2/First some anatomy. Palatine tonsils (or faucial to the cool kids) sit in the oropharynx between the two palatine arches: the palatoglossus arch in front and the palatopharyngeus arch in back. These are easily visible on physical exam.
3/These archs are actually just mucosa draped over the palatoglossus and palatopharygeus musculature, like kids drape sheets over themselves to dress up for Halloween.
4/The palatine tonsils sit nestled in between these two arches in a space called the tonsillar fossa. The pillars are like the bed and blankets--and the tonsils are tucked in between
5/Tonsils are made up triangular folds w/crevices in between, called crypts. This anatomy increases tonsillar surface area to expose it to as many of the oropharyngeal antigens as possible. Just below the surface are many lymph node germinal centers to examine the antigens
6/The lymphatic channels from these germinal centers are valveless (in adults—I don’t do kids 😉). This allows for immediate transport of antigens. This makes sense, as you want to be aware of any bad antigen entering your oropharynx as soon as possible
7/Tonsillitis occurs when there is an infection of the tonsils, usually strep pneumo. Inflammatory debris is made in the crypts and excreted out, creating the white patches seen on physical exam
8/On CT, this inflammatory change causes enlargement of the tonsils and hyper-enhancement of the crypts. This results in the classic tiger-stripe appearance of tonsillitis.
9/An abscess occurs when one of these crypts gets obstructed and its inflammatory exudate turns into pus under pressure.
10/But the pus doesn’t stay in the tonsil. It’s under pressure, like a volcano. If it’s plugged, the lava will find a way out b/c of the pressure. Lava will flow out any cracks/pores in the rock. In the tonsil, pores are the valveless lymphatics that allow the pus to flow out
11/Trying to keep the pus in the tonsil is like trying to keep water in a bathtub when the drain is open. It will always pour out. Similarly, in adults, the pus never stays in the tonsil—it pours out the valveless lymphatics into the tonsillar fossa/peritonsillar space.
12/Once the pus is in the tonsillar fossa, it becomes a peritonsillar abscess. It does not have to go through the superior constrictor musculature to be considered a peritonsillar abscess
13/So, in adults, the answer to the question “Tonsillar or peritonsillar abscess?” is the same answer my kid knows to give when asked, “Which parent do you love the most?” The answer: both!
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1/ I always say, "Anyone can see the bright spot on diffusion images—what sets you apart is if you can tell them why it’s there!”
If you don't why a stroke happened, you can't prevent the next one!
Can YOU tell a stroke’s etiology from an MRI?
Here’s a thread to show you how!
2/First a review of the vascular territories.
I think the vascular territories look a butterfly—w/the ACA as the head/body, PCA as the butt/tail, and MCA territories spreading out like a butterfly wings.
3/Of course, it’s more complicated than that.
Medially, there are also small vessel territories—the lenticulostriates & anterior choroidal.
I think they look like little legs, coming out from between the ACA body & PCA tail.
1/Asking “How old are you?” can be dicey—both in real life & on MRI!
Do you know how to tell the age of blood on MRI?
Here’s a thread on how to date blood on MRI!
After reading this, when you see a hemorrhage, your guess on its age will always be in the right vein!
2/If you ask someone how to date blood on MRI, they’ll spit out a crazy mnemonic about babies that tells you what signal blood should be on T1 & T2 imaging by age.
But mnemonics are crutch—they help you memorize, but not understand
If you understand, you don’t need to memorize
3/If you look at the mnemonic, you will notice one thing—the T1 signal is all you need to tell if blood is acute, subacute or chronic.
T2 signal will tell if it is early or late in each of those time periods—but that type of detail isn’t needed in real life
@TheAJNR 2/Since the prehistoric days of medicine (1979!), we knew that some brain tumor patients treated w/radiation (XRT) initially declined, but then get better.
Today, we see this on imaging, where it looks worse early, but then gets better.
Now we call this pseudoprogression.
@TheAJNR 3/Why does this happen?
XRT induces a lot of inflammatory changes—from initiating the complement cascade to opening the blood brain barrier (BBB)
It’s these inflammatory changes that make the imaging look worse.