𝕯𝖗Дℒα乃ⓐ$†ヨ℞ㄷΓΦϖ⟭⟬⁷ Profile picture
STEM PhD, biopharma insider, favors objective analysis of data rather than pundits, entrenched virologists, and DISC-narrative flagbearers' opinions. 𖤐無神論𖤐 반일
Mar 22, 2021 12 tweets 5 min read
Wow! My following went up literally by like 25% in one day. Please if you could all take a gander at my sticky, that would be great, whereby I am (AFAIK) the only person to do off-the-cuff math illustrating how bogus a 100% natural #furincleavagesite is in #SARSCoV2origins threadreaderapp.com/thread/1339682…

and remember, the FCS itself is ectopic, and NO OTHER member of that sarbecovirus clade has one.

See Page 7: ncbi.nlm.nih.gov/corecgi/tilesh…
Jan 13, 2021 14 tweets 5 min read
The left: Politicians lie all the time, so we should just ignore Trump and Pompeo's legitimate claims of election fraud, the origins of the coronavirus, and CCP's dire influence.

ALSO THE LEFT: BELIEVE ALL WOMEN, BELIEVE ALL SCIENTISTS, BELIEVE BIDEN, KAMALAH, & CNN Yes! Politicians DO lie. But they lie about certain things in certain circuitous ways. They might make a campaign promise and then flake. Or they might feign forgetting. Or they might say "that depends on what your definition of 'is' is".

*skillful* plausible deniability is KEY
Dec 17, 2020 12 tweets 4 min read
1)"The only measurement of a mutation rate in a β-coronavirus suggests that this site will accumulate ~10^–6 mutations in each round of replication. Each replication cycle takes ~10 hr, and so there are 10^3 cycles/year. "

ncbi.nlm.nih.gov/pmc/articles/P…
i've read 10^-6 in another paper 2) but the argument that there are 1000 cycles of replication/year is *only* true if one considers 10^4 being ~8760, *and* only 1 viral mRNA replicates.
there are numerous other issues with this math.

No wonder "codon shuffle" ncbi.nlm.nih.gov/pmc/articles/P…
is the only mutation tool
Oct 29, 2020 5 tweets 2 min read
For example, SARS-CoV-2’s genome closely resembles that of a bat coronavirus, but a small section of the
genome called the “polybasic cleavage site,” believed to provide a selective advantage for disease transmission, would have been expected to evolve over time but instead is present in the earliest sequences of the virus. Investigators can further determine whether the infecting agent’s genome so closely resembles a given reference
strain that a period of limited or no replication is likely. Such socalled “frozen evolution,” when an infecting