Post

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

@DrJackKruse

Jul 30, 2025 • 11 tweets • 9 min read • Read on X

Scrolly

https://twitter.com/ValerieAnne1970/status/1950434524740939907

While the tweet is correct, my tribe knows Dr. B is someone's advice to avoid. He has no idea about the quantum biology of NO. His advice was so bad I had to cancel a member's membership over this.

https://twitter.com/ValerieAnne1970/status/1950434524740939907

2. NO to excess is a danger. I saw this first hand in New Orleans during HIV.

3. UV sunlight stimulates vitamin B12 synthesis, red blood cell production, NO release (lowering blood pressure, increasing venous oxygen), and stem cell activity, with corrin-bound NO modulating its bioactivity as a GOE legacy against oxygen toxicity.

The Coulomb force (∇·E = ρ/ε₀) amplifies skin’s electrostatic fields, optimizing these processes, with potential significance in Nodes of Ranvier (nerve conduction) and retinal vasculature (blood flow).

In California for example, nnEMF, smog, and lifestyle factors disrupt this photobiology, reducing 25(OH)D, B12, and NO, driving many diseases like atopy, eczema and autism (1 in 24 children, 2025). Maternal disruptions in this system induce many epigenetic changes, amplifying transgenerational risks versus Florida. ICD-10 code use in California W90.0 supports nnEMF’s role in how excess NO can harm.

4. Corrins bind nitric oxide (NO), quenching its bioactivity, making sure it acts locally and globally. No enhances blood flow but it decreases ATP production and this legacy action is tied to the GOE to protect us from oxygen toxcity. The real action of NO is to increase the heat sink in cells. This has massive effects on the semiconduction inside of cells. There has to be a big story here for the Coulomb force and skin, brain, eyes. It might be a bigger deal in the Nodes of Ranvier and in retinal vascular system in diseases like ALS.

5. Sunlight’s Photobiological Effects on B12 & NO are antagonistic by design to make its effects focal.

Vitamin B12 Synthesis: UV sunlight (particularly UVB, ~290–320 nm) can stimulate the synthesis of vitamin B12 (cobalamin) in the skin and/or gut microbiome by enhancing the activity of photolyase-like enzymes or influencing bacterial cobalamin production.

While direct evidence is limited, I have asked @SabinehazanMD and @dralexisjazmyn to study it because centralized gastro MDs and PhDs of th emocrobiome won't because it breaks their paradigm of current religious belief.

First principles teach the Savage that UV exposure increases B12 levels in some species (e.g., algae), suggesting the first two domains of life use many photobiological pathways. This must involve quantum excitation of corrin ring electrons, explaining why the human gut is filled with aromatic amino acids and things like serotonin, though the mechanism in humans requires further study because the NIH does not want this truth out. If people got in the sun it would negate the need for the FDA and BigHarma. This is why it is not studied.

Red Blood Cell Synthesis: UV sunlight enhances erythropoiesis (red blood cell production) by stimulating bone marrow activity, via NO-mediated signaling in the renin angiotensin loop via vitamin D-induced upregulation of erythropoietin (EPO). Studies show UV exposure increases hematocrit in all animals studied, and this supports my thesis, but remarkably no PhD can get funding to study humans because the results would destroy the power base of BigHarma and the FDA.

6. NO as a Photoswitch: NO electrifies the gut barrier via splanchnic vasodilation, modulates ROS/RNS, and links to ATP/ROS balance, acting as a rapid, local signal per Shannon’s information theory.

Gut Dysbiosis and Endothelial Dysfunction: Lack of full-spectrum sunlight (UV, blue-green) reduces NO, causing gut barrier breakdown, dysbiosis, and systemic diseases (e.g., PAD, glaucoma, eczema, autism), spreading via mitochondrial colonies.

HIF-1 and PER2 Link: HIF-1, tied to PER2, regulates circadian clocks and SIRT3 under hypoxia, influenced by sunlight, with evolutionary roots in the KT event favoring mammals over dinosaurs.

Red Light Potential: Intense red light may mimic sunlight’s PER system effects, offering a therapeutic avenue.

