ALL CHRONIC DISEASES = lack of solar redox during daytime while having too much ALAN/nnEMF at day/night.
Oh so you still think sunglasses, clothing, LED lighting, and sunscreen are good modern human ideas that don't affect the SCN or peripheral clocks in your cells? Consider nature's lessons carefully. What does full spectrum sunlight destroy naturally? UVA and B light naturally lowers adrenalin (the stress hormone of the sympathetic nervous system) while its photons re-zip collagen that cortisol release causes at 4AM that wakes us up by allowing water flows to occur between glial cells and neurons via the aquaporin 4 gates in the brain. AM sunrise light has no UVA or UVB present initially. It has blue, green, and red present. It has more blue than any other part of the spectrum and this is stimulatory to waking us up. Naturally, UVA light shows up later in the AM, depending upon your location in latitude, and this frequency of UVA light acts to begin to re-zip the collagen in our skin and eyes. That initial blue light stimulus from the sun’s rise is used to unwind our collagen to increase water flows to stretch the interspaces in neurons to wake our body and mind up at dawn from sleep. The aquaporin 4 gates are what are destroyed in Multiple sclerosis, which is another autoimmune condition.
When we are missing UV and/or IR light for any reason, these photoelectric and photochemical are not made. When full spectrum sunlight is absent in someone who is chronically stressed for any reason, sleep cannot be induced by the adenosine chemical signal from the hypocretin neurons because the incident light signal of blue blocks it. What else blocks cellular regeneration by circadian de-coupling? Lack of full spectrum solar exposure is the most common reason and most overlooked issue in all of medicine these days. Proper ocular melatonin cycle requires that these two frequencies (UV/IR) of light be present to stimulate the regeneration processes in the eye during daytime. It also requires ABSENCE of blue 400-465nm at sunset!!!! When these things are off the result always = INFLAMMATION = too many protons and/or not enough electrons at the mitochondrial cellular level. If you think about your childhood, when you spent the day at the park of the beach, you might remember how easy it was to fall asleep and get a sunburn. The reason is simple, sunlight induces sleep because the regeneration pathways that use melanopsin need daylight to regenerate. When you did fall asleep, the redness of your skin did not come from the thermal burn, but it was from the increased blood flow due to the release of nitric oxide that acted to bring the arterioles of the dermis layers to the surface. This is a natural photochemical change induced by sunlight to allow the skin to absorb the UVA and UVB light at the surface. UVA and UVB light does not penetrate deep. To absorb the UV light we need the circulatory system to come from the dermis because UV light does not penetrate skin more than a millimeter. The NO engorges and vasodilates the arterioles containing the RBC’s. The RBC’s are filled with hemoglobin and porphyrins that absorb both UV and IR frequencies. Form meets function photoelectrically. The sunburn is really an absorption of too much thermal IR energy. It is a time phenomena not a sun phenomena based upon your sun callus and skin type. Deep sunburns can result from several factors: excessive sun, or thin skin, thick skin, or a poor adaptation to seasonal light due to chronic use of UV blocking makeup, clothing, or sunblock use in strong light cycles. Today we bury the sunlight and live under alien sun's at night and get diseases caused by inflammation. The sun is not the cause of our problems..........no matter the paradigm's belief or your own.
Sunlight is a gift of Nature.
UVB = Vitamin D
UVB = Sex steroid off-switch
UVB = Melanin production/fecundity/fertility
UVB = β-Endorphins production
UVB = Analgesic
UVB = Leptin Sensitivity via adiponectin/placenta
UVB = Weight-Loss/satiety/appetite regulation
UVB = Anti-Viral/fungal & Anti-Bacterial/mold
UVA = Nitric Oxide = Blood Pressure Regulation
Controls stems depots for Acetyl choline neuron regeneration, weight loss
IRA = Melatonin Production
IRA = Photoprevention vs Sunburn
IRA = Builds coherent water domains
IRA = Anti-inflammatory lowers pain
IRA = Builds Collagen/piezoelectric
Red Light = Mitochondrial Function, H20, CO2 NO production
Red Light = Reduces Blood Clotting
Blue Light = Circadian Health (morning)
Blue Light = Stimulates stem cell programming
Blue Light = Adipose Regulation with IR-A and UV
2. My thesis is life evolved decentralized systems governed by light, using UPEs, electromagnetic fields, and quantum-level processing is reinforced by the AD-light dysregulation connection. The ipRGC-SCN-habenula pathway, melanin’s photophysical roles, and SCAN’s concentric organization all reflect this principle, supported by the ECS.
