Common Physiological Misconceptions: (thread)
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🔹Fluid responsiveness predicts clinical benefit from fluid administration
🔹High lactate = under-resuscitated
🔹Venous Congestion effects on organs (e.g Renosarca) are different according to cause (CHF vs Pulm HTN)
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🔹Fluid responsiveness predicts clinical benefit from fluid administration
🔹High lactate = under-resuscitated
🔹Venous Congestion effects on organs (e.g Renosarca) are different according to cause (CHF vs Pulm HTN)
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🔹Significant hypoxemia from alveolar disease should cause dyspnea
🔹PEEP tirtration benefit comes from improvement in hypoxemia
🔹There is an absolute plateau pressure number that causes VILI regardless of driving pressure
🔹Significant hypoxemia from alveolar disease should cause dyspnea
🔹PEEP tirtration benefit comes from improvement in hypoxemia
🔹There is an absolute plateau pressure number that causes VILI regardless of driving pressure
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🔹Harm with rapid sodium correction is symmetrical (from either hypo or hypernatremia)
🔹Insulin therapy goal in DKA is to lower glucose
🔹Ringers Lactate exacerbates hyperkalemia
🔹Metabolic alkalosis = overdiuresis
🔹Harm with rapid sodium correction is symmetrical (from either hypo or hypernatremia)
🔹Insulin therapy goal in DKA is to lower glucose
🔹Ringers Lactate exacerbates hyperkalemia
🔹Metabolic alkalosis = overdiuresis
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🔹T Wave inversions on EKG = ongoing ischemia
🔹PSAx D-sign or McConnell sign = Pulmonary Embolism
🔹Diffuse ST depression (with ST elevation in aVR) = Left Main occlusion
🔹Right Ventricular failure is usually volume responsive
🔹T Wave inversions on EKG = ongoing ischemia
🔹PSAx D-sign or McConnell sign = Pulmonary Embolism
🔹Diffuse ST depression (with ST elevation in aVR) = Left Main occlusion
🔹Right Ventricular failure is usually volume responsive
