Lea Alhilali, MD Profile picture
Dec 7, 2022 18 tweets 9 min read Read on X
1/Have disagreements between radiologists on the degree of cervical canal stenosis become a pain in the neck?!
Here’s a #tweetorial on cervical stenosis grading that’s easy, reproducible & evidence based
#medtwitter #spine #neurosurgery #radres #neurorad #meded #FOAMed #FOAMrad Image
2/In the lumbar spine, it is all about the degree of canal narrowing & room for nerve roots. In the cervical spine, we have another factor to think about—the cord. Cord integrity is key. No matter the degree of stenosis, if the cord isn’t happy, the patient won’t be either Image
3/Cord flattening, even w/o canal stenosis, can cause myelopathy. No one is quite sure why. Some say it’s b/c mass effect on static imaging may be much worse in dynamic positions, some say it’s repetitive microtrauma, & some say micro-ischemia from compression of perforators Image
4/Let’s start w/canal stenosis. Measurements have been proposed (<10mm), but this is cumbersome & introduces reader variability. Think functionally. Cord swims in CSF, like a fish in water. Like a fish, it needs room to swim. How much room is in the fish bowl determines stenosis Image
5/Mild stenosis is when your fish bowl decorations take up 1 side of the bowl. Not great, but fish can still swim. Moderate means your decorations take up both sides—swimming is really affected. Severe means you went all out w/decorations & there isn’t any more room for the fish Image
6/The sides of the fish bowl are the ventral & dorsal CSF. So mild canal stenosis is when either the ventral or dorsal CSF is effaced, but the other side of the fish bowl is still empty. Not ideal, but the fish can still swim Image
7/For moderate canal stenosis, both sides of the fish bowl have been filled. So both the ventral and dorsal CSF have been effaced. Now the room to swim has been notably limited Image
8/Finally, in severe canal stenosis, the bowl is completely filled and no CSF is seen. There is no room for the fish in this scenario. Similarly, there is no room for the cord and it is compressed. Not only is there no swimming, the fish has been crushed. Image
9/This classification is to all other classifications like a goldfish is to all other pets—super easy & simple. It’s also evidence based. It's the Muhle classification. It has excellent reproducibility. It hasn’t been correlated w/pain, but it's been correlated w/SSEP & outcomes Image
10/But canal stenosis isn’t enough. Cord flattening can cause myelopathy regardless of degree canal stenosis. It’s like being punched in the face—no matter how far away the hit comes from, it still hurts. Cord flattening is like being punched—it hurts even in mild stenosis Image
11/Think of the canal like a parking space. Even if no one encroaches on your space, if someone opens their door & dings your car, your car is still damaged and you are still mad. Your parking space may still be wide open, but you still have a nick in your door. Image
12/Cord flattening has 3 degrees. Either it’s not there, there, or so bad it causes cord damage. Think of it like a fight. Cord deformity w/o signal is like someone pushing you to start a fight---you can still walk away. Cord deformity w/signal is a punch to the face—it’s on! Image
13/Here are examples:

Cord deformity w/o signal (Grade 2, someone pushing and trying to start something)

Deformity w/cord signal (Grade 3, fight has already started & the cord already has a black eye!) Image
14/Remember, this is independent of the degree of canal stenosis. You can have cord deformity and signal even in lesser degrees of canal stenosis. Remember--cord flattening can cause cord damage regardless of the degree of canal stenosis. Image
15/This is the Kang system, and it was created to bring the idea of cord flattening into the rating of cervical spine stenosis, since flattening/deformity contribute to myelopathy regardless of stenosis. Image
16/Why don’t we just use the Kang & forget Muhle? Well, the problem w/Kang is that if there’s no cord signal, many degrees of canal stenosis are equal. Here, both mild stenosis w/flattening & severe stenosis w/flattening are equal in Kang, but clearly one is much more at risk Image
17/So we use both. For every level, we rate the degree of canal stenosis according to Muhle & the degree of cord flattening according to Kang. Remember—there is no perfect classification system. Sometimes you need combine. Image
18/So remember both canal and cord matter in the cervical region! Degree of stenosis is important, but even w/o it, cord flattening can have you swimming w/the fishes. So hopefully, you will take to these rating systems like a fish to water! Image

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Jul 10
1/Nothing strikes fear into the heart of a radiologist like the question,“Is it safe to do an MRI on this pt w/an implanted device?”