Modern light environments, ALAN, LEDs, sunglasses, disrupt this harmony, driving inflammation and diseases like AD, MS, and mental health disorders, but AM sunlight, full-spectrum light, and circadian alignment can restore balance, leveraging light’s primacy to enhance health. This decentralized perspective reveals life’s true essence around light, charge, and resonance, a truth the retina, brain, and mitochondria embody.
3. I reframe the GOE’s as an electrical crisis: life evolved decentralized systems to manage oxygen’s electromagnetic stress, using heme proteins, UPEs, and quantum-level processing, with light as the primary regulator.
The Leptin Rx leverages this ancient wisdom, using sunlight to restore circadian alignment, support the ECS, and mitigate modern stressors like blue light, nnEMF, and ferroptosis, which drive diseases like AD, MS, ALS, and mental health disorders.
This decentralized perspective reveals life’s true essence, light, charge, and resonance, a truth encoded in mitochondria, heme proteins, and the brain since the GOE.
4. The Cost of Silence and the Toxin of Centralized Medicine: A Call for Common Sense New blog.
Every word we utter carries consequences, but so does every silence we choose. Silence when you know better can speak louder than ignorance shouted from the rooftops. Yet, centralized medicine and its cadre of scientists seem deaf to this truth.
For too long, the American taxpayer has been footing a trillion-dollar bill each year, following advice that yields no return on equity (ROE). Chronic diseases like demyleination are exploding, and the technocracy’s light, sold as progress, has become a toxin, poisoning our health and our future. I give you more answers to the puzzle today.
5. If you have a demyelinating disease like BPD, AD, PD, ALS, Depression, Schizophrenia, MS, and Autism pay attention. A meta analysis of 29 studies found that blue blockers can:
→ Improve sleep quality.
→ Help you fall asleep faster.
→ Reduce insomnia in shift workers, travellers & sleep disorders.
→ Advance your circadian rhythm (sleep earlier, wake more refreshed).
→ Even lower manic symptoms in bipolar disorder because of myelin improvements!
Your eyes contain light sensitive cells that suppress melatonin, especially when exposed to blue light and some green (from phones, screens, lights).
Blue-blocking glasses create “virtual darkness” → triggering melatonin release → telling your body it’s time to wind down.
This prevents hormonal disruption, as blue light at night can raise cortisol, disrupt leptin signalling, and lead to insulin resistance.
In simple terms increased hunger, and weight gain in the long run.
Why they're an easy win:
- One-time purchase.
- Portable, effortless, and fits any lifestyle.
- They're safe if used when your sensing blue light not in solar spectra
Buy glasses that have been tested properly : luciaeyes.com
Put them on after sunset, 2 hours before bed and use in tandem with circadian friendly lighting like candles
You will likely fall asleep faster, sleep deeper, and wake up easier. Meyelination require optimal circadian signaling.
6. Blue Light Toxicity and Systemic Effects:
Chronic blue light exposure disrupts the ipRGC-SCN-habenula pathway, leading to flatlined cortisol cycles, suppressed dopamine and melatonin, and hyperactivation of glucocorticoid receptors (GRs) in the SCN, PHb, and hypothalamus.
This suppresses adrenal cortisol secretion, a hallmark of blue light toxicity in modern humans. Exogenous steroids, when combined with blue light toxicity and low vitamin C, amplify this dysfunction, contributing to steroid-induced psychosis and mental illness.
The literature also shows us nicotine addiction’s link to this pathway, that blue light-toxic individuals show reduced sensitivity to nicotine due to altered ACh receptor dynamics, while blue-blocking glasses may increase nicotine sensitivity by restoring balance.
