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1/ It’s funny about that pesky furin cleavage site that makes SARS-CoV-2 particularly infectious to humans. The possibility that it got there through natural recombination with other viruses exists, but it’s obviously very low—so low that no other SARS-like virus…
2/ (subgenus Sarbecovirus) actually has it, even though 200-300 of them have been identified. Meanwhile, the likelihood that the FCS got into the virus either through genetic engineering or serial passage in cell lines or animal models is quite high. Why? Because…
3/ Scientists at the Wuhan Inst Virology and their research partners in the U.S. and Singapore signaled their intention of doing just those experiments in the 2018 DEFUSE proposal to DARPA. Even though that particular proposal was not funded, we know for sure that…
4/ A large part of the research program at the WIV consisted of genetically engineering SARS-like viruses to see precisely which mutations/changes would cause them to be more infectious to humans. So the desire to insert a FCS into a virus combined with our knowledge…
5/ that this is exactly the kind of work WIV scientists were doing makes it more likely than not that they did it. The problem with those scientists and journalists whose primary motivation is to discredit the research origin hypothesis is that they will keep coming up…
6/ with the same answer every time no matter what the facts show, whereas those who are truly interested in knowing the origins of this killer pandemic often come up with different answers. The discussion of the furin cleavage site and how it got there is a leading…
7/ example of this distorted intellectual process and how even our best “experts” can engage in very biased thinking. #COVID19 #Wuhan
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