_aestheticprimal_ Profile picture
Jul 21, 2025 15 tweets 5 min read Read on X
The protein hack that can add INCHES to your height
(and why "20g protein" labels are misleading)

Most people think protein is just protein. They're dead wrong.

and it isn’t just about animal > plant protein

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Here's what they don't tell you:

Not all proteins are created equal. Even different cuts of the SAME animal have completely different amino acid profiles.

That "20g protein" on your label? Useless without knowing WHICH amino acids you're getting.

this is the silent height killer
your growth plates are primarily made of 4 specific amino acids:

glycine, glutamic acid, (hydroxy)proline, alanine

except for glutamic acid, these are found in LOW amounts in regular muscle meat (chicken breast, steak, etc.)

but HIGH amounts in collagenous foods Image
here's the shocking math:

average person consumes only 3g of Glycine per day.

increasing Glycine intake by just 10g daily = 200% INCREASE in collagen synthesis.

if you know the role collagen plays in height growth, you’d realize how crucial this is. Image
Collagen is so crucial for deciding how tall you become, it composes a majority of the tissues that compose your height

30% of growth plates = collagen
40% of bones = collagen
60% of spinal discs = collagen

low glycine = CATASTROPHIC for height Image
The brutal truth:

Unless you're eating pig nose, chicken joints, and beef tendons DAILY, you are not getting in enough Glycine.

Your expensive grass-fed steak? Chicken thigh? Whey protein powder?

They DON'T have the amino acids your growth plates need. Image
Even worse..

Muscle meat, protein powder or even plant proteins don’t just LACK the amino acids your body needs to grow taller

It’s also high in the amino acid which SLOWS DOWN height growth:

Tryptophan. Image
When animals are exposed to a low-tryptophan diet, their biomarkers improve IMMENSELY

A low-Tryptophan diet has been shown to increase:

- serum T3
- growth plate size
- GH by 2-3fold
- size of pineal and pituitary gland

All are CRUCIAL for height Image
This is why smart people are turning to Beef Gelatin.

Instead of choking down plates of connective tissue and joints every day…

Mix gelatin powder with water/coffee.
Drink it.
Done.

You now have the amino acids your body needs, to grow taller.
The difference is night and day:

❌ Regular protein powder: Too high in tryptophan
❌ Chicken breast: Low glycine, proline, alanine
❌ Even quality steak: Missing the collagen precursors
✅ Beef gelatin: Loaded with growth plate amino acids

The type of protein you eat, is crucial for height
What about supplementing Glycine directly?

I love Glycine as supplement, but…

Overprocessed, tends to contain a lot of traces of industrial chemicals.
Lacks anti-inflammatory mead acid
No Alanine, (Hydroxy)proline or Glutamic acid

It’s a suboptimal variant of Beef gelatin, which is more expensive per gram
The protocol is simple:

1-2 scoops of high-quality beef gelatin daily
Mix with water or coffee
Best taken with high protein meals or during coffee breaks

Within weeks, you'll notice the difference in recovery, joint health, and probably even faster heightgrowth. Image
But heightgrowth is not only limited to puberty itself

The intervertrebal discs are made out of collagen for 60% and make up for around 16,5% of your total height.

Loss of Collagen in these discs is why people tend to shrink with age.

But with gelatin powder you can GAIN height by gaining Collagen in these discs by around 1 or even 2 inches.Image
Your genetics load the gun, nutrition pulls the trigger.

You might have incredible height potential locked in your DNA

But your flawed amino acid intake us keeping you away from your true height-potential
Don't leave inches on the table.

Your growth plates won't stay open forever. Every day you're glycine deficient is a day of lost potential.

Obtain Beef Gelatin powder here:

Make sure to like and retweet.aesthetic-alimentum.com/products/beef-…

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More from @aestheticprimal

Jun 22
Shilajit is NOT a Testosterone booster, it is a mitochondrial enhancer, which is where it truly shines.

1g in morning = amazing workout + brain energy

but why?

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DBPs in Shilajit (and why they're so interesting)

Most people talk about how Shilajit contains a lot of trace minerals and Fulvic acid.