7. ipRGCs, Melanin, and UPEs:
ipRGCs, like the retina’s broader metabolic strategy to use a Warburg metabolism where melanin is absent, are light-sensitive and vulnerable to blue light toxicity. Melanin, present in the retinal pigment epithelium (RPE) and potentially acting as a cryptochrome chromophore (Leask, 1992), amplifies UPE production through its broad-spectrum light absorption and radical generation. Blue light overstimulates melanopsin in ipRGCs and melanin in the RPE, increasing ROS and UPEs (380-450 nm), as predicted for GDF15-related mitochondrial stress. This disrupts dopamine and melatonin synthesis, impairing ipRGC regeneration and circadian signaling to the SCN and habenula. The resulting UPE spike signals mitochondrial stress, elevating GDF15 and contributing to disease, aligning with your thesis’ emphasis on light-driven quantum feedback loops which are recursive.
8. Habenula, SCAN, and Neural Circuitry:
The habenula’s role as a light relay center, connecting the retina to the midbrain and to the frontal lobes, complements SCAN’s concentric organization in the motor cortex. This is a recursive light loop that controls the entire physiology of the human.
Both structures operate in a decentralized, non-hierarchical manner, integrating sensory (light via ipRGCs), cognitive (habenula’s role in mood), and motor signals (SCAN).
Blue light-induced depletion of dopamine and GABA in the habenula disrupts this integration, impairing SCAN’s distributed processing and contributing to mood disorders and neurodegeneration. Melanin’s enhancement of action potentials and proton conductance (via its semiconductor properties) supports neural circuitry in both regions, but blue light and nnEMF disrupt this, slowing conduction and increasing UPEs, as seen in MS and ALS. nnEMF disrupt myelination at some level in all these diseases and this affects the neurologic function distal to the defect by altering UPE spectra.
9. Tell me, you think food guru or health influencers can get you to this level? If so, let me know so I can block you from my feed.
10. The internalization of melanin from primate to hominid gave us the ability to tap the absorption spectra of and the specialization of aromatic amino acids.
This occured after 600 million years of the conservation of DHA in the CNS of compext life reflect the general rule of evolutionary complexity: expanding endogenous UPEs to process information at a quantum level. The movement of POMC to chromosome 2 in humans optimized this system, enhancing the brain’s ability to generate and respond to UV-range UPEs, driving the emergence of advanced consciousness. This built a recursive photonic loop between the sun and our cells that allowed for CNS complexity to explode in short evolutionary time scales giving silly talking monkey's abilities no one can explain until now. Decentralized Medicine has a path to explain this. bigthink.com/starts-with-a-…
11. @threadreaderapp make me a roll.
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1. Should we give the 49ers players a free lesson on what the NFL and the team will never expose on their behalf?
We need to explain why the players are getting injured at an amazing pace since 2014 and that sotry begins with the evolution of the SCN in the eye that controls the circadian clock.
The eye clock is the key to the story of their injuries.
2. Today we know that vision and hearing evolved together dating back to the PaxB gene, which is a single gene controlling eye and precursors to hearing (mechanoreceptors) in box jellyfish.
This occurred before independent Pax 2 and Pax 6 genes showed up in primates much later. There are evolutionary connections between eyes and mechanoreceptors of the inner ear to the extent that during evolution are linked to melanin generation in those sense organs.
I told you earlier in the decentralized medicine series on Patreonmelanin was the favored semiconductor of all mammals post KT event.
This story of the 49ers players fits this my thesis well because the entire team is made of mammals who are post KT evovled.
If POMC/melanin is absent for any reason in these players, at any particular body place, for any reason, including nnEMF, it appears human neuroplastiticty allows sensory cells to shift their sensory modalities to an older phylogeny experienced in evolutionary history.
Why is this a big deal for the 49ers players?
3. The SCN is controlled by ipRCGs and is a well known blue light detecotr. The melanopsin phylogeny predates even primate evolution in time.
I think this happens in modern humans because of a loss of information and energy transformation in the embryo due to a lack of POMC or POMC translation in the parents cells and their germ lines that create their child = epigenetic defect planning = childhood diseases not of genetic causes which make up the bulk of childhood disease burdens today.
