2/Calvarium & sinuses act as important protectors of your intracranial contents, most importantly, your brain. They are like a built in helmet to protect you from linebackers of life
3/The sinuses are actually even better than a helmet. They are like the crumple zone of a car, but for your brain. They can be crushed inwards, absorbing energy and keeping it from impacting your brain
4/The crumple zone of the frontal sinus is like a sandwich. First, the top bread, is the anterior wall. The meat/filling is the sinus itself & sinus drainage pathway. Finally, the bottom bread is the posterior wall. Each of these can be affected by trauma.
5/First is the anterior wall. Usually, a significant trauma is required to break the anterior wall of the frontal sinus, as it forms the frontal bar, supporting the facial buttresses.
6/You can think of an anterior wall fx like a fender bender. The front of the car is damaged, but everything else is intact. Fixing a fender bender is all about cosmetics. Anterior wall fxs are fixed if they are depressed bc having a dent in your forehead is very noticeable.
7/Next is a fx that affects the meat, or the sinus itself. Any medial fx that affects the ability of the sinus to drain into the ethmoids or significantly disrupts the muscosal elements will make the sinus unable to drain and susceptible to mucocele formation.
8/Fxs that obstruct the drainage pathway or mucosal elements are like car accidents that wreck your engine. A car needs to drive & a sinus needs to drain. If they can’t, they are worthless. So a car goes to the junk yard & a sinus gets obliterated. Tx is sinus obliteration.
9/Finally, posterior wall fxs. Posterior wall fxs are dangerous if they disrupt the dura & therefore cause a communication between the brain & the outside—bc this can cause infection. Signs that the dura have been violated are extra-axial blood or pneumocephalus.
10/Posterior wall fxs are like an accident that breaks your windshield. Now all that air is free to flow into the car—like the air from the sinus, potentially containing bacteria, can flow into the intracranial compartment
11/You will never get that seal back. The treatment is cranialization—kind of like turning the car into a convertible.
12/Here is the algorithm for treating frontal sinus fxs.
Anterior wall only = fixation for cosmetic.
Fx affecting sinus drainage = junk the sinus/obliteration.
Fx of the posterior wall w/risk for infxn = take the top off/cranialization.
13/Based on the treatment algorithm, radiology reports must reflect findings that change management.
Depression/comminution of ant wall fx=need for ORIF
Findings of violation of the dura=need for cranialization
Findings of nasofrontal duct obstruction=need for obliteration
14/So here is a summary of the frontal sinus fractures and their management. Hopefully, when it comes to frontal sinus fractures, this tweetorial will help you save face and stay a head of the game!
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Brain MRI anatomy is best understood in terms of both form & function.
Here’s a short thread to help you to remember important functional brain anatomy--so you truly can clinically correlate!
2/Let’s start at the top. At the vertex is the superior frontal gyrus. This is easy to remember, bc it’s at the top—and being at the top is superior. It’s like the superior king at the top of the vertex.
3/It is also easy to recognize on imaging. It looks like a big thumb pointing straight up out of the brain. I always look for that thumbs up when I am looking for the superior frontal gyrus (SFG)
@TheAJNR 2/Everyone knows about the spot sign for intracranial hemorrhage
It’s when arterial contrast is seen within a hematoma on CTA, indicating active
extravasation of contrast into the hematoma.
But what if you want to know before the CTA?
@TheAJNR 3/Turns out there are non-contrast head CT signs that a hematoma may expand that perform similarly to the spot sign—and together can be very accurate.
1/My hardest thread yet! Are you up for the challenge?
How stroke perfusion imaging works!
Ever wonder why it’s Tmax & not Tmin?
Do you not question & let RAPID read the perfusion for you? Not anymore!
2/Perfusion imaging is based on one principle: When you inject CT or MR intravenous contrast, the contrast flows w/blood & so contrast can be a surrogate marker for blood.
This is key, b/c we can track contrast—it changes CT density or MR signal so we can see where it goes.
3/So if we can track how contrast gets to the tissue (by changes in CT density or MR signal), then we can approximate how BLOOD is getting to the tissue.
And how much blood is getting to the tissue is what perfusion imaging is all about.