But the most interesting compounds that can be found in Shilajit are the DBPs, which are also known as:
Dibenzo-alpha-pyrones

let me explain👇Image
Why are these DBPs so beneficial?

there are 3 big reasons

(boring science warning)

NUMBER ONE:
they're 'Quinone-like' compounds

DBPs are technically not Quinones, because they do not have the structure of a Quinone (their core structure is a fused tricyclic lactone and not a cyclic dione)

But they act like quinones in redox reactions, electron transport, and mitochondrial function due to how they cycle between three redox states (reduced, semiquinone radical, and oxidized/quinone-like forms).

Accepting electrons from Complex I and II →  then carrying those electrons to Complex III → in Complex III the electrons are donated and carried to Complex IV where the electrons are handed off to Oxygen preventing electrons to accumulate at Complex III and excess ROS to be produced

NUMBER TWO:
They work in tandem with CoQ10

CoQ10 shuttles electrons between Complex I/II & III but can be easily oxidized back from Ubiquinol (active CoQ10) back into Ubiquinon (oxidized form) under stress.

DBPs prevent the oxidation of Ubiquinol into Ubiquinon, and keep CoQ10 in its reduced form, protecting it from oxidative stress.

What does this mean?
Ubiquinol stays active for a longer time duration in the ETC (electron transport chain) causing less electron leakage and increased ATP production

Ubiquinol is kept in the reduced form and its ability to neutralize free radicals is extended because of how Ubiquinol acts as antioxidant

Mitochondrial membrane potential is now better preserved

Energy production stays stable and efficient under stress, heavy exercise or even prolonged period of focus and attention

Can handle more exercise, more work and even more emotional stress

(more on how Shilajit/DBPs enhance brain energy through improved mitochondrial function of the PFC later in this thread)

NUMBER THREE:
Mitophagy benefits of DBPs

Mitophagy is the process where your cells detect/recycle damaged/dysfunctional mitochondria through autophagy which prevents excessive ROS/oxidative stress and ensures that inefficient mitochondria are cleared away, keeping energy production efficient.

This helps to prevent fast aging, low energy and diseases.

So how does Shialjit enhance mitophagy through DBPs?

One of the primary DBPs in Shilajit is:
‘3,8-dihydroxy-6H-dibenzo[b,d]pyran-6-one’

this DBP is chemically similar to Urolithin A

It stabilizes PINK1 on damaged mitochondria, which then helps to recruit ‘Parkin” which then makes the mitochondria a visible target for mitophagy by attaching ubiquitin chains to the many proteins that sit on the outer membrane of the mitochondria.Image
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Read 6 tweets
Mar 30
Current Dopamine enhancement stack

extra fire to crush objectives (and actually chase the right objectives)

- 5mL Cerebrolysin (EOD)
- 2-4 IU Intranasal insulin (almost daily)
- 2g+ royal jelly (daily)
- 10-15mg 9-me-bc (daily)

below is why👇 Image
Cerebrolysin seems to enhance Dopaminergic transmission mainly through improving redox balance (total antioxidant capacity), and also seemingly through activation of GDNF receptor complex.

Evidence on Cerebrolysin and Dopmaine itself has not been studied much, but from personal experience, the 'Dopamine baseline seems to be much much higher.

when it comes to sourcing it, it's very different per region, Rupharma is decent, but even Indiamart is a better alternative

You have to dig a bit to properly source itImage
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Intranasal Insulin seems to be something very niche, until you realize that there's a tremendous amount of scientific literature on it, just go on google scholar and type in 'intranasal insulin' and you'll see.

Seems to be mainly mediated through improving redox balance in the brain, just like Cerebrolysin

no, intranasal insulin does not cause any insulin resistance (as far as we know)

@doctormorphh has written some great threads on it, go check it out.

When it comes to obtaining it, again completely dependent on region, but it requires a little bit of digging (they literally have Insulin everywhere, you just need an atomizer for intranasal delivery)Image
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Read 5 tweets
Mar 20
There's a guy on instagram doing an n=1 experiment using bees stings to grow his hairline back.

it's working exceptionally well, for a REASON, it tackles ALL the underlying mechanisms of balding.

So what's the science behind the most effective hairloss cure?👇
Bee venom against fibrosis/scar tissue formation in the scalp:

Male pattern baldness is a slow process of scar tissue formation in the scalp (fibrosis), eventually when the amount of scar tissue starts accumulating more and more, it starts to suffocate the hair follicles, and they die off.

