listen to Pirogene's Nobel speech. Notice what he says about time? Two for two.
It is not all about energy even though it appears to be. Same thing why it is not all about food either. Food is not medicine without consioderation of timing. It is about how the system experiences time. The paper is not quite right. This is the decentralized truth. Details matter. They demand your focus.
Timing controls how energy flows in the matter in cells because that is NATURE's decentralized truth.
10. My work in quantum biology is a very difficult read for many. This science has patterns & references to physics and physical chemistry, which usually repel centralized humanists/transhumanists. But do not be afraid to enter my realm. Even if you do not understand everything, my work will sharpen your appetite for knowledge. This is why I will never dumb my work down for the centralizd dumbed down masses. They need what I know and I am going to force it down their throats. I want you to remain hungry for the wisdom Nature provides to cut your umbilical cord to centralized healthcare.
There is a well-known theorem of minimum entropy production derived by Prigogine, which states that entropy exported from a system reaches a minimum, or becomes zero, at thermodynamic equilibrium and at steady states close to thermodynamic equilibrium. Prigogine's theorem is a direct consequence of Onsager's reciprocity relationship which holds at steady states close to thermodynamic equilibrium. The principle of internal entropy compensation, is in addition to, and implies the principle of minimum entropy production, and may even be valid in regimes far from thermodynamic equilibrium.
He had some very interesting things to say about TIME in his work and Nobel Prize speech. I think they are key to understanding circadian biology and timing in dencentralized networks in cells. All of our time reversible equations in physics created by Newton, Einstein, and Schrödinger describe a simplification of what actually occurs in nature. We live our lives with eyes blinkered, dismissing reality as the exception to our neatly formed approximations of what time really is. nobelprize.org/prizes/chemist…
11. Prigogine defined dissipative structures and their role in thermodynamic systems far from equilibrium, a discovery that won him the Nobel Prize in Chemistry in 1977. In summary, Ilya Prigogine discovered that the importation and dissipation of energy into chemical systems could result in the emergence of new structures (hence dissipative structures) due to internal self-reorganization. In his 1955 text, Prigogine drew connections between dissipative structures and the Rayleigh-Bénard instability, and the Turing mechanism. Prigogine's theorem is germane to these ideas.
Mitochondria are dissipative structures in cells, but not the only ones inside of cells. They transform energy and create order from the disorder in light energy they use to operate. The water mitochondria create is probably the single most important dissipative structure that life is based upon in cells. According to Prigogine, determinism loses its explanatory power in the face of irreversibility and instability in dissipative systems. This is a major departure from the approach of Newton, Einstein and Schrödinger, all of whom expressed their theories in terms of deterministic equations.
Indeterminism is the opposite of determinism and is related to chance. Chance is related to probability. In science, most specifically quantum theory in physics links directly to probability and not cause and effect. Now go listen to what I said two weeks ago in Prague.......ABOUT TIME. @BTCPrague
@MaxGulhaneMD 12. I'm re writing the biology books using the physics of time. That is where the truth lies. Few see where I am going. It is time they do.
13. You dont need diverse research.......that is corporate cronyism DEI BS.
They want to dilute science just like they diluted PEER reivew to keep us all from the truth.
You have to make the acurate diagnosis when the public is sick.
BigHarma wants to censor real speech in science because it goes to the hear tof their monopoly and they sit next to the money printer = Cantillon effect 101.
@MaxGulhaneMD 14. We need concentration on science that focus specificially on Time.
15. Just how ignorant are the centralized experts.......unfathomable.
Are they any other reactions like Vitamin A in mammals? Can I introduce you to Vitamin D?
Production of Vitamin D in the Skin is electromagnetic like a ripe fruit falling from a tree.
During exposure to sunlight, the ultraviolet B photons with energies between 290 and 315 nm are absorbed by provitamin D3 (7-dehydrocholesterol) in the skin. This absorption results in a photolysis of the B-ring of provitamin D3 resulting in the formation of previtamin D3.
However, because previtamin D3 is thermodynamically unstable, it quickly undergoes an isomerization (rearrangement) of its triple bond system to form vitamin D3. Do you see right here in the sentence that the words unstable & quickly are used. These words imply timing is involved, don't they? Has centralized science accounted for this?
This photo-isomerization process is enhanced in skin cells because the previtamin D3 is synthesized in the cell membrane of mammals when it is fully electrified. When this happens it restricts its movement making sure UVB photons interact with the cholesterol molecule thereby accelerating the transformation of previtamin D3 to vitamin D3. Once vitamin D3 is formed in the skin cell membrane, it is photoelectrically release like when an apple falls from a tree to the ground. This is the fractal aspect of nature from the nano to the macro levels no one visualizes. When the chemical is photo readied by UVB light conversion it is no longer restricted in its movement and freely translocates into the extracellular space to find its way into the dermal capillary bloodstream where it is bound to a specific vitamin D-binding protein
16. A picture above is a schematic representation of the photochemical and thermal events that result in the synthesis of vitamin D3 in the skin, and the photodegradation of previtamin D3 and vitamin D3 to biologically inert photoproducts.
