1/4 Tell me ? How much anecdotal evidence do we all need to get Statins off the market ? Here goes : FB Forum: " I stopped my statins a couple of weeks ago when I was having trouble walking. My muscles got better almost immediately. One leg is pretty much back to normal
2/4 but the other