This entire process is mediated by a ton of hormonal inputs, inflammatory cytokines and hypoxic markers, but they all pretty much exert their effect on speeding up male pattern baldness by upregulating TGF-beta1.

TGF-beta1 is the master regulator of Fibrosis (scar tissue formation), its activation leads to:
→ fibroblast activation
→ ECM deposition
→ myofibroblast differentiation

Reducing expression of TGF-beta1 is key when it comes to putting a halt to male pattern baldness.

And Bee stings reduce TGF-beta1 expression exceptionally well through Melittin by inhibiting NF-κB activation (through blocking IκB degradation) and suppressing the consequent release of various pro-inflammatory cytokines like TNF-alpha, IL-6 and IL-beta1 as well as through the direct interference of Melittin with the TGF-beta1 receptor itself.

By blocking the expression of TGF-beta1, Bee stings prevent scar tissue formation from occurring, reducing the exact mechanism responsible for Norwooding.

study references:
mdpi.com/1420-3049/21/9…
worldscientific.com/doi/abs/10.114…
pmc.ncbi.nlm.nih.gov/articles/PMC11…
pmc.ncbi.nlm.nih.gov/articles/PMC46…Image
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Bee venom promotes blood flow in scalp:

Bee venom locally upregulates VEGF, a growth factor which dictates the process of angiogenesis, this is the process where new capillaries (very small blood vessels) are formed locally.

VEGF is already produced by cells in the hair follicle during the Anangen (growth phase) to enhance vascularization which helps with improving blood flow to the hair follicles that delivers the necessary oxygen and nutrients to grow healthy (and stay alive).

VEGF its expression is higher when the hair follicle is in the Anagen (growth) phase, starts declining in the Catagen (regression) phase, and eventually becomes very low during the Telogen (rest) phase.

Bee venom its ability to potently enhance local VEGF expression ultimately helps with improving vasculature of the scalp and allows for hair follicles to get the necessary nutrients needed for growth.

study references:
jstage.jst.go.jp/article/bpb/39…
pubmed.ncbi.nlm.nih.gov/38393162/
mdpi.com/2072-6651/13/1…Image
Read 6 tweets
Jan 22
There’s nothing with a more neurotoxic profile and a dimming effect on the soul like viewing yourself as a loser has.

viewing yourself as a loser actually has a scientific term → ‘social defeat stress’

you essentially view yourself as a victim to your external environment (lack of control equates to learned helplessness) and at the bottom of the social hierarchy.

This feeling of not being in control of situations, or lacking a strong future vision, weakens activity in the prefrontal lobes (mainly mPFC, vmPFC and dlPFC).

These prefrontal cortexes normally have an inhibitory effect on the Amygdala, but when this inhibition fades due to negative self-talk and surrender to external environment, it causes the gut-microbiome its alpha-diversity to decrease through HPA axis activation.

result?

reduced SCFA production, with Butyrate in particular.

Butyrate is crucial for:

- HDAC inhibition (intelligence, memory, focus, motivation)
- androgen receptor expression
- reducing estrogen receptor expression through hyperacetylation
- increasing natural Testosterone production through the HPG
- thyroid hormone uptake

Back in 2022, a bunch of researchers showcased how they were able to completely alter the social dynamic between two mice by simply altering their gut-microbiome through antibiotics, targeting C. Butyricum in particular (one of the main bacteria that produces Butyrate)

The researchers could turn an ‘alpha’ mouse into a ‘beta’ mouse, making it subordinate to the mouse that was previously ‘beta’ in the social dynamic.

It was found that recolonization of the gut-microbiome of said mouse that ‘turned beta’ with C. Butyricum could restore that mouse’s status, and make him ‘alpha’ again.

The researchers thought this was because of the huge role that SCFA’s, with butyrate in particular, play in activity of the prefrontal cortex (mPFC, vmPFC and dlPFC) due to Butyrate its function as HDACi.

With insufficient SCFA/Butyrate levels, it’s almost guaranteed that the function of the prefrontal cortex is going to be impaired in some way.

