2/ and presents with excruciating unilateral orbital, supraorbital, and/or temporal pain that lasts 15-180 minutes if left untreated. Patients also have ipsilateral autonomic symptoms during a cluster attack.
3/ Although the exact pathophysiology is unclear, the trigeminovascular pathway is important for the unilateral trigeminal distribution of pain, the trigeminal-autonomic reflex is important for producing ipsilateral cranial autonomic symptoms,
4/ and the hypothalamus is important for the circadian and circannual pattern of cluster attacks, and may play a role in generating a cluster attack.
5/ Treatment of cluster headache is divided into 3 categories: acute treatment, which acutely stops an attack, intermediate treatment, which temporarily reduces the severity and/or frequency of attacks while preventive treatment takes time to take effect,
6/ and preventive treatment, which reduces the severity and frequency of attacks for a longer time. Please take a look at the infographic I have created to take with you in your studies. Happy learning!
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Check out my illustration on the course of the oculomotor nerve with its associated sites of injury!
Patients with an oculomotor (CN III) nerve palsy can present with binocular horizontal, vertical, or oblique diplopia and ptosis. (1/3)
In addition to extraocular muscle weakness, one may find a dilated pupils (mydriasis) due to unopposed sympathetic innervation to the pupil. Having an understanding of the course of the oculomotor nerve, and where injury can occur, leads to the production of a (2/3)
differential diagnosis, ordering appropriate diagnostic studies to confirm the cause of oculomotor nerve injury, and implementing appropriate management to best serve the patient. I hope you all enjoy it! (3/3).