7-Dehydrocholesterol (7-DHC) a seasonal non visual photoreceptor in the skin is converted to previtamin D3 by the action of solar ultraviolet B radiation. Once formed, previtamin D3 is transformed into vitamin D3 by a heat-dependent (ΔH) process. When heat is involved it implies so is timing.
Vitamin D3 exits the skin into the dermal capillary blood system and is bound to a specific vitamin D-binding protein (DBP). When previtamin D3 and vitamin D3 are exposed to solar ultraviolet B radiation, they are converted to a variety & myriad of photoproducts that have little or no activity on calcium metabolism. This picture was found in Holick MF (1995) Vitamin D: Photobiology, metabolism, and clinical applications. In: DeGroot LJ, et al. (eds.) Endocrinology, 3rd edn, pp. 990–1013. Philadelphia: W.B. Saunders.
When human skin is exposed to sunlight, it is the solar ultraviolet B photons with energies between 290 and 315 nm that are responsible for causing the photolysis of 7-dehydrocholesterol to previtamin D3. This photochemical process occurs in the plasma membrane of keratocytes; as a result, the thermodynamically unstable cis, cis isomer of previtamin D3 is rapidly transformed by a rearrangement of double bonds to form vitamin D3.
Approximately 50% of previtamin D3 is converted to vitamin D3 within 2 hours. See time plays another role in biology of mammals. As vitamin D3 is formed in the membrane, its open flexible structure is likely jettisoned from the plasma membrane into the extracellular space. Once vitamin D3 enters the extracellular fluid space, it is attracted to the vitamin D binding protein (DBP) in the circulation, and thus enters the dermal capillary bed.
Is there another layer of complexity linked to time?
Why does Vitamin D receptor vary in thermal reactions? It turns out evolution uses realtivity and quantum mechanics in the Vitamin d receptor types. This is why there is existence of different forms of the 1,25-dihydroxyvitamin D3(1,25-(OH)2D3) receptor in mammals. The classic nuclear receptor for 1,25-(OH)2D3 is associated with nuclear genomic activity and their is another uncharacterized membrane receptors for both 1,25-(OH)2D3and 1,25-dihydroxyprevitamin D3(1, 25-(OH)2preD3) associated with non-genomic activity in mammals. One can get through the nuclear membrane and the others cannot. Timing yet again is why this occurs.
Look at the picture below. Stringing the lessons together yet?
TIME IS EVERYWHERE WHEN YOUR PERSPECTIVE GETS DOWN TO THE NANO LEVEL.
@MaxGulhaneMD 17. I hate ignorant people who think they are wise and I challenge them like a great white shark challenges their prey. I do it for the public's health.
Centralized healthcare is the biggest predator on the Planet for longevity.
18. How to get past the nuclear membrane once you make Vitamin D in the skin electromagnetically?
Sunlight energy is also used to sulfate cholesterol FIRST to get the Vitamin D train rolling in the skin to the blood to get to nDNA. When it is sulfated it becomes water soluble in blood. When it isn't sulfated, your liver responses to protect the nucleus in cells and it upregulates LDL production to act like a sponge to protect the AMO physics inside of cells. So, when your centralized dermatologist tells you to stay out of the sun and take the secosteroid Vitamin D from a plant or animal it is also packaged in a seed oil because it is normaly a fat soluable compound. Few realize that the simple act of taking D3 pills and living indoors behinds glass walls is raising your LDL cholesterol constantly. bit Harma knew it and this is why Big Pharma taught every doctor since they found the evidence in the 1950s military documents that they could profit massively by training doctors ignorant of light's effects to put their patients on statins. They knew to blame it on biochemistry gone awry and they baited the PhDs with the papers on the melavoant pathway and Q cycle. When you review the history of the evidence you know they are playing you like a fiddle. You are their mouse and they are the cat. Few see the game plan, and less of you get it.
Cholesterol and Vitamin D3 are nearly identical in chemical structure. Most people do not know this. Sunlight naturally sulfates these things (pic below). The only difference in both bio-molecules is a single double bond in the second ring of the cholesterol backbone. Remember light is BURIED at the electronic level in all things including cholesterol and Vitamin D. Your pills are sold to you soaked in seed oil do not. this is done by design because BigHarma knows what deuterium vs hydrogen can do. They've know about the effects of deuterium in drug delivery because of the the physical chemistry trials they have done. They never disclosed this to their curriculums to medical students or pharmacy students by design. This is why they want centralized pharmacy like WalMart, CVS, and Walgreens versus compounding pharmacies. When the drugs are ready made and all one needs to do is count your chemistry skills atrophy. A compounding pharmacist is an alchemist of physical chemistry.. They are more apt to realize what Pfizer and Moderna are really up to in New Jersey factories. I told @nayibbukele this and I put that in my law for El Salvador.