Results:

- less discipline
- lack of willpower
- reduced goal-achieving capabilities
- surrender to reptile brain (prefrontal cortex exerts inhibitory control over these)
- ADHD-like behavior
- excess rumination of thoughts/overthinking (negative spiral)

-

mPFC activity is the most accurate biomarker to predict spot in the social hierarchy

high mPFC activity is endlessly more correlated with being 'alpha' than Testosterone ng/dL levels

-

The prefrontal cortex is extremely active when choosing non-pleasurable things for long-term gain over short term gratification.

strong willpower, discipline and goal-achieving capabilities are the symptoms of a highly developed and active mPFC, vmPFC and dlPFC.

and the gut-microbiome is arguably the biggest influence on these brain regions after mindset.

this is why ‘alpha’ mice became ‘beta’ after their gut-microbiomes were attacked (something currently happening to 99% of people through processed foods and silicone dioxide-filled supplements)Image
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your gut-microbiome, almost acts as your second DNA, this is not only because of how tightly it correlates with epigenetic expression by altering histone actetylation/deacetylation, it's way deeper than that.

there's a reason people even experience personality changes from fecal microbiome transplants (not just intestine transplantation)

the contents of the gut shape who you are, and you're quite literally what you eat (no this shouldn't be dumbed down to micronutrient intake only, think of miRNA's that have been shown to alter genomic expression in the organism, and way more than that)

x.com/aestheticprima…

sciencedirect.com/science/articl…
Read 5 tweets
Dec 14, 2025
GDNF is a neurotrophic factor your body produces that acts as natural FERTILIZER of the Dopaminergic system.

- Dopamine synthesis
- Dopamine neuron size and health
- Dopamine release

Optimize GDNF before you stimmaxx the F out of yourself

THREAD🧵 Image
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Neurotrophic factors are proteins that support the growth, survival and differentiation of neurons, examples include:

- NGF (nerve-growth factor)
- BDNF (brain-derived neurotrophic factor)
- NT-3/4/5 (Neurotrophins 3/4/5)
- GDNF (glian-derived neurothropic factor)

GDNF is the neurotrophic protein that is most targeted towards the Dopaminergic system, this protein acts like a fertilizer of your Dopaminergic system improving it from all angles.Image
1. Enhanced Dopamine synthesis
GDNF upregulates the mRNA expression of Tyrosine Hydroxylase, the rate-limiting step in Dopamine synthesis.

2. Improves count, size and health of Dopaminergic neurons
Dopamine itself is mainly produced in the Dopaminergic neurons, the size and amount of these neurons, as well as their health basically decides your Dopaminergic baseline.

3. Improves Dopamine release
Dopamine has to be released from the pre-synaptic endings of a neuron to bind to the synapse and have an effect.
These pre-synaptic endings are also called terminals.
GDNF promotes release from these terminals and also increases the size and count of terminals, so Dopamine release becomes more efficient in the long-term.

4. Protection of Dopaminergic system
GDNF protects against oxidative stress, toxins and everything that could damage the Dopaminergic neurons, terminals, receptors etc.
Mainly by activating anti-apoptotic signalling pathwaysImage
Read 7 tweets
Nov 19, 2025
Height-growth DOESN'T have to stop AFTER the growth plates have closed, and the science points towards that.

There's more than enough proof showcasing that height-growth IS possible after growth plate closure.

THREAD🧵 Image
Let's start by looking at the fingers.

Which still shows increases in length even after the growth plates have fully fused

This study found that after the age of 21 (age where the growth plates have fused) the length of the finger continues to increase in size with the normal aging process.

This increase in finger size was much higher in males that worked manual labour, which is a direct argument for Wolff’s law and how mechanical stress influences bones, even at later ages.

This increase in finger length is tiny, but the fact that the length keeps increasing, even when the growth plates have closed down, goes to show that bone doesn’t just lengthen only from the growth plates.Image
The size of the fingers/hands KEEPS increasing AFTER all the growth plates in the hands have fully fused, and the hands/fingers actually keep growing in size even 4 YEARS after the growth plates have fully closed.

With some increases in size even going as high as 5 whole centimeters (2 inches for my american friends), that’s not miniscule, it’s actually quite significant, and the bone growth happens AFTER the plates were closed.

How would this be possible when the growth plates were closed?

The only answer you can give to this question, is by accepting that bone DOES lengthen even when the growth plates are closed, there’s no room for disagreement.Image
Read 8 tweets

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