1/5 THE BIG FAT LIE about Cholesterol may well kill millions of people without ever interfering with their rights, their beliefs, or their backgrounds. The cholesterol juggernaut has figuratively rolled over the world’s population and shows how government agencies /2
2/5 and heart associations, have forced a drastic change in dietary habits with mostly manufactured evidence, and how those dietary changes, along with dangerous drugs such as statins, can cause far more disease, disability, and death than the illness they are supposed /3
3/5 to prevent. This may seem perplexing to some, especially if you have not been brought up in the world of statistical shenanigans, but the only real way one can counter the corrupt propaganda and the misleading preaching about the alleged cholesterol menace is to discover /4
4/5 all that is known about the subject and second, to refute the relevant statistical malpractice that has been foisted on the general public. To do this, it is necessary to show, in detail, the misinterpretations, the misrepresentations, and lies told by the /5
5/5 anti-cholesterol forces.
To succeed requires a willingness to peruse patiently, to evaluate open-mindedly, and to arrive at conclusions based on what FACTS there are available without succumbing to the implied promise of extending one’s life for an extra 8+ days.
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1/8 Please don't be fooled. There is NO such thing as a Statin “side effect”. Statin toxicity is a direct and predictable effect of blocking MEVALONATE by inhibiting the enzyme HMG-CoA Reductase, which makes it. /2
2/8 Statin poisoning is a sign that certain cells cannot mutate by increasing REDUCTASE and are unable to overcome the direct CELL TOXIC effect of the Mevalonate blockade of Statins. Pharmaceutical companies knew this back in 1980, but failed to mention this to the FDA. /3
3/8 The Journal of Biochemical Chemistry reported that when HMG-CoA Reductase is inhibited by Statins, Mevalonate formation is blocked and cultured cells DIE. Every Statin user is a victim of this process. /4
1/10 For years people have been led to believe that cholesterol is some kind of evil fatty substance that somehow gets into the bloodstream, where it doesn’t belong, and clogs arteries. Since everyone is being taught to fear cholesterol we should know that cholesterol is /2
2/10 actually a crystal of solid alcohol known as a steroid - specifically a sterol. The word cholesterol comes from Ancient Greek chole- 'bile' stereos 'solid', followed by the chemical suffix -ol for an alcohol. Cholesterol is vital in the membranes of cells /3
3/10 where it regulates the exchange of nutrients and waste products. It is critical in growth and development of the brain and nervous system, and acts as a conductor of nerve impulses. It is a necessary component in bile acids which promote the digestion of foods. /4
1/16 I’m more than happy to work with @DoctorTro
on heart health. His dietary advice and focus on preventing heart disease are spot on. But I cannot accept the claim that statins are plaque busters. /2
2/16 It is based on flawed, industry-funded studies that don’t hold up to scientific scrutiny
Statins’ mechanism of action tells a different story. Statins increase blood glucose, cause apoptosis in smooth muscle cells lining the endothelium, damage miochondria, and induce /3
3/16 oxidative stress. How can a drug with these effects shrink plaque ? It defies logic. Worse, statins block Vitamin K2 synthesis, a key cofactor for matrix gla-protein, which PROTECTS arteries from calcification. Statins also impair selenoprotein production, like /4
1/4 Claiming “no ideological conflicts” is a lazy way to dodge the real debate. The argument over statins isn’t ideology, it’s about their actual effects. Statins block the mevalonate pathway, which doesn’t just lower cholesterol but also starves cells of compounds needed
/2
2/4 for signaling and membrane health. This can lead to myositis, memory issues, and calcification in arteries that makes heart disease worse. Rosuvastatin, which you seem to like, can still mess with brain cholesterol enough to cause problems like brain fog. /3
3/4 On top of that, statins raise blood glucose leading to insulin resistance, which fuels inflammation, the root of heart disease they’re supposed to fight. /4
1/6 @DoctorTro Your tourniquet analogy is a stretch, and honestly, it feels dismissive of the real risks statins pose. Tourniquets are a temporary, external fix to stop bleeding, while statins cause systemic, biochemical changes with long-term consequences like muscle damage, /2
2/6 neurological effects, heart failure, autoimmune issues, and even increased artery calcification. Tourniquets don’t alter cellular pathways or cause widespread cell death, but statins, by depriving cells of mevalonate, can kill cells and lead to serious effects like
/3
3/6 weakened muscles, higher infection risk from lowered LDL, worsening heart failure in vulnerable patients, autoimmune conditions, and calcified arteries that can raise cardiovascular risk. /4
1/8 I certainly do have an opinion on this article, because this post highlights the oversimplified advice often given about statins like Rosuvastatin. (Crestor). Suggesting that supplementing with CoQ10 alone can mitigate statin-induced damage is reckless and overlooks the /2
2/8 broader biochemical disruptions these drugs cause. Statins inhibit HMG-CoA reductase, which not only reduces CoQ10 levels but also blocks other essential isoprenoids like dolichol and prenylated proteins. These compounds are vital for cell signaling, membrane integrity, /3
3/8 and overall cellular function, and their depletion can lead to cascading health issues that a single supplement like CoQ10 cannot fully address. The notion that CoQ10 is a catch-all solution dismisses the complexity of these pathways and the potential for serious, /4