19. The AMO physics of these two biomolecules are nearly identical with one exception. HYDROGEN. This gives Vitamin D3 one less hydrogen atom than the closed ring of cholesterol has. If you spent anytime reading my Tensegrity 6 blog about my original hacks of the periodic table I did 20 years ago it should make you think a lot more carefully about atomic details.
One hydrogen is the only difference at the atomic scale between them, but from the quantum thermodynamic perspective you learn that there is a shit ton of energy buried in hydrogen's bond at the electronic and vibrational level. These are the bonds that food gurus and biochemists do not see, much less understand, so they ignore it at your peril. Here is why the public health goes awry because of a lack of focus on really what does matter in a cell. Here you will see the wisdom of my lessons in the Tensegrity 6 blog at play. Look it all the charge density sunlight adds to the system that a pill cannot. See, never divorce biophysics from biochemistry. If you do, you lose and it is through this crack that chronic disease epidemics begin.
20. The photonic energy in sunlight is used to oxidize hydrogen sulfide to make sulfate. This process sulfates everything at our surfaces and just below the surface and this makes many things water soluable and lowers charge density. Sunscreen was innovated by BigHarma to block sulfation by design. When you block sulfation you create a need for more drugs because you lower the tissues in energy and this exacerbates the timing mismatch in mtDNA. This fuels more mitochondrial mutations and creates disease phenotypes which fuels their centralized business models A cursory review of the what sulfation does to photovoltaics tells you how Nature uses sulfation to get past a highly charged membrane. It discharges the charge density to ruin the capacitance of the Vitamin D. This is how you enter the nDNA environment like a ghost. Nature has more magics it uses in sunlight. Melanin, Vitamin D, cholesterol, heparin, and nitric oxide all respond to the UV part of the spectrum when it is in sunlight. Few people know that the UV, and blue green part of the spectrum work in unison to very locally vasodilate blood vessels in the skin. I showed people the the melanopsin 2014 paper 1000's of times that showed blue light is capable of dialating blood vessels but not one person ask me about the AMO physics of the interaction.
21. This area interested me as a neurosurgeon because I was looking for a mechanism to explain the queer things that happen in brain bleeds called subarachnoid hemorrhages. We see them in trauma and in aneurysm ruptures. I have always felt that a lack of light operating in unison is behind most of the cerebrovascular pathologies that neurosurgeons treat. It took me ten years to figure it out, but altered light signaling is why AVMs and aneurysms rupture. It is also why some people develop SAH with minor trauma. I know I have been up all night taking care of these patient now and I am tired. I sacrificed my time for money to pay off legal bills to keep this info flowing to you clowns.
22. They are all symptoms of low redox power in the brain. Nitric oxide (NO) is a well-known “gasotransmitter” in the literature because a Nobel Prize was given for its discovery in 1992. It is a gas that acts focally and locally when it is released. It does not have global effects. This tells you something about TIMING too. It acts RAPIDLY, not CHRONICALLY. Vitamin D actions act CHRONICALLY.
This tells you that NO signaling is likely behind many light induced chronic diseases. Electriified membranes absorb gas and turn it into many other biomolecules. Life figured this out 3.8 billion years ago. NO is a gaseous signaling molecule that has a unique ability to induce a relaxation of the artery wall and a resulting drop in blood pressure. Recalll from my "Kruse for Dummies" lecture what I said about unique signals. They are how information is transferred in biological systems by Claude Shannon's theory of information.
Every one else is playing checkers looking at biochemical pathways while I am playing 4D chess because I understand how light operates in physical chemistry. Without full spectum sunlight, endothelial dysfunction and peripheral arterial damage should be EXPECTED by the decentralized clinician. It is not. No one in medical curriculums looks at the data in BigHarma literature to see this data much less understand the clinical significance.
UV light increases NO which lowers ATP and raises ROS/RNS. Both are incredibly important. It is the key photoswitch of mammals.
23. A lack of NO production at our integument and eye surfaces link PAD directly to cardiovascular dysfunction. The local effect become generalized in the entire organ as the lack of NO production gets worse under ALAN or nnEMF influence.
Peter Attia still does know this and he is printing money selling you statins and jabs.
This is why PAD is always linked to cardiovascular disease. The link is the aberrant use fo the electromagentic spectrum to communicate to create NO. Modern light and RF and cell radiation impairs production of NO from arginine by eNOS.
This, in turn, induces high blood pressure by causing endothelial dysfunction. Mitochondria are intimately involved in importing nitorgen into tissues to create the substrates that eventually become NO when sunlight is present. Nitrogen substrates are not created from the direct synthesis by eNOS.
