I’m incredibly thankful for that day back in March 2015 when I got an A1c of 6.1 for the second year in a row.
Why?
It led to my finding #lowcarb as a possible means to prevent the onset diabetes…
2/ Shortly after starting, I had immediate family members with health issues be inspired to do likewise.
One of those immediate family members had an A1c of 8.1 at the time. Two were hypertensive, and taking medication for it. And these are just the immediate family members…
3/ off the top of my head, I think there are roughly 2 dozen people between both close friends and family who are sustained lower carb now than they were before For weight loss, or other reasons of health gain.
To be sure, it hasn’t worked for everyone, nor would I expect that.
4/ I also have friends who tried it, didn’t like it, but found other means of reaching their health goals.
As always, I don’t believe any diet is superior to all others.
However, I think most of us agree the Standard American Diet is one of the worst.
5/ With all that said, lower carb dieting has easily been the most successful sustained lifestyle change within my circle of friends and family. And I’ll concede I am especially glad in having been a part of bringing this awareness to them.
6/ While I’m very proud of our research endeavors around #cholesterol, I have a lot of personal appreciation for how finding this unintuitive pathway to reducing my risk of #T2D would ultimately have a ripple effect in improving the health of so many others closest to me. 🙏🙏🙏
• • •
Missing some Tweet in this thread? You can try to
force a refresh
2/ naturally, you’re discussing a topic that’s very near and dear to my research, @NutritionMadeS3. I’m certainly very interested in metabolic health as it relates to lipid profiles — particularly the “triad” of high LDLc, high HDLc, and low TG. (See mdpi.com/2218-1989/12/5…)
3/ But we should agree to recognizing a common problem. Rather than take several tweets in this thread to explain it, let me open with our conversation from last month where I posed this question for you regarding #LMHRs and #ASCVD at several points:
1/3 Okay, here's my daily breakdown for the #IsItSaturatedFat Experiment.
I'm going to consume 1,000 calories for @KerrygoldUSA! 🧈🧈🧈
That's 140g of butter per day!
2/3 However, I'll be likewise supplementing 728 calories from dextrose, thus consuming much of my remaining dietary energy from carbs.
Per the #LEM, I believe this will have a substantial lowering impact on my LDL-C relative to the increasing influence via ⬇️LDLr of butter
3/3 Note I'm performing this experiment in the spirit of both learning and keeping it fun. ☮️
While I'll concede I might have been caught off guard for a moment yesterday, I'm now thankful this can provide us a new opportunity for discovery and furthering the discussion. 🔬🔬🔬
Yes, I'm doing a new N=1 -- and it's going to be a biggie!
My good friend and colleague, @DrNadolsky completed his recent #MakingLMHR experiment concluding the added 2 sticks of butter as the reason for his LDL increase.
2/ He's already conceded he's left out all the context on aiming for #LMHR profile, the relevance of RER, and the #LEM (so no need for people to keep pinging me on the IG video). I've chatted with him privately and we'll leave it at that. 👍
2/ We've had an ongoing debate on how much (or little) #saturated fat consumption is responsible for high #LDL#cholesterol levels we typically see for #LMHRs.
Whereas I (we) believe #LEM to have greater relevance overall.