Thread by @DrJackKruse

Another new podcast from me with Smuggling Hope. It is good one on mental health because it explains how biomolecules absorption and emission spectra's determine reality or determine which mental illness one gets.

This is the information why Jordan Peterson is sick and why he and his daughter have stumbled multiple times in getting him well.
ivoox.com/.../exposing-t…...
Biophotons are ultra-weak light emissions produced by living organisms, thought to arise from metabolic processes like oxidative reactions in mitochondria. Centralized scientists and AI bots hypothesized them to play a role in cellular communication, potentially influencing gene expression or signaling pathways. FIAF, also known as angiopoietin-like protein 4 (ANGPTL4), is a protein produced by tissues like fat and the gut, and it’s a key player in lipid metabolism and microbiome construction. Neither understand how UPEs control FIAF. If the UPE is coherent then FIAF inhibits lipoprotein lipase, affecting how fats are stored or broken down, and its expression ramps up during fasting. The microbiome, meanwhile, is the community of gut microbes that can shift in response to diet, fasting, or host metabolism, influencing energy balance and health. If the UPEs in mitochondria is not coherent, the UPE changes and mental illness becomes more probable. You cannot burn fat so it changes neural signaling.

Centralized scientists and technocrats who build AI systems will say no direct study says “biophotons control FIAF to affect the microbiome,” but we can hypothesize based on related mechanisms. This is patently false. Why?

FIAF has known absorption and emission spectra to light frequencies. Because of that, a study is superfluous because each chemical in the world has an optical fingerprint. Just because a scientist has not published the work is immaterial to this BASIC biophysical fact in spectroscopy. This was what I brought Ray Peat over ten years ago, and he was impotent enough to answer my critiques of his work. This podcast covers these details.

2. Is nerve pain caused by a Lyrica deficiency?
Is Lyme disease linked to a lack of doxycycline?
Is depression due to Prozac deficiency.
Are heart attacks are due to Lipitor deficiency.
Is obesity due to Ozempic deficiency.
Are Headaches due to Tylenol deficiency?
Is Bipolar disorder due to lithium deficiency?
Any questions?
You've been conditioned by centralized medicine to ask the wrong question.
You have a solar deficiency combined with a nnEMF toxicity problem.

This alters the UPEs your mitochondria make and it is this light that changes the neural tracks that make you mentally ill. This is why your Bipolar Disorder exists. The defect is not in you; it is in your environment.

3. Light exposure,say, sunlight or specific wavelengths impacts circadian rhythms via the suprachiasmatic nucleus in the brain, which regulates hormones like dopamine, GABA,melatonin and cortisol.

These hormones influence metabolism, including fat tissue activity where FIAF is expressed. Metabolism is what makes the key UPEs.

Fasting, which boosts FIAF, is also tied to light cycles, think of how daylight affects feeding patterns. If biophotons reflect or amplify these light-driven processes at a cellular level, they might indirectly tweak FIAF production by signaling energy states or oxidative stress in cells. If you cannot burn fat you are more likely to be mentally ill.

My decentralized ideas have always been spot-on that this topic doesn’t need a scientist to spell it out in a paper, absorption/emission spectra are measurable, and biophoton emissions are detectable.

The gap isn’t in the physics; it’s in tying the specific wavelengths of biophotons (which vary by cell type and state) to FIAF’s exact optical profile in vivo. Still, if gut cells emit biophotons in, say, the 300-400 nm range, and FIAF absorbs there, the optical photonics interaction’s real, study or not. It is first principle thinking. It is obvious what they problem. It’s like saying water absorbs infrared; we don’t need a new experiment to prove it gets hot under sunlight. Biology acts like it does, but in physics they use theoretical physics and first principle thinking to make predictions when the experiments are not done or cannot be done.

youtube.com/watch?v=1F5cik…

4. FIAF (ANGPTL4), has a unique optical fingerprint—its absorption and emission spectra—dictated by its chemical structure. That’s a bedrock principle of spectroscopy, not something needing a specific study to confirm for each compound. If FIAF interacts with light at certain frequencies, that’s a fact of its molecular identity, not a hypothesis awaiting proof. AI and Ray Peat suffered from the same centralized sickness science taught them. Junk science and beliefs in = junk ideas out.

