2/A key concept in these fxs is dental occlusion. The jaw is meant to chew. To crush food, teeth need to come as close together as possible—occlusion. Each tooth needs to meet up with its counterpart that fits with it, so no room is left for food—and food will be crushed.
3/Occlusion can be lost w/a fx. The importance of dental occlusion makes mandibular fxs different from other fxs. Usually, we want to fix a bone so that it lines up again. But for the mandible, we want to fix it so the TEETH line up again—so chewing will work.
4/Another important concept is multiplicity. It used to be taught that the mandible was a ring w/the skullbase—& you can’t break a ring in one place, so every mandible fx had to have a 2nd one. But this is only true ~50% of time—but it is still worth it to look for the 2nd fx.
5/Because of the shape of the mandible and the typical forces applied to it, mandibular fractures usually occur at the same few locations. But the names of these fxs are not always intuitive—at least not without a tweetorial to help explain them! 😉
6/Well, perhaps I lied a little. The most common fx does have an intuitive name. An angle fx starts at…wait for it…the angle and extends into socket of the 3rd molar. If you can’t remember an angle fx starts at the mandibular angle, then not even this tweetorial can help you!
7/A subcondylar fracture starts from the notch between the condyle and coronoid process, called the sigmoid notch and extends into the posterior ramus. Don’t call this a ramus fx bc a ramus fx goes straight horizontally through the ramus!
8/Here are examples of subcondylar fxs. A key finding in subcondylar fxs is that it separates the condyle from the rest of the mandible. It can be difficult to see the fx running through the sigmoid notch & ramus on 2D images—3D images can be helpful to see the fx anatomy.
9/I remember subcondylar fxs bc they separate the condyle from the rest of the mandible. If you isolate someone, you make them feel SUBpar—so SUBcondylar fxs make the condyle feel subpar bc they separate it from the rest of the mandible!
10/Condylar head/neck fxs are easy to remember—they are through, well, the condylar head or neck. These are fx/dislocations. Pterygoid muscles attach to the head & pull medially. So when the head is no longer attached to the mandible, pterygoids are unopposed & pull it medially.
11/So if you ever see a condylar head displaced medially, you know it is a condylar head or neck fracture bc this is where the pterygoids are unopposed and pull medially!
12/Body fxs are through the body of the mandible and are named for the tooth socket that they involve. So you would say “A mandibular body fx through the FILL IN THE BLANK TOOTH socket.”
13/Body fxs through the canine are given the special name “parasymphaseal” or “mental” fx bc they are near the mental foramen where the inferior alveolar nerve exits. Menton means “chin” in French, so if they are a body fx anteriorly near the chin, they are “mental” fxs
14/So now you know your mandibular fractures & you have a plan even when the patient gets punched!
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If the patient is symptomatic & the greatest stenosis from the plaque is >70% of the diameter of normal distal lumen, patient will likely benefit from carotid endarterectomy
But that doesn’t mean the remaining patients are just fine!
3/Yes, carotid plaques resulting in high-grade stenosis are high risk
But assuming that stenosis is the only mechanism by which a carotid plaque is high risk is like assuming that the only way to kill someone is by strangulation.
1/My hardest thread yet! Are you up for the challenge?
How stroke perfusion imaging works!
Ever wonder why it’s Tmax & not Tmin?
Do you not question & let RAPID read the perfusion for you? Not anymore!
2/Perfusion imaging is based on one principle: When you inject CT or MR intravenous contrast, the contrast flows w/blood & so contrast can be a surrogate marker for blood.
This is key, b/c we can track contrast—it changes CT density or MR signal so we can see where it goes.
3/So if we can track how contrast gets to the tissue (by changes in CT density or MR signal), then we can approximate how BLOOD is getting to the tissue.
And how much blood is getting to the tissue is what perfusion imaging is all about.
1/”That’s a ninja turtle looking at me!” I exclaimed. My fellow rolled his eyes at me, “Why do I feel I’m going to see this a thread on this soon…”
He was right! A thread about one of my favorite imaging findings & pathology behind it
2/Now the ninja turtle isn’t an actual sign—yet!
But I am hoping to make it go viral as one. To understand what this ninja turtle is, you have to know the anatomy.
I have always thought the medulla looks like a 3 leaf clover in this region.
The most medial bump of the clover is the medullary pyramid (motor fibers).
Next to it is the inferior olivary nucleus (ION), & finally, the last largest leaf is the inferior cerebellar peduncle.
Now you can see that the ninja turtle eyes correspond to the ION.
3/But why are IONs large & bright in our ninja turtle?
This is hypertrophic olivary degeneration.
It is how ION degenerates when input to it is disrupted. Input to ION comes from a circuit called the triangle of Guillain & Mollaret—which sounds like a fine French wine label!
1/I always say you can tell a bad read on a spine MR if it doesn’t talk about lateral recesses.
What will I think when I see your read? Do you rate lateral recess stenosis?
Here’s a thread on lateral recess anatomy & a grading system for lateral recess stenosis
2/First anatomy.
Thecal sac is like a highway, carrying the nerve roots down the lumbar spine.
Lateral recess is part of the lateral lumbar canal, which is essentially the exit for spinal nerve roots to get off the thecal sac highway & head out into the rest of the body
3/Exits have 3 main parts.
First is the deceleration lane, where the car slows down as it starts the process of exiting.
Then there is the off ramp itself, and this leads into the service road which takes the car to the roads that it needs to get to its destination