Nature provided the clue to me why humans got rid of Vitamin C for glutathione in this AMO physics dance. When Vitamin C is missing in subcutaneous tissues, glutathione become more reactive with locally produced NO. This mimics a radical pair/triad effect we see in avian compass navigation.
Here is more evidence of magnetochemistry in humans being used. When glutathione and nitric oxide are powered by terrestrial sunlight this allowed humans to produce S-nitrosoglutathione (GSNO).
I think this is why human primates lost the majority of their integumentary hair and absorbed more melanin from the hair follicle to the interior. This is why Peter is bald as he works out in his blue lit gym with airpods in his ears and wants you to believe a 100 push ups some how saves you from all this? That is lunatic level thinking, in my opinion based on the science of NO.
This allowed the skin to become a better charge capacitor for the brain and heart by allowing the skin to become a depot station to store massive amounts of nitric oxide. This is why human immune T cells are so common in the skin and why leptin was placed in SQ fat. This is where anti-phospholipid sysndrome and all other auto-immune conditions come from.
Other primates do not have these phenotypes even thought their genomes are close to identical. This tells me magnetochemistry timing induced this evolutionary change. We never need genes to change this. We used timing to change the metabolic pathways in the skin using hair loss and removal of Vitamin C from the radical triad mechanism to do it.
As a consequence of this dance using more light on the skin, keratinocytes were able to sense more visible light combinations with purple, blue, and green light to easily photocatalyze the release of NO from glutathione. Not only does this cause a relaxation of the blood vessels, but it also frees up glutathione to react with hydrogen sulfide gas to produce sulfate to make every other chemical in the skin water soluable to get access to body parts to have global effects in distal organs. This is why PAD is linked to so many diseases.
This is how light develops its abscopal effects. No one has figured out how this all works in humans but this is how I have seen it for 20 plus years. To date no one has published a thing using my ideas. But I can explain why subtraction of Vitamin C in humans links to hair loss. This explains most of the integumentary and ocular diseases we see today. It also explains obesity, too. It explained to me why humans get aneuysms and AVMs.
People need to wake the fuck up and stop making stupid people famous in healthcare. We need the decentralized truth spread like a virus.forum.jackkruse.com/threads/why-do…
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1. Should we give the 49ers players a free lesson on what the NFL and the team will never expose on their behalf?
We need to explain why the players are getting injured at an amazing pace since 2014 and that sotry begins with the evolution of the SCN in the eye that controls the circadian clock.
The eye clock is the key to the story of their injuries.
2. Today we know that vision and hearing evolved together dating back to the PaxB gene, which is a single gene controlling eye and precursors to hearing (mechanoreceptors) in box jellyfish.
This occurred before independent Pax 2 and Pax 6 genes showed up in primates much later. There are evolutionary connections between eyes and mechanoreceptors of the inner ear to the extent that during evolution are linked to melanin generation in those sense organs.
I told you earlier in the decentralized medicine series on Patreonmelanin was the favored semiconductor of all mammals post KT event.
This story of the 49ers players fits this my thesis well because the entire team is made of mammals who are post KT evovled.
If POMC/melanin is absent for any reason in these players, at any particular body place, for any reason, including nnEMF, it appears human neuroplastiticty allows sensory cells to shift their sensory modalities to an older phylogeny experienced in evolutionary history.
Why is this a big deal for the 49ers players?
3. The SCN is controlled by ipRCGs and is a well known blue light detecotr. The melanopsin phylogeny predates even primate evolution in time.
I think this happens in modern humans because of a loss of information and energy transformation in the embryo due to a lack of POMC or POMC translation in the parents cells and their germ lines that create their child = epigenetic defect planning = childhood diseases not of genetic causes which make up the bulk of childhood disease burdens today.
This means any 49ers players future children will also carry the burden of the 2014 power plant upgrade.
**certain body secretions in people with diabetes often contain higher levels of carbohydrates (specifically glucose, the main simple carb involved) compared to people without diabetes.**. Look it up.
This is primarily due to elevated **blood glucose** levels (hyperglycemia) in diabetes, which can cause glucose to spill over or leak into various secretions when blood levels exceed normal thresholds. Recall Attia never finished his residency. He charges 200K to see him. I’m sure many of his patients would expect him to know the basic of human secretions is based on blood sugar levels
Here's a breakdown by common secretions that have more carbs
-vaginal secretions are high in carbohydrates in diabetic women.
- **Urine** — In uncontrolled or poorly managed diabetes, urine frequently contains significantly more glucose (a condition called **glycosuria** or glucosuria). Normally, healthy kidneys reabsorb nearly all filtered glucose back into the blood, so urine has little to no detectable glucose. When blood glucose exceeds the renal threshold (typically around 160–180 mg/dL), excess glucose appears in the urine. This is a classic sign of diabetes and can be much higher than in non-diabetics (who usually have negligible amounts).