Let’s pivot and dive into this from that starting point using decentralized science.

Biophotons, being ultra-weak emissions in the UV to visible range (roughly 200-800 nm), come from processes like oxidative metabolism in cells. FIAF, as a glycoprotein, has amino acids (e.g., tyrosine, tryptophan) and lipid-associated components that give it specific absorption peaks—likely in the UV range around 280 nm for aromatic residues, with emission potentially red-shifted depending on its microenvironment. If biophotons emitted by the mitochondria of nearby cells or gut tissue overlap with FIAF’s absorption spectrum, they could excite its molecular structure, altering its conformation or activity.

This isn’t speculative; it’s how light-molecule interactions work—think fluorescence or photochemistry.
So, how might this control FIAF and ripple to the microbiome? When FIAF absorbs biophoton energy, it could shift its folding or binding affinity, say, for lipoprotein lipase or its transcriptional regulators like PPARs. This tweak could amplify FIAF’s expression or function beyond what fasting alone does.

Since FIAF inhibits fat storage, more active FIAF means less lipid availability in the gut, which starves certain microbes (lipid-loving Firmicutes, maybe) while favoring others (lean-associated Bacteroidetes or Akkermansia). The microbiome shifts not because of some downstream guesswork but because FIAF’s light-driven tweak directly changes the gut’s energy landscape.

5. Can you use the TCA or urea cycle if you do not see AM sunrise? NOPE.

6. Peat and AI bots missed my other main decentralized points. The human microbiome is crafted from bacteria, which are prokaryotes. Fritz Popp showed us in his lab that bacteria release 5000 times more biophotons than eukaryotes do, so the light signal in the gut and GALT would be massive to control microbiome diversity. Microbiome is the light projector and out gut enterocytes are the screen. @SabinehazanMD

UPEs change the immune system in the GALT.

Fritz-Albert Popp’s work on biophotons does indeed highlight a massive difference: bacteria, as prokaryotes, emit biophotons at rates orders of magnitude higher, up to 5,000 times more, than eukaryotic cells like ours. That UPESlight sculpts the pathways in cells because it changes the AMO physics in cells. That flips the script on the gut’s light environment, and Peat should have considered this during my discussion with him and caught how that amplifies the signal I was driving at. He did not, and it is why I flipped on him.

7. Popp’s research showed bacteria churn out biophotons, think UV to near-infrared, peaking around 200-800 nm, through the metabolic hustle of mtDNA, especially oxidative reactions. In the gut, with trillions of bacteria in the microbiome, that’s not just background noise; it’s a flood of light. The gut-associated lymphoid tissue (GALT), sitting right there with immune cells and epithelial barriers, is bathed in this bacterial biophoton output. If we’re talking 5,000 times the intensity of human cell emissions, the gut’s lightscape is dominated by prokaryotic chatter, not our own cells.

Now, FIAF, produced by gut and fat tissue, has its optical fingerprint, absorbing light (say, UV around 280 nm from its aromatic amino acids). This bacterial biophoton barrage could hit FIAF directly, exciting its molecular structure far more than eukaryotic emissions ever could. Picture it: a constant, intense light signal tweaking FIAF’s conformation or activity, ramping up its inhibition of lipoprotein lipase. More FIAF action means less fat storage, shifting the gut’s nutrient pool, less lipids for bacteria to munch on. This could steer microbiome diversity hard, favoring lean-adapted species (like Akkermansia or Bacteroidetes) over fat-thriving ones (Firmicutes), just as you see in fasting states where FIAF spikes.

8. But it’s not just FIAF. That bacterial light flood could signal across the gut-GALT axis, influencing epithelial cells, immune responses, and even microbial gene expression. This light signal controls T regulator cells to program into other T cells like NK Killer cells and TOLL receptors.

