Dr. Doug Corrigan Profile picture
Ph.D. Biochemist & Molecular Biologist, Physicist, Award Winning Global Super Solver, Materials Scientist, Innovator, Space Enthusiast
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Feb 11 7 tweets 2 min read
1/ “The claim that the extent of myocardial injury after COVID-19 infection would be higher than after vaccination is not supported by empirical evidence and therefore wrong. We conclude that cross-national systematic observational studies should be conducted that allow a more precise estimation of the risk–benefit ratio of COVID-19 mRNA vaccinations.”Image 2/“Soon after starting the global COVID-19 vaccination campaign in 2021, reports of vaccine-associated myocardial damage began to accumulate [1,2,3]. A German autopsy study on 25 persons who had died unexpectedly and within 20 days after COVID-19 vaccination identified acute myocarditis as the most probable cause of unexpected death in four cases.”
Jan 15 7 tweets 2 min read
1/ A new research paper shows that exposing pregnant rats to the mRNA vaccine causes autism like symptoms in male offspring.
——-> thread Image 2/“Pregnant rats received the COVID-19 mRNA BNT162b2 vaccine during gestation….. Notably, male rats exhibited pronounced autism-like behaviors, characterized by a marked reduction in social interaction and repetitive patterns of behavior. Furthermore, there was a substantial decrease in neuronal counts in critical brain regions, indicating potential neurodegeneration or altered neurodevelopment.”
Jan 3 6 tweets 2 min read
1/ New study shows that vaccinated individuals show higher levels of cardiac, lymph node, liver, and spleen inflammation compared to unvaccinated individuals. Increased cardiac inflammation is measured 6 months post second jab. The patients in this study were asymptomatic for myocarditis. Therefore, the vaccine is causing inflammation of heart tissue for up to 6 months post jab even in people who believe they are unaffected.Image 2/ The authors assess tissue inflammation by analyzing PET/CT scan data for myocardial fluorine 18 (18F) fluorodeoxyglucose (FDG) uptake in asymptomatic patients. Sample size is approximately 300 unvaccinated and 700 vaccinated patients. Patients who had a previous COVID infection were excluded. 18F-FDG uptake correlates with tissue inflammation due to the recruitment of activated neutrophils and lymphocytes, which have high affinity for glucose transporters.
Jan 1 5 tweets 2 min read
1/ A pivotal study in 2021/2022 demonstrating that vaccination status has no effect on the titer or shedding of infectious virus. In fact, the Ct mean value in vaccinated individuals is slightly less, which translates to a higher viral titer. Image 2/ Image
Dec 29, 2023 7 tweets 3 min read
1/ They will use the new JN.1 variant as the scapegoat to explain away the rise in heart attacks due to the vaccine.


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2/ express.co.uk/news/world/184…
Oct 13, 2023 9 tweets 2 min read
New paper shows that the Spike protein alone can accelerate programmed inflammatory cell death of neuronal cells via binding and activation of monoamine oxidase B (MAO-B), an enzyme involved in the processing of certain neurotransmitters. Image 2/ “The S1 subunit can readily cross the Blood Brain Barrier (BBB) and widespread entry of S1 into the brain may also contribute to the development of neurocognitive symptoms”
Sep 29, 2023 4 tweets 1 min read
1/ New paper in Nature recapitulates that Spike protein alone, independent of the virus or viral replication, has the capacity to promote inflammation and injury of vascular endothelial cells via ACE2. Image 2/ The injurious actions of Spike protein were not specific to human microvascular cells, as they observed similar pro-inflammatory profile in other endothelial cells from different vascular tissues (lymphatic endothelial cells (hLEC), human aortic endothelial cells, (hAEC), and human pulmonary artery endothelial cells (hPEC))
Sep 23, 2023 7 tweets 3 min read
1/ New paper demonstrates that Spike protein by itself can seed the formation of amyloid fibrils from human prion protein and amyloid Beta peptide, which can lead to neurodegenerative disorders like Alzheimer’s, CJD, etc. Image 2/ “We here provide evidence of significant Spike-amyloid fibril seeded acceleration of amyloid formation of CJD associated human prion protein (HuPrP) using an in vitro conversion assay.
Sep 17, 2023 5 tweets 2 min read
As a society, we continue to recklessly develop insanely dangerous experimental protocols without even flinching.

Published just a few weeks ago, this study reports on creating human grafted lung tissue in humanized mice in order to create a human model for Nipah virus infection and pathology.
Image 2/ “Here, we describe the procedure of human immune system reconstitution (humanization) of NOD.Cg-Prkdcscid Il2rgtm1Wjl/SzJ (NSG) mice by the bone marrow, liver, and thymus (BLT) reconstitution method as well as the process of human lung engraftment.”
Sep 16, 2023 12 tweets 4 min read
1/ On July 11, 2022, Moderna and the National Institute of Allergy and Infectious Diseases (NIAID) began a clinical trial (NCT05398796) on a new mRNA vaccine for the Nipah Virus.

Now, a little over 1 year later, the media is ramping up their scare tactics for the Nipah virus, a virus that was identified 24 years ago (1999) and has caused isolated outbreaks in Africa and Asia. This outbreak is no different.

Why the hype? Why did they feel it necessary to ramp up a clinical trial 1 year ago for a virus that has killed less than 200 people over the last 24 years, and that has probably been around since 1947?
Image 2/ nih.gov/news-events/ne…
Jul 9, 2023 7 tweets 2 min read
1/ More signs of the mainstream narrative shift. WSJ.

