Lea Alhilali, MD Profile picture
Feb 13, 2023 12 tweets 6 min read Read on X
1/Do you know all the aspects of, well, ASPECTS?

Many know the anterior circulation #stroke scoring system—but posterior circulation (pc) ASPECTS is often unknown

Here’s a #tweetorial to help you remember pc-ASPECTS
#medtwitter #neurotwitter #meded #neurorad #neurology #FOAMed Image
2/Many know anterior circulation ASPECTS. It uses a 10 point scoring system to semi-quantitate the amount of the MCA territory infarcted on non-contrast head CT.

If you need a review: here’s my tweetorial on ASPECTS: Image
3/But it’s only useful for the anterior circulation. Posterior circulation accounts for ~25% of infarcts. Even w/recanalization, many of these pts do poorly bc of the extent of already infarcted tissue. So there’s a need to quantitate the amount of infarcted tissue in these pts Image
4/Posterior circulation ASPECTS, or pc-ASPECTS for short, is also a 10 point system—but for the vertebrobasilar circulation. Patients get points for each region NOT infarcted on the initial CT. Image
5/Similar to anterior ASPECTS, points are GOOD

It’s like a city—when a region infarcts, it is like the lights go out in that city region (literally, tissue darkens on CT)

In ant & post ASPECTS, you are counting the regions where the lights are still on—so high ASPECTS is good Image
6/Which regions get scored?

The biggest prognostic factors in posterior strokes are time & amount of already infarcted tissue.

So my mnemonic to remember pc-ASPECTS regions is:

Time & Mortal Brain decide Posterior Circulation Outcomes
(mortal brain= infarcted brain) Image
7/How do you assign points?

It’s a little different than anterior ASPECTS, where every region just got one point

For pc-ASPECTS, think of it like a mother. She only has 2 arms. So if there are two kids, they have to split the arms—only one arm can be wrapped around each kid Image
8/So unpaired structures, like the brainstem, are like having only one kid—you get both arms wrapped around you = 2 arms = 2 points.

If the structures are paired, like the cerebellum, it’s like having two kids--they must split the arms. Each gets only 1 arm = 1 point Image
9/So lets go through our regions using the mnemonic.

Each thalmus is worth only 1 point, bc they’re paired & have to share their mother’s arms.

The unpaired midbrain is worth 2 points (gets both arms) Image
10/Pons is unpaired, so it is worth 2 points.

But the paired cerebellar hemispheres & occipital cortex are each only worth 1 point per side, as each side claims only one of the mother’s arms Image
11/While there’s evidence that higher pc-ASPECTS means poorer outcome, there’s no consensus about what pc-ASPECTS score definitively confers a UFO (unfavorable outcome).

But the consensus about what confers a UFO in anterior circulation ASPECTS may change soon as well! Image
12/So now you know the regions & scoring for pc-ASPECTS!

Remember, the anterior circulation isn’t the only aspect of ASPECTS! Image

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More from @teachplaygrub

Jun 6
1/Raise your hand if you’re confused by the BRACHIAL PLEXUS!

I could never seem to remember or understand it—but now I do & I’ll show you how!

A thread so you will never fear brachial plexus anatomy again! Image
2/Everyone has a mnemonic to remember brachial plexus anatomy.

I’m a radiologist, so I remember one about Rad Techs.

But just remembering the names & their order isn’t enough.

That is just the starting point--let’s really understand it Image
3/From the mnemonic, we start with the roots—the cervical nerve roots.

I remember which roots make up the brachial plexus by remembering that it supplies the hand.

You have 5 fingers on your hand so we start with C5 & we take 5 nerve roots (C5-T1). Image
Read 20 tweets
Jun 4
1/Having trouble remembering what to look for in vascular dementia on imaging?

Almost everyone w/memory loss has infarcts. Which are important?

The latest @theajnr SCANtastic has what you need to know:

ajnr.org/content/46/5/1…Image
@TheAJNR 2/Vascular cognitive impairment, or its most serious form, vascular dementia, used to be called multi-infarct dementia.

It was thought dementia directly resulted from brain volume loss from infarcts, w/the thought that 50-100cc of infarcted related volume loss caused dementia Image
@TheAJNR 3/But that’s now outdated. We now know vascular dementia results from diverse pathologies that all share a common vascular origin.

It’s possible to lose little volume from infarct & still result in dementia.

So if infarcts are common—which contribute to vascular dementia? Image
Read 20 tweets
Jun 2
1/Having trouble remembering how to differentiate dementias on imaging?

Is looking at dementia PET scans one of your PET peeves?

Here’s a thread to show you how to remember the imaging findings in dementia & never forget! Image
2/The most common functional imaging used in dementia is FDG PET. And the most common dementia is Alzheimer’s disease (AD).

On PET, AD demonstrates a typical Nike swoosh pattern—with decreased metabolism in the parietal & temporal regions Image
3/The swoosh rapidly tapers anteriorly—& so does hypometabolism in AD in the temporal lobe. It usually spares the anterior temporal poles.

So in AD look for a rapidly tapering Nike swoosh, w/hypometabolism in the parietal/temporal regions—sparing the anterior temporal pole Image
Read 16 tweets
May 27
1/Feel perplexed by the lumbosacral plexus??

This plexus doesn’t have to be so complex-us

Here’s what you need to know from this month’s @Radiographics!



@cookyscan1 @RadG_editor doi.org/10.1148/rg.240…Image
@RadioGraphics @cookyscan1 @RadG_Editor 2/The lumbosacral plexus is like a love story

The lumbar & sacral plexuses met & fell in love

They loved each other so much they came together to create the nerves to the lower extremities! Image
@RadioGraphics @cookyscan1 @RadG_Editor 3/Lumbosacral plexus is essentially formed by the nerves from L1-S4 (with some other small contributions)

Remember this bc the plexus is to the lower extremitieis and L & 1 look legs and S & 4 look like feet! Image
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May 6
1/Have disagreements between radiologists on the degree of cervical canal stenosis become a pain in the neck?

Worried about sticking your neck out & calling severe cervical stenosis?

This month’s @theAJNR SCANtastic has the latest about Cspine MRI!

ajnr.org/content/46/4/7…Image
@TheAJNR 2/In the lumbar spine, it is all about the degree of canal narrowing & room for nerve roots.

In the cervical spine, we have another factor to think about—the cord.

Cord integrity is key. No matter the degree of stenosis, if the cord isn’t happy, the patient won’t be either Image
@TheAJNR 3/Cord flattening, even w/o canal stenosis, can cause myelopathy.

No one is quite sure why.

Some say it’s b/c mass effect on static imaging may be much worse dynamically, some say repetitive microtrauma, & some say micro-ischemia from compression of perforators Image
Read 16 tweets
May 2
1/Do radiologists sound like they are speaking a different language when they talk about MRI?

T1 shortening what? T2 prolongation who?

Here’s a translation w/an introductory thread to MRI. Image
2/Let’s start w/T1—it is #1 after all! T1 is for anatomy

Since it’s anatomic, brain structures will reflect the same color as real life

So gray matter is gray on T1 & white matter is white on T1

So if you see an image where gray is gray & white is white—you know it’s a T1 Image
3/T1 is also for contrast

Contrast material helps us to see masses

Contrast can’t get into normal brain & spine bc of the blood brain barrier—but masses don’t have a blood brain barrier, so when you give contrast, masses will take it up & light up, making them easier to see. Image
Read 20 tweets

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