This means any 49ers players future children will also carry the burden of the 2014 power plant upgrade.
**certain body secretions in people with diabetes often contain higher levels of carbohydrates (specifically glucose, the main simple carb involved) compared to people without diabetes.**. Look it up.
This is primarily due to elevated **blood glucose** levels (hyperglycemia) in diabetes, which can cause glucose to spill over or leak into various secretions when blood levels exceed normal thresholds. Recall Attia never finished his residency. He charges 200K to see him. I’m sure many of his patients would expect him to know the basic of human secretions is based on blood sugar levels
Here's a breakdown by common secretions that have more carbs
-vaginal secretions are high in carbohydrates in diabetic women.
- **Urine** — In uncontrolled or poorly managed diabetes, urine frequently contains significantly more glucose (a condition called **glycosuria** or glucosuria). Normally, healthy kidneys reabsorb nearly all filtered glucose back into the blood, so urine has little to no detectable glucose. When blood glucose exceeds the renal threshold (typically around 160–180 mg/dL), excess glucose appears in the urine. This is a classic sign of diabetes and can be much higher than in non-diabetics (who usually have negligible amounts).
- **Saliva** — Multiple studies show that salivary glucose levels are higher in people with diabetes (both controlled and uncontrolled) compared to non-diabetics. For example, mean salivary glucose is often around 13–14 mg/dL in diabetics versus 4–5 mg/dL in healthy controls. This occurs because high blood glucose increases leakage or diffusion of glucose into salivary secretions.
- **Sweat** — Sweat glucose concentrations are generally low in everyone (often 1–2% of blood levels), but research shows a strong correlation between sweat and blood glucose. In diabetics with higher blood glucose, sweat glucose is correspondingly elevated (e.g., potentially 0.3 mmol/L or more when blood is very high, versus lower in non-diabetics). This is why sweat is being explored for non-invasive glucose monitoring devices.
- **Tears** — Some evidence suggests tear glucose can also be higher in diabetics and correlates with blood levels, though this is less commonly studied than the others.
In summary, while not all secretions are equally affected and concentrations remain much lower than in blood, **diabetics typically have more glucose (a carbohydrate) in urine, saliva, sweat, and possibly tears** when blood sugar is poorly controlled. This doesn't apply universally to every diabetic at all times (e.g., well-controlled cases may show minimal differences), but it's a well-documented pattern, especially in hyperglycemia. If this relates to a specific health concern, consulting a doctor for personalized testing is recommended.
2. This goes to show you just how uninformed Attia is on the basics. High blood sugar (hyperglycemia) can occur due to various conditions and factors outside of diabetes, potentially leading to similar effects on carbohydrate (primarily glucose) levels in body secretions like vaginal secretions, urine, saliva, sweat, and tears. Physical or emotional stress: Acute stress from illness, infection, injury, trauma, surgery, tech screen abuse, cell phone use triggers the release of hormones like cortisol and epinephrine, which raise blood sugar to provide energy for the "fight or flight" response. High-intensity workouts can cause a short-term spike in blood sugar as the body releases stored glucose for fuel, especially in non-diabetics unaccustomed to such activity. Poor sleep quality from blue light and Earpod use disrupts hormonal balance, leading to insulin resistance and elevated glucose levels. Attia is known to believe that doing 100 push ups limits nnEMF risk. Look it up. He told this to Chris Williamson on a live IG post.
3. What else did Attia miss in his answer to Epstein? Cushing's syndrome: Excess cortisol production (often from adrenal tumors or prolonged steroid use) impairs insulin function and raises blood sugar.
Polycystic ovary syndrome (PCOS): This common hormonal disorder in women causes insulin resistance, leading to chronic hyperglycemia.
Acromegaly: Overproduction of growth hormone from a pituitary tumor reduces insulin sensitivity and elevates glucose.
Other hormonal issues: Conditions like hyperthyroidism or pheochromocytoma (a tumor causing excess catecholamines) can also contribute.
In my decentralized framework, this is exactly how the system is wired. The nipple-areola complex acts as a quantum-optical sensor, and the infant’s saliva is the fiber-optic cable delivering a real-time UPE (ultra-weak photon emission) status report from the child's mitochondria to the mother’s "manufacturing plant."
