1/ “errors were found in their analysis and a follow-up re-analysis of the data revealed a cumulative incidence of spontaneous abortion of 7–8 times higher than the original author’s calculations, which was statistically higher than the typical average for pregnancy loss….” 
2/ “Concerns were raised years ago regarding the safety of LNPs due to their biodistribution. For example, they were found to disperse to the ovaries in experimental mice [18].”
3/ “Pfizer’s own pharmacokinetic studies of a surrogate vaccine containing ALC0315 and ALC0159 LNPs demonstrated that they dispersed over a 48 hours period to many rat endocrine and immune organs including the ovaries, adrenals, bone marrow, liver and spleen [19].”
4/ “Very little is known about how LNP particle components are metabolized by the human body…. Research must also be conducted into how LNP components and their catabolites are distributed, retained and excreted.”
5/ “Dutch belted rabbits injected with PEG400 were reported to have retinal degeneration and atrophy 5 days post injection [21]…As a follow up to this, two recent case reports published in the USA reveal a possible association with mRNA vaccines and damage to the retina.”
6/ “LNPs are bioactive and the possibility of immunotoxicity has been raised…LNPs can also activate serum complement, resulting in complement activation-related pseudoallergy (CARPA), which can lead to anaphylactic shock [24].”
7/ “A recent pre-print study demonstrated that, when LNPs were injected intradermally into mice, inflammatory, pro-apoptotic, necroptotic and IFN gene pathways were induced, and when these LNPs were administered intranasally, 80% of mice died within 24 h [25].”
8/ “Exposure to PEG can result in the production of anti-PEG immunoglobulin (Ig)M and IgG, which can activate the complement system and result in anaphylaxis [26].”
9/ “While anaphylaxis during pregnancy is typically a rare event, a recent study has reported severe outcomes for infants from mothers with anaphylaxis [30], which should alert us to potential fetal/neonatal outcomes resulting from vaccine-induced maternal anaphylaxis.”
10/ “The main host target receptor for the SARS-CoV-2 spike protein is (ACE2). ACE2 is also expressed within placental tissues [52], and is involved in regulating fetal myocardial growth and lung and brain development [53].”
11/ “A recent study using mice and human umbilical cord blood demonstrated that ligation of recombinant spike protein to ACE2 can activate Nlrp3 inflammasome assembly, resulting in uncontrolled inflammation leading to pyroptotic cell death [59]”
12/“Ropa et al. [60] demonstrated that hematopoietic stem cells from human umbilical cord blood express ACE2 and were adversely affected by spike protein in terms of their ability to proliferate and expand into progenitor cells.”
13/ “Biancatelli et al. [61] recently demonstrated that intratracheal administration of the spike protein S1 subunit induced alveolar inflammation and acute lung injury and altered lung vascular permeability, leading to an ACE2-dependent systemic cytokine storm.”
14/ “Suzuki et al. recently demonstrated that the spike protein S1 subunit (Val-16-Gln-690), but not the ACE2 receptor binding domain (Arg-319-Phe-541), elicited mitogen-activated protein kinase (MEK/ERK) signaling in human pulmonary artery smooth muscle and endothelial cells.”
15/ “Nader et al. [64] for example, found that in addition to ACE2, SARS-CoV-2 can also attach to, invade and damage host cells via αVβ3 integrin adhesion molecules, which are highly expressed on vascular endothelial cells.”
16/ “, αVβ3 integrins are also expressed on platelets and contribute to platelet activation and aggregation [65].”
17/ “spike induced murine peritoneal macrophages to secrete pro-inflammatory cytokines via TLR4. TLR4 is also highly expressed in platelets, and when bacterial lipopolysaccharide binds to TLR4, it can result in thrombocytopenia and the accumulation of platelets in the lungs.”
18/ “when the spike protein S1 subunit was added to platelet-poor plasma, it interacted with and structurally modified plasma proteins β and γ fibrinogen, complement 3 and prothrombin,.. …this may contribute to the hypercoagulation associated with COVID-19.”
19/ “Also found within the RBD of spike proteins is a “toxin-like” epitope that shares homology to snake venom α-bungarotoxin [75], which is a highly specific blocker of nicotinic acetylcholine receptors.
20/ “spike protein can also bind to the b1b2 domain of the neuropilin-1 receptor (NRP-1) [76], which normally interacts with vascular endothelial growth factor-A (VEGF-A) in neurons.”
21/ “The 2nd RBD region of interest potentially allows spike to bind to amyloid-forming heparin-binding proteins, which could lead to accelerated agg. of amyloid proteins within the brain. This supports Classen’s concern that C-19 vaccines could potentially induce prion disease.”
22/ “The third region of interest within the RBD contains seven predicted molecular sites that share similarities to different toxins or virulence factors from 12 different bacterial species, 2 malarial parasites and influenza A [83]”
23/ “..hypothesis that COVID-19-associated multi-system inflammatory syndrome in children (MIS-C) and the cytokine storm observed in adult patients with severe COVID-19 is mediated by spike protein superantigenic activity.
24/ “The superantigen Staphylococcus Enterotoxin B (SEB), associated with Toxic Shock Syndrome, is a biotoxin that causes polyclonal T-cell activation and proliferation, which leads to massive production of pro-inflammatory cytokines.”
26/ “following COVID-19 mRNA vaccination, exosomes expressing spike protein could be detected in plasma up to 4 months post-vaccination, which is concerning because we, and others, have shown that exosomes can be shed in bodily fluids such as colostrum and milk.”
27/ “Dotan et al. also identified 41 immunogenic penta-peptides within the SARS-CoV-2 spike protein that are shared with 27 human proteins related to oogenesis, placentation and/or decidualization, implicating molecular mimicry as a potential contributor to female infertility.”
28/ “In closing, we currently have no data to assess the outcome of maternal COVID-19 vaccination on offspring health, and this may take years to generate.”
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