"If your idea is correct, the RNA π is containing decent amount of intact spike gene connected to T7 promoter. I have found several papers which is describing that T7 promoter works efficiently in mammalian cells. This is a big disaster." #WarpedSpeed
The test kits the government mandated people to take were contaminated with bacteria, just as the ππ the mandated people take were. Is EVERYHING from the U.S. government #Dirrty?
How the SARS-CoV-2 spike protein chimera persists in the body chronically and activates a dual mTOR response, and remodels the immune system.
Including Annelise Bocquetβs Janus mTOR theory, my SAPIR model and published papers given to Grok. π§΅
π₯ Toxic Phoenix Hypothesis π₯
The spike protein of SARS-CoV-2
(whether from π¦ or mRNA π) behaves like a
"Toxic Phoenix"
a mythic creature of self-consuming π₯ and regenerative venom.
It ignites a destructive π₯ in the host
(acute hyper-mTOR activation, cytokine storm, oxidative inferno),
it appears to π or be cleared, only to rise renewed and more toxic from its own ashes through persistent immune sanctuaries, microbiome reprogramming, and self-amplifying loops.
Each rebirth is more insidious than the last, turning the hostβs own biological pathways into fuel for chronic pathologies. >
π₯
The TP theory is an extension of my #ItsAlive theory on the π§ Frankenstein nature of SARS2, positing that short gene sequence analogs in the lab-created spike protein can cause similar pathologies in C0VID infected patients as the original organisms do.
I.e. when you put an an abby normal π§ of a murderer into the backbone of several corpses, you shouldnβt be surprised when it goes berserk and starts killing the townsfolk after you revive it.
Because these gene analogs come from diverse progenitors:
HIV, SEB, venoms,
they cause heterogeneous symptoms and pathological mechanisms in the body.
This is why C0VID and the C0VID π have different symptoms/adverse events in different people and different persistent sequelae. > x.com/janiesaysyay/sβ¦
Self-Amplifying Cardiac Inflammation After mRNA π
Updated π§΅ (published research + autopsies + Grok)
The C0VID mRNA-LNP platforms (BNT162b2 & mRNA-1273) were deliberately engineered with specific modifications to maximize antigen expression, evade innate immunity, and produce a stable immunogen.
These exact choices create extraordinary durability for both the mRNA cargo and the synthetic spike protein β and they are the root of the platformβs π« vulnerability.
π
2 proline substitutions (K986P + V987P = S-2P) act as a molecular π
They sit in the S2 subunit and create rigid kinks that block the natural pre-to-post-fusion refolding.
This keeps the spike in its prefusion shape for better antibodies β but also makes the synthetic spike far more resistant to degradation and unfolding. >
π
Every uridine is replaced with N1-methylpseudouridine (m1Ξ¨) β the single most powerful durability factor.
m1Ξ¨ fools TLR7/8 and RIG-I/MDA5 sensors, suppresses inflammation, boosts translation efficiency, stabilizes mRNA structure, and resists RNase enzymes.
What was meant to solve short half-life now lets mRNA persist for weeks to years. π‘οΈ >
Together these aggregate to form #WhiteClotSyndrome #CalimariClots #CalamariClots which have a tougher tensile strength, like EPDM rubber.
(@Greg21143362)
Ultimate Spike Detox canβt thoroughly remove the clots.
>
The carcinogenic potential of the SARS2 spike protein
in the π« due to fibrosis and inflammation via
upregulation of TYMP - STAT 3.
π§΅
Covid causes lung fibrosis, fibrosis is a precursor for cancer in the lungs.
Looking at this preprint and other published research with Grok, on SARS-CoV-2 and the platelet endothelial growth factor enzyme, Thymidine Phosphorylase, TYMP a mechanism for lung cancer post C0VID develops. >
They are π because of the disastrous pandemic response directed by Fauci.
Fauci gave conflicting medical advice and censored
life-saving early πs in order to shield DoD bi0weapons programs from scrutiny and enable a novel warp speed mRNA π >