This isn't just convenience; it is a biophysical "handshake" that ensures the survival of the post Cambrian hardware mammals need to thrive.
1. Saliva as the "Optical Bio-Feed"
Saliva is a highly structured, mineral-rich fluid. In my thesis, it serves as the medium for optical information transfer:
The Child’s Signal: When a calf or child is sick, their internal "optical smog" increases. The VUV (Vacuum Ultraviolet) and chaotic UPEs produced by their congested Complex II are transmitted through the saliva. Congestion usually is due to reverse electron flow or deuterium.
The Mother’s Sensor: The areola is one of the most melanized and innervated tissues in the mammalian body. Melanin here doesn't just protect against the sun; it acts as a broadband transducer like an optical scanner in the grocery store. It "reads" the frequency of the biophotons in the saliva.
2. The Backflow "Optical Loop"
Research into the "infant-led" backflow confirms that when a child suckles, a vacuum is created that pulls saliva back into the mother's mammary ducts.
Information Sensing: The mother’s immune-sensing cells in the ductal walls "listen" to the ROS/RNS signatures and UPE entropy in that saliva.
The Response: If the child’s saliva "shouts" of deuterium congestion at cytochrome two because succinate is elevated or viral "optical noise," the mother’s brain (via the SCN-hypothalamic oxytocin posterior pituitary pathway) triggers a change in milk composition. She begins to "distill" more NPD1, Iodine, and IgA antibodies into the milk to act as a "quantum reset" for the child’s mitochondria.
3. The "Rotating Mom" Phenomenon (Allomaternal Nursing)
Why do calves or elk rotate moms? In my framework, this is "Frequency Matching":
Metabolic Matching: A calf may instinctively seek a different "frequency" of milk if its own mother’s melanin-metal hardware is jammed (perhaps she spent too much time in a "dirty" environment like inside a barn under fake light).
Information Diversity: By "sampling" different mothers, the young mammal is essentially "crowd sourcing" optical coherence. They are looking for the milk with the lowest deuterium/highest DHA-D3-Iodine ratio to stabilize their own emerging Faraday cage.
Modern humans with nnEMF toxicity who cannot breast feed their children due to a lack of production should be considering what elk do when they are faced with the same problem. This concept is foreign to humans because they do not observe nature carefully enough.
4. Milk as a "Re-Cambrian-ization" Serum
Mother's milk is the ultimate low-deuterium, high-DHA, solar-coded fuel.
Deuterium Depletion: Milk fat (cream) is naturally low in deuterium, helping the child build a "clean" 14 Angstrom tunneling gap in their developing mitochondria.
Melanocortin Programming: The act of nursing at sunrise/sunset ensures the child’s Leptin-Melanocortin pathway is synced to the mother’s, preventing the "Proterozoic regression" that leads to neolithic disease later in life.
The ranch memories you have are of a Quantum Ecosystem in action. The child’s saliva "tells" the mother's nipple exactly how the child's internal "mercury lamp" (deuterium) is burning. The mother then alters the "Optical Duty Cycle" of her milk to quench the fire.
Does this explain why formula-fed infants (drinking high-deuterium, seed-oil-heavy milk) are essentially being "pushed into the Proterozoic mud" from birth, as they lack this bidirectional optical feedback loop? Yes, it does but few seem to care about it.
This lesson also has deep information for the diseased breast too. Men can help their women with diseased breasts by kissing their nipples religiously to transfer information to their women about how they feel for them. This will be highly stimulatory and healing.
Cells and Stars have a lot on common. When they fail they have mechanisms that feedback on themselves that lead to the possibility of future survival if conditions are met. In a star when it burns through its fuel source its core gets to iron and the star begins to emit microwaves. The microwave radiation interacts with the D shell electrons of Fe and it blows up in a super nova so the atoms it creates in this destruction can be recycled to something with more life in the cosmos. Cells in our body use a similar idea in apoptosis, autophagy, and ferroptosis.
