1️⃣ When a rhythm comes from the ventricles, it travels slowly across muscle instead of racing through the Purkinje system. That’s why VT is wide, fast, and regular.
2️⃣ Next trick: look at the bundle-branch pattern.
- VT that looks like LBBB usually started in the right ventricle.
- VT that looks like RBBB usually started in the left ventricle.
Impulse travels away from its origin as if the opposite bundle is blocked.
3️⃣ VT comes from the RVOT =most common idiopathic VT
It has LBBB shape with a strong inferior axis (big positive in II, III, aVF).
These patients often feel palpitations during exercise or stress.
4️⃣ If the QRS looks RBBB with an inferior axis, think LVOT i.e near the aortic valve or left ventricular outflow.
5️⃣ There’s a special one called fascicular VT.
Young patient, no structural disease, heart suddenly takes off at 150–180 bpm.
ECG: RBBB pattern + left axis deviation.
And here’s the magic: it usually stops with Verapamil.
6️⃣ In patients with an old MI or cardiomyopathy, the ventricles have scar tissue.
That scar becomes a little electrical maze.
The circuit keeps spinning in the same path--) monomorphic VT with a stable QRS every beat.
7️⃣ Torsades de Pointes is a different beast.
The QRS keeps changing direction it literally "twists."
You’ll almost always find a long QT before the episode.
8️⃣ When the QRS is polymorphic but the QT is normal, think acute ischemia, especially if there are ST changes.
9️⃣ There’s even a VT that forms its own race track inside the bundle branches: bundle-branch reentry VT.
Very wide QRS, looks like a classic BBB, but going way too fast
🔟 So here’s a quick summary clinicians rely on:
◾Monomorphic VT: usually scar or idiopathic outflow/fascicular origin
◾Polymorphic VT: usually ischemia, long QT,..
The ECG won’t pinpoint the exact millimeter, but it gets you surprisingly close to the neighborhood.
Aortic Stenosis (AS): essential clinical pearls every clinician should know🧵 🧵 🧵
A concise thread on pathophysiology, diagnosis, prognosis, and management.
1/ Aortic stenosis is a progressive valvular disease causing obstruction to left ventricular outflow.
2/ Main causes of AS:
🔵 Degenerative calcific disease remains the leading cause in older adults.
🔵Bicuspid aortic valve is the most common cause in younger patients
🔵Rheumatic disease remains an important cause in developing regions.
Right Ventricular (RV) Infarction: A Must-Know ECG Pattern.
RV infarction often coexists with inferior MI but is frequently overlooked. Missing it can be dangerous, as treatment differs from LV infarcts!
Here’s how to detect it on ECG ⬇️
1️⃣ When to Suspect RV Infarction?
🔹 Any inferior MI (ST ↑ in II, III, aVF)
🔹 Hypotension + JVD + clear lungs
🔹 Bradycardia, AV block
A simple rule that works well in practice: inferior MI + hypotension = look for RV involvement
Why a standard ECG isn’t enough
The usual 12-lead ECG doesn’t adequately see the right ventricle. That’s why right-sided leads matter.
You mirror the chest leads to the right side (V3R–V6R), keeping V1 as it is.
Abdominal aortic aneurysm (AAA) is one of the most dangerous silent conditions in medicine.
Most patients don’t know they have it.
Many never get a second chance once it presents.
🧵
1/ An AAA is a localized dilation of the abdominal aorta, reaching at least 150% of its normal diameter.
It sounds simple, but the implication is serious:
as the vessel enlarges, the wall weakens, and rupture becomes inevitable if it progresses.
2/ The real danger of AAA isn’t just the disease.
It’s the way it behaves.
It stays quiet for years…
and then presents as a catastrophe.
Interpreting an #ECG can seem overwhelming at first, but with a structured approach, it becomes far more manageable.
In this thread, I’ll walk you through how to analyze an ECG like a professional, step by step.
Let’s begin.🧵
Step 0: Initial Checks 1. Paper Speed
The standard ECG paper speed is 25 mm/s, meaning:
◾1 small square = 0.04 s
◾1 large square = 0.20 s (5 small squares) 2. Calibration
This determines the amplitude of the waves:
⏺️ 1 mV = 10 mm (i.e., 2 large squares vertically.
Step 1: Heart Rate (HR)
If rhythm is regular:
Use the formula
Heart Rate = 300 / number of large squares between R waves
Alternatively, memorize the sequence:
300 → 150 → 100 → 75 → 60 → 50
Estimate HR by seeing where the next R wave falls in this pattern.
Acute coronary syndrome management extends far beyond revascularization.
Optimal outcomes depend on a structured, evidence-based strategy before, during, and after intervention.
A concise summary of the 2025 guideline approach: 🧵
1/ Dual antiplatelet therapy (DAPT) remains foundational in ACS.
In patients undergoing PCI, ticagrelor or prasugrel is preferred over clopidogrel due to superior ischemic protection.
2/ In NSTE-ACS with a planned invasive strategy delayed beyond 24 hours, early administration of clopidogrel or ticagrelor may be considered to reduce major adverse cardiovascular events.