Coulomb Force and Electrification: Electrostatic fields (∇·E = ρ/ε₀) amplify photobiological signals in skin, gut, and neural tissues.

Orange County Context: nnEMF and malillumination exacerbate these disruptions, driving transgenerational risks in offspring in California.

Actionable Insights: Strategies to restore photobiology I teach ad nauseum on my Patreon blogs. patreon.com/DrJackKruse

7. What PhD Bryan is clueless on and you can see it Dr. Attia's reasoning too? This is why peripheral arterial disease is always/often associated with light induced diseases via the abscopal effect. The link is the aberrant use of the electromagentic spectrum to communicate to create NO to act as the photoswitch between electric and the magnetochemistry of free radicals and this creates the UPE spectra that control biochemistry actions.

RNS, include nitric oxide (NO), peroxynitrite, and nitroxylanion, is also an important class of oxidative bio-signal molecules. The corrins and B12 quench all this assuming you are doing life right.

Nitric oxide synthases (NOS) are a group of enzymes that produce NO from L-arginine (L-Arg), O2 and NADPH. Skeletal muscle contains three NOS subtypes, which are neuronal NOS (nNOS), inducible NOS (iNOS) and endothelial NOS (eNOS)

8. Modern lighting and the AC power grid affect all RNS and ROS production but really destroy NO production. RF/microwave cell radiation impairs production of NO from arginine by eNOS. This, in turn, induces oxygen variations in the cytosol and matrix by altering blood pressure and partial pressures of oxygen in a cell to induce endothelial dysfunction in the target organ.

Mitochondria are intimately involved in importing nitrogen into tissues to create the substrates that eventually become NO when sunlight is present. This is why Bryan is talking up foods with nitrates and nitrites. People who live in shit holes are super sensitive to them because they live in shit light. Bryan does not have this sophistication understanding. You must. Nitrogen substrates are not created from the direct synthesis by eNOS.

Nature provided the clue to me why humans got rid of Vitamin C for glutathione in this AMO physics dance. When Vitamin C is missing in subcutaneous tissues, glutathione become more reactive with locally produced NO. This mimics a radical pair or triad effect we see in the magnetic avian compass navigation. Here was more evidence for me that magnetochemistry in humans being used and timing was more important than I'd ever imagined.

9. When glutathione and nitric oxide are powered by terrestrial sunlight this allowed humans to produce S-nitrosoglutathione (GSNO). I think this is why human primates lost the majority of their integumentary hair to absorb more solar light to power their new territory in the frontal regions and it added a ton of charge density to the gut. This is why is shortened and got smaller. The better the skin absorbed solar photons the more melanin from the hair follicle migrated to the interior and the more NO was made in the skin and gut.

This allowed the skin & gut to become a MASSIVE charge capacitor for the brain and heart by allowing the skin to become a depot station to store massive amounts of nitric oxide. This is why human immune T cells are so common in the skin and why leptin was placed in SQ fat. Other primates do not have these phenotypes even thought their genomes are close to identical.

This told me the primate body plan was changed using magnetochemistry and not genetic therapy all indiced by the timing of photoswitching in the skin. Solar light completely induced this evolutionary change in our primate cousins. Everything I believed was thrown out the window from medicial school that day 20 yrs ago. We never need genes to change this.

10. We used timing to change the metabolic pathways in the skin using hair loss and removal of Vitamin C from the radical triad mechanism to do it. As a consequence of this dance using more light on the skin, keratinocytes were able to sense more visible light combinations with purple, blue, and green light to easily photocatalyze the release of NO from glutathione. Not only does this cause a relaxation of the blood vessels, but it also frees up glutathione to react with hydrogen sulfide gas to produce sulfate to make every other chemical in the skin water soluable to get access to body parts to have global effects. This is how light develops its abscopal effects in your tissues and each tissue zip code matters. The optical density is designed to change and free radicals are what is behind our biological kaleidoscope.

11. No one has figured out how this all works in humans but this is how I have seen it for 20 plus years and I got a big head start.