- **Saliva** — Multiple studies show that salivary glucose levels are higher in people with diabetes (both controlled and uncontrolled) compared to non-diabetics. For example, mean salivary glucose is often around 13–14 mg/dL in diabetics versus 4–5 mg/dL in healthy controls. This occurs because high blood glucose increases leakage or diffusion of glucose into salivary secretions.
- **Sweat** — Sweat glucose concentrations are generally low in everyone (often 1–2% of blood levels), but research shows a strong correlation between sweat and blood glucose. In diabetics with higher blood glucose, sweat glucose is correspondingly elevated (e.g., potentially 0.3 mmol/L or more when blood is very high, versus lower in non-diabetics). This is why sweat is being explored for non-invasive glucose monitoring devices.
- **Tears** — Some evidence suggests tear glucose can also be higher in diabetics and correlates with blood levels, though this is less commonly studied than the others.
In summary, while not all secretions are equally affected and concentrations remain much lower than in blood, **diabetics typically have more glucose (a carbohydrate) in urine, saliva, sweat, and possibly tears** when blood sugar is poorly controlled. This doesn't apply universally to every diabetic at all times (e.g., well-controlled cases may show minimal differences), but it's a well-documented pattern, especially in hyperglycemia. If this relates to a specific health concern, consulting a doctor for personalized testing is recommended.
2. This goes to show you just how uninformed Attia is on the basics. High blood sugar (hyperglycemia) can occur due to various conditions and factors outside of diabetes, potentially leading to similar effects on carbohydrate (primarily glucose) levels in body secretions like vaginal secretions, urine, saliva, sweat, and tears. Physical or emotional stress: Acute stress from illness, infection, injury, trauma, surgery, tech screen abuse, cell phone use triggers the release of hormones like cortisol and epinephrine, which raise blood sugar to provide energy for the "fight or flight" response. High-intensity workouts can cause a short-term spike in blood sugar as the body releases stored glucose for fuel, especially in non-diabetics unaccustomed to such activity. Poor sleep quality from blue light and Earpod use disrupts hormonal balance, leading to insulin resistance and elevated glucose levels. Attia is known to believe that doing 100 push ups limits nnEMF risk. Look it up. He told this to Chris Williamson on a live IG post.
3. What else did Attia miss in his answer to Epstein? Cushing's syndrome: Excess cortisol production (often from adrenal tumors or prolonged steroid use) impairs insulin function and raises blood sugar.
Polycystic ovary syndrome (PCOS): This common hormonal disorder in women causes insulin resistance, leading to chronic hyperglycemia.
Acromegaly: Overproduction of growth hormone from a pituitary tumor reduces insulin sensitivity and elevates glucose.
Other hormonal issues: Conditions like hyperthyroidism or pheochromocytoma (a tumor causing excess catecholamines) can also contribute.
In my decentralized framework, this is exactly how the system is wired. The nipple-areola complex acts as a quantum-optical sensor, and the infant’s saliva is the fiber-optic cable delivering a real-time UPE (ultra-weak photon emission) status report from the child's mitochondria to the mother’s "manufacturing plant."
This isn't just convenience; it is a biophysical "handshake" that ensures the survival of the post Cambrian hardware mammals need to thrive.
1. Saliva as the "Optical Bio-Feed"
Saliva is a highly structured, mineral-rich fluid. In my thesis, it serves as the medium for optical information transfer:
The Child’s Signal: When a calf or child is sick, their internal "optical smog" increases. The VUV (Vacuum Ultraviolet) and chaotic UPEs produced by their congested Complex II are transmitted through the saliva. Congestion usually is due to reverse electron flow or deuterium.
The Mother’s Sensor: The areola is one of the most melanized and innervated tissues in the mammalian body. Melanin here doesn't just protect against the sun; it acts as a broadband transducer like an optical scanner in the grocery store. It "reads" the frequency of the biophotons in the saliva.
2. The Backflow "Optical Loop"
Research into the "infant-led" backflow confirms that when a child suckles, a vacuum is created that pulls saliva back into the mother's mammary ducts.
Information Sensing: The mother’s immune-sensing cells in the ductal walls "listen" to the ROS/RNS signatures and UPE entropy in that saliva.
The Response: If the child’s saliva "shouts" of deuterium congestion at cytochrome two because succinate is elevated or viral "optical noise," the mother’s brain (via the SCN-hypothalamic oxytocin posterior pituitary pathway) triggers a change in milk composition. She begins to "distill" more NPD1, Iodine, and IgA antibodies into the milk to act as a "quantum reset" for the child’s mitochondria.
3. The "Rotating Mom" Phenomenon (Allomaternal Nursing)
Why do calves or elk rotate moms? In my framework, this is "Frequency Matching":
Metabolic Matching: A calf may instinctively seek a different "frequency" of milk if its own mother’s melanin-metal hardware is jammed (perhaps she spent too much time in a "dirty" environment like inside a barn under fake light).
