BEIGHTON CRITERIA VIDEO: is somewhat disappointing because the model could not perform all the demonstrations. Her elbow hyperextensions were great.
The model was older, and when people age they lose their youthful ability to do all the hyperextensions. I would say that some of the degrees of the various hyperextensions degrade starting after 30 years old.
Another factor that must be taken into account is that Hypermobility can present as a mosaic in some persons. Their knees well demonstrate Hypermobility, but not the elbows.
There are other factors to regard when determining Hypermobility. A reliable question seems to be to. ask if the person could do the “back-bridge” as a child. Another is to ask the mother if the child could put feet to ears when younger.
A useful demonstration is to have the person put hand behind back and to raise fingers toward neck. Many hypermobiles can get fingertips up to top of the shoulders.
Another clue is to ask female patients about hypermobile functions in their sisters. If the patient is male, I ask about Hypermobility in both father and mother. Genetic inheritance certainly seems dominant within female lineages.
The ability to put palms on the floor with knees locked is a slam dunk. This is able to be done by many hypermobiles who have never entered a yoga studio.
The idea that yoga provides practitioners with perpetual Hypermobility qualities is something I am unsure of, and which someone should answer; if they have taste for a longitudinal study that will take decades to complete.
The whole yoga-Hypermobility relationship is arguable. What is the chicken, what is the egg? I have met plenty of hypermobile males and asked them how they were regarded the first time they took yoga. Their first response has usually been a wide smile.
Amongst males, a key question is whether they have dislocated any major joints. Hypermobile males often have had dislocations, and have done so in benign sports activities wherein it is unusual for non-hypermobiles to incur dislocations.
My own opinion is the degrees of hypermobile ranges of motion & dramatic displays of hypermobileness of several body parts provide no scientific evidence of amount of underlying organ dysfunctions & dysautonomias that the patient currently harbors or might harbor in the future.
Connective tissues pervade the entire body and well beyond the ability of clinicians to regard at surface of the skin & via views of ranges of motion of joints with lax ligaments. All internal tissues contain a matrix of connective tissue; known by God & Mother Nature.
Efforts by advocacy organizations to parse different hypermobiles into arbitrary categories of an alphabet soup of HSD, hEDS, EDS, etc. seems to me to just confuse the situation. Each patient is a custom package of protoplasm with a unique combination of heredity and environment.
What physicians have done & will continue to do is try to outguess Mother Nature: history, observations, and study clues provide empirical evidence for a case study of N=1. Successful application of therapies is similarly based on wisdom & empirical clinical knowledge hard won.
A major fallacy of modern medicine is most human ills can be cured if only the correct combination of synthetic chemicals can be discovered. The importance of these synthetics has exceeded value of the natural energies that exist to heal disordered humans & other animals.

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30 Nov
RESTLESS LEGS: the question becomes, why would the autonomic nervous system evoke involuntary leg movements when people are reclined and drifting into sleep; in those with POTS?
POTS highly associated with connective tissue Hypermobility & manifests as hypotension/tachycardia upon sudden upright posture; as body blood column descends. Perhaps Mitral Valve regurgitation is involved + laxity/sluggishness of autonomic-driven constriction of vein walls.
Blood returns from legs to heart vis superior-directed propulsion of lower blood column by means of leg muscle contractions; restless legs provide such muscle contraction propulsions par excellence.
Read 24 tweets
29 Nov
A video for people to test for hypermobility.
An experience I have with patients who have stigmata of hypermobility is that I will comment on their widespread joint Hypermobility & their rejoinder commonly is, “of course I do Yoga”.
When patients tell me that my heart sinks. I was excited to tell them that I think that I have an explanation for several of their seemingly disparate symptoms in several of the dysautonomia & MCAS spheres.
Read 10 tweets
29 Nov
MCAS ALARM SYSTEMS: etiology & treatment speculations. The idea that there might be primary causations of Mast Cell Activation Syndrome (MCAS) raises interesting ideas for treatments.
Human bodies have certain tissues that warn of danger. Ligaments pulled asunder result in inability to walk & run so stretched ligaments hurt. Infections cause acidosis & death; arousing protective white cells. Nerves sense dangers of heat & pressure and signal pain.
Mast cells release histamine, which in occasional & moderate amounts can promote & participate in the healing physiological process called inflammation. Inflammation announces that the body is attempting to heal.
Read 33 tweets
24 Nov
CANNABIS FOR FIBROMYALGIA is something I have studied. In a threadreaderapp essay that I am working on, I will discuss the concept.
CANNABIS FOR FIBROMYALGIA
In order to understand the use of Cannabis for pain there are certain principles that first need to be understood.
The first Principle is that, like any substance used medicinally, Cannabis first needs to be somewhat purified so that standardized dosages
can be measured, identified & used rationally & medicinally.
Read 43 tweets
18 Nov
TMD: Quite an analysis; which neatly confuses reasonable understanding of Temporal Mandibular Disorder (TMD). My next retweet of this analysis will bring etiology & treatment of TMD to a more understandable & clinically manageable level of understanding. Use of Botox intriguing.
TMD is a biomechanical disorder. & commonly found associated with Hypermobility Syndrome. Lay people commonly refer to this disorder as TMJ (disorder of the Temporal Mandibular Joint). The secret to understanding this order is to observe numerous people with this disorder.
My clinical findings in dozens of patients with TMJ & Hypermobility Syndrome (called Hypermobility Spectrum Disorder; HSD) is a thoracolumbar functional scoliosis (remits reclined when gravity is absented) with one shoulder lower & head tilted toward the lower shoulder side.
Read 21 tweets
18 Nov
Doctors & clinical scientists of the late 1800’s had a view that the dynamic of disease was not so much the nature of microorganism but the terrain of the organism. Preventive health practices of diet, exercise, and clean environments were the major ways to keep from becoming ill
These days there needs to be an added consideration, safe environments. Violence, illicit drugs, and thousands of synthetic chemicals added to the environment each year are causing human organisms to live without safety.
The precepts of Psychoneuroimmunology teach us that when either of these spheres, Mind, Nervous System, and Immune System, become overwhelmed, the mutual interrelationships can shift the organism into imbalance, disrupt homeostasis, and initiate disease and disorder.
Read 6 tweets

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