To date no one has published a thing using my ideas. But I can explain why subtraction of Vitamin C in humans links to hair loss.

It also explains why balding people all have bad surface topology. None of the photoswitches work. This explains most of the integumentary and ocular diseases we see today. It also explains obesity, too. It also explains autoimmunity.

As a throw in for my neurosurgery life it finally explained to me why humans get aneuysms and AVMs and the gut barrier goes boom when it happens.

• • •

Missing some Tweet in this thread? You can try to force a refresh

More from @DrJackKruse

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

@DrJackKruse

Feb 5

https://x.com/DrJackKruse/status/2019454199256207483

1. Should we give the 49ers players a free lesson on what the NFL and the team will never expose on their behalf?

We need to explain why the players are getting injured at an amazing pace since 2014 and that sotry begins with the evolution of the SCN in the eye that controls the circadian clock.

The eye clock is the key to the story of their injuries.

https://x.com/DrJackKruse/status/2019454199256207483

2. Today we know that vision and hearing evolved together dating back to the PaxB gene, which is a single gene controlling eye and precursors to hearing (mechanoreceptors) in box jellyfish.

This occurred before independent Pax 2 and Pax 6 genes showed up in primates much later. There are evolutionary connections between eyes and mechanoreceptors of the inner ear to the extent that during evolution are linked to melanin generation in those sense organs.

I told you earlier in the decentralized medicine series on Patreonmelanin was the favored semiconductor of all mammals post KT event.

This story of the 49ers players fits this my thesis well because the entire team is made of mammals who are post KT evovled.

If POMC/melanin is absent for any reason in these players, at any particular body place, for any reason, including nnEMF, it appears human neuroplastiticty allows sensory cells to shift their sensory modalities to an older phylogeny experienced in evolutionary history.

Why is this a big deal for the 49ers players?

3. The SCN is controlled by ipRCGs and is a well known blue light detecotr. The melanopsin phylogeny predates even primate evolution in time.

I think this happens in modern humans because of a loss of information and energy transformation in the embryo due to a lack of POMC or POMC translation in the parents cells and their germ lines that create their child = epigenetic defect planning = childhood diseases not of genetic causes which make up the bulk of childhood disease burdens today.

This means any 49ers players future children will also carry the burden of the 2014 power plant upgrade.

Read 38 tweets

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

@DrJackKruse

Jan 31

https://x.com/DrJackKruse/status/2017638218741780967

Here is the irony: Attia advice was wrong.

**certain body secretions in people with diabetes often contain higher levels of carbohydrates (specifically glucose, the main simple carb involved) compared to people without diabetes.**. Look it up.

This is primarily due to elevated **blood glucose** levels (hyperglycemia) in diabetes, which can cause glucose to spill over or leak into various secretions when blood levels exceed normal thresholds. Recall Attia never finished his residency. He charges 200K to see him. I’m sure many of his patients would expect him to know the basic of human secretions is based on blood sugar levels

Here's a breakdown by common secretions that have more carbs

-vaginal secretions are high in carbohydrates in diabetic women.

- **Urine** — In uncontrolled or poorly managed diabetes, urine frequently contains significantly more glucose (a condition called **glycosuria** or glucosuria). Normally, healthy kidneys reabsorb nearly all filtered glucose back into the blood, so urine has little to no detectable glucose. When blood glucose exceeds the renal threshold (typically around 160–180 mg/dL), excess glucose appears in the urine. This is a classic sign of diabetes and can be much higher than in non-diabetics (who usually have negligible amounts).

- **Saliva** — Multiple studies show that salivary glucose levels are higher in people with diabetes (both controlled and uncontrolled) compared to non-diabetics. For example, mean salivary glucose is often around 13–14 mg/dL in diabetics versus 4–5 mg/dL in healthy controls. This occurs because high blood glucose increases leakage or diffusion of glucose into salivary secretions.

- **Sweat** — Sweat glucose concentrations are generally low in everyone (often 1–2% of blood levels), but research shows a strong correlation between sweat and blood glucose. In diabetics with higher blood glucose, sweat glucose is correspondingly elevated (e.g., potentially 0.3 mmol/L or more when blood is very high, versus lower in non-diabetics). This is why sweat is being explored for non-invasive glucose monitoring devices.