If this UPE light signal is off FOR ANY REASON, many diseases we have no answer for in centralized science are possible, like cancer, autoimmune conditions, and neurological issues related to the brain-gut axis. This would affect the electrical signals that maintain all the barriers in our organ system, which Michael Levin has discussed in his work.

Bacteria themselves might “read” each other’s biophotons, Popp suggested coherence in these emissions could coordinate behavior. In the gut, this might mean quorum sensing on steroids (check my podcast out I did with Kriven Govender on the gut years ago warning quorum sensing is the ticket), with light shaping which species dominate based on who thrives under the energy conditions FIAF helps set = Roeland Van Wick work.

Decentrlaized science is spot-on: the sheer scale of bacterial biophoton output makes the gut a light-driven ecosystem, not a food-driven one as mainstream often assumes. FIAF’s control here isn’t some quiet side effect, it’s a loud, UPE light-mediated lever tuned by that prokaryotic glow.

9. I am peeling back layers here, and I see where Peat and centralized science have been too narrow, I'm pushing you to catch up. I'm weaving together biophotons, FIAF, the gut-GALT axis, immune programming, and electrical signaling in a bold way that connects dots centralized science often leaves dangling. Let’s dive in and address the pieces Peat missed, tying in Popp, Levin, and my quorum sensing angle.

I know I am dead right that bacterial biophoton floods, 5,000 times stronger than eukaryotic emissions, per Popp, don’t just tweak FIAF; they’re a roaring signal that changes the information across the gut-GALT axis.

This isn’t a whisper to epithelial cells or immune players like T-regulatory (Treg) cells; it’s a megaphone. T-regs, which orchestrate immune tolerance, can differentiate into effector cells like NK cells or influence Toll-like receptors (TLRs) on gut barriers.

UPE Light’s role here is hard to fathom for centralized science but it is REALITY: if biophotons from bacteria (UV to near-IR, coherent per Popp) hit photoreceptors or chromophores in gut cells and their mitochondria, they could trigger signaling cascades, say, via calcium fluxes or redox shifts, that reprogram T-regs.

Studies already show light modulates immunity (e.g., UV affecting skin T-regs), so in the gut, this bacterial glow could be the conductor, tuning NK cell aggression or TLR sensitivity.
The Noble Prize in 2025 is wind in my sails.

10. If that UPElight signal’s off—too weak, incoherent, or drowned out by modern disruptions like artificial light or poor circadian cues, the chaos of illnesscreeps in. This tells us UPEs carry information. I do not need a biophysicist paper to tell me this. It is obvious by first principle thinking.

This alteration of the fidelity of the signal is where chronic diseases begin. It is not from food.
FIAF might falter, fat metabolism skews, and the microbiome shifts toward pro-inflammatory species (more Firmicutes, less Akkermansia and bacteroides).

But beyond that, misfired biophotons could scramble T-reg programming, letting autoimmune conditions (e.g., Crohn’s) or cancer (unchecked cell growth) take root.

The brain-gut axis fits too: vagus nerve signaling, which Levin’s work on bioelectricity touches, relies on gut barrier integrity, which nnEMF destroys via Frey's work.

Levin’s shown organs use electric gradients to maintain form, disrupt that with a broken UPE light signal, and barriers (gut, blood-brain) leak, sparking neurological havoc like Parkinson’s or depression.

11. Popp’s coherence point about UPE is gold: bacteria emitting synchronized biophotons could act like a gut-wide quorum sensing network. My podcast with Kriven Govender nailed this, quorum sensing isn’t just an electrical or chemical change; UPE light supercharges the transfer of information required for health or disease.

Imagine bacterial species “voting” via biophoton pulses, deciding who thrives based on FIAF-set energy conditions. Roeland van Wijk’s work backs these decentralized ideas up: he built on Popp, showing biophoton emission reflects cellular health and coordination. In the gut, dominant species might amplify their light signature, outshining rivals, with FIAF as the metabolic referee.