In my decentralized framework, the brain and breast in cancer do not operate in the same fashion regarding IDH protection because their "optical hardware" and evolutionary duty cycles are fundamentally different. Neurons are not epithelium, but breast tissue is.
While the brain relies on IDH mutations from its DHA-rich antenna to create UPEs to work and eventually help create some VUV smog from complex two back up to clean the dirty chemistry in cancer, the breast mitigates oncogenic risk through a different mechanism:
It uses the DHA-Iodine-Melanin triad.
1. The IDH Paradox: Why the Brain Needs It, But the Breast Doesn't
The brain is the ultimate "quantum sensor" that can feedback on itself and it must maintain 24/7 DHA coherence. DHA allows for this.
The Brain (DHA Antenna): When Complex II jams, the resulting VUV emission from Deuterium threatens to shatter the DHA antenna. The IDH mutation occurs as an emergency "filter" to deplete deuterium and lower the VUV entropy. DHA, as a PUFA explodes like the star and in its destructions NPD1 and Elovanoids along with UPEs are made which are highly anti-inflammatory. The released UPEs feed back on IDH and mutate it in such a specific way that the brain is able to make more deuterium depleted water because of this interaction than it could before to help self sustain its survival. This is how most low grade gliomas begin. This process does not happen in GBM transformation due to a lack of DHA in the brain.
The Breast (The Glandular Sink): Breast tissue is not a primary "spectrometer" like the brain. Instead of a mutation-driven shunt (IDH), the breast uses Iodine as its primary "quantum ground." In the breast, the "De-Cambrian-ization" of its mitochondria is mitigated by the concentration of iodine in the Ductal-Lobular Units. 2. How the Breast Mitigates Risk: The Iodine Shield
If the brain uses the IDH mutation to "buffer" the VUV smog itsdeuterium squeeze, but the human breast uses Iodine to "quench" the fire in the cytochrome 2 Fe-S clusters that begin the disease from reverse electron flow from cytochrome 2 dysfunction.The Delta-Iodolactone Mechanism: Iodine is required to form iodo-lipids (delta-iodolactone). These act similarly to Bazan’s Elovanoids in the brain, but they are specifically tuned to inhibit the Warburg Effect in glandular tissue.
Melanin & The Nipple: The high concentration of melanin in the areola/nipple is not a "decoration." It is the Optical Port that harvests solar UV/IR to control the metal flux (Cu, Fe) in the underlying breast tissue to make sure the TCA and urea cycle are the primary pathways used to avoid cytochrome 2 congestion and Fenton reactions of Fe-S couples.
The Sink: The breast is designed as a "DHA sink" (for lactation). It mitigates VUV damage by using Melanin and Iodine to sequester the "dirty" Iron noise created from a lack of sun, nnEMF, or polarized light. nnEMF for the breast is the stimulus that leads to ferrotoptosis destruction of the Fe-S couples and this mimics the process that happens in a star. When Iodine is missing, the breast cannot "ground" its own self created UPE field, leading to DCIS or invasive carcinoma without the IDH "slow-burn" protection seen in the brain. 3. The Unified "De-Cambrian" Failure
Both cancers represent a Proterozoic Regression, but the "breakdown" follows the tissue's specific metal hierarchy:Brain Cancer (GBM/LGG): A failure of the DHA-VDR-IDH loop. The brain tries to mutate (IDH) to survive the VUV fire.
Breast Cancer: A failure of the Melanin-Iodine-DHA loop. Without Iodine to "buffer" the Iron D-shell electrons, the breast tissue undergoes a rapid phenotypic regression and this is how cancer begins.
The synthesis of both molecules is tied to the Melanin-Metal hardware:
The Sun: UVB/IR input on the skin stimulates the Leptin-Melanocortin pathway. This manages the Copper (Cu) and Manganese (Mn) levels required to build the antioxidant "Mn-SOD shield."
The Translation: Coherent UPE signals (from healthy mitochondria) then tell the cell to cleave the lipids needed for NPD1 or gamma iodolactone
.
The Result: You have a "protected" post Cambrian cell that can use oxygen via the TCA/urea cycle without producing the ionizing VUV UPEs that destroys the genome.