- **Tears** — Some evidence suggests tear glucose can also be higher in diabetics and correlates with blood levels, though this is less commonly studied than the others.

In summary, while not all secretions are equally affected and concentrations remain much lower than in blood, **diabetics typically have more glucose (a carbohydrate) in urine, saliva, sweat, and possibly tears** when blood sugar is poorly controlled. This doesn't apply universally to every diabetic at all times (e.g., well-controlled cases may show minimal differences), but it's a well-documented pattern, especially in hyperglycemia. If this relates to a specific health concern, consulting a doctor for personalized testing is recommended.

https://x.com/DrJackKruse/status/2017638218741780967

2. This goes to show you just how uninformed Attia is on the basics. High blood sugar (hyperglycemia) can occur due to various conditions and factors outside of diabetes, potentially leading to similar effects on carbohydrate (primarily glucose) levels in body secretions like vaginal secretions, urine, saliva, sweat, and tears. Physical or emotional stress: Acute stress from illness, infection, injury, trauma, surgery, tech screen abuse, cell phone use triggers the release of hormones like cortisol and epinephrine, which raise blood sugar to provide energy for the "fight or flight" response. High-intensity workouts can cause a short-term spike in blood sugar as the body releases stored glucose for fuel, especially in non-diabetics unaccustomed to such activity. Poor sleep quality from blue light and Earpod use disrupts hormonal balance, leading to insulin resistance and elevated glucose levels. Attia is known to believe that doing 100 push ups limits nnEMF risk. Look it up. He told this to Chris Williamson on a live IG post.

3. What else did Attia miss in his answer to Epstein? Cushing's syndrome: Excess cortisol production (often from adrenal tumors or prolonged steroid use) impairs insulin function and raises blood sugar.

Polycystic ovary syndrome (PCOS): This common hormonal disorder in women causes insulin resistance, leading to chronic hyperglycemia.

Acromegaly: Overproduction of growth hormone from a pituitary tumor reduces insulin sensitivity and elevates glucose.

Other hormonal issues: Conditions like hyperthyroidism or pheochromocytoma (a tumor causing excess catecholamines) can also contribute.

Read 5 tweets

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

@DrJackKruse

Jan 31

https://x.com/DrJackKruse/status/2016990657450299837

In my decentralized framework, this is exactly how the system is wired. The nipple-areola complex acts as a quantum-optical sensor, and the infant’s saliva is the fiber-optic cable delivering a real-time UPE (ultra-weak photon emission) status report from the child's mitochondria to the mother’s "manufacturing plant."

This isn't just convenience; it is a biophysical "handshake" that ensures the survival of the post Cambrian hardware mammals need to thrive.

1. Saliva as the "Optical Bio-Feed"

Saliva is a highly structured, mineral-rich fluid. In my thesis, it serves as the medium for optical information transfer:
The Child’s Signal: When a calf or child is sick, their internal "optical smog" increases. The VUV (Vacuum Ultraviolet) and chaotic UPEs produced by their congested Complex II are transmitted through the saliva. Congestion usually is due to reverse electron flow or deuterium.
The Mother’s Sensor: The areola is one of the most melanized and innervated tissues in the mammalian body. Melanin here doesn't just protect against the sun; it acts as a broadband transducer like an optical scanner in the grocery store. It "reads" the frequency of the biophotons in the saliva.

2. The Backflow "Optical Loop"

Research into the "infant-led" backflow confirms that when a child suckles, a vacuum is created that pulls saliva back into the mother's mammary ducts.
Information Sensing: The mother’s immune-sensing cells in the ductal walls "listen" to the ROS/RNS signatures and UPE entropy in that saliva.
The Response: If the child’s saliva "shouts" of deuterium congestion at cytochrome two because succinate is elevated or viral "optical noise," the mother’s brain (via the SCN-hypothalamic oxytocin posterior pituitary pathway) triggers a change in milk composition. She begins to "distill" more NPD1, Iodine, and IgA antibodies into the milk to act as a "quantum reset" for the child’s mitochondria.

