Probably the first thing that occurs to most people
AMVL prolapse is less common than PMVL prolapse
Here you can see the highly eccentric, posteriorly directed jet of MR
This is the AP3Ch view showing the same abnormality
2. Systolic Anterior Motion (SAM)
SAM of the AMVL tends to (not always) produce posteriorly directed MR; important to remember that SAM is not the only reason a HCM patient may have MR
PLAX here is not mine, it's from an excellent lecture (on YouTube) by Dr @KyleWKlarich
Here you can see the posteriorly directed MR jet on colour Doppler imaging
Here is the still image
3. Ischaemic MR due to posterior leaflet restriction
Inferolateral / inferoposterolateral MI often produces a regional LV abnormality that affects the posteromedial papillary muscle & affects PMVL >> AMVL. The apical PM displacement tethers the PMVL, causing MR
PLAX view here
It may look like there's AMVL prolapse, but if you look closely you'll see the PMVL barely moves
Issue here is PMVL tethering due to inferolateral MI, causing apical displacement of PM pap muscle, thus tethering the leaflet
As a result, we see this highly eccentric, posteriorly directed MR jet
Here's a still frame of that MR jet
4. Atrial functional MR (AF-MR)
Huge atrial enlargement. There are multiple postulated mechanisms for MR in patients with AF - one is posterior leaflet tethering
Again, posteriorly directed MR jet seen here in AP4Ch view
MR still image...
A nice review on AF-MR in JACC Imaging details mechanisms of MR in such patients
Superb medical student on placement
Keen, energetic, friendly, smart
She wants to go into neurosurgery
I thought I'd better give her some advice
After what my wife went through in her training
I thought the advice was for the future
Little did I realise it had already started
What I had planned on saying:
You're going to receive a lot of unsolicited advice about your intended career
Most of it will be from men
Most will mean well, but will suggest a 'more family-friendly' career choice
Yet, even whilst a student, the 'advice' has started:
'Neurosurgery, are you sure?'
'It's very long & arduous training'
'It's not the most family friendly career'
'You might feel like that now, but in 10yrs you'll probably regret it'
'It's harder to find a partner when you're working all the time'
'GP is a better choice for balance'
Another nice example recently of the importance of a systematic approach to assessment of mitral regurgitation during TOE
Indication was known MR, assess suitability for TEER
Mid oesophageal 4Ch & 5Ch views...wondering if we have the right patient! No real MR to see...
The segmentation approach in the bicommisural view is a very reliable and easy-to-do method
Start at the lateral side of the valve with X-plane (Philips) / MultiD (GE) and you have A1-P1 coaptation on the right side...do this with 2D only & then with colour Doppler too
Then move to the middle of the valve with your cursor, cutting through A2 in bicomm view so you see A2-P2 coaptation on the right side
It takes time to read the paper, read the supplementary appendix, analyse the results, think about them etc!
Some thoughts...π§΅
On Sunday I wrote a thread about asymptomatic severe AS and what we knew already from RECOVERY & AVATAR RCTs and what the guidelines currently advocate
Transthoracic echocardiography (TTE), when performed with care and diligence, can reveal a lot about the valve. TOE isn't necessary in all cases to determine leaflet pathology.
A worked example below:
In the PLAX view, you can assess the scallops of the leaflets
In a true PLAX view with aortic valve clearly visible, you mostly see the A2-P2 interface. Here, you can see a clear & large prolapse of the posterior leaflet
If you tilt upwards towards the PLAX RV outflow (pulmonary valve) view you see mostly the A1-P1 interface
Here, you can see the valve looks slightly different & no prolapse is seen