Not fake news— Jamain Stephens, a football player at California University of Pennsylvania has died at the age of 20 of #COVID19 complications.

➡️ This was why football was cancelled— in addition to myocarditis found in 15% of Big 10 players (not 30%)
profootballtalk.nbcsports.com/2020/09/09/col…
2) Earlier study: “cardiac M.R.I.s, an expensive and sparingly used tool, revealed an alarmingly high rate of myocarditis — heart inflammation that can lead to cardiac arrest with exertion — among college athletes who had recovered from the coronavirus...”
3) ...”The survey found myocarditis in close to 15% of athletes who had the virus, almost all of whom experienced mild or no symptoms, perhaps shedding more light on the uncertainties about the short- & long-term effects the virus may have on athletes.” nytimes.com/2020/08/23/spo…
4) Another study: “And [myocarditis] is not age-specific: In The Lancet, doctors recently reported on an 11-year-old child with multisystem inflammatory syndrome (MIS-C)—a rare illness—who died of myocarditis and heart failure.”
5) ...”At autopsy, pathologists were able to identify coronavirus particles present in child’s cardiac tissue, helping to explain the virus’ direct involvement in her death.”
6) “In fact, researchers are reporting the presence of viral protein in the actual heart muscle, of six deceased patients.” scientificamerican.com/article/covid-…
7) “Of note is the fact that these patients were documented to have died of lung failure, having had neither clinical signs of heart involvement, nor a prior history of cardiac disease.”
8) “Ossama Samuel, associate chief of cardiology at Mount Sinai Beth Israel in New York, told me about a cluster of younger adults developing myocarditis, some of them a month or so after they had recovered from COVID-19.”
9) “One patient, who developed myocarditis four weeks after believing he had recovered from the virus, responded to a course of steroid treatment only to develop a recurrence in the form of pericarditis (an inflammation of the sac surrounding the heart).”
10) “A second patient, in her 40s, now has reduced heart function from myocarditis, and a third—an athletic man in his 40s—is experiencing recurring and dangerous ventricular heart rhythms, necessitating that he wear a LifeVest defibrillator for protection.”
11) “His MRI also demonstrates fibrosis and scarring of his heart muscle, which may be permanent, and he may ultimately require placement of a permanent defibrillator.”
12) Unclear now. There’s been a retraction by some sources. It’s currently unconfirmed cause of death.

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More from @DrEricDing

11 Sep
How long do you need to self isolate if you test #COVID19 positive? It depend:

📌symptomatic: 10 days from symptom onset & extra 3 days w/o symptoms.

📌asymptomatic: 10 days from date of PCR (+)test

➡️ And if we had cheap antigen home testing, we’d have even faster isolation. Image
2) How do we know this? From this study. “SARS-CoV-2 appears to be most contagious around the time of symptom onset and infectivity rapidly decreases thereafter to near-zero after about 10 days in mild-moderately ill patients” academic.oup.com/cid/advance-ar… Image
3) However authors point out, “and 15 days in severely-critically ill and immunocompromised patients”.
Read 5 tweets
10 Sep
COVID & BRAIN damage—half of patients report neurological symptoms, including headaches, confusion and delirium. A new study offers the first clear evidence that, in some people, the coronavirus invades brain cells. 🧵#COVID19 nytimes.com/2020/09/09/hea…
2) #SARSCoV2 seems to suck up all of the oxygen nearby, starving neighboring cells to death. It’s unclear how virus gets to the brain or how often it sets off this trail of destruction.
“If the brain does become infected, it could have a lethal consequence” says @VirusesImmunity.
3) “In the new study, Dr. Iwasaki and her colleagues documented brain infection in three ways: in brain tissue from a person who died of Covid-19, in a mouse model and in organoids — clusters of brain cells in a lab dish meant to mimic the brain’s three-dimensional structure.”
Read 7 tweets
9 Sep
I knew in January, but nobody wanted to listen. Our leaders have blood on their hands. We have to go forward from here. #COVID19
2) "I wanted to always play it down. I still like playing it down because I don't want to create a panic."

"plenty of young people" are vulnerable -- this is also completely opposite what Trump and Scott Atlas keeps saying.
3) Trump also said all this in Feb / March very early.
Read 6 tweets
8 Sep
AEROSOL TRANSMISSION—we need to keep hammering this. Key takeaways by @jljcolorado to avoid places:

📌if it's crowded

📌if cannot keep distance

📌if it's a long time

📌if low ventilation

📌if not wearing masks

📌if people singing, yelling or also breathing hard. #COVID19
2) Link to NPR audio. npr.org/2020/09/07/910…
3) Also, @linseymarr points out that the key danger of aerosols are that they can be “Wafted by air currents and travel around the room and be inhaled by other people” — and this 6 feet distance indoors is not enough protection. And stay afloat 20 min - 4 hours, says @kprather88.
Read 6 tweets
7 Sep
I remember West Nile Virus hit the US, especially in NYC. I was a summer trainee working at @nycHealthy’s mosquito and bird sentinel surveillance program, when @DrTomFrieden was at the helm of NYCDOHMH. Chased down lots of mosquito 🦟 larvae in Queens, Brooklyn and Staten Island.
2) It was the best summer of my life. NYC health Dept gave us HRTP interns working in the field unmarked blue Ford Crown Victoria with NYC city officials plates—identical to many unmarked police cars. Bonus: We never got any parking tickets.
3) I was studying public health at @JohnsHopkins at the time. @JohnsHopkinsPHS sponsored part of my summer cost of living. We learned so much about field epidemiology—and how it is a shoe leather science, not some abstract computer modeling of infectious disease. Opened my eyes.
Read 4 tweets
6 Sep
📍HUGE—a new study in New Orleans, Louisiana estimates there was a local infection fatality ratio (IFR) of 1.63%—much higher than thought for #COVID19. the IFR is among all infected, unlike CFR mortality in just those diagnosed. Antibody in May only 6.9%.🧵wwwnc.cdc.gov/eid/article/26…
2) seroprevalence was highest (9.8%) in Blacks, followed by multiracial (7.1%), Asians (5.5%), & Whites (4.5%). Hispanics had 5.3% seroprevalence.

➡️ The IFR was statistically similar for Whites (1.58%), Blacks (1.72%), and multiracials (1.40%) but lower for Asians (0.61%).
3) “No COVID-19–related data on Hispanic persons were collected by the Louisiana Department of Public Health during the study period.”

➡️ oh cmon @LADeptHealth!
Read 8 tweets

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