Viral Persistence In The Central Nervous System Reservoir. Depletion of CD4 cells. Sound familiar? It should. I am talking about HIV Associated Neurocognitive Decline and folk here and yonder have turned a blind eye to how it is prevented.
Additionally, I participated in a podcast discussion. You can listen here sandscan.me/sand.mp3.
We are ignoring the criticality of rapid initiation of antivirals, with the time being within one year of the Estimated Date of Seroconversion. We are ignoring the appreciable decline in the organs required to metabolize/excrete antivirals due to ACE-2 receptor presence.
Having a really hard time understanding why the government and medical establishment believe that not only myself but others are deserving of living in a society of cognitively declining individuals when they have the ability to do something about it.
I don't know about you but I feel like a worker at a Tyson plant watching the chickens run around with their damn head cutoff.
• • •
Missing some Tweet in this thread? You can try to
force a refresh
#LongCovid I have previously shared my thoughts on Paxlovid and after a Spaces last night, decided to put more effort into a Tweet. A 🧵
With the prescribing of Paxlovid, there has been a turning of a blind eye to HIV medicine and we are paying for it dearly.
The first mistake made was that there was an assumption made that each individual had the same log copies of virus in one mL of blood and cleared the virus at the same rate. Impossible.
With the overwhelming evidence of viral persistence, antivirals are the answer. However, folk here and yonder have turned a blind eye to HIV medicine and have created a massive mess. A 🧵
Before the first therapeutic was used, every single person was infected with the "wild type" variant of SARS-Cov-2. That no longer became the case when individuals were discharged from the hospital without full viral clearance.
The individual who was administered the therapeutic developed resistance and was then able to expel air to another individual who acquired resistance.
You fast track the development of a point of care viral load test that will accomplish the following: 1. Monitoring of Disease Progression. 2. Monitoring Treatment Efficacy. 3. Research into the length and level of suppression required to prevent forward transmission.
You develop a resistance panel that will allow for determining if an individual has been infected with the wild type virus or one for which resistance has developed either through abrupt treatment discontinuation or acquired from another individual.
We have an amazing opportunity to decide that we are going to learn from history in order to save that which our economy relies: our intellectual capital.
As I have mentioned multiple times, there are a number of compelling reasons why we should borrow from HIV medicine in the management of SARS-Cov-2 Associated Neurocognitive Decline. Let's discuss.
As an advocate for those battling a chronic SARS-Cov-2 infection, I find tweets such as this to be quite disgusting and believe they demonstrate a gross misunderstanding of pathophysiology.
Despite it being 2022, we have individuals attempting to determine the epidemiology of "Long Covid" through the use of surveys and not point of care viral load tests.
One can look at the three distinct groups of individuals living with HIV to arrive at the conclusion that the prevalence is much greater.
Join me tonight as we discuss the criticality of onboarding antivirals as quickly as possible so as to prevent further decompensation.
Can you imagine suffering from even more profound neurological impairment and can do nothing about it because your kidneys are burned up and your liver is impaired? That is exactly where millions are heading.