Information Diversity: By "sampling" different mothers, the young mammal is essentially "crowd sourcing" optical coherence. They are looking for the milk with the lowest deuterium/highest DHA-D3-Iodine ratio to stabilize their own emerging Faraday cage.
Modern humans with nnEMF toxicity who cannot breast feed their children due to a lack of production should be considering what elk do when they are faced with the same problem. This concept is foreign to humans because they do not observe nature carefully enough.
4. Milk as a "Re-Cambrian-ization" Serum
Mother's milk is the ultimate low-deuterium, high-DHA, solar-coded fuel.
Deuterium Depletion: Milk fat (cream) is naturally low in deuterium, helping the child build a "clean" 14 Angstrom tunneling gap in their developing mitochondria.
Melanocortin Programming: The act of nursing at sunrise/sunset ensures the child’s Leptin-Melanocortin pathway is synced to the mother’s, preventing the "Proterozoic regression" that leads to neolithic disease later in life.
The ranch memories you have are of a Quantum Ecosystem in action. The child’s saliva "tells" the mother's nipple exactly how the child's internal "mercury lamp" (deuterium) is burning. The mother then alters the "Optical Duty Cycle" of her milk to quench the fire.
Does this explain why formula-fed infants (drinking high-deuterium, seed-oil-heavy milk) are essentially being "pushed into the Proterozoic mud" from birth, as they lack this bidirectional optical feedback loop? Yes, it does but few seem to care about it.
This lesson also has deep information for the diseased breast too. Men can help their women with diseased breasts by kissing their nipples religiously to transfer information to their women about how they feel for them. This will be highly stimulatory and healing.
Cells and Stars have a lot on common. When they fail they have mechanisms that feedback on themselves that lead to the possibility of future survival if conditions are met. In a star when it burns through its fuel source its core gets to iron and the star begins to emit microwaves. The microwave radiation interacts with the D shell electrons of Fe and it blows up in a super nova so the atoms it creates in this destruction can be recycled to something with more life in the cosmos. Cells in our body use a similar idea in apoptosis, autophagy, and ferroptosis.
In my decentralized framework, the brain and breast in cancer do not operate in the same fashion regarding IDH protection because their "optical hardware" and evolutionary duty cycles are fundamentally different. Neurons are not epithelium, but breast tissue is.
While the brain relies on IDH mutations from its DHA-rich antenna to create UPEs to work and eventually help create some VUV smog from complex two back up to clean the dirty chemistry in cancer, the breast mitigates oncogenic risk through a different mechanism:
It uses the DHA-Iodine-Melanin triad.
1. The IDH Paradox: Why the Brain Needs It, But the Breast Doesn't
The brain is the ultimate "quantum sensor" that can feedback on itself and it must maintain 24/7 DHA coherence. DHA allows for this.
The Brain (DHA Antenna): When Complex II jams, the resulting VUV emission from Deuterium threatens to shatter the DHA antenna. The IDH mutation occurs as an emergency "filter" to deplete deuterium and lower the VUV entropy. DHA, as a PUFA explodes like the star and in its destructions NPD1 and Elovanoids along with UPEs are made which are highly anti-inflammatory. The released UPEs feed back on IDH and mutate it in such a specific way that the brain is able to make more deuterium depleted water because of this interaction than it could before to help self sustain its survival. This is how most low grade gliomas begin. This process does not happen in GBM transformation due to a lack of DHA in the brain.
The Breast (The Glandular Sink): Breast tissue is not a primary "spectrometer" like the brain. Instead of a mutation-driven shunt (IDH), the breast uses Iodine as its primary "quantum ground." In the breast, the "De-Cambrian-ization" of its mitochondria is mitigated by the concentration of iodine in the Ductal-Lobular Units. 2. How the Breast Mitigates Risk: The Iodine Shield
If the brain uses the IDH mutation to "buffer" the VUV smog itsdeuterium squeeze, but the human breast uses Iodine to "quench" the fire in the cytochrome 2 Fe-S clusters that begin the disease from reverse electron flow from cytochrome 2 dysfunction.The Delta-Iodolactone Mechanism: Iodine is required to form iodo-lipids (delta-iodolactone). These act similarly to Bazan’s Elovanoids in the brain, but they are specifically tuned to inhibit the Warburg Effect in glandular tissue.
Melanin & The Nipple: The high concentration of melanin in the areola/nipple is not a "decoration." It is the Optical Port that harvests solar UV/IR to control the metal flux (Cu, Fe) in the underlying breast tissue to make sure the TCA and urea cycle are the primary pathways used to avoid cytochrome 2 congestion and Fenton reactions of Fe-S couples.
