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Marion Holman

@holmanm

13 Feb

@TMackeown

1/4 What a great question (thanks @TMackeown ). "Given that most of the deaths/serious effects of Covid-19 are in the elderly, who are also the most likely to have been on statins for some time, do you think there is a connection between the lowering of LDL/ApoB100 and the

2/4 susceptibility to infection - not just from MRSA ?"
If you google Statins/Covid you will find that statins are most efficacious in every way 😂 (We would expect that wouldn't we ?). However if we turn to independent researchers we find the truth.

3/4 See the link below

bmj.com/content/368/bm…
It gets worse because Statins act through the reversible block of (HMG-CoAR), thereby reducing cholesterol synthesis, 7-dehydrocholesterol and vitamin D production.

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Marion Holman

@holmanm

12 Feb

1/6 In 1984 Merck discovered a way to accelerate mechanism based cell alterations with a Statin by giving rats a second cholesterol lowering drug called a “bile resin”. The combination lowered cholesterol better than Lovastatin alone (Grrrreat !).

2/6 It elevated Reductase more than Lovastatin alone, and it made more abnormal-appearing ER than Lovastatin. In case you are wondering ER = endoplasmic reticulum. This drug combination is what reportedly killed the majority of Baycol users.

3/6 The liver has a very high demand for cholesterol to make bile that is made in the liver and then stored in the gall bladder for digestion of fat. The bile is then recycled back to the liver from the intestines for reuse.

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Marion Holman

@holmanm

12 Feb

1/4 Oh how we have been played ! Brown and Goldstein studied the defective genetics of the LDL receptor in people with FH who have extremely high cholesterol. It was for these patients that the FDA gave Dallas an IND (Investigational New Drug) permission to test Lovastatin,

2/4 Brown and Goldstein studied the defective genetics of the LDL receptor in people with FH who have extremely high cholesterol. It was for these patients that the FDA gave Dallas an IND (Investigational New Drug) permission to test Lovastatin, and not the general public.

3/ 4 A compilation of FH is that painful deposits of oxidised cholesterol would improve this condition. Notably, two of these six FH patients with cholesterol levels over 500 didn’t even have CVD !!

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Marion Holman

@holmanm

9 Feb

1/4 What infuriates me about this paper is there is NO overwhelming evidence to support lowering LDL-C to reduce cardiovascular morbidity and mortality. What prompted this paper is the fact that the Pharmaceutical industry had new drugs to sell i.e. PCSK9 inhibitors.

2/4 Far more profitable than Statins, and the reason why they now confess that Statins are toxic, to promote their new and more profitable PCSK9’s. The problem is the new PCSK9’s are no better than Statins. The pharma Industry has known for the past 40+ years the damage

3/4 that Statins do, but have been in denial. Here they are stupid enough to make the admission about Statin Toxicity and its mechanisms. If you look at the Fourier trial for PCSK9’s you will see that more people died in the treatment arm of the trial than in the placebo group.

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Marion Holman

@holmanm

19 Jan

1/23 Thoughts on Covid Vaccine: First of all, up until this point there has never been a successful vaccine for coronaviruses in humans, due to a problem typical of coronavirus vaccine development called antibody dependent enhancement or ADE.

2/ In preliminary animal trials for previous coronavirus vaccines (SARS and MERS, for instance), animals were vaccinated and seemed to exhibit a robust antibody response, but upon exposure to the wild virus, they developed a paradoxical immune enhancement leading to severe

3/ organ inflammation (especially in their lungs), and they died. Paradoxical immune response in coronavirus vaccines has also taken place in human trials, which occurred during testing of the failed RSV vaccines of the 1950s. Alarmingly, there are some statistical indications

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Marion Holman

@holmanm

10 Jan

1/4 Rather than stop their use altogether, the modality of the health profession is to simply change Statins when dangerous symptoms such as muscle pain are reported. Statins are genotoxic & cause cell mutations. It is inevitable that everyone taking Statins long term will

2/4 develop one or more of the direct effects of statin use. Statins block the Mevalonate pathway inside cells. It is from that pathway that molecules called isoprenoids are made from DNA production. Cholesterol is but one of the many isoprenoids blocked by Statins. .

3/4 All of the isoprenoids made inside cells from Mevalonate (a by-product of food) are required for cell development. NONE are expendable. There is a cure for Statin poisoning, it's called MEVALONATE. Cells normally make Mevalonate, but does this normal function return

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