Do questions about pacemakers & MRIs send your heart racing?

Never fear again! Here’s a thread on how to navigate implanted devices & MRI! Image
2/MRI & CT are like nuclear & coal power, respectively. Everyone knows CT is worse for you & usually MRI is very safe & better for your body

But like nuclear power, when things go bad in MRI, they can go very wrong. Flying chairs into the magnet wrong. So, people are afraid. Image
3/The trouble is from the magnetic attractive forces. There are 3 ways these attractions can wreak havoc. First is translation.

Magnet literally pulls an object, like a chair, towards itself. This is the strongest attraction—like two lovers who literally can’t stay apart. Image
Read 19 tweets
Jun 29
1/I always say you can tell a bad read on a spine MR if it doesn’t talk about lateral recesses.

What will I think when I see your read? Do you rate lateral recess stenosis?

Here’s a thread on lateral recess anatomy & a grading system for lateral recess stenosis Image
2/First anatomy.

Thecal sac is like a highway, carrying the nerve roots down the lumbar spine.

Lateral recess is part of the lateral lumbar canal, which is essentially the exit for spinal nerve roots to get off the thecal sac highway & head out into the rest of the body Image
3/Exits have 3 main parts.

First is the deceleration lane, where the car slows down as it starts the process of exiting.

Then there is the off ramp itself, and this leads into the service road which takes the car to the roads that it needs to get to its destination Image
Read 21 tweets
Jun 26
1/Time is brain! But what time is it?

If you don’t know the time of stroke onset, are you able to deduce it from imaging?

Here’s a thread to help you date a stroke on MRI! Image
2/Strokes evolve, or grow old, the same way people evolve or grow old

The appearance of stroke on imaging mirrors the life stages of a person—you just have to change days for a stroke into years for a person

So 15 day old stroke has features of a 15 year old person, etc. Image
3/Initially (less than 4-6 hrs), the only finding is restriction (brightness) on diffusion imaging (DWI)

You can remember this bc in the first few months, a baby does nothing but be swaddled or restricted

So early/newly born stroke is like a baby, only restricted Image
Read 10 tweets
Jun 1
1/Having trouble remembering how to differentiate dementias on imaging?

Is looking at dementia PET scans one of your PET peeves?

Here’s a thread to show you how to remember the imaging findings in dementia & never forget! Image
2/The most common functional imaging used in dementia is FDG PET. And the most common dementia is Alzheimer’s disease (AD).

On PET, AD demonstrates a typical Nike swoosh pattern—with decreased metabolism in the parietal & temporal regions Image
3/The swoosh rapidly tapers anteriorly—& so does hypometabolism in AD in the temporal lobe. It usually spares the anterior temporal poles.

So in AD look for a rapidly tapering Nike swoosh, w/hypometabolism in the parietal/temporal regions—sparing the anterior temporal pole Image
Read 16 tweets
May 1
1/Do radiologists sound like they are speaking a different language when they talk about MRI?

T1 shortening what? T2 prolongation who?

Here’s a translation w/an introductory thread to MRI. Image
2/Let’s start w/T1—it is #1 after all! T1 is for anatomy

Since it’s anatomic, brain structures will reflect the same color as real life

So gray matter is gray on T1 & white matter is white on T1

So if you see an image where gray is gray & white is white—you know it’s a T1 Image
3/T1 is also for contrast

Contrast material helps us to see masses

Contrast can’t get into normal brain & spine bc of the blood brain barrier—but masses don’t have a blood brain barrier, so when you give contrast, masses will take it up & light up, making them easier to see. Image
Read 20 tweets
Apr 30
1/Does your ability to remember temporal lobe anatomy seem, well, temporary?

Or are you feeling temporally challenged when it comes to this complex region?

Here’s a thread to help you remember the structures of the temporal lobe! Image
2/Temporal lobe can be divided centrally & peripherally.

Centrally is the hippocampus.

It’s a very old part of the brain & is relatively well preserved going all the way back to rats.

Its main function is memory—getting both rats & us through mazes—including the maze of life Image
3/Peripherally is the neocortex.

Although rats also have neocortex, theirs is much different structurally than humans.

So I like to think of neocortex as providing the newer (neo) functions of the temporal lobes seen in humans: speech, language, visual processing/social cues Image
Read 12 tweets

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