The Sink: The breast is designed as a "DHA sink" (for lactation). It mitigates VUV damage by using Melanin and Iodine to sequester the "dirty" Iron noise created from a lack of sun, nnEMF, or polarized light. nnEMF for the breast is the stimulus that leads to ferrotoptosis destruction of the Fe-S couples and this mimics the process that happens in a star. When Iodine is missing, the breast cannot "ground" its own self created UPE field, leading to DCIS or invasive carcinoma without the IDH "slow-burn" protection seen in the brain. 3. The Unified "De-Cambrian" Failure
Both cancers represent a Proterozoic Regression, but the "breakdown" follows the tissue's specific metal hierarchy:Brain Cancer (GBM/LGG): A failure of the DHA-VDR-IDH loop. The brain tries to mutate (IDH) to survive the VUV fire.
Breast Cancer: A failure of the Melanin-Iodine-DHA loop. Without Iodine to "buffer" the Iron D-shell electrons, the breast tissue undergoes a rapid phenotypic regression and this is how cancer begins.
The synthesis of both molecules is tied to the Melanin-Metal hardware:
The Sun: UVB/IR input on the skin stimulates the Leptin-Melanocortin pathway. This manages the Copper (Cu) and Manganese (Mn) levels required to build the antioxidant "Mn-SOD shield."
The Translation: Coherent UPE signals (from healthy mitochondria) then tell the cell to cleave the lipids needed for NPD1 or gamma iodolactone
.
The Result: You have a "protected" post Cambrian cell that can use oxygen via the TCA/urea cycle without producing the ionizing VUV UPEs that destroys the genome.
Bazan’s Docosanoids (Brain/Retina): These cleave from DHA to form a "Faraday cage" of 22 carbons. Their purpose is to quench the VUV (Vacuum Ultraviolet) smog emitted by deuterium in the high-density neural environment.
3. UPE isn't mere waste; from quantum biology, these photons (or associated fields) may mediate non-local signaling, akin to coherence in radical pairs or bystander effects.
Intensity/spectrum reflect metabolic flux:
Aerobic paths (TCA) produce more ROS/UPE than anaerobic (glycolysis); stress shifts spectra (e.g., UV-linked UPE from glycolysis/peptides). Melanin optimizes by calibrating inputs—solar photons tune metal-ROS-UPE, enabling adaptive switches via redox/epigenetic relays (e.g., NAD+/SIRT1, from the Decentralized Medicine series of blogs on Patreon).
2. What is the rescue plan? Remember the famous now deleted Bessent tweet about DJT/Treasury plan to confiscate Bitcoin for the US Bitcoin Reserve? That was updated once the backlash on the tweet went out. Now it is about using Tether to centralize gold as they implode the Dollar and they will confiscate the Gold.
I've argued for 10 years that Bitcoin is a superior form of "trust-minimized" money compared to gold due to the high costs of verifying gold's authenticity.
This is the ability to own and verify an asset without relying on a third party (like a Central bank or Treasury of a government). Historically, gold's "trustless" nature was its greatest strength, but Savages now know this strength has been lost because most gold now sits in centralized vaults. This is why DJT won't let anyone audit Fort Knox. Why audit what you plan to steal?
The Cost of Validation for gold is steep so no one in the USA will want to pay that freight so now we are on the honor system for the Treasury.
Anyone who has owned gold knows that verifying that a gold bar before a sale/audit is real and pure requires specialized equipment, chemical tests, or expensive third-party audits. Because this is so difficult to do, you must have an inherit "trust" the vault or institution holding it for them. + Treasury and Bessent play. What did they do in 2025. They brought their middle man in. Tether. Go check if I am bullshitting you. Tether has bought more gold in the last 18 months than they have bought Bitcoin. Why? They are storing what the thieves in the industrial miliatry surveillence state will take down the road when the retards are sidetracked by circus maximus of some other psy-ops.
Why isn't Tether buying Bitcoin in this case? Anyone with a simple computer or smartphone can instantly verify the entire history and authenticity of their Bitcoin by running a node or using a block explorer. This "validation" is nearly free, making it more decentralized and harder to seize. Tether should be buying T bills but instead is buying Gold Reserves for the Zionist bankers to steal soon. Got it, my Savages.
Bitcoiners should know and remember their history better. This gameplan was used to before in 1933 during the Great Deperession to make it easy for governments to confiscate it.....Remember FDR's EO?
The U.S. did this already with Executive Order 6102 in 1933.
Looting and Centralization are the play. For thousands of years, physical gold was frequently stolen or seized by empires. To protect it, it was eventually moved into highly secure, centralized vaults (like those at the Federal Reserve Bank of New York or the Bank of England under control of the Treasury Head.
If the steal the gold this will tank markets including Bitcoin and then the Treasury will come in an sell gold at astronomical prices to buy Bitcoin at crashed Prices. This is how the Rockefeller and Rothschild Banks plan to do this.
