Lea Alhilali, MD Profile picture
Jun 3, 2022 7 tweets 5 min read Read on X
1/”Tell me where it hurts.” How back pain radiates can tell you where the lesion is—if you know where to look!
A 🧵about how to remember lumbar radicular pain distributions. #medstudenttwitter #medtwitter #radres #FOAMed #FOAMrad #neurorad #Meded #backpain #spine #Neurosurgery Image
2/Let’s start with L1. L1 radiates to the groin. I remember that b/c the number 1 is, well, um…phallic. So the phallic number 1 radiates to the groin. Image
3/Let’s skip to L3 for a second. I remember L3 is to the knee—easy, it rhymes! Image
4/Ok, back to L2. Two is the number between 1 and 3, so the distribution of L2 is between the distributions of L1 and L3—and between the groin and knee is the thigh. L2 radiates to the thigh. It’s not the catchiest way to remember it, but it works. Image
5/L4 radiates to the calf. I remember this bc the number 4 looks like the calf, with the top part of the 4 looking like a bulging gastroc & the bottom part of the four is the rest of the calf connecting to the ankle. Don’t we all wish we had bulging gastrocs like the number 4! Image
6/L5 radiates to the big toe. So I have the little rhyme “Five is to the big guy!” L5 is also foot drop. So I remember big guys are heavy, and heavy gravity = drop. If I hear the history “foot drop,” I never stop looking until I have traced out the entire L5 nerve root. Image
7/Finally, S1 radiates to the side of the foot. I remember this bc both S1 & Side start w/S.
So now you know where in the lumbar spine to look when a patient says the pain radiates down the leg & hopefully remembering the lumbar radicular distributions won’t cause you any pain! Image

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More from @teachplaygrub

Jan 27
1/The hardest thread yet! Are you up for the challenge?

How stroke perfusion imaging works!

Ever wonder why it’s Tmax & not Tmin?

Here’s what to know from @theAJNR SCANtastic!

ajnr.org/content/47/1/28Image
@TheAJNR 2/Perfusion imaging is based on one principle: When you inject CT or MR intravenous contrast, the contrast flows w/blood & so contrast can be a surrogate marker for blood.

This is key, b/c we can track contrast—it changes CT density or MR signal so we can see where it goes. Image
@TheAJNR 3/So if we can track how contrast gets to the tissue (by changes in CT density or MR signal), then we can approximate how BLOOD is getting to the tissue.

And how much blood is getting to the tissue is what perfusion imaging is all about. Image
Read 19 tweets
Jan 19
1/Do you get a Broca’s aphasia trying remember the location of Broca's area?

Does trying to remember inferior frontal gyrus anatomy leave you speechless?

Don't be at a loss for words when it comes to Broca's area

Here’s a 🧵to help you remember the anatomy of this key region! Image
2/Anatomy of the inferior frontal gyrus (IFG) is best seen on the sagittal images, where it looks like the McDonald’s arches.

So, to find this area on MR, I open the sagittal images & scroll until I see the arches. When it comes to this method of finding the IFG, i’m lovin it. Image
3/Inferior frontal gyrus also looks like a sideways 3, if you prefer. This 3 is helpful bc the inferior frontal gyrus has 3 parts—called pars Image
Read 13 tweets
Dec 5, 2025
1/They say form follows function!

Brain MRI anatomy is best understood in terms of both form & function.

Here’s a short thread to help you to remember important functional brain anatomy--so you truly can clinically correlate! Image
2/Let’s start at the top. At the vertex is the superior frontal gyrus. This is easy to remember, bc it’s at the top—and being at the top is superior. It’s like the superior king at the top of the vertex. Image
3/It is also easy to recognize on imaging. It looks like a big thumb pointing straight up out of the brain. I always look for that thumbs up when I am looking for the superior frontal gyrus (SFG) Image
Read 12 tweets
Dec 1, 2025
1/To call it or not to call it? That is the question!

Do you feel a bit wacky & wobbly when it comes to calling normal pressure hydrocephalus on imaging?

You don’t want to overcall it, but you don’t want to miss it either!

Let me help you out w/a thread about imaging in NPH! Image
2/First, you must understand the pathophysiology of “idiopathic” or iNPH.

It was first described in 1965—but, of the original six in the 1965 cohort, 4 were found to have underlying causes for hydrocephalus.

This begs the question—when do you stop looking & call it idiopathic? Image
3/Thus, some don’t believe true idiopathic NPH exists.

After all, it’s a syndrome defined essentially only by response to a treatment w/o ever a placebo-controlled trial.

However, most believe iNPH does exist--but its underlying etiology is controversial. Several theories exist Image
Read 19 tweets
Nov 21, 2025
1/Time to go with the flow!

Hoping no one notices you don’t know the anatomy of internal carotid (ICA)?

Do you say “carotid siphon” & hope no one asks for more detail?

Here’s a thread to help you siphon off some information about ICA anatomy! Image
2/ICA is like a staircase—winding up through important anatomic regions like a staircase winding up to each floor Lobby is the neck.

First floor is skullbase/carotid canal. Next it stops at the cavernous sinus, before finally reaching the rooftop balcony of the intradural space.Image
3/ICA is divided into numbered segments based on landmarks that denote transitions on its way up the floors.

C1 is in the lobby or neck.

You can remember this b/c the number 1 looks elongated & straight like a neck. Image
Read 10 tweets
Nov 4, 2025
1/The 90s called & wants its carotid imaging back!

It’s been 30 years--are you still on NASCET?

Feeling vulnerable about plaque vulnerability?

This month’s @theAJNR SCANtastic has what you need to know about carotid plaque

ajnr.org/content/46/10/…Image
2/Everyone knows the NASCET criteria:

If the patient is symptomatic & the greatest stenosis from the plaque is >70% of the diameter of normal distal lumen, patient will likely benefit from carotid endarterectomy

But that doesn’t mean the remaining patients are just fine! Image
3/Yes, carotid plaques resulting in high-grade stenosis are high risk

But assuming that stenosis is the only mechanism by which a carotid plaque is high risk is like assuming that the only way to kill someone is by strangulation. Image
Read 13 tweets

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