Share this page!

Marion Holman Profile picture
Twitter logo
9 Feb, 4 tweets, 1 min read
1/4 What infuriates me about this paper is there is NO overwhelming evidence to support lowering LDL-C to reduce cardiovascular morbidity and mortality. What prompted this paper is the fact that the Pharmaceutical industry had new drugs to sell i.e. PCSK9 inhibitors.
2/4 Far more profitable than Statins, and the reason why they now confess that Statins are toxic, to promote their new and more profitable PCSK9’s. The problem is the new PCSK9’s are no better than Statins. The pharma Industry has known for the past 40+ years the damage
3/4 that Statins do, but have been in denial. Here they are stupid enough to make the admission about Statin Toxicity and its mechanisms. If you look at the Fourier trial for PCSK9’s you will see that more people died in the treatment arm of the trial than in the placebo group.
4/4 It’s not about Cholesterol. Never has been ! ALSO, look at the DISCLOSURES ! and see that Amgen, the makers of Repatha are funding the study.

ahajournals.org/doi/full/10.11…

• • •

Missing some Tweet in this thread? You can try to force a refresh
　

Keep Current with Marion Holman

Marion Holman Profile picture

Stay in touch and get notified when new unrolls are available from this author!

Read all threads

This Thread may be Removed Anytime!

PDF

Twitter may remove this content at anytime! Save it as PDF for later use!

Try unrolling a thread yourself!

how to unroll video
  1. Follow @ThreadReaderApp to mention us!

  2. From a Twitter thread mention us with a keyword "unroll"
@threadreaderapp unroll

Practice here first or read more on our help page!

More from @holmanm

Marion Holman Profile picture
12 Feb
1/6 In 1984 Merck discovered a way to accelerate mechanism based cell alterations with a Statin by giving rats a second cholesterol lowering drug called a “bile resin”. The combination lowered cholesterol better than Lovastatin alone (Grrrreat !).
2/6 It elevated Reductase more than Lovastatin alone, and it made more abnormal-appearing ER than Lovastatin. In case you are wondering ER = endoplasmic reticulum. This drug combination is what reportedly killed the majority of Baycol users.
3/6 The liver has a very high demand for cholesterol to make bile that is made in the liver and then stored in the gall bladder for digestion of fat. The bile is then recycled back to the liver from the intestines for reuse.
Read 6 tweets
Marion Holman Profile picture
12 Feb
1/4 Oh how we have been played ! Brown and Goldstein studied the defective genetics of the LDL receptor in people with FH who have extremely high cholesterol. It was for these patients that the FDA gave Dallas an IND (Investigational New Drug) permission to test Lovastatin,
2/4 Brown and Goldstein studied the defective genetics of the LDL receptor in people with FH who have extremely high cholesterol. It was for these patients that the FDA gave Dallas an IND (Investigational New Drug) permission to test Lovastatin, and not the general public.
3/ 4 A compilation of FH is that painful deposits of oxidised cholesterol would improve this condition. Notably, two of these six FH patients with cholesterol levels over 500 didn’t even have CVD !!
Read 4 tweets
Marion Holman Profile picture
19 Jan
1/23 Thoughts on Covid Vaccine: First of all, up until this point there has never been a successful vaccine for coronaviruses in humans, due to a problem typical of coronavirus vaccine development called antibody dependent enhancement or ADE.
2/ In preliminary animal trials for previous coronavirus vaccines (SARS and MERS, for instance), animals were vaccinated and seemed to exhibit a robust antibody response, but upon exposure to the wild virus, they developed a paradoxical immune enhancement leading to severe
3/ organ inflammation (especially in their lungs), and they died. Paradoxical immune response in coronavirus vaccines has also taken place in human trials, which occurred during testing of the failed RSV vaccines of the 1950s. Alarmingly, there are some statistical indications
Read 23 tweets
Marion Holman Profile picture
10 Jan
1/4 Rather than stop their use altogether, the modality of the health profession is to simply change Statins when dangerous symptoms such as muscle pain are reported. Statins are genotoxic & cause cell mutations. It is inevitable that everyone taking Statins long term will
2/4 develop one or more of the direct effects of statin use. Statins block the Mevalonate pathway inside cells. It is from that pathway that molecules called isoprenoids are made from DNA production. Cholesterol is but one of the many isoprenoids blocked by Statins. .
3/4 All of the isoprenoids made inside cells from Mevalonate (a by-product of food) are required for cell development. NONE are expendable. There is a cure for Statin poisoning, it's called MEVALONATE. Cells normally make Mevalonate, but does this normal function return
Read 4 tweets
Marion Holman Profile picture
10 Jan
1/4 Here we go again ! There is no such thing as a "side effect any more". Statins merely "UNMASK" underlying conditions, in a similar way that Reece's Peanut Butter cups may unmask the latent condition of T2 Diabetes.😃
2/4 In this study, we demonstrate that in vitro exposure of spinal Motoneurons to selected statins, especially fluvastatin, resulted in marked cell loss. Shorter term exposures to fluvastatin resulted in neuritic degeneration.
3/4 The results of this study provide evidence that specific pathophysiological mechanisms may underlie reports of neuromuscular disease exacerbation with statin exposure.

Clinical observations have suggested that statin exposure may UNMASK 🤔 or
Read 4 tweets
Marion Holman Profile picture
3 Jan
1/10 When examining Statins, it should be noted that they have been shown to damage Mitochondria. Article – ncbi.nlm.nih.gov/pmc/articles/P…. Mitochondrial damage can account for all of the relevant adverse effects of Statins – muscle weakness and damage, loss of memory/cognitive function
2/10 Diabetes, depletion of glutathione, etc. An interesting feature of mitochondrial injury is that a threshold of damage must be reached before a disease state results (as described in this article – ncbi.nlm.nih.gov/pmc/articles/P…).
3/10 So, if your study period isn’t long enough, Statins truly will have the same number of adverse effects as sugar pills because in a short period of time, the subjects have not crossed their tolerance threshold for mitochondrial damage and no disease state (or adverse events)
Read 10 tweets

Did Thread Reader help you today?

Support us! We are indie developers!

This site is made by just two indie developers on a laptop doing marketing, support and development! Read more about the story.

Become a Premium Member ($3/month or $30/year) and get exclusive features!

Become Premium

Too expensive? Make a small donation by buying us coffee ($5) or help with server cost ($10)

Donate via Paypal Become our Patreon

Thank you for your support!

Follow Us on Twitter!

Like this author's thread?

Get emails when @holmanm has new unrolls!

Check out other threads from @holmanm!

Read all threads by @holmanm