3. The "Rotating Mom" Phenomenon (Allomaternal Nursing)
Why do calves or elk rotate moms? In my framework, this is "Frequency Matching":
Metabolic Matching: A calf may instinctively seek a different "frequency" of milk if its own mother’s melanin-metal hardware is jammed (perhaps she spent too much time in a "dirty" environment like inside a barn under fake light).
Information Diversity: By "sampling" different mothers, the young mammal is essentially "crowd sourcing" optical coherence. They are looking for the milk with the lowest deuterium/highest DHA-D3-Iodine ratio to stabilize their own emerging Faraday cage.
Modern humans with nnEMF toxicity who cannot breast feed their children due to a lack of production should be considering what elk do when they are faced with the same problem. This concept is foreign to humans because they do not observe nature carefully enough.

4. Milk as a "Re-Cambrian-ization" Serum

Mother's milk is the ultimate low-deuterium, high-DHA, solar-coded fuel.
Deuterium Depletion: Milk fat (cream) is naturally low in deuterium, helping the child build a "clean" 14 Angstrom tunneling gap in their developing mitochondria.
Melanocortin Programming: The act of nursing at sunrise/sunset ensures the child’s Leptin-Melanocortin pathway is synced to the mother’s, preventing the "Proterozoic regression" that leads to neolithic disease later in life.
The ranch memories you have are of a Quantum Ecosystem in action. The child’s saliva "tells" the mother's nipple exactly how the child's internal "mercury lamp" (deuterium) is burning. The mother then alters the "Optical Duty Cycle" of her milk to quench the fire.
Does this explain why formula-fed infants (drinking high-deuterium, seed-oil-heavy milk) are essentially being "pushed into the Proterozoic mud" from birth, as they lack this bidirectional optical feedback loop? Yes, it does but few seem to care about it.

This lesson also has deep information for the diseased breast too. Men can help their women with diseased breasts by kissing their nipples religiously to transfer information to their women about how they feel for them. This will be highly stimulatory and healing.

Cells and Stars have a lot on common. When they fail they have mechanisms that feedback on themselves that lead to the possibility of future survival if conditions are met. In a star when it burns through its fuel source its core gets to iron and the star begins to emit microwaves. The microwave radiation interacts with the D shell electrons of Fe and it blows up in a super nova so the atoms it creates in this destruction can be recycled to something with more life in the cosmos. Cells in our body use a similar idea in apoptosis, autophagy, and ferroptosis.

In my decentralized framework, the brain and breast in cancer do not operate in the same fashion regarding IDH protection because their "optical hardware" and evolutionary duty cycles are fundamentally different. Neurons are not epithelium, but breast tissue is.
While the brain relies on IDH mutations from its DHA-rich antenna to create UPEs to work and eventually help create some VUV smog from complex two back up to clean the dirty chemistry in cancer, the breast mitigates oncogenic risk through a different mechanism:

It uses the DHA-Iodine-Melanin triad.

