Thread by @BethanyCherisse

Remember that in the animal trials, the animals did not become sick or die from the va**ine. They died later upon exposure to a wild virus, as a result of the cytokine storm effect.

A cytokine storm, also known hypercytokinemia, is “a condition that consists of an immune reaction involving a positive feedback loop between immune cells and cytokines”.

Simply put, it is when the body’s immune system overreacts to an infection or ‘intruder’, such as a v*rus.

When the body is flooded with an excessive release of cytokines, it results in a 'storm'. The reaction spirals out of control, triggering the activation of too many immune cells.
Often responsible for neurological damage, this overreaction can be fatal. ncbi.nlm.nih.gov/pmc/articles/P…

In relation to v*ruses, retired neurosurgeon Dr Russell Blaylock MD indicated that it is not v*ruses themselves that are directly responsible for causing harm, but a resulting overreaction of the body that may occur when mounting an inflammatory response to combat the infection.

In 2015 Dr Blaylock wrote:

“Studies have shown that in many cases the virulence of a v*rus is actually based on its ability to trigger immune overreaction. In essence, it is your immune system that makes you sick.”

Blaylock explained that often when this occurs, it's linked to an abnormality in immune system functionality, from a portion of the immune system being “weakened” - usually the cellular immune system. The “intact parts of the immune system” then “overreact to correct the defect".

In multiple animal studies conducted during the first and only phase of trials in an attempt to develop a va**ine for SARS, findings were that the va**ine actually made the animals more susceptible to the coron*v*rus,’, rather than protecting against them.

“Caution in proceeding to application of a SARS - CoV va**ine in humans is indicated”- Te et al., 2012

Yasui et al., (2012) reported severe pneumonia in mice va**inated against SARS, who then subsequently became infected with SARS.

Further study of a double-inactivated SARS va**ine found increased eosinophilic pro-inflammatory responses in va**inated mice: “Importantly, aged animals displayed increased eosinophilic immune pathology in the lungs and were not protected against significant v*rus replication.”

In attempting to create a va**ine for felines for infections, including against dengue and a cat coronav*rus known as feline infectious peritonitis virus (FIPV), it was found that cats va**inated against FIPV got sicker than cats which were not va**inated.

The v*rus-specific antibody increased the v*rus uptake by macrophage, due to the phenomenon known as “immune enhancement”.

Another trial which utilised ferrets did result in the production of antibodies. HOWEVER. When those ferrets were then afterwards exposed to a wild v*rus, the antibodies proceeded to attack their own bodies, creating a cytokine storm, causing the majority to die of organ failure.

Synthetic creation of antibodies for one serotype of a virus can result in the enhancement of infection upon subsequent exposure to a new serotype. The potential for enhanced disease on future exposure to the virus, post va**ination, is profound.

That is before even taking into consideration long-term effects such as autoimmune diseases and neurological damage, adverse reactions to the lipid vessel used to encase the RNA, and other serious issues such as blood clots and myocarditis, which are being reported in droves.

The following article outlines the serious risks of this “counterproductive, even dangerous, immune system reaction”, referred to as “immune enhancement”.

pnas.org/content/pnas/1…

The best line of defence against respiratory illnesses is prevention. The secondary response should be to boost the NATURAL immune responses. NOT induce a synthetic response, that will cause the body to go into excitatory overdrive, creating a potentially lethal reactive storm.