Lea Alhilali, MD Profile picture
Nov 21, 2022 17 tweets 8 min read Read on X
1/Ready for a throwdown? MMA fights get a lot of attention, but MMA (middle meningeal art) & dural blood supply doesn’t get the attention it deserves.

A #tweetorial on dural vascular #anatomy

#neurosurgery #neurorad #Neurointervention #radres #medtwitter #neurotwitter #meded Image
2/Everyone knows about the blood supply to the brain. Circle of Willis anatomy is king and loved by everyone, while the vascular anatomy of the blood supply to the dura is the poor, wicked step child of vascular anatomy that is often forgotten Image
3/But dural vascular anatomy & supply are important, especially now that MMA embolizations are common for chronic recurrent subdurals. It also important for understanding dural arteriovenous fistulas as well. Image
4/Although we talk about individual vessels feeding the dura, it should actually be thought of more as a vascular network. Anastomoses among the dural vessels are common and plentiful, as is often seen with external carotid networks. Image
5/The largest & most important dural vessel is the middle meningeal artery or MMA. It arises from the internal maxillary artery or IMAX. I remember that b/c Mortal Kombat & other MMA type fighting is commonly shown in IMAX theaters. Image
6/MMA enters at foramen spinosum.

At the skullbase, foramen ovale & spinosum together look like a high heel shoe footprint

Spinosum is the heel of the footprint. I remember this b/c that’s the high heel spike and SPinosum & SPike sound alike. I always look for this footprint Image
7/After spinosum, the MMA takes a sharp, corkscrew-like turn lateral & anterior following the curvature of the middle cranial fossa.

This gives it a very characteristic angiographic appearance—always look for the sharp turn.

I remember that the artery SPINs after SPINosum Image
8/MMA immediately gives off a tiny petrous branch and then splits into anterior (frontal) & posterior (parietal) divisions. I think it looks like an MMA fighter celebrating their victory with their two arms in the air Image
9/Post division is smaller & has branches covering the posterior convexity. Its territory is draped over the back of the calvarium the way MMA fighters drape flags over their backs after winning. So it covers the back of the calvarium like the flag covers the MMA fighter’s back Image
10/Ant division is larger & has branches that anastomose to the contralateral MMA. You can remember this b/c opposing MMA fighters touch gloves before the fight, and gloves are out in front. So ant division touches the opposite side like opponents touching gloves before a fight Image
11/Anterior division passes under the pterion, a junction of four calvarial bones. This renders it vulnerable to trauma & resulting epidural hemorrhage. This is easy to remember—the forward facing or anterior part of an MMA fighter (his face) is very vulnerable to injury Image
12/Posterior meningeal artery is much smaller than the MMA. It arises from the ascending pharyngeal artery and supplies the dura to the posterior fossa. It also has anastomoses with the posterior division of the MMA. Image
13/You can remember its origin bc TONSILS are in the PHARYNX, so the ascending PHARYNGEAL supplies the dural around the cerebellar TONSILS (posterior fossa) Image
14/Anterior meningeal artery is also much smaller than the MMA. It arises from both the anterior and posterior ethmoidal arteries. It supplies the dura of the anterior cranial fossa. It has many anastomoses with the frontal branches of the MMA Image
15/You can remember its origin bc the anterior meningeal artery supplies the dura overlying the ethmoids, so it would make sense it arises from the ethmoidal arteries Image
16/Uniquely, as it ascends, the anterior meningeal artery actually runs in the wall of the anterior superior sagittal sinus. It is the only named artery to run in the wall of a sinus. Image
17/Now you know the anatomy of the major arterial supply to the dura & their territories. So the next time someone questions you about dural blood supply, you can attack it MMA style! Image

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More from @teachplaygrub

Aug 19
1/Do questions about brainstem anatomy cause you to suddenly get a case of locked in syndrome?!

Do you try to localize the lesion or just wait for the MR?

Wait no more!

