ECG Leads:
- Body act as a conductor of electricity; the recording electrodes in the arms, legs, and chest wall show the differences in voltage (potential) among electrodes
- Different views of the same event leads to different ECG patterns
ECG Limb Leads:
- 6 Limb leads (extremity leads) and 6 chest (precordial)
- 3 bipolar limb leads: I, II, III
- 3 augmented unipolar: aVR, aVL, and aVF
- 6 precordial leads: V1-V6
Einthoven’s Triangle:
Lead I: LA - RA
Lead II: LL - RA
Lead III: LL - LA
Remember: Lead I + Lead III = Lead II
Should double check if the R-lead in lead II = sun of the R-waves in lead I and lead III (1 + 3 = 2)
ECG Leads:
- Unipolar leads record the electrical voltage at one location relative to an electrode with close to 0 potential
- aVR + aVL + aVF = 0
ECG Limb Leads
- V1: 4th intercostal space (right)
- V2: 4th intercostal space (left)
- V3: Mid-way b/w V2-V4
- V4: Mid-clavicle line in 5th intercostal space
- V5: Anterior axillary line
- V6: Mid-axillary line
*Placement is key, but can be highly variable*
Let me know what you think and stay tuned for the next ECG thread!
• • •
Missing some Tweet in this thread? You can try to
force a refresh
General Principles:
- Positive deflection: wave of depolarization towards positive pole of that lead
- Negative deflection: wave of depolarization towards negative pole of that lead
- Biphasic deflection: wave of depolarization is perpendicular to a lead
Normal Sinus P-wave:
- Atrial depolarization that marks spontaneous depolarization of pacemakers cells in the right atrium
- Should be negative P-wave in aVR and upright in lead II
- Can communicate 'sinus rhythm with 1:1 AV conduction'
Conduction System:
- SA node (pacemaker cells) have specialized conduction tissue
- SA node is located in the RA near the opening of the SVC
- Stimulation occurs from right atrium to left atrium
- Simultaneous atrial contractions allows for blood filling into LV and RV
Conduction:
- AV Junction: Located at the base of the inter-atrial septum and extends into the inter-ventricular septum; the proximal portion is the AV node and distal portion is bundle of His
- Left & Right Bundle branches depolarize the myocardium via Purkinje fibers
1) Hypertensive Encephalopathy
- Cerebral edema is induced by markedly elevated blood pressures
- Dysregulation of auto-regulatory capabilities of the brain
- Characterized by headache, irritability, and altered mental status
- Treatment of choice: Nitroprusside/Labetalol
2) Reversible Posterior Leukoencephalopathy Syndrome (PRES)
- MRI may reveal white matter edema in the parito-occipital regions
Fenoldopam:
- Used mainly by anesthesiologists to control BP intra-operatively
- Selective peripheral dopamine-1 receptor agonist approved for the management of severe HTN
- Arterial vasodilator w/ relatively short half-life
- Contraindicated w/ glaucoma b/c can raise ICP
Nicardipine:
- Dihydropyridine calcium channel blocker that inhibits vascular smooth muscle contraction
- Little to no activity on the AV or sinus node
- Does not raise ICP and reduces cerebral ischemia
- Contraindicated w/ advanced HF, acute MI, and renal failure
Therapy:
- The presence of acute/rapidly progressive end-organ damage and not the absolute BP determines whether the situation is an emergency
- Goals should be based on mean arterial pressure (MAP) with close monitoring in ICU setting with arterial line
Therapy:
- In general, should reduce no more than 25% of MAP in the first 24 hours; after this time will be more gradual and allow auto-regulatory mechanisms to reset
- Exceptions: more aggressive BP reduction in aortic dissection, post-operative bleeding, and pulmonary edema