Lea Alhilali, MD Profile picture
Neuroradiologist @HRInstitute_AZ. @BarrowNeuro. Striving to make learning neuroimaging and anatomy fun. If I can make you laugh, I can make you learn.

Sep 12, 18 tweets

1/Do you feel there’s a back-log of findings in a spine MRI report?

Everyone talks about discs & facets, but not everyone talks about the endplates

Do you?

Do you need to talk about degenerative changes (Modic changes) of the endplates?

Here’s thread w/all you need to know!

2/Over 30 years ago, Modic et al. found there were 3 types of degenerative endplate changes:

(1) T2 bright changes (indicating edema, Modic 1)
(2) T1 bright changes (indicating fat, Modic 2)
(3) T1 & T2 dark changes (indicating sclerosis, Modic 3)

But what do they mean?

3/Let’s start w/Modic 1.

These are bright on T2, indicating edema

On pathology, it’s what you’d expect w/edema: inflammation, vascular granulation tissue, & high cellular turnover

Vascular granulation tissue means these can enhance on post contrast images—mimicking discitis!

4/Modic 2 changes seem to be when the pendulum swings the other direction.

These are bright on T1, indicating fat.

They show fatty conversion of the marrow & low cellular turnover.

I think of them as the middle-aged dad bod of degenerative change—lots of fat & slow to change!

5/Modic 3 changes seem to be the endgame.

These are dark on T1 & T2, indicating sclerosis—which is what you see on pathology: dense fibrous tissue & sclerosis.

It’s like the endplates get fossilized—once you turn into rock, you are never going back.

6/I remember these bc the forces at work in the endplates are like the famous astrology signs—almost.

Instead of earth, wind, & fire—it’s earth (sclerosis), fat, & fire (inflammation)

7/Although, it can also be earth, WIND, & fire

Just remember: when we eat a big fatty meal, we tend give off some, well, gas or WIND.

So why is inflammation type 1, fat type 2, and sclerosis type 3?

8/Modic watched patients longitudinally & found inflammation turned to fat but never the other way around.

So he hypothesized these are the stepwise chronological changes of the endplates in response to stress & degeneration

They mirror the same way we respond to stress & conflict

9/Endplates are like two neighbors w/a wall (disc) separating them.

When the wall is too thin, you get conflict! This is what happens when disc degenerates!

Your first response is anger & yelling at them to keep it down!

Just like the endplates, you first get inflamed

10/When anger & inflammation don’t resolve it, you give up, get depressed & start stress eating.

Same w/the endplates! You will transition to from inflammation to low turnover & fat!

11/Finally, you try to resolve the conflict by reinforcing the barrier between you by adding some rock

Same w/the endplates. They start to increase the bony sclerosis between them.

12/But why are degenerative endplate changes important?

Most important reason is that Modic 1 changes are associated w/pain.

It makes sense, as inflammation tends to be painful, as opposed to fatty atrophy.

This pain can sometimes be treated w/basivertebral nerve ablation

13/So what causes these Modic 1 inflammatory changes?

Sadly, after >30 years, we still don’t really know.

Theories include microtrauma, low grade infection, or an autoimmune response.

Basically, you can remember the things that get YOU inflamed

14/First is microtrauma

Just like little annoyances from a coworker eventually get you inflamed, same happens w/endplates

Microtraumas constantly happens. So there’s a frustrated healing response—bc there’s no break for endplates to fully heal, so you get inflammation

15/Next is low grade infection

Low grade bacteria have been cultured from Modic 1 changes.

It’s just like how you are more like to get super annoyed when you are already feeling sick!

However, this theory is not widely accepted & many dismiss it.

16/Finally is autoimmune.

Nucleus pulposis is notochord. It’s shielded from the immune system early in development—meaning the immune system never recognizes it as its own

It’s like having a brother that is separated from you at birth, so you never get to know him

17/When the disc fissures & exposes it, it gets attacked as foreign by the immune system

It’s the same way you would get inflamed if you met your long lost brother. You wouldn’t know him & you would freak out!

18/So now you know the types of degenerative endplate changes, why their important, & the theories as to why they happen.

Now you can use this when you go BACK to work on reading spine MRIs!

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