"If your idea is correct, the RNA π is containing decent amount of intact spike gene connected to T7 promoter. I have found several papers which is describing that T7 promoter works efficiently in mammalian cells. This is a big disaster." #WarpedSpeed
The test kits the government mandated people to take were contaminated with bacteria, just as the ππ the mandated people take were. Is EVERYHING from the U.S. government #Dirrty?
π Another brilliant research paper from Pretorius et al.
#DiamondsOnTheSolesOfHerShoes
It's interesting to hypothesize what these findings might mean in light of the C0VID π's mRNA/spike protein persistence in the body, and its accompanying dose of billions of lipid nanoparticles in systemic circulation. >
The scientists used single cell multi-omics programming, antibody and cytokine profiles to uncover:
"a distinct myeloid priming program induced by Type I interferons after the first mRNA dose, and a broader boosting response involving both Type I and Type II interferons after the second mRNA dose or Ad26.COV2.S." >
They compared different expressions of genes in monocytes and dendritic cells and found that interferon stimulated genes (ISGs) were upregulated. >
2 interesting papers on blood signatures for
Sporadic CJD.
Researchers found 38 different DNA methylation sites related to sCJD.
9 of these sites were replicated in a further study, with sites in or close to genes: FKBP5, AIM2, UHRF1, KCNAB2 successfully replicated, specific to sCJD.
These were specific to sCJD vs other prion disease, or AD and found only in the blood, not brains of sCJD patients, suggesting a possible blood biomarker.
> tinyurl.com/bdnn7ysr
"Top 10 gene ontologies enriched in genes overlapping DMPs as identified using MetaCore (p value thresholdβ=β0.1)." -Fig. 2
>
"Loss of DNA methylation at 2 sites on the AIM2 promoter correlates with disease severity.
AIM2 is a key component of the inflammasome pathway...drives IL-1Ξ²"
>