Don't Let Your Lives "Narrow"!!
I've been an orthopedic surgeon for 30 years.
The thing I watch happen to people, more than any injury or surgery, is refer to as the narrowing.
Most of my patients have no idea it's happening to them. They think it's just aging. It's not. 🧵
The narrowing is the slow shrinking of what your body will let you do... or what you assume your body can or should be doing at your age.
You used to carry four grocery bags. Now you take two. You used to sit on the floor with the grandkids. Now you sit on the couch and watch. You used to take the stairs two at a time. Now you hold the rail.
If you're under 80 and you lie down for an injection... you shouldn't need a two armed lift to sit back up. Granted... in some instances you will... but I'm speaking about the "healthy" 60-70 something that can't because they never recognized this loss of ability and never did something about it.
Nobody decides to narrow their lives intentionally. You don't wake up one morning and choose a smaller life.
Your body quietly loses some capacity, and your daily choices adjust to the loss, and within a few years the smaller version is your new normal.
And the memory of the larger version gets filed away under "when I was younger."
Patients tell me about the narrowing every single day. They just don't use that word.
They say: "I can't do what I used to do."
They say: "I'm getting older."
They say: "That's just what happens at my age."
And they say it as if it's a law of physics.
Yes! Some decline is real and unavoidable. VO2 max drops. Max heart rate drops. Strength drops. Power drops faster than strength. Quickness drops. Proprioception drops. I see these changes in the gym all the time. Especially power and quickness.
But... The unavoidable decline is only a small fraction of what most people are actually losing.
The rest, the bigger part, the part that turns a sixty-year-old into a frail seventy-year-old, is not aging.
It is disuse.
The cruelest part of the narrowing is that people normalize it. They don't question it. They talk about capacities they've lost as if losing them was scheduled.
They simply adjust their lives and activities to their list of limitations.
When patients see me running, climbing, lifting at 62, a common response is: "Well, you're different. You're built for this."
I was not uniquely built for this. I built myself for this. Every week, on purpose, for decades.
What they see as genetic luck is accumulated work.
Most of my patients could be doing most of what I do.
Not all of them. Some have real structural problems. Some have diseases and disabilities. Some are genuinely limited.
But the majority are limited by the decade or two of training they did not do.
Once the narrowing starts, it accelerates on its own.
You stop lifting heavy things. Your muscles lose fast-twitch fibers. You get weaker. You lift even less. You lose more. The loss feels like aging. You accept it. The loop tightens.
Again... aging brings some changes. VO2 max declines about 10% per decade in sedentary adults. Strength declines slowly starting in the forties. Power declines about twice as fast as strength after fifty. Bone density drops. Balance degrades.
Get over it... you still have agency and lost abilities to recover.
The slope and severity of every one of those declines is profoundly modifiable with training. The sedentary decline curves are not the human decline curves. They are the untrained decline curves.
Trained adults in their seventies routinely outperform untrained adults in their fifties.
The gap between what aging takes from you and what disuse takes from you is huge. Most people can't tell the two apart, because nobody showed them the difference.
They live inside a body that is losing capacity fast, and they attribute the whole thing to a calendar.
I have watched patients in their seventies start from positions that would make a physical therapist flinch, and rebuild themselves into versions of themselves their own families barely recognized.
Not by becoming athletes. By doing small, specific work, consistently, for months and then years.
The body remains responsive to training well into the seventies and eighties. This is one of the best-established findings in the literature on aging.
Almost nobody's doctor tells them about it.
The patients who reverse the narrowing are not the ones with the best genetics, or the best knees, or the best circumstances.
They are the ones who decided to do something.
Something made them stop accepting the losses as inevitable, and they started doing things differently from that day forward.
What have you already stopped doing?
Not what you can't do, exactly. What have you quietly stopped doing over the last five or ten years, without ever making a real decision about it?
And more importantly: did your body actually tell you to stop, or did you assume you needed to?
one of the most common finding in my office is that patients have stopped doing things their bodies could still do, on the basis of assumptions that were never tested.
They basically narrowed preemptively.
Most of the narrowing in your life right now is reversible. I have watched it happen in thousands of patients. It is not a miracle. It is just the body doing what the body does when you start asking it to do something again.
The door you thought had closed is usually still open.
I am not a one-off. I am a sixty-two-year-old who decided not to let my life narrow, and who did the specific work to back the decision up, for long enough that the work is now visible from the outside.
You can do this too. At any age I am likely to be talking to.
Start where you are. Start this week.
Are Most Rotator Cuff Tears Actually Tears?
This topic is far, far more complicated than most think... and it's certainly far more complicated than the discussion in most office visits portrays. 1/ If you have a rotator cuff tear on an MRI report, almost everything you have been told about it is more wrong than right.
A short thread on what the cuff actually is, why most "tears" aren't what you think, and why surgery is rarely the right first step.
2/ The rotator cuff is a stabilizer, not a lifter. The deltoid does most of the work to lift the arm. The cuff keeps the head of the humerus centered while the deltoid does the work.