1. The IDH Paradox: Why the Brain Needs It, But the Breast Doesn't

The brain is the ultimate "quantum sensor" that can feedback on itself and it must maintain 24/7 DHA coherence. DHA allows for this.
The Brain (DHA Antenna): When Complex II jams, the resulting VUV emission from Deuterium threatens to shatter the DHA antenna. The IDH mutation occurs as an emergency "filter" to deplete deuterium and lower the VUV entropy. DHA, as a PUFA explodes like the star and in its destructions NPD1 and Elovanoids along with UPEs are made which are highly anti-inflammatory. The released UPEs feed back on IDH and mutate it in such a specific way that the brain is able to make more deuterium depleted water because of this interaction than it could before to help self sustain its survival. This is how most low grade gliomas begin. This process does not happen in GBM transformation due to a lack of DHA in the brain.
The Breast (The Glandular Sink): Breast tissue is not a primary "spectrometer" like the brain. Instead of a mutation-driven shunt (IDH), the breast uses Iodine as its primary "quantum ground." In the breast, the "De-Cambrian-ization" of its mitochondria is mitigated by the concentration of iodine in the Ductal-Lobular Units.
2. How the Breast Mitigates Risk: The Iodine Shield
If the brain uses the IDH mutation to "buffer" the VUV smog itsdeuterium squeeze, but the human breast uses Iodine to "quench" the fire in the cytochrome 2 Fe-S clusters that begin the disease from reverse electron flow from cytochrome 2 dysfunction.The Delta-Iodolactone Mechanism: Iodine is required to form iodo-lipids (delta-iodolactone). These act similarly to Bazan’s Elovanoids in the brain, but they are specifically tuned to inhibit the Warburg Effect in glandular tissue.
Melanin & The Nipple: The high concentration of melanin in the areola/nipple is not a "decoration." It is the Optical Port that harvests solar UV/IR to control the metal flux (Cu, Fe) in the underlying breast tissue to make sure the TCA and urea cycle are the primary pathways used to avoid cytochrome 2 congestion and Fenton reactions of Fe-S couples.
The Sink: The breast is designed as a "DHA sink" (for lactation). It mitigates VUV damage by using Melanin and Iodine to sequester the "dirty" Iron noise created from a lack of sun, nnEMF, or polarized light. nnEMF for the breast is the stimulus that leads to ferrotoptosis destruction of the Fe-S couples and this mimics the process that happens in a star. When Iodine is missing, the breast cannot "ground" its own self created UPE field, leading to DCIS or invasive carcinoma without the IDH "slow-burn" protection seen in the brain.
3. The Unified "De-Cambrian" Failure
Both cancers represent a Proterozoic Regression, but the "breakdown" follows the tissue's specific metal hierarchy:Brain Cancer (GBM/LGG): A failure of the DHA-VDR-IDH loop. The brain tries to mutate (IDH) to survive the VUV fire.
Breast Cancer: A failure of the Melanin-Iodine-DHA loop. Without Iodine to "buffer" the Iron D-shell electrons, the breast tissue undergoes a rapid phenotypic regression and this is how cancer begins.

https://x.com/DrJackKruse/status/2016990657450299837

2. Integration with Melanin and the Sun

The synthesis of both molecules is tied to the Melanin-Metal hardware:
The Sun: UVB/IR input on the skin stimulates the Leptin-Melanocortin pathway. This manages the Copper (Cu) and Manganese (Mn) levels required to build the antioxidant "Mn-SOD shield."

The Translation: Coherent UPE signals (from healthy mitochondria) then tell the cell to cleave the lipids needed for NPD1 or gamma iodolactone
.
The Result: You have a "protected" post Cambrian cell that can use oxygen via the TCA/urea cycle without producing the ionizing VUV UPEs that destroys the genome.

Bazan’s Docosanoids (Brain/Retina): These cleave from DHA to form a "Faraday cage" of 22 carbons. Their purpose is to quench the VUV (Vacuum Ultraviolet) smog emitted by deuterium in the high-density neural environment.

3. UPE isn't mere waste; from quantum biology, these photons (or associated fields) may mediate non-local signaling, akin to coherence in radical pairs or bystander effects.

Intensity/spectrum reflect metabolic flux:

Aerobic paths (TCA) produce more ROS/UPE than anaerobic (glycolysis); stress shifts spectra (e.g., UV-linked UPE from glycolysis/peptides). Melanin optimizes by calibrating inputs—solar photons tune metal-ROS-UPE, enabling adaptive switches via redox/epigenetic relays (e.g., NAD+/SIRT1, from the Decentralized Medicine series of blogs on Patreon).

patreon.com/DrJackKruse

Read 7 tweets

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

@DrJackKruse

Jan 29

https://twitter.com/GhostOfStoneyX2/status/2016730195701489736

Plan B in El Salvador is all about the Tether gold play. This is how they want to rescue things for the surveillance state.

https://twitter.com/GhostOfStoneyX2/status/2016730195701489736

2. What is the rescue plan? Remember the famous now deleted Bessent tweet about DJT/Treasury plan to confiscate Bitcoin for the US Bitcoin Reserve? That was updated once the backlash on the tweet went out. Now it is about using Tether to centralize gold as they implode the Dollar and they will confiscate the Gold.

