1/ 🙏 Happy Accident 🧵

I’m incredibly thankful for that day back in March 2015 when I got an A1c of 6.1 for the second year in a row.

Why?

It led to my finding #lowcarb as a possible means to prevent the onset diabetes…
2/ Shortly after starting, I had immediate family members with health issues be inspired to do likewise.

One of those immediate family members had an A1c of 8.1 at the time. Two were hypertensive, and taking medication for it. And these are just the immediate family members…
3/ off the top of my head, I think there are roughly 2 dozen people between both close friends and family who are sustained lower carb now than they were before For weight loss, or other reasons of health gain.

To be sure, it hasn’t worked for everyone, nor would I expect that.
4/ I also have friends who tried it, didn’t like it, but found other means of reaching their health goals.

As always, I don’t believe any diet is superior to all others.

However, I think most of us agree the Standard American Diet is one of the worst.
5/ With all that said, lower carb dieting has easily been the most successful sustained lifestyle change within my circle of friends and family. And I’ll concede I am especially glad in having been a part of bringing this awareness to them.
6/ While I’m very proud of our research endeavors around #cholesterol, I have a lot of personal appreciation for how finding this unintuitive pathway to reducing my risk of #T2D would ultimately have a ripple effect in improving the health of so many others closest to me. 🙏🙏🙏

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More from @realDaveFeldman

Jul 8
2 x 2 poll — answer all four.

1/ looking to a population that has this combination of lipid levels:

⬇️⬇️ very low LDL-C & ApoB
+
⬆️⬆️ very high HDL-C
+
⬇️⬇️ very low triglycerides

… you would expect to see what prevalence of heart disease compared to the general population?
2/ looking to a population that has this combination of lipid levels:

⬇️⬇️ very low LDL-C & ApoB
+
⬇️⬇️ very low HDL-C
+
⬆️⬆️ very high triglycerides

… you would expect to see what prevalence of heart disease compared to the general population?
3/ looking to a population that has this combination of lipid levels:

⬆️⬆️ very high LDL-C & ApoB
+
⬇️⬇️ very low HDL-C
+
⬆️⬆️ very high triglycerides

… you would expect to see what prevalence of heart disease compared to the general population?
Read 5 tweets
Jul 6
1/ Interesting thread via @NutritionMadeS3. I invite everyone to read through his before reading the remaining of mine here below…
2/ naturally, you’re discussing a topic that’s very near and dear to my research, @NutritionMadeS3. I’m certainly very interested in metabolic health as it relates to lipid profiles — particularly the “triad” of high LDLc, high HDLc, and low TG. (See mdpi.com/2218-1989/12/5…)
3/ But we should agree to recognizing a common problem. Rather than take several tweets in this thread to explain it, let me open with our conversation from last month where I posed this question for you regarding #LMHRs and #ASCVD at several points:
Read 9 tweets
Jun 30
1/3 Okay, here's my daily breakdown for the #IsItSaturatedFat Experiment.

I'm going to consume 1,000 calories for @KerrygoldUSA! 🧈🧈🧈

That's 140g of butter per day!
2/3 However, I'll be likewise supplementing 728 calories from dextrose, thus consuming much of my remaining dietary energy from carbs.

Per the #LEM, I believe this will have a substantial lowering impact on my LDL-C relative to the increasing influence via ⬇️LDLr of butter
3/3 Note I'm performing this experiment in the spirit of both learning and keeping it fun. ☮️

While I'll concede I might have been caught off guard for a moment yesterday, I'm now thankful this can provide us a new opportunity for discovery and furthering the discussion. 🔬🔬🔬
Read 4 tweets
Jun 29
1/ 🚨New N=1 Experiment: #IsItSaturatedFat🚨
🙏retweet🙏

Yes, I'm doing a new N=1 -- and it's going to be a biggie!

My good friend and colleague, @DrNadolsky completed his recent #MakingLMHR experiment concluding the added 2 sticks of butter as the reason for his LDL increase. Image
2/ He's already conceded he's left out all the context on aiming for #LMHR profile, the relevance of RER, and the #LEM (so no need for people to keep pinging me on the IG video). I've chatted with him privately and we'll leave it at that. 👍

3/ However -- this actually affords us a huge new opportunity.

@DrNadolsky's claim isn't just his own, it's just about everyone else's outside the low carb community.

Simply stated: high #LDL #cholesterol seen in #LMHRs is predominantly due to high consumption of saturated fat.
Read 6 tweets
Jun 28
1/ I want to give huge props to @DrNadolsky for doing this #MakingLMHR experiment!

Yes, I know some of my followers may be blocked -- so I'm including an image as well.

If you're interested in how this experiment came about -- read on... 🧵 Image
2/ We've had an ongoing debate on how much (or little) #saturated fat consumption is responsible for high #LDL #cholesterol levels we typically see for #LMHRs.

Whereas I (we) believe #LEM to have greater relevance overall.

Which led to the experiment👇
3/ Could @DrNadolsky emulate the #LMHR phenotype while *not* being fat adapted? (or at least, to get his #LDL #cholesterol that high?)

He consumed two sticks of butter a day to test this! 🧈🧈

Yes, the experiment went more toward my prediction, so alas, I won't be flying there.
Read 7 tweets

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