COSTOCHONDRITIS is common (mostly women) in Hypermobility syndrome. Costo- means rib (bone). Chondro- means cartilage (connective tissue). Each rib is joined to the sternum by cartilage, which is a place where biomechanical stress is focused. Set up for stress begins in the spine
The spine of most women with Hypermobility I have examined (thousands) has a functional scoliosis: curved when upright & straight when reclined & sitting. This is easily examined in the exam room, but of course better discerned when person is unclothed.
Reason for the functional scoliosis is that spine is, in great part, cartilage; each disk is a specialized ligament. In women with Hypermobility, the pelvic girdle, platform for the upright spine, is commonly unstable & tilted. Gravity evokes the functional scoliosis.
Functional scoliosis is a way for the body to attempt to maintain symmetry when upright & to provide equality of load from side to side & equal load on the paired muscles that support the upright body.
Sitting posture seems to prevent the functional scoliosis curve in hyper-mobile women with scoliosis because while sitting the Ischial prominences, seat bones, contact the seat equally & maintain stability of the pelvic girdle platform. These are my own theories & observations.
FUNCTIONAL SCOLIOSIS (discussed above) is partly etiologic for Costochondritis, Gastroparesis, Panic Attacks, & POTS. All these conditions are highly correlated in hypermobile persons. Let me explain my theory. But first one more clinical exam finding about functional scoliosis.
In persons with scoliosis a useful exam is to have person bend over like a Halloween cat & flex their spine. Next, examiner repeatedly raises up & squats down to view horizon of patient’s back. What is often seen is a bump (Adams Sign) on one side of spine where ribs are higher.
Adam Sign often occurs on side of thoracic spine where spine curve is concave or at level where the curve goes from concave to convex, which is often at bottom of the barrel of ribs, at T-12. The concept of a “barrel of ribs” (my term) is key to understanding Chostochondritis.
Thoracic spine, ribs, costochondral junctions, & sternum all form a configuration not unlike a wooden barrel, except that thorax has slats that run horizontal, not vertical like in a wooden barrel. When spine is beset with a functional scoliosis, this skews barrel configuration.
A twisted thoracic spine & skewed Thorax barrel places biomechanical stress on all the costochondral junctions. Cartilage (chondral) is flexible. Ribs (bone) are not flexible. In those with Costochondritis strain in the junctions elicits stress & local inflammation = pain.
Chostochondritis pain is often mistaken, by patients & doctors, for “heart attack”. Countless times I have seen ER work ups fail to show an heart attack. Yet moderate pressure on exact rib-chondral junction exactly replicates pain. Once observed, diagnostic confirmation is easy.
Costochondritis is easily diagnosed by a SPECT Scan, a nuclear medicine scan that shows white cells gathered at the exact junction. Treatment is readily accomplished by easy & safe injection of a small deposit of cortisone into the costochondral junction, just under the skin.
Costochondritis is effectively aborted by the corticosteroid injection, but the underlying bio-mechanical disorder, a functional scoliosis is not. That is information that has been discussed in other of my threadreaders.
Earlier, I noted relationships of functional scoliosis to Gastroparesis, Panic Attacks, and POTS. These are all dysautonomias, and in my next threadreader I will discuss those relationships. These theories & findings are my own original inventions.
Finally, functional scoliosis is highly associated with asymmetric shoulder girdles. This biomechanical disorder begets crossed muscle syndromes in the shoulders & cervical spine: etiologies of other dysautonomias known as Thoracic Outlet Syndrome & Migraine, other threadreaders.
Autonomic nervous system tissues closely tract osseous system. Sympathetic/Parasympathetic tracts run in & beside vertebral spine. Impingements of autonomic nerves arouse pain & dysfunctions, but not nerve cell death. Are you beginning to understand Super-Syndrome Fibromyalgia?

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More from @BadgleyLaurence

8 Dec
SACROILIAC JOINT DISORDER causes people to sit like this. In a threadreader I published yesterday I discussed the reason people choose to sit like this.
These are exact postures chosen by hypermobile patients with unilateral sacroiliac joint disorder & low back pain on that side. One leg bolsters the other so as to hold up the hemi-pelvis on side the SIJ is subluxing; because it hurts to sit on that buttock.
Another posture that attends unilateral sacroiliac joint disorder in hypermobile people is the Trendelenberg posture. This is to sit flexed forward with elbows on both knees. When brought to their attention, many are apologetic, “I know I shouldn’t slouch”.
Read 14 tweets
8 Dec
HYPERMOBILE YES! These are exact postures chosen by hypermobile patients with unilateral sacroiliac joint disorder & low back pain on that side. One leg bolsters the other so as to hold up the hemi-pelvis on side the SIJ is subluxing; because it hurts to sit on that buttock.
Another posture that attends unilateral sacroiliac joint disorder in hypermobile people is the Trendelenberg posture. This is to sit flexed forward with elbows on both knees. When brought to their attention, many are apologetic, “I know I shouldn’t slouch”.
The Trendelenberg posture is in fact the wisdom of the body finding the most comfortable position; in this seated bent over position the femurs leverage each sacroiliac joint into an anatomical neutral station; so joint ligaments are not stretched.
Read 16 tweets
5 Dec
MYTH OF FIBROMYALGIA?
Idea that Fibromyalgia is a myth persists. In 2009, I published (“Practical Pain Management”) announcement of cause. I sent Dr. Tennant, editor, a manuscripts entitled “Fibromyalgia-Finally The Cause”.
Dr. Tennant chose to change title to, “Sacroiliac Joint Disorder”; I was a country doctor without academic credentials. The article continues on-line. Gratefully, Dr. Tennant did not change the article content. He sent me a note, something like, “you will help many”.
I suspect the world medical community continues of one mind that the cause of Fibromyalgia is unknown, & that many doctors continue to question authenticity of such a disorder. Naysayers are simply ignorant.
Read 33 tweets
30 Nov
RESTLESS LEGS: the question becomes, why would the autonomic nervous system evoke involuntary leg movements when people are reclined and drifting into sleep; in those with POTS?
POTS highly associated with connective tissue Hypermobility & manifests as hypotension/tachycardia upon sudden upright posture; as body blood column descends. Perhaps Mitral Valve regurgitation is involved + laxity/sluggishness of autonomic-driven constriction of vein walls.
Blood returns from legs to heart vis superior-directed propulsion of lower blood column by means of leg muscle contractions; restless legs provide such muscle contraction propulsions par excellence.
Read 24 tweets
29 Nov
BEIGHTON CRITERIA VIDEO: is somewhat disappointing because the model could not perform all the demonstrations. Her elbow hyperextensions were great.
The model was older, and when people age they lose their youthful ability to do all the hyperextensions. I would say that some of the degrees of the various hyperextensions degrade starting after 30 years old.
Another factor that must be taken into account is that Hypermobility can present as a mosaic in some persons. Their knees well demonstrate Hypermobility, but not the elbows.
Read 16 tweets
29 Nov
A video for people to test for hypermobility.
An experience I have with patients who have stigmata of hypermobility is that I will comment on their widespread joint Hypermobility & their rejoinder commonly is, “of course I do Yoga”.
When patients tell me that my heart sinks. I was excited to tell them that I think that I have an explanation for several of their seemingly disparate symptoms in several of the dysautonomia & MCAS spheres.
Read 10 tweets

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