I've argued for 10 years that Bitcoin is a superior form of "trust-minimized" money compared to gold due to the high costs of verifying gold's authenticity.

This is the ability to own and verify an asset without relying on a third party (like a Central bank or Treasury of a government). Historically, gold's "trustless" nature was its greatest strength, but Savages now know this strength has been lost because most gold now sits in centralized vaults. This is why DJT won't let anyone audit Fort Knox. Why audit what you plan to steal?

The Cost of Validation for gold is steep so no one in the USA will want to pay that freight so now we are on the honor system for the Treasury.

Anyone who has owned gold knows that verifying that a gold bar before a sale/audit is real and pure requires specialized equipment, chemical tests, or expensive third-party audits. Because this is so difficult to do, you must have an inherit "trust" the vault or institution holding it for them. + Treasury and Bessent play. What did they do in 2025. They brought their middle man in. Tether. Go check if I am bullshitting you. Tether has bought more gold in the last 18 months than they have bought Bitcoin. Why? They are storing what the thieves in the industrial miliatry surveillence state will take down the road when the retards are sidetracked by circus maximus of some other psy-ops.

Why isn't Tether buying Bitcoin in this case? Anyone with a simple computer or smartphone can instantly verify the entire history and authenticity of their Bitcoin by running a node or using a block explorer. This "validation" is nearly free, making it more decentralized and harder to seize. Tether should be buying T bills but instead is buying Gold Reserves for the Zionist bankers to steal soon. Got it, my Savages.

Bitcoiners should know and remember their history better. This gameplan was used to before in 1933 during the Great Deperession to make it easy for governments to confiscate it.....Remember FDR's EO?

The U.S. did this already with Executive Order 6102 in 1933.

Looting and Centralization are the play. For thousands of years, physical gold was frequently stolen or seized by empires. To protect it, it was eventually moved into highly secure, centralized vaults (like those at the Federal Reserve Bank of New York or the Bank of England under control of the Treasury Head.

If the steal the gold this will tank markets including Bitcoin and then the Treasury will come in an sell gold at astronomical prices to buy Bitcoin at crashed Prices. This is how the Rockefeller and Rothschild Banks plan to do this.

If you know your history this is how the same guys did the scam during the Napoleaonic Wars. They manipulated the market with a psy-ops. In 1812 it was the Battle of Waterloo.

WAKE THE FUCK UP.

If you knew this history would would not be so gullible.

https://x.com/DrJackKruse/status/2016961157823688910

3. Learn the lesson.

https://x.com/DrJackKruse/status/2016961157823688910

Read 5 tweets

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

@DrJackKruse

Jan 29

https://twitter.com/DrJackKruse/status/2013641720785686687

1. New lesson today from my forum for the Savages to consider. This tweet below is the primer.

https://twitter.com/DrJackKruse/status/2013641720785686687

2. Question asked in last 6 weeks: Jack, My breast cancer recurrence has occured in the left auxiliary node. Currently taking Verzenio and tamoxifen. Declined ovarian suppression. Starting Dr. Makis protocol soon.

x.com/drjackkruse/st…

How can I monitor my axillary lymph nodes without using ultrasound, given your concerns about its disruption to quantum coherence in tissues? Especially since my traditional blood tumor markers (CA 15-3 and CA 27.29) have consistently tested negative?

Aside from the tests listed below, are there any additional laboratory studies you recommend prioritizing for tomorrow with Dr. G?

BUN/creatinine ratio
Vitamin D
Liver function
HsCRP

3. Back round info on U/S: westonaprice.org/health-topics/…

Read 15 tweets

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

@DrJackKruse

Jan 28

1. Today's lesson from my forum is on hyperhydrosis and dysautonimia.

QUESTION: Hi everyone,

Starting this thread to document my improvements or lack thereof, as I get closer to the Equator and further away from nnEMF.

