CROHN’S & TMJ: A patient asked if these disorders are causally related? The answer is “no”. However, in my opinion, there is an association based on underlying genetically-determined tissue disorders.
Hypermobility Spectrum Disorder tissue variations seem to potentiate evolution of these conditions: TMJ=spinal flexibility ➡️ asymmetric shoulders ➡️ unilateral Masseter m. (TMJ etiology). I consider Crohn’s a dysautonomia; aroused by lax-SIJ impingement of the presacral plexus.
A common association is TMJ (Temporal Mandibular Disorder) & IBS in same patient. Tie-in is Hypermobility Spectrum Disorder (HSD). Ligament (connective tissue) laxity exposes upright body tower to gravitational stress deforming the tower; lose joints sublux & impinge nerves.
IBS is a true dysautonomia, & Crohn’s is symptoms of escalated intensity of same pathophysiology; along a bowel-tissue-spectrum of disorders leading to Ulcerative Colitis. Hyper-elastic tissues arose autonomic neural impingements + mast cell degranulations (see med literature).
Insights & clinical impressions herein are of my own inventions; gained via an iterative evidence-based method of studying tissue & organ clinical systems; not intended for orthodox medical diagnoses. My threadreader library items propound upon these ideas.
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