Eduardo R Argaiz Profile picture
Dec 28 9 tweets 5 min read
Ambulatory Hemodialysis Unit Rounds:

Called to see a patient with hypotension: BP 76/40,🧠 OK, CRT 5 seconds

1st step ➡️🛑Ultrafiltration + 300 ml bolus. BP 90/60

Pt is a middle aged ♂️ w ESRD and T2DM

1/9 🧵
Now 3 kg above Dry Weight.

UF Volume so far: Only 600 ml

🔎📁 Previos HD sessions with no hypotensive episodes

1 week with URI symptoms, 2 days with dyspnea on exertion

On exam: No leg edema, Clear 🫁, JVP hard to assess (hx of multiple CVCs and central vein stenosis)

2/9
#POCUS:

Pericardial Effusion, Normal LV function, looks like there is some RV colapse

Plethoric IVC, Portal Pulsatility 39%.

A-Pattern on LUS, Small bilateral pleural effusions

🚨⬇️BP + Collapsing RV + Venous Congestion (IVC + Portal Pulsatility) suggests Tamponade!

3/9
Hepatic Vein Doppler shows characteristic D wave reversal!

Again: This is strongly suggestive of Tamponade!!🚨

4/9
Also..LV looks very empty. LV walls can get too close together and obstruct the flow of blood!

Obstruction causes blood to speed up! (think of thumb on a hose).

Velocity is 3.5 m/s (very fast) with a gradient of 50 mmHg! This is Hemodynamically significant obstruction!

5/9
In order to improve obstruction, we need to increase preload of the LV! Pericardiocentesis will achieve this (by relieving RV collapse)

In the meantime, a fluid bolus can be a temporizing measure.

More on the physiology of LV obstruction here:

6/9
Gave an extra 300 ml fluid and arranged ICU admission!

Fluid did help: BP now 105/69, gradient improved from 50 to 24 mmHg!

However, liver congestion got worse: Portal Vein Pulsatility now 62%. This is not good, especially since LFTs came back ⬆️⬆️⬆️

7/9
Pericardiocentesis was performed, this fixed everything!

BP now 153/76.

Portal Vein is no longer pulsatile

Hepatic vein shows no D wave reversal

Gradient is now 4.8 mmHg (Not hemodynamically significant!)

8/9
Learning Points:

🔷#POCUS can help in the evaluation of intradialytic hypotension
🔷Venous congestion ≠ Volume Overload
🔷Tamponade ➡️ Low LV preload ➡️ Obstruction (if predisposing conditions)
🔷Fluids worsen congestion, but can be an adequate temporizing measure

/END

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More from @ArgaizR

Nov 22
#AKIConsultSeries:👨w T2DM➡️🏥 for fever, dysuria and CVA tenderness. On arrival: ⬇️BP, ⬆️Glucose, ⬆️AGMA. Dx UTI + DKA. Tx: Abx + Insulin Pump + 4 L Crystalloid + NE

After resus, pt still oliguric, Cr 3.2. NE 0.7 ug/kg/min,🧠confused, BP 85/62, HR 123, 2L O2. CRT 4 sec

1/12
Given DKA, giving additional fluids is tempting. But before we do this, its easy to do a quick assessment of fluid tolerance #POCUS

#LUS shows some B-lines (bilat)
#IVC plethoric w no respiratory collapse
#VExUS shows very pulsatile portal vein 🚨🤔

2/12
Pulse pressure is low (23!): This suggest a low cardiac output state!

Also, there are signs of fluid intolerance!

#EchoFirst: Window is suboptimal, but we see a Hyper-dynamic LV w small cavity and a turbulent flow (green color). There was no systolic RV failure

3/12
Read 12 tweets
Sep 17
Pt seen in ambulatory clinic with worsening kidney function

While the patient is sitting down (90 degrees), you notice neck pulsations!

Are they arterial or venous??

1/4 🧵
It is single peak (but not sharp)

The most striking feature is the inward movement

The breath of movement is diffuse

These are signs of venous pulsations!

Very helpful table from @AndreMansoor 👇



2/4
Thankfully we have #POCUS in clinic! I believe #POCUS can really help you improve your classic physical exam skills as it gives you immediate feedback!

Quick #VExUS reveals plethoric IVC, reverse S wave on Hepatic Vein, >100% portal vein pulsatility and mono-phasic IRVD!

3/4
Read 4 tweets
Aug 20
Young pt ➡️ 🏥 worsening shortness of breath

PMH: ESRD. Only 1 HD session/week. However, residual urine volume has now decreased substantially

On exam: BP 134/94, 2L O2,🧠✅, elevated JVP, decreased 🫁 sounds at bases, No murmurs, very mild edema. Functional left BC AVF

1/13
Careful examination of neck veins reveals no pulsations, even with pt sitting up 🤔

What could explain the absence of venous pulse? 2/13
Answer is all of the above. JVP examination can be complicated in pts with ESRD.

In the absence of pulsations, I find #POCUS much helpful. Let's enhance our physical examination of congestion:

3/13
Read 13 tweets
Jul 10
#AKIConsultSeries Middle-aged male ➡️🏥 for painful knee and fever. Now in shock 🚨

📂Chart review: PMH EtOH Cirrhosis, right knee arthroplasty.

It is always a good practice review previous PACS images🩻: Nodular liver, colateral vessels and prosthetic right knee

1/11
On exam: BP 72/48, HR 82, O2Sat 95%.
CRT 7 sec, 🧠somnolent, confused. No edema, no obvious ascites.

Warm, swollen and erythematous knee: Tap with obvious purulent fluid🧫

Cr 2.8 mg/dl (baseline 0.5), K 6.7, Urine 🔬: hyaline casts, some urothelial cells

2/11
Loos like hemodynamic AKI (AKA Pre-renal)

Usual causes in Cirrhosis:

🔷Distributive: Septic, "Hepatorenal physiology" 🔷Hypovolemic: Laxatives, vomiting, large volume paracentesis
🔷Congestive: Porto-pulmonary HTN, Co-existing cardiomyopathy

3/11
Read 11 tweets
Dec 23, 2021
Patient with flank pain, hematuria and significant leg edema

#POCUS 🧵

1/10
Lung Ultrasound #LUS 2/10
#IVC long axis 3/10
Read 10 tweets
Dec 8, 2021
A tale of two hearts: Physiological observations on AV shunts and congestion 🧵

These are 2 patients on IHD I saw in the outpatient clinic

🔷 Both with severe venous congestion (#VExUS = 3)
🔷 Both with tortuous brachiocephalic AV fístula

1/11
What I found remarkable was the diametrically opposed effects of manual AVF compression on JVP! 🤯

🔴 Patient A: AVF Compression improves venous congestion
🔵 Patient B: AVF Compression worsens venous congestion

2/11
🔴 Patient A: SLE + Lupus Nefritis ➡️ ESRD in HD

#echofirst: Plethoric IVC, good LVEF, paradoxical septal motion, ventricular interdependence, severe RV/RA dilation, torrential TR

3/11
Read 13 tweets

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