Lea Alhilali, MD Profile picture
Dec 12, 2022 20 tweets 10 min read Read on X
1/”I LOVE spinal cord syndromes!” is a phrase that has NEVER, EVER been said by anyone.
Never fear—here is a #tweetorial on all the incomplete #spinalcord syndromes!
#medtwitter #neurotwitter #neurology #neurosurgery #neurorad #radres #meded #FOAMed #FOAMrad #radtwitter #spine
2/Spinal cord anatomy can be complex. On imaging, we can see the ant & post nerve roots. We can also see the gray & white matter. Hidden w/in the white matter, however, are numerous efferent & afferent tracts—enough to make your head spin.
3/Lucky for you, for the incomplete cord syndromes, all you need to know is gray matter & 3 main tracts. Anterolaterally, spinothalamic tract (pain & temp). Posteriorly, dorsal columns (vibration, proprioception, & light touch), & next to it, corticospinal tracts—providing motor
4/I think the layout looks like a guy doing jumping jacks. Gray matter centrally is the head. Spinothalamic tracts coming forward & anterior are the arms, while posteriorly, corticospinal tracts are the legs. Finally, all the way back, at the bottom, dorsal columns are the feet
5/This way of remembering spinal anatomy makes sense. The gray matter should be the head—because where most of all your gray matter stored? In your head! So gray matter is the head of the jumping jacks guy.
6/Extremities are the sensory tracts—makes sense, most sensation comes from the extremities. Arms/hands give you the most sensation (pain, burning=spinothalamic tract). Feet can feel big things, but mostly, they are there to help you balance (mainly proprioception=dorsal columns)
7/Corticospinal tracts look like the legs, and this makes sense—legs are what propel you forward and move you. So the legs are for motion = motor = corticospinal tract
8/An important point: The spinothalamic tract actually crosses in the gray matter to supply the OPPOSITE side.

So it does a fancy jumping jack. After tracts come together in the middle, they switch sides—like someone turning around 180 when they do jumping jacks
9/First and most important cord syndrome is anterior or ventral cord syndrome. On imaging, you get a classic “owl eye” appearance.

I remember this is anterior cord syndrome bc someone w/owl eyes would be your lookout—and look outs are always in front or ANTERIOR.
10/Anterior cord syndrome is usually from anterior spinal artery occlusion, which supplies the anterior 2/3rds of the cord.

Usually it is from embolism/atherosclerosis, but can also be from occlusion of the feeder of the ASA that come from the aorta (aortic dissection/surgery)
11/In anterior cord syndrome, you’ve lost 2rds of your body! Only the feet remain (dorsal columns).

What do feet do? Mainly feel the ground (proprioception, vibration) and a little sensation (light touch). That is all that is left w/anterior cord syndrome.
12/Central cord syndrome is usually from trauma. Either true trauma or chronic trauma related to compression of the cord from degenerative changes/canal stenosis

(see my tweetorial on degenerative compression here: )
13/What is effected? Getting hit centrally is like getting punched centrally—in the stomach. Immediately your arms cross to protect your stomach & they get hit.

This is what happens in central cord syndrome, the crossing tracts of the arms (spinothalamic tracts) get hit.
14/Hitting the crossing spinothalamic tracts gives you the classic cape distribution of pain in central cord syndrome.

This is easy to remember bc the spinothalamic tracts are the arms/hands—so it makes sense that if they get hit, you have pain along both arms/hands!
15/Dorsal cord syndrome is where only the dorsal columns are involved. It looks like a little white mountain on imaging. Differential is extensive—and contains some real zebras.

I just remember that if I see a mountain in the cord, there is a mountain of differentials!
16/Going back to our jumping jack guy—dorsal cord syndrome is having your feet cut off. You can still walk if you want—you have legs that you can fit into peg legs. But w/o feet to feel the ground, you will never quite have the same balance (loss of proprioception)
17/Finally is hemi cord syndrome or Brown Sequard to the cool kids. Half of the cord is injured—usually from a penetrating trauma (knife, gunshot) that severs the cord in half.
18/Now it gets complicated--this is the only unilateral syndrome. You have lost both your leg & your foot on that side. So you can’t move your leg on that side—it’s gone. Not even a stump to put a peg leg on. And w/o your foot, it is hard to feel the ground & balance on that side
19/But you lose your PAIN sensation on the OPPOSITE side, b/c the spinothalamic tracts does its own 180 jumping jack & actually innervates the other side.

I remember this bc Spino Thalamic = Slip Through to the other side.
20/Now you know all the incomplete spinal cord syndromes and the jumping jack man anatomy behind them. So while you may never love them—at least you will always remember them!

• • •

Missing some Tweet in this thread? You can try to force a refresh
 

Keep Current with Lea Alhilali, MD

Lea Alhilali, MD Profile picture

Stay in touch and get notified when new unrolls are available from this author!

Read all threads

This Thread may be Removed Anytime!

PDF

Twitter may remove this content at anytime! Save it as PDF for later use!

Try unrolling a thread yourself!

how to unroll video
  1. Follow @ThreadReaderApp to mention us!

  2. From a Twitter thread mention us with a keyword "unroll"
@threadreaderapp unroll

Practice here first or read more on our help page!

