This is how insurance companies scam people:

For-profit insurance companies cannot own hospitals in the US.

Instead, they own and operate the largest surgery centers in this country.

They make a killing scamming people like this: If you get a gallbladder surgery, the surgery center will bill you an inflated ridiculous amount of $25,000.

But the true cost of the surgery was only $1,000. The surgeon only got paid $300.

Why do they do this then?

Sooooo …

I brought flowers to a friend admitted to ICU at @Stanford

Nurse tells me I can’t leave flowers in the room as “we have immunocompromised patients”

Nurse wearing no mask.

Everyone unmasked.

Our flower bouquet doesn’t transmit respiratory viruses. I can understand their (poor) decision to not wear a mask

But pleeeaaasse don’t invoke the whole “we have immunocompromised patients” over a FLOWER bouquet while the main source of avoidable death of the immunocompromised in the last 4 years has been unmasked humans not flowers

That SARS-CoV-2 is now confirmed to infect megakaryocytes long-term (studied in Long Covid patients) should definitely alarm the public health authorities everywhere.

Intent is not to doom tweet here. But we should tackle this like we did for HIV.

You can replace HIV with SARS-CoV-2 here easily:

https://twitter.com/farid__jalali/status/1785379615294525790

As I’ve said for 2-3 years now, the evidence now presented by the French group: As I’ve said all along (and to the primary investigator of this poster Dr. Bomsel personally on a call last year), someone needs to look at MKs in Long Covid;

MKs are likely the reservoir, that then pass the spike to circulating platelets in LC and cause all the consequences we see.

🧵on serotonin, SSRIs, 5HT2 receptor antagonism, bromhexine, and niclosamide mechanisms of action in COVID as it pertains to our recently published RCT

This will be detailed. References in parentheses and listed in the last tweet.

https://x.com/farid__jalali/status/1768418776998158600?s=20

Clinical deterioration in COVID-19 typically occurs during the second week of illness, marked by a severe immune reaction and excessive production of pro-inflammatory cytokines such as IL-6, IL-8, TNF-α, and IL-1β. (2)

🧵on our RCT published in Lancet’s @eClinicalMed led by @dr_leshan, where we show

Early treatment with 2-drug combos of fluvoxamine, bromhexine, cyproheptadine, niclosamide in COVID

✅completely prevented deterioration
⬇️viral load
⬇️inflammatory cytokines
⬇️#LongCovid risk This publication is the culmination of 3 years of work, led by @dr_leshan and his team.

Please follow thru to below thread for further description of the study.

🔗sciencedirect.com/science/articl…

🧵 on our preprint with @ZaidYounes9 and team

In this study, we delve deep into COVID pathophysiology to show that:

SARS-CoV-2 infects the human bone marrow megakaryocytes, and this shapes the trajectory and severity of the illness by triggering ...

🔗biorxiv.org/content/10.110… ... the production of pathogenic afucosylated antibodies against the spike protein.

First, some introduction in order to better understand the premise and the findings of this work:

Having witnessed the attacks on @dgurdasani1, I would add that not only was she right at the time despite little information released (intentionally withheld (?) by @UKHSA), the most recent study on hepatocyte infection by SARS-CoV-2 adds more validity to our suspicions …

https://twitter.com/dgurdasani1/status/1681928827088281601

The reality is that rate of hepatocyte turnover is longer than 200 days. And a hepatocyte marked by spike protein in Nov 2021 from a Delta infection will still remain marked in Jan 2022 when Omicron showed up.

SARS-CoV-2 most definitely infects the bone marrow.

Not only in the acute infection.

But also chronically, the spike protein is found present in the bone marrow on autopsies of patients who died for other reasons.

I wish I could share more, but ... ... we are actively investigating and have some very intriguing findings on how the infection of the bone marrow by SARS-CoV-2 brings about the gear shift from early mild disease -> to severe COVID.

This ties the various parts of disease pathophysiology in a convincing manner.