Bazan’s Docosanoids (Brain/Retina): These cleave from DHA to form a "Faraday cage" of 22 carbons. Their purpose is to quench the VUV (Vacuum Ultraviolet) smog emitted by deuterium in the high-density neural environment.
3. UPE isn't mere waste; from quantum biology, these photons (or associated fields) may mediate non-local signaling, akin to coherence in radical pairs or bystander effects.
Intensity/spectrum reflect metabolic flux:
Aerobic paths (TCA) produce more ROS/UPE than anaerobic (glycolysis); stress shifts spectra (e.g., UV-linked UPE from glycolysis/peptides). Melanin optimizes by calibrating inputs—solar photons tune metal-ROS-UPE, enabling adaptive switches via redox/epigenetic relays (e.g., NAD+/SIRT1, from the Decentralized Medicine series of blogs on Patreon).
2. What is the rescue plan? Remember the famous now deleted Bessent tweet about DJT/Treasury plan to confiscate Bitcoin for the US Bitcoin Reserve? That was updated once the backlash on the tweet went out. Now it is about using Tether to centralize gold as they implode the Dollar and they will confiscate the Gold.
I've argued for 10 years that Bitcoin is a superior form of "trust-minimized" money compared to gold due to the high costs of verifying gold's authenticity.
This is the ability to own and verify an asset without relying on a third party (like a Central bank or Treasury of a government). Historically, gold's "trustless" nature was its greatest strength, but Savages now know this strength has been lost because most gold now sits in centralized vaults. This is why DJT won't let anyone audit Fort Knox. Why audit what you plan to steal?
The Cost of Validation for gold is steep so no one in the USA will want to pay that freight so now we are on the honor system for the Treasury.
Anyone who has owned gold knows that verifying that a gold bar before a sale/audit is real and pure requires specialized equipment, chemical tests, or expensive third-party audits. Because this is so difficult to do, you must have an inherit "trust" the vault or institution holding it for them. + Treasury and Bessent play. What did they do in 2025. They brought their middle man in. Tether. Go check if I am bullshitting you. Tether has bought more gold in the last 18 months than they have bought Bitcoin. Why? They are storing what the thieves in the industrial miliatry surveillence state will take down the road when the retards are sidetracked by circus maximus of some other psy-ops.
Why isn't Tether buying Bitcoin in this case? Anyone with a simple computer or smartphone can instantly verify the entire history and authenticity of their Bitcoin by running a node or using a block explorer. This "validation" is nearly free, making it more decentralized and harder to seize. Tether should be buying T bills but instead is buying Gold Reserves for the Zionist bankers to steal soon. Got it, my Savages.
Bitcoiners should know and remember their history better. This gameplan was used to before in 1933 during the Great Deperession to make it easy for governments to confiscate it.....Remember FDR's EO?
The U.S. did this already with Executive Order 6102 in 1933.
Looting and Centralization are the play. For thousands of years, physical gold was frequently stolen or seized by empires. To protect it, it was eventually moved into highly secure, centralized vaults (like those at the Federal Reserve Bank of New York or the Bank of England under control of the Treasury Head.
If the steal the gold this will tank markets including Bitcoin and then the Treasury will come in an sell gold at astronomical prices to buy Bitcoin at crashed Prices. This is how the Rockefeller and Rothschild Banks plan to do this.
If you know your history this is how the same guys did the scam during the Napoleaonic Wars. They manipulated the market with a psy-ops. In 1812 it was the Battle of Waterloo.
WAKE THE FUCK UP.
If you knew this history would would not be so gullible.
2. Question asked in last 6 weeks: Jack, My breast cancer recurrence has occured in the left auxiliary node. Currently taking Verzenio and tamoxifen. Declined ovarian suppression. Starting Dr. Makis protocol soon.
How can I monitor my axillary lymph nodes without using ultrasound, given your concerns about its disruption to quantum coherence in tissues? Especially since my traditional blood tumor markers (CA 15-3 and CA 27.29) have consistently tested negative?
Aside from the tests listed below, are there any additional laboratory studies you recommend prioritizing for tomorrow with Dr. G?