My issue is a form of Dysautonomia driven by a small gain of function mutation in the gene encoding for Nav1.7.

The results is a persistent Na+ leak in the neurons where Nav1.7 is expressed, resulting in hyperexcitability of these neurons.

This hyperexcitability leads to the following symptoms: sympathetic overactivity, hyperhidrosis, gut hypersensitivity, more prone to visceral anxiety, bronchoconstriction, etc.

I know the decentralized medicine perspective says this is an environment problem and not a genetic problem.
But I'll only be able to confirm this once I get my environment right and get rid of these symptoms.

Best,
Alex

How can my neurons help Alex?

2. ANSWER:
Relationship Between Hyperhidrosis and Dysautonomia

Hyperhidrosis is frequently recognized as a specific symptom of a broader autonomic dysfunction.

In cases involving the upper neck:

1. Localized Sweating: Irritation of the sympathetic fibers around the vertebral artery often causes sweating or flushing on only one side of the face.
2. Systemic Dysautonomia: If the compression affects the brainstem's ability to regulate the whole body, you might experience more generalized symptoms like heart palpitations, temperature dysregulation, or "drop attacks" (sudden weakness).
3. Other relationships to be explored are found below

A. Vertebrobasilar Insufficiency (VBI): The bony bridge can compress or "kink" the vertebral artery, especially during head rotation. This reduces blood flow to the brainstem, which houses the primary control centers for the autonomic nervous system. This can manifest as dizziness, fainting (syncope), and nausea, all signs of dysautonomia.

B. Barré-Liéou Syndrome: This is a specific cluster of symptoms caused by irritation of the posterior cervical sympathetic chain (the nerves that control "automatic" functions like sweating and heart rate) due to cervical spine issues. Symptoms often include unilateral facial sweating, flushing, blurred vision, and ear ringing (tinnitus). Tinnitus brings the link to melanin dysfunction in the stria medullaris as I have laid out painstakingly on 7 Patreon blogs. It signifies an nnEMF etiology to the Hyperhidrosis and dysautonomia.

C. Trigemino-Autonomic Activation: When the C1 nerve or the vertebral artery is irritated by the ponticulus posticus, the signal is processed in the Trigeminocervical Complex. This can trigger a "reflex" in the autonomic system, leading to craniofacial hyperhidrosis (sweating on the face/forehead), nasal congestion, or eye-watering. This can also be stimulated by demyelination in this region by melanin POMC defects, DHA defects in the central retinal pathways, or polarization toxicity that affects the nerve complex that links these two disease. Both, hyperhidrosis and dysautonomia are located in two distinct but interconnected systems: the Central Autonomic Network (CAN) in the brainstem and the Peripheral Sympathetic Chain in the neck. Hyperhidrosis in this scenario is typically a "positive" neurological phenomenon (overactivity) caused by irritation of the Superior Cervical Sympathetic Ganglion (SCG) and the Periarterial Carotid Plexus (lots of POMC).

3. ANSWER CONTINUES

The broader "dysautonomia" (dizziness, heart rate changes, nausea) stems from a defect in the Lower Brainstem, specifically the Nucleus Tractus Solitarius (NTS) and the Ventrolateral Medulla. These are the primary control centers for blood pressure and heart rate, located in the medulla oblongata of the brainstem. When these brainstem centers are deprived of oxygen-rich blood, like we see when Fe is forced into the +3 state over the +2 state, they fail to regulate the autonomic system correctly. This is why nnEMF can cause this syndrome. This results in the "mismatch" symptoms of dysautonomia, such as postural dizziness, syncope (fainting), or "drop attacks" where the legs suddenly give out.

Because these symptoms are often positional, it is highly recommended to speak with a radiologist specialist about a Digital Motion X-ray (DMX) or a CT Angiogram to see how your vertebral artery behaves when you move your neck.

Read 16 tweets

Share this page!

Enter URL or ID to Unroll

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

Try unrolling a thread yourself!

More from @DrJackKruse

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆

Did Thread Reader help you today?

Don't want to be a Premium member but still want to support us?

Send Email!