We believe there are three hypotheses that could explain the infection among the swimming teams:
A. The virus may be present in the water of the swimming pool.
B. The virus might be circulating in the swimming camp due to inadequate ventilation...
3. C. The virus could remain stable at the pool as a result of chemical reactions, especially involving chlorine.
A. Regarding the presence of the virus in water which is also chlorinated, the likelihood is nearly zero.
4. B. Regarding the presence of the virus in the swimming camp,
it is difficult to answer in the absence of reliable information on ventilation systems.
This hypothesis seems to us the most probable.
5) The 3rd very interesting hypothesis is based on the studies carried out by @ukhadds
6) We highly recommend his study.
"Differences in airborne stability of SARS-CoV-2 variants of concern is impacted by alkalinity of surrogates of respiratory aerosol" royalsocietypublishing.org/doi/10.1098/rs…
7) The study showed that differences in SARS-CoV-2 aero-stability correlate with alkalinity, not salt concentration. Acidic air increases virus stability, while alkaline conditions rapidly inactivate it, highlighting importance of ventilation in reducing airborne transmission.
8) Finally, there is this older study :
"Swimming pool, respiratory health, and childhood asthma: Should we change our beliefs?"
which showed that chlorinated pools may negatively impact respiratory health, especially in asthmatic and elite swimmers ... onlinelibrary.wiley.com/doi/10.1002/pp…
9) ...due to exposure to chlorine by-products. Effects on recreational and infant swimmers are less clear, requiring further research to establish causal links between pool attendance and asthma development.
10) Regardless of the contributing factors, these infections among elite swimmers highlight that the virus is still spreading rapidly across the globe.
Thanks for reading and great thanks to @ChristinaVirgil for her questions and shared information.
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NIPAH VIRUS
Jumping to conclusions during outbreaks only fuels panic and misunderstanding; NOT EVERY VIRUS OUTBREAK is a PANDEMIC.
We raised the alarm about Nipah in 2023, but it’s essential not to confuse an epidemic related to this virus, ...
2) ...like the one in India, with a pandemic risk or a situation like COVID-19.
Nipah virus is transmitted primarily from animals, especially bats, to humans. It spreads through direct contact with infected animals, their secretions, or contaminated foods.
3) Human-to-human transmission occurs only through close contact, such as touching or caring for an infected person, as well as sharing fluids like saliva or blood.
We cannot equate a virus transmitted through fluids with a respiratory virus like SARS-CoV-2.
"70% to 90% of our CELLS are completely RENEWED in less than 5 YEARS!
So If we’re mostly "New", why are so many still struggling with SARS-CoV-2?
We will tackle this question, which is more complex than it seems, in several posts.
2) First, let's say a few words about cell renewal.
70% to 90% of our cells are renewed over time. This turnover occurs in various cell types, including skin, blood, and immune cells, allowing the body to replace damaged or aged cells. sciencefocus.com/the-human-body…
3) For instance, red blood cells have a lifespan of about 120 days, while skin cells regenerate every few weeks.
Even with significant cell renewal, aging persists due to changes in our DNA. sciencefocus.com/the-human-body…
2) Co-infection occurs when a person is infected by more than one virus at the same time. This can lead to interesting and sometimes complex interactions between the viruses
3) In one study about SARS-CoV-2, researchers found that when different versions of this virus infect the same person, they can mix their genetic material through a process called recombination. This means that the new version of the virus can have traits from both parent viruses
For more than three years, we have been emphasizing that the envelope protein is an essential component and that we cannot limit our focus to just the spike protein. We are now uncovering its role in long COVID.
3) The Envelope (E) protein of SARS-CoV-2 plays a crucial role for several reasons:
▶️ Virus Assembly and Release: The E protein is essential for the virus's assembly and budding from infected cells, contributing to its stability and infectivity.
In 2022, COVID-19 was the second leading cause of death globally, with the repercussions of the virus far from over. The lasting impact of the pandemic continues to affect lives around the world.
Recent research may have identified a key factor contributing to long COVID: microscopic clots intertwined with immune system debris in the blood. These unusual structures were found to be nearly 20 times more prevalent ...
2) ...in long COVID patients compared to healthy individuals. The clots, associated with neutrophil extracellular traps (NETs), suggest a potential biological marker for persistent symptoms.
3) These microclots could impede blood flow in small vessels, leading to issues like brain fog, fatigue, and shortness of breath.