Here’s a thread about the brainstem Rule of Four to help you localize brainstem lesions! Image
2/The hallmark of a brainstem lesion/syndrome is:

(1) Ipsilateral cranial nerve deficit

(2) Contralateral body deficit (be it weakness, sensory loss, or ataxia) Image
3/You can remember this because often your head has certain feelings that opposite the feelings in your heart/body.

Similarly, the cranial nerve deficit can be the opposite of the body deficit

This split between head and body is key for recognizing brainstem syndromes Image
Read 12 tweets
Aug 16
1/Is your understanding of medial temporal anatomy, well, temporary?

If only there was a way to make hippocampal anatomy memorable!

Here is a thread of the basics of hippocampal anatomy that will hopefully stay in your hippocampus! Image
2/Its name “hippocampus” comes from its shape on gross anatomy.

Early anatomists thought it looked like an upside down seahorse—w/its curved tail resembling the tail of a seahorse.

Hippocampus literally means seahorse. Image
3/In cross section, it has a spiral appearance, leading to its other name, Cornu Ammonis, translated Ammon’s Horn.

Ammon was an Egyptian god w/spiraling rams horns.

The hippocampal subfields are abbreviated CA-1, CA-2, etc, w/CA standing for “Cornu Ammonis” Image
Read 17 tweets
Aug 9
1/Tired of stressing if a brain tumor is progressing?

Wish you had some insurance about calling tumor recurrence?

Here’s the cheat sheet you NEED for the best signs of tumor progression! Image
2/Just when treatment thinks it’s got tumor trapped at cliff, tumor is able to get away

Think how you would get away if you were chased to a cliff’s edge.

These are same signs of tumor progression! Image
3/Here's how both you and the tumor can escape:

1. Jump off into the water:
Tumor heads to the water—the ventricular surface

Subependmyal enhancement is very specific for tumor progression (93% sensitivity), but it isn’t commonly seen (38% sensitive). Image
Read 8 tweets
Aug 7
1/Tired of always speculating about MR spectroscopy?

If you've ever looked at an MR spectroscopy & thought: "I have no idea what I’m looking at!"--then this cheat sheet is for you!

Here's a thread on the 4 basic rules you need to understand the spectrum of basic spectroscopy! Image
2/First you need to know the peaks.

There are 3 main peaks: Choline, Creatine, NAA

Remember the order bc a spectrum looks like mountain peaks & it is cold in the mountains.

And CHOld CREATures NAp or hibernate in the mountains Image
3/First peak is Choline

It's a marker of membrane turnover

You can remember this because membranes coat or “CHOat” the cell Image
Read 11 tweets
Aug 2
1/Wish that your knowledge of autoimmune encephalitis was automatic?

Do you feel in limbo when it comes to the causes of limbic encephalitis?

Do you know the patterns of autoimmune encephalitis?

Here’s a thread with some hints to help you figure it all out! Image
2/Two pearls:
(1) Most common pattern is limbic encephalitis
(2) Small cell can cause any autoimmune pattern.

You can also remember the causes by the demographic:
🔸Young man: testicular
🔸Older: Small cell
🔸Woman with psychiatric symptoms: breast Image
3/Limbic encephalitis is the most common pattern

But it has many, many different causes

Remember--limbic involvement is shaped like a question mark!

So for limbic encephalitis, the cause remains a question bc the differential is so broad

Must question & clinically correlate! Image
Read 7 tweets
Jul 23
1/To call it or not to call it? That is the question!

Do you feel a bit wacky & wobbly when it comes to calling normal pressure hydrocephalus on imaging?

You don’t want to overcall it, but you don’t want to miss it either!

Let me help you out w/a thread about imaging in NPH! Image
2/First, you must understand the pathophysiology of “idiopathic” or iNPH.

It was first described in 1965—but, of the original six in the 1965 cohort, 4 were found to have underlying causes for hydrocephalus.

This begs the question—when do you stop looking & call it idiopathic? Image
3/Thus, some don’t believe true idiopathic NPH exists.

After all, it’s a syndrome defined essentially only by response to a treatment w/o ever a placebo-controlled trial.

However, most believe iNPH does exist--but underlying etiology is controversial. Several theories exist Image
Read 19 tweets

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