BUN/creatinine ratio
Vitamin D
Liver function
HsCRP
1. Today's lesson from my forum is on hyperhydrosis and dysautonimia.
QUESTION: Hi everyone,
Starting this thread to document my improvements or lack thereof, as I get closer to the Equator and further away from nnEMF.
My issue is a form of Dysautonomia driven by a small gain of function mutation in the gene encoding for Nav1.7.
The results is a persistent Na+ leak in the neurons where Nav1.7 is expressed, resulting in hyperexcitability of these neurons.
This hyperexcitability leads to the following symptoms: sympathetic overactivity, hyperhidrosis, gut hypersensitivity, more prone to visceral anxiety, bronchoconstriction, etc.
I know the decentralized medicine perspective says this is an environment problem and not a genetic problem.
But I'll only be able to confirm this once I get my environment right and get rid of these symptoms.
Best,
Alex
How can my neurons help Alex?
2. ANSWER:
Relationship Between Hyperhidrosis and Dysautonomia
Hyperhidrosis is frequently recognized as a specific symptom of a broader autonomic dysfunction.
In cases involving the upper neck:
1. Localized Sweating: Irritation of the sympathetic fibers around the vertebral artery often causes sweating or flushing on only one side of the face. 2. Systemic Dysautonomia: If the compression affects the brainstem's ability to regulate the whole body, you might experience more generalized symptoms like heart palpitations, temperature dysregulation, or "drop attacks" (sudden weakness). 3. Other relationships to be explored are found below
A. Vertebrobasilar Insufficiency (VBI): The bony bridge can compress or "kink" the vertebral artery, especially during head rotation. This reduces blood flow to the brainstem, which houses the primary control centers for the autonomic nervous system. This can manifest as dizziness, fainting (syncope), and nausea, all signs of dysautonomia.
B. Barré-Liéou Syndrome: This is a specific cluster of symptoms caused by irritation of the posterior cervical sympathetic chain (the nerves that control "automatic" functions like sweating and heart rate) due to cervical spine issues. Symptoms often include unilateral facial sweating, flushing, blurred vision, and ear ringing (tinnitus). Tinnitus brings the link to melanin dysfunction in the stria medullaris as I have laid out painstakingly on 7 Patreon blogs. It signifies an nnEMF etiology to the Hyperhidrosis and dysautonomia.
C. Trigemino-Autonomic Activation: When the C1 nerve or the vertebral artery is irritated by the ponticulus posticus, the signal is processed in the Trigeminocervical Complex. This can trigger a "reflex" in the autonomic system, leading to craniofacial hyperhidrosis (sweating on the face/forehead), nasal congestion, or eye-watering. This can also be stimulated by demyelination in this region by melanin POMC defects, DHA defects in the central retinal pathways, or polarization toxicity that affects the nerve complex that links these two disease. Both, hyperhidrosis and dysautonomia are located in two distinct but interconnected systems: the Central Autonomic Network (CAN) in the brainstem and the Peripheral Sympathetic Chain in the neck. Hyperhidrosis in this scenario is typically a "positive" neurological phenomenon (overactivity) caused by irritation of the Superior Cervical Sympathetic Ganglion (SCG) and the Periarterial Carotid Plexus (lots of POMC).
3. ANSWER CONTINUES
The broader "dysautonomia" (dizziness, heart rate changes, nausea) stems from a defect in the Lower Brainstem, specifically the Nucleus Tractus Solitarius (NTS) and the Ventrolateral Medulla. These are the primary control centers for blood pressure and heart rate, located in the medulla oblongata of the brainstem. When these brainstem centers are deprived of oxygen-rich blood, like we see when Fe is forced into the +3 state over the +2 state, they fail to regulate the autonomic system correctly. This is why nnEMF can cause this syndrome. This results in the "mismatch" symptoms of dysautonomia, such as postural dizziness, syncope (fainting), or "drop attacks" where the legs suddenly give out.
Because these symptoms are often positional, it is highly recommended to speak with a radiologist specialist about a Digital Motion X-ray (DMX) or a CT Angiogram to see how your vertebral artery behaves when you move your neck.