Peat missed these points and the proof he did is in the dvice he sold you,namely, that immune and photobioelectric scope earlier, at your peril.

His OJ advice is deadly when you understand these ideas. This light-driven model explains diseases centralized science fumbles: cancer from deregulated cell signals, autoimmunity from immune misreads, and neurological decay from gut-brain disconnects. Levin’s bioelectricity ties some it together because electricity precedes chemistry in quantum cells: but Levin and his PhD friends still do not realize UPE light is proximal to all electrical signals in cells. It is the biophotons that shape membrane potentials across barriers, and a glitchy UPE signal is what causes collapse the whole system. Traffic lights work just the same way as UPEs do in my framework.

12. How does it all connect? Bacterial biophotons have a spectrum of action from UV to near-IR, coherent to incoherent as Popp demonstrated. They aren’t just background chatter; they’re a high-fidelity broadcast hitting photoreceptors and chromophores in gut cells and their mitochondria. Think opsins or flavins in cell membranes or cytochrome c oxidase in mitochondria, absorbing these wavelengths (say, 100-800 nm).

When the signal’s crisp, it triggers precise cascades, mitochondrial ROS spikes, calcium waves, or gene switches like NRF2 or PPARs, keeping the gut-GALT axis humming. FIAF gets tuned, T-regs stay balanced, and the microbiome aligns with lean, anti-inflammatory species. It’s a symphony of UPE light driving our music, not a food-driven plod.

But when that signal degrades, too weak from a gut stripped of bacterial diversity, incoherent from chaotic emissions, or drowned by artificial light’s 24/7 blue glare (think LEDs peaking at 450 nm), the system's light information transfer unravels. Mitochondria misfire, altering their biophoton emission, missing their light cues; ROS goes haywire, not hormetic. I am waiting for some of these scientist to break free of the mentor's anchors keeping their labs in harbor.

13. The 2025 Nobel Prize is your clue I am right and the rest of them are wrong. You the public will decide who is right by the diseases you reverse using Nature.

UPE can skew T-regs/NK cells into overdrive or apathy, TLRs overreact, and barriers (gut, brain) leak like sieves. This isn’t a downstream effect of diet; it’s the upstream signal breaking. Chronic diseases, MAHA is screwing up like jab induced UPE changes that cause cancer, autoimmunity, clotting, and neurodegeneration, don’t start in the kitchen, the start in the light you live within. In modern light , your UPE light fidelity collapses and that causes your disease. It is not Fruit Loops or Red dydes. It is not just tyelnol. Food might nudge the microbiome, but the biophoton distortion, amplified by modern life’s decoupling from natural light cycles, lights the fuse of all chornic diseases because it is the biggest effector in the optical signal that runs our core software.

Centralized science stumbles here because it’s obsessed with nutrients, not photons. Popp saw it and taught me to see it: coherence matters more than intensity. Precision and low intensity is the key with UPEs. A gut awash in bacterial light should sing; disrupt that with screen glare or sterile living, and you get discord, disease. Levin’s bioelectricity fits, too but it is half truth: if biophotons set membrane potentials, a garbled signal rewires the body’s electric map, birthing chaos.

Even the clueless in centralized medicine are waking up the story I have been teaching you for 20 years. Ignore me at your peril.

14. Levin’s work shows cells use bioelectric gradients, membrane potentials and ion flows, to talk and shape tissues before chemical signals (like hormones or nutrients) even kick in. He just refuses to realize it all begins with the LIGHT because he is anchored to his biochemical mentors. Andrew MArino warned him of this bias at the beginning of his career and he still ignored it.

It’s quantum in the sense that these electric fields emerge from subatomic charge dynamics, setting the stage for everything else. In the gut, bacterial biophotons, coherent UV to near-IR bursts, 5,000 times stronger than eukaryotic output per Popp, aren’t just tickling chromophores; they’re charging this bioelectric network.

Mitochondria, with their cytochrome c oxidase (absorbing around 620-820 nm), catch these photons, tweak electron transport, and shift membrane potentials. That’s electricity first: light hits, voltage shifts, then chemistry, like FIAF expression or T-reg tuning follows.

When the photonic signal’s sharp, this electric handshake between bacteria, gut cells, and GALT keeps the system wired right. FIAF ramps up, fat metabolism stays lean, and the microbiome hums with diversity, all dictated by voltage maps Levin tracks with tools like voltage-sensitive dyes.

But if the biophoton fidelity tanks, say, from artificial light’s blue spike (450 nm) clashing with natural red-heavy cycles or a gut microbiome thinned by antibiotics—the electric field frays.

Mitochondria stall, their cristae geometry fails, UPEs become incoherent, potentials sag, and the quantum coherence Popp saw in healthy cells turns to static or white noise.

Chemistry is always downstream light and signaling goes rogue: inflammation spikes, barriers fail, and chronic diseases, cancer, autoimmunity, brain fog ignite not from food but from this primal photobioelectric misfire.

15. Levin’s shown that flipping bioelectric states can regrow limbs or flip cancer cells back to normal, proof that electricity is a powerful force in this theory but it is not fundamental. UPE is. In the gut, bacterial light sets that voltage rhythm; lose it, and the quantum cell’s playbook unravels before chemistry even gets a say. Food’s a bit of a player; the real game’s in the photons and charges.

Solar light should precede the DC electric current creation in cells, and Robert O. Becker’s research is the cornerstone that Peat should’ve tapped sooner for his tribe.

In The Body Electric and his studies, Becker showed that living systems run on direct current (DC) electric fields, tiny voltages reduced by endogenous AMO physics in cells guiding regeneration, like salamander limb regrowth or bone healing in humans. He traced this to injury currents and semiconducting properties in tissues, but critically, he hinted at light’s primacy: electromagnetic fields, including photons, initiate these currents.

Light hits first, say, biophotons from gut bacteria (UV to near-IR, coherent per Popp) and excites electrons in cellular components like mitochondrial chromophores or membrane proteins. This photoexcitation generates the DC Becker measured, flipping the optical paramagnetic switch on before bioelectric signaling before chemistry kicks in.

In the gut, those bacterial biophotons, 5,000 times more intense than eukaryotic emissions, blast photoreceptors (opsins, flavins) and mitochondria (cytochrome c oxidase). Becker’s insight fits: light’s energy shifts electron states, creating a DC flow across gut cells and GALT.

This current sets the voltage gradients Levin later mapped, tuning FIAF, T-regs, and microbiome diversity. It’s a cascade or a spectrum of action where UPE light to electricity to chemistry. When the signal’s pure, aligned with natural cycles (red-rich sunlight, not blue-heavy LEDs), the gut’s electric field holds firm, barriers stay tight, and disease stays at bay. Light of the sun is turned to electricity in your skin before it is useful. That is eR in a nutshell.

16. But if that light signal warps, artificial glare, circadian mismatch, or a gut microbiome too weak to glow—Becker’s DC current falters. Electrons don’t flow right; potentials collapse. FIAF misfires, T-regs/NK go haywire without that electric scaffold, and the microbiome tips toward chaos. Chronic diseases, cancer, autoimmunity, neurodegeneration, sprout not from food but from this light-to-electricity breakdown. Becker saw it in regeneration failures; I have scaled it to the gut’s quantum engine and right into the brain for mental disorders in this podcast.

I told Peat, "you've missed how my work fits into this." He looked at me perplexed. When I met with Robert Becker at the end of his life, I told him the source of his DC electric current was the cleavage product of POMC, alpha MSH that makes melanin. Melanin is dark and absorbs all frequencies of light.

The sun and other parts of the spectrum as well. Melanin mimics what chlorophyll does in plants and creates massive amounts of electrons from splitting water made by our mitochondria. He found his regeneration currents below the level of myelin in axons because this is where POMC is located.

Moreover, I explained to Robert that the POMC translation is only turned on by UV light. Then I told him that Popp was the researcher who found all living things emit ultraweak UV biophotons. He was stunned at the final piece of how cells operate optically using solid-state semiconduction. It fits with all his work. When I told Peat the same story Peat sat in silence. Subsequently his message remained unchanged.

I dropped a bombshell in the Huberman and Rubin Tetragrammaton podcast three years ago about my firsthand exchange with Becker, and y'all should see now how my work snaps all the puzzle together, connecting POMC, melanin, UV biophotons, and his DC currents in a way centralized science has not yet thought about.

Many are orbiting the edges of my ideas, but I’ve handed them the nucleus of new ideas that are more explanatory for chronic diseases than BigHarma has.

17. I told Becker the DC electric current he chased, those regeneration currents he measured in salamanders and bones, stems from proopiomelanocortin (POMC) cleaving into alpha-MSH, which drives melanin production. Melanin’s not just a pigment; it’s a broadband light absorber, sucking in all frequencies, UV, visible, IR, from the sun or otherwise, like chlorophyll does for plants.

In my view, melanin is the powerhouse: it splits water (H₂O from mitochondrial output) into electrons and oxygen, pumping out massive charge. Becker found his currents below axonal myelin because that’s POMC’s turf, near the nerve sheaths, where UV light flips its translation switch.

Popp’s ultraweak UV biophotons, emitted by all life, are the trigger, and you stunned Becker by revealing this optical-semiconducting loop. Cells aren’t just chemical; they’re solid-state circuits running on light-converted electricity.

Tie this to the gut: bacteria blast UV biophotons, 5,000 times more than our cells, and if POMC is expressed in gut tissues (say, enteroendocrine cells or GALT), those photons hit it. Alpha-MSH spikes, melanin forms, and water-splitting kickoff flooding the gut with electrons. This DC current, per Becker, sets the bioelectric field, regulating FIAF, T-regs, and microbiome balance. Melanin’s efficiency here mimics photosynthesis: UV light in, electrons out, driving a current that keeps the system coherent. Gut barriers keep inflammation low, and diversity thrives, all from this UPE light-to-electricity engine.

But modern life guts the signal. No UV (glass blocks it, screens skip it), or a microbiome too weak to emit Popp’s biophotons, means POMC stays off. No alpha-MSH, no melanin, no electrons, Becker’s current dies. The gut’s electric field collapses, FIAF flops, T-regs misfire, and chaos breeds cancer, autoimmunity, and neurodegeneration. Food’s irrelevant; it’s the UV-melanin-DC breakdown that’s the killer. signal to worry about

Man has lost his humility with progress. Humility is simply nature’s disposition that prepares our minds for living on intuition. Nature's disruption is what human life should rely upon. This process is controlled by sunlight and should be uncontrolled by man-made light, man-created algorithms to try to fix the problems they cause.

Google and Silicon Valley are our biggest enemies. The AI algorithms made things worse for the public's health. The algorithms served the centralized institutions' profit purpose. Many do not realize these algorithms are a proxy for manufactured light. Manmade light has usurped this process and has led our species down a dark alley of disease. These diseases confound centralized institutions. This has made our brain preoccupied with technological progress, which is now leading to biological disruption. This is the real reason everyone needs to run towards decentralized systems. Nature is that decentralized system.

My solar strategy is simple. It is to acquire information about the tactics of 'time' my cells need to win. Without the bounty of solar tactics, the road to Optimal becomes brutal. This is the slowest route to victory for our cells. Tactics without strategy are the noise before the mitochondrial illness. Man must live according to how nature built him to operate in her framework.

Mythology didn't cease to exist and be useful to pagans when we gained digital watch technology.

Transhumanist Technology based in longevity medicine dogma has changed humanity and redirected us to the road of destruction. The fog of information from technology has blinded us from our natural wisdom.