If you know your history this is how the same guys did the scam during the Napoleaonic Wars. They manipulated the market with a psy-ops. In 1812 it was the Battle of Waterloo.
WAKE THE FUCK UP.
If you knew this history would would not be so gullible.
2. Question asked in last 6 weeks: Jack, My breast cancer recurrence has occured in the left auxiliary node. Currently taking Verzenio and tamoxifen. Declined ovarian suppression. Starting Dr. Makis protocol soon.
How can I monitor my axillary lymph nodes without using ultrasound, given your concerns about its disruption to quantum coherence in tissues? Especially since my traditional blood tumor markers (CA 15-3 and CA 27.29) have consistently tested negative?
Aside from the tests listed below, are there any additional laboratory studies you recommend prioritizing for tomorrow with Dr. G?
BUN/creatinine ratio
Vitamin D
Liver function
HsCRP
1. Today's lesson from my forum is on hyperhydrosis and dysautonimia.
QUESTION: Hi everyone,
Starting this thread to document my improvements or lack thereof, as I get closer to the Equator and further away from nnEMF.
My issue is a form of Dysautonomia driven by a small gain of function mutation in the gene encoding for Nav1.7.
The results is a persistent Na+ leak in the neurons where Nav1.7 is expressed, resulting in hyperexcitability of these neurons.
This hyperexcitability leads to the following symptoms: sympathetic overactivity, hyperhidrosis, gut hypersensitivity, more prone to visceral anxiety, bronchoconstriction, etc.
I know the decentralized medicine perspective says this is an environment problem and not a genetic problem.
But I'll only be able to confirm this once I get my environment right and get rid of these symptoms.
Best,
Alex
How can my neurons help Alex?
2. ANSWER:
Relationship Between Hyperhidrosis and Dysautonomia
Hyperhidrosis is frequently recognized as a specific symptom of a broader autonomic dysfunction.
In cases involving the upper neck:
1. Localized Sweating: Irritation of the sympathetic fibers around the vertebral artery often causes sweating or flushing on only one side of the face. 2. Systemic Dysautonomia: If the compression affects the brainstem's ability to regulate the whole body, you might experience more generalized symptoms like heart palpitations, temperature dysregulation, or "drop attacks" (sudden weakness). 3. Other relationships to be explored are found below
A. Vertebrobasilar Insufficiency (VBI): The bony bridge can compress or "kink" the vertebral artery, especially during head rotation. This reduces blood flow to the brainstem, which houses the primary control centers for the autonomic nervous system. This can manifest as dizziness, fainting (syncope), and nausea, all signs of dysautonomia.
B. Barré-Liéou Syndrome: This is a specific cluster of symptoms caused by irritation of the posterior cervical sympathetic chain (the nerves that control "automatic" functions like sweating and heart rate) due to cervical spine issues. Symptoms often include unilateral facial sweating, flushing, blurred vision, and ear ringing (tinnitus). Tinnitus brings the link to melanin dysfunction in the stria medullaris as I have laid out painstakingly on 7 Patreon blogs. It signifies an nnEMF etiology to the Hyperhidrosis and dysautonomia.
C. Trigemino-Autonomic Activation: When the C1 nerve or the vertebral artery is irritated by the ponticulus posticus, the signal is processed in the Trigeminocervical Complex. This can trigger a "reflex" in the autonomic system, leading to craniofacial hyperhidrosis (sweating on the face/forehead), nasal congestion, or eye-watering. This can also be stimulated by demyelination in this region by melanin POMC defects, DHA defects in the central retinal pathways, or polarization toxicity that affects the nerve complex that links these two disease. Both, hyperhidrosis and dysautonomia are located in two distinct but interconnected systems: the Central Autonomic Network (CAN) in the brainstem and the Peripheral Sympathetic Chain in the neck. Hyperhidrosis in this scenario is typically a "positive" neurological phenomenon (overactivity) caused by irritation of the Superior Cervical Sympathetic Ganglion (SCG) and the Periarterial Carotid Plexus (lots of POMC).
3. ANSWER CONTINUES
The broader "dysautonomia" (dizziness, heart rate changes, nausea) stems from a defect in the Lower Brainstem, specifically the Nucleus Tractus Solitarius (NTS) and the Ventrolateral Medulla. These are the primary control centers for blood pressure and heart rate, located in the medulla oblongata of the brainstem. When these brainstem centers are deprived of oxygen-rich blood, like we see when Fe is forced into the +3 state over the +2 state, they fail to regulate the autonomic system correctly. This is why nnEMF can cause this syndrome. This results in the "mismatch" symptoms of dysautonomia, such as postural dizziness, syncope (fainting), or "drop attacks" where the legs suddenly give out.
Because these symptoms are often positional, it is highly recommended to speak with a radiologist specialist about a Digital Motion X-ray (DMX) or a CT Angiogram to see how your vertebral artery behaves when you move your neck.