More from @teachplaygrub

Jun 10
1/Do you know all the aspects of, well, ASPECTS?

Many know the anterior circulation stroke system—but posterior circulation (pc) ASPECTS is often left behind

25% of infarcts are posterior circulation

Do you know pc-ASPECTS?!

Here’s a thread to help you remember pc-ASPECTS Image
2/Many know anterior circulation ASPECTS.

It uses a 10-point scoring system to semi-quantitation the amount of the MCA territory infarcted on non-contrast head CT

If you need a review: here’s my thread on ASPECTS:
Image
3/But it’s only useful for the anterior circulation.

Posterior circulation accounts for ~25% of infarcts

Even w/recanalization, many of these pts do poorly bc of the extent of already infarcted tissue

So there’s a need to quantitate the amount of infarcted tissue in these pts Image
Read 8 tweets
May 29
1/Waving the white flag when it comes to white matter anatomy?

Turns out white matter anatomy isn’t black & white!

This months @theAJNR SCANtastic is the white knight you need to rescue you!

Here’s the white matter anatomy you NEED to know!

ajnr.org/content/45/5/5…
Image
2/Gray matter or cortical functional anatomy is well known.

Everyone knows the motor & sensory strips. Most know Broca’s & Wernicke’s

But most forget that white matter has similar functional topography & just bc it’s white matter doesn’t mean it doesn’t have function! Image
3/But too often we don’t refer to this white matter functional anatomy.

Instead we use general terms like “corona radiata”

But that’s the equivalent of using the word “body.”

Just like the body has many different systems in it, so does the corona radiata! Image
Read 12 tweets
May 21
1/Having trouble remembering what you should look for in vascular dementia on imaging?

Almost everyone worked up for dementia has infarcts. Which ones are important?

Here’s a thread on the key findings in vascular dementia! Image
2/Vascular cognitive impairment, or its most serious form, vascular dementia, used to be called multi-infarct dementia.

It was thought dementia directly resulted from brain volume loss from infarcts, w/the thought that 50-100cc of infarcted related volume loss caused dementia Image
3/But that’s now outdated. We now know vascular dementia results from diverse pathologies that all share a common vascular origin.

It’s possible to lose little volume from infarct & still result in dementia.

So if infarcts are common—which contribute to vascular dementia? Image
Read 21 tweets
May 20
1/Correlate clinically!

It’s harder than you think in THALAMUS—where its size is small & but the clinical symptoms are large.

Here’s a thread to help you remember the main thalamic syndromes & their locations! Image
2/Thalamus is a dense network of nuclei & tracts connected to almost everything in the brain. So almost any symptom can be correlated to it.

So saying “thalamus” as the answer when asked where a lesion is located is always reasonable—even w/o knowing what the symptoms are! Image
3/Think of the thalamus like the internet service provider or ISP for the brain.

Like an ISP, everywhere is connected through it. And like an ISP, things go bad when it goes down.

But just like an ISP, the problems created depend on where in the network the outage is located. Image
Read 23 tweets
May 17
1/ I always say, "Anyone can see the bright spot on diffusion images—what sets you apart is if you can tell them why it’s there!”

If you don't why a stroke happened, you can't prevent the next one!

Can YOU tell a stroke’s etiology from an MRI?

Here’s a thread to show you how! Image
2/First a review of the vascular territories.

I think the vascular territories look a butterfly—w/the ACA as the head/body, PCA as the butt/tail, and MCA territories spreading out like a butterfly wings. Image
3/Of course, it’s more complicated than that.

Medially, there are also small vessel territories—the lenticulostriates & anterior choroidal.

I think they look like little legs, coming out from between the ACA body & PCA tail. Image
Read 25 tweets
May 14
1/Got the diagnosis when it comes to vessel stenosis?

Or is your knowledge narrow when it comes to vessel narrowing?

When it comes to vasospasm, do you know why it happens or what to look for?

Here is the thread you NEED to unravel why vessels twist up! Image
2/Vasospasm results from subarachnoid hemorrhage (SAH) & a buildup of multiple factors

It’s like how you can handle 1 nag from your boss on Monday—but after nagging all week, you break down on Friday!

Same w/vasospasm—it doesn’t happen until the end of the week after SAH! Image
3/So what is nagging that causes the vessel to shut down?

When the body breaks down blood from SAH, it releases free heme

And this free heme causes a cascade of negative consequences, call heme-related inflammation

So free heme is the annoying boss! Image
Read 21 tweets

Did Thread Reader help you today?

Support us! We are indie developers!


This site is made by just two indie developers on a laptop doing marketing, support and development! Read more about the story.

Become a Premium Member ($3/month or $30/year) and get exclusive features!

Become Premium

Don't want to be a Premium member but still want to support us?

Make a small donation by buying us coffee ($5) or help with server cost ($10)

Donate via Paypal

Or Donate anonymously using crypto!

Ethereum

0xfe58350B80634f60Fa6Dc149a72b4DFbc17D341E copy

Bitcoin

3ATGMxNzCUFzxpMCHL5sWSt4DVtS8UqXpi copy

Thank you for your support!

Follow Us!

:(