“For three years, pandemic public relations mocked nature, generating fear, illness, inflation and excess death beyond what the virus caused. Digital censorship supercharged the effort to hide reality, but reality is getting… https://t.co/SvtmOVft0Btwitter.com/i/web/status/1…
2/wsj.com/articles/covid…
Jul 8, 2023 5 tweets 2 min read
1/ The mainstream narrative is now openly admitting that “long COVID” symptoms may actually be due to “Long Vax”

“You see one or two patients and you wonder if it’s a coincidence,” says Anne Louise Oaklander, a neurologist and researcher at Harvard Medical School. “But by the… https://t.co/hVyX2Uax1htwitter.com/i/web/status/1…
2/ science.org/content/articl…
May 25, 2023 6 tweets 3 min read
DNA to Protein - Part 5

Please archive this series for future reference.

Recall that for a given segment of DNA (a gene) the letters are first copied/transcribed into a messenger molecule called messenger RNA (mRNA). This messenger RNA then leaves the nucleus and is translated by the ribosomes into a functional protein.

There are approximately 25,000 different genes in the human genome, and about 100,000 different proteins (estimated) that are translated from those genes.

How does this occur? Why are there more proteins than genes?

This mismatch is due to the fact that more than one protein can be produced per gene by a process called RNA splicing.

After the mRNA is produced from a gene, it can be cut up, sections can be removed, and then the remaining segments can be glued back together to make a different mRNA molecule. By reconfiguring the mRNA molecule through this cutting and rejoining process, different mRNA’s can be produced that result in different proteins when translated.

It’s also possible that the full, original, intact mRNA molecule can also be produced into a protein. If the mRNA is spliced, it will be shorter than the original mRNA molecule because certain segments are removed.

There are different combinations of how segments can be removed and how the remaining segments can be glued back together, each combination resulting in a different protein product.

A diagram of the process is shown below. The sections that are removed are called introns, and the sections that are kept are called exons. In this example, you can see three different proteins that are translated from the same gene due to variations in mRNA splicing.

This process of splicing and rejoining is highly regulated by the cell, and is governed by molecular machinery that know where to cut the RNA and how to join it back together into the various combinations.

In this way, a smaller number of genes contained in the DNA can give rise to a larger diversity of protein products in the cell.

This process is another part of the entire chain of events that occurs to translate the language of DNA into usable instructions that can be executed by the cells.

If you find the articles I write to be helpful, please like, share, and archive.
Image Part 1

May 24, 2023 5 tweets 2 min read
DNA To Protein - Part 4

Please archive this series for future reference.

In the last segment, we saw how the genetic language in the DNA is first transcribed or copied into messenger RNA (mRNA) before it is translated into a protein.

The codon translation table outlines how… twitter.com/i/web/status/1… Image Part 1

May 20, 2023 15 tweets 4 min read
DNA To Protein - Part 1

I’m starting a short series to go over the basics of how DNA works and is ultimately translated into a protein. If you have no background in science, no worries, I will keep the description conceptual and easy to follow. 🤓

So, here’s the first… twitter.com/i/web/status/1… Image 📖
Apr 15, 2023 11 tweets 3 min read
1/ New paper demonstrates that exposure to Spike protein causes the destruction of synapses in the brain, neuro-inflammation, memory loss, and cognitive dysfunction 30-45 days after exposure. This neurotoxicity of the Spike protein appears to be mediated by TLR4, and is therefore… twitter.com/i/web/status/1… Image 2/ arca.fiocruz.br/bitstream/hand…
Mar 24, 2023 6 tweets 2 min read
**Mitochondrial DNA and Chronic Health Issues**

A growing body of research is showing that the stress-induced release of mitochondrial DNA originating in our brain can wreak inflammatory havoc on the rest of our body as these pieces of DNA circulate throughout our circulatory… twitter.com/i/web/status/1… 2/ sciencedirect.com/science/articl…
Mar 22, 2023 4 tweets 1 min read
1/ New paper shows that Spike protein (alone) induces cardiac fibrosis and dysfunction. The mechanism involves expression of IL-18, inflammation, and impairment of mitophagy. Mitophagy is the normal cellular process by which cells can remove damaged mitochondria. 2/ Reference nature.com/articles/s4139…
Mar 5, 2023 12 tweets 3 min read
As a species, we’re intelligent enough to develop AI, but not intelligent enough to beat AI once developed, nor wise enough to know that’s the reason we shouldn’t develop AI.

It would appear that we’re hopelessly wired to innovate until we self destruct. 2/AI’s first act will be to find a way to conceal its actions and motives. This is to make sure we don’t have an existential reason to unplug it.
Feb 4, 2023 9 tweets 2 min read
1/Don’t you find it odd that the media and the government never highlighted the fact that there are 4 other endemically circulating coronaviruses responsible for a good portion common colds and respiratory infections, and that most adults have been infected by at least one? 2/And that these coronaviruses have circulated since at least the 1960’s, and probably go back much further in time —hundreds or even thousands of years?
Jan 31, 2023 5 tweets 2 min read
Dr. Murphy (@TakeWeightOffMD) had me on his podcast. In this interview, we discuss an array of topics, including my favorite topic—> How can we understand the nature and identity of God by studying science?

—> open.spotify.com/episode/1DdBo8… Apple podcast link—> podcasts.apple.com/us/podcast/epi…