2/In the lumbar spine, it is all about the degree of canal narrowing & room for nerve roots. In the cervical spine, we have another factor to think about—the cord. Cord integrity is key. No matter the degree of stenosis, if the cord isn’t happy, the patient won’t be either
3/Cord flattening, even w/o canal stenosis, can cause myelopathy. No one is quite sure why. Some say it’s b/c mass effect on static imaging may be much worse in dynamic positions, some say it’s repetitive microtrauma, & some say micro-ischemia from compression of perforators
4/Let’s start w/canal stenosis. Measurements have been proposed (<10mm), but this is cumbersome & introduces reader variability. Think functionally. Cord swims in CSF, like a fish in water. Like a fish, it needs room to swim. How much room is in the fish bowl determines stenosis
5/Mild stenosis is when your fish bowl decorations take up 1 side of the bowl. Not great, but fish can still swim. Moderate means your decorations take up both sides—swimming is really affected. Severe means you went all out w/decorations & there isn’t any more room for the fish
6/The sides of the fish bowl are the ventral & dorsal CSF. So mild canal stenosis is when either the ventral or dorsal CSF is effaced, but the other side of the fish bowl is still empty. Not ideal, but the fish can still swim
7/For moderate canal stenosis, both sides of the fish bowl have been filled. So both the ventral and dorsal CSF have been effaced. Now the room to swim has been notably limited
8/Finally, in severe canal stenosis, the bowl is completely filled and no CSF is seen. There is no room for the fish in this scenario. Similarly, there is no room for the cord and it is compressed. Not only is there no swimming, the fish has been crushed.
9/This classification is to all other classifications like a goldfish is to all other pets—super easy & simple. It’s also evidence based. It's the Muhle classification. It has excellent reproducibility. It hasn’t been correlated w/pain, but it's been correlated w/SSEP & outcomes
10/But canal stenosis isn’t enough. Cord flattening can cause myelopathy regardless of degree canal stenosis. It’s like being punched in the face—no matter how far away the hit comes from, it still hurts. Cord flattening is like being punched—it hurts even in mild stenosis
11/Think of the canal like a parking space. Even if no one encroaches on your space, if someone opens their door & dings your car, your car is still damaged and you are still mad. Your parking space may still be wide open, but you still have a nick in your door.
12/Cord flattening has 3 degrees. Either it’s not there, there, or so bad it causes cord damage. Think of it like a fight. Cord deformity w/o signal is like someone pushing you to start a fight---you can still walk away. Cord deformity w/signal is a punch to the face—it’s on!
13/Here are examples:
Cord deformity w/o signal (Grade 2, someone pushing and trying to start something)
Deformity w/cord signal (Grade 3, fight has already started & the cord already has a black eye!)
14/Remember, this is independent of the degree of canal stenosis. You can have cord deformity and signal even in lesser degrees of canal stenosis. Remember--cord flattening can cause cord damage regardless of the degree of canal stenosis.
15/This is the Kang system, and it was created to bring the idea of cord flattening into the rating of cervical spine stenosis, since flattening/deformity contribute to myelopathy regardless of stenosis.
16/Why don’t we just use the Kang & forget Muhle? Well, the problem w/Kang is that if there’s no cord signal, many degrees of canal stenosis are equal. Here, both mild stenosis w/flattening & severe stenosis w/flattening are equal in Kang, but clearly one is much more at risk
17/So we use both. For every level, we rate the degree of canal stenosis according to Muhle & the degree of cord flattening according to Kang. Remember—there is no perfect classification system. Sometimes you need combine.
18/So remember both canal and cord matter in the cervical region! Degree of stenosis is important, but even w/o it, cord flattening can have you swimming w/the fishes. So hopefully, you will take to these rating systems like a fish to water!
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MMA fights get a lot of attention, but MMA (middle meningeal art) & dural blood supply doesn’t get the attention it deserves.
A thread on dural vascular anatomy!
2/Everyone knows about the blood supply to the brain.
Circle of Willis anatomy is king and loved by everyone, while the vascular anatomy of the blood supply to the dura is the poor, wicked step child of vascular anatomy that is often forgotten
3/But dural vascular anatomy & supply are important, especially now that MMA embolizations are commonly for chronic recurrent subdurals.
It also important for understanding dural arteriovenous fistulas as well.
2/Aneurysm rupture is a devastating even, as it results in subarachnoid hemorrhage & complications such as hydrocephalus, vasospasm, infarcts, & death.
Preventing it by treating aneurysms before they rupture is key. But you also don’t want to overtreat.
3/To remember what features make an aneurysm more likely to rupture, think what makes that guy at the bar that you angered more likely to rupture & start a fight.
What makes him more likely to rupture are the same things that make aneurysms more likely to rupture
1/Need help reading spine imaging? I’ve got your back!
It’s as easy as ABC!
A thread about an easy mnemonic you can use on every single spine study you see to increase your speed & make sure you never miss a thing!
2/A is for alignment
Look for: (1) Unstable injuries
(2) Malalignment that causes early degenerative change. Abnormal motion causes spinal elements to abnormally move against each other, like grinding teeth wears down teeth—this wears down the spine
3/B is for bones.
On CT, the most important thing to look for w/bones is fractures. You may see focal bony lesions, but you may not
On MR, it is the opposite—you can see marrow lesions easily but you may or may not see edema associated w/fractures if the fracture is subtle