That distinction explains why a cuff defect or "tear" is often compatible with full or nearly full arm function.
3/ The rotator cuff in most of you is built like a suspension bridge.
A thick band of fibers called the rotator cable carries most of the load. The thinner area inside the cable, called the rotator crescent, is stress-shielded.
The supraspinatus tear the MRI described often sits in this stress-shielded area. Therefore... even if an MRI says the supraspinatus is torn... you may not have lost any function. And... fixing or repairing these can cause more problems than you have now. The cable doesn't want to be lateralized and repaired to the humerus.
Knee Osteoarthritis... Why Rest and Wait For Surgery is Not the Answer 1/ You have more agency than you have been told you do. The X-ray is not the verdict. The trajectory is not fixed. There are many things you can do... and they will influence the trajectory of the disease course more than any injection or medication will.
A short thread.
2/ The X-ray is a poor predictor of your symptoms.
I have friends with bone-on-bone changes on their imaging who run and lift with me. I have patients with mild radiographic findings who can barely climb stairs. The correlation between imaging and pain is famously weak.
The X-ray describes the structure of the knee. But... Function isn't defined solely by structure.
What predicts your symptoms and your trajectory is not the X-ray. It is the muscle around your knee, your body's metabolic state, the chronic inflammatory burden you are carrying, your aerobic conditioning, and your ability to keep loading the tissue intelligently.
APOE4 is the strongest common genetic risk factor for late-onset Alzheimer's. About 25% of the population carries at least one copy.
It's a risk modifier... not a guarantee. And the levers that change the trajectory are mostly things you can actually do something about.
A short thread...
2/ The basics.
APOE comes in three versions: E2, E3, and E4. You inherit one from each parent.
E3/E3 (60% of people): average risk
E3/E4 (~22%): one copy, 2-3x lifetime AD risk
E4/E4 (2-3%): two copies, much higher risk, often a decade earlier
E2/anything: usually below average risk.
3/ The reason E4 matters: it shuttles lipids less efficiently than E3 or E2. Carriers tend to have higher LDL and ApoB levels, slower clearance of remnant lipoproteins, earlier amyloid deposition, and greater vulnerability to insulin resistance, poor sleep, and vascular injury.
The biology/risk is very real; however, so is your ability to modify your risk.
Knee Osteoarthritis Thread 4: Metabolic Health and the Knee.
Your metabolic health will directly impact how your knee feels more than you can imagine. Your metabolic health directly influences how quickly your arthritis progresses and how your cartilage responds to the stress it's under.
This is the part of the knee OA conversation that most patients never hear.
Here's a useful fact to set the stage for this discussion: Hand osteoarthritis is far more common in people with obesity.
Hands don't bear weight. So if OA were purely a mechanical wear-and-tear issue, this wouldn't make sense.
But... It does make sense if OA is driven, in part, by systemic inflammation, which circulates everywhere, including into non-weight-bearing joints.
Insulin resistance, elevated blood sugar, visceral fat accumulation, and chronic low-grade inflammation all produce pro-inflammatory molecules — cytokines, adipokines — that circulate through the bloodstream and infiltrate the synovial membrane of the knee.
Once inside the joint, they shift the balance toward breakdown rather than cartilage repair.
I'm a 62-year-old orthopedic surgeon, trail runner, climber, and cyclist. This is my Midlife Athlete's Playbook. I've combined what I've learned from 30+ years of treating active adults, and from training through my own 50s and 60s. The physiology of aging is real, but most of the decline people accept is optional.
Aging brings real physiological changes. But lack of specific age-appropriate training brings far worse ones. A few things I've learned from three decades of treating active adults and from my own body.
After 30, muscle mass declines 3 to 8% per decade. After 50, it accelerates to 1-2% per year. The biology is real: anabolic resistance increases, satellite cell populations decline, and the signaling that drives hypertrophy becomes less efficient. But muscle still responds to progressive load at every age. The response is harder to earn, which means the training has to be more deliberate, not less.
Coming Back After Injury, Surgery, or Illness... Why It's Easy To Set Yourself Up For Breaking Again.
1/ I've helped hundreds of athletes return to activity after injury, surgery, or serious illness.
The return is almost never linear.
The sooner you accept that, the better the return will be.
A thread on the actual data — and how to try to come back without breaking yourself a second time. 🧵
2/ Most returning athletes underestimate two things:
How fast you detrain.
and...
How uneven the losses are across different systems.
Your aerobic base, strength, tendons, bone, and metabolism all lose capacity — but not at the same rate. And they don't come back at the same rate either.
3/ VO2max goes first.
Coyle 1984 (J Appl Physiol): highly trained athletes lost 7% of VO2max at 21 days of cessation, 16% at 56 days.
Mujika & Padilla's 2000 Sports Med review: ~4% at 3 weeks, ~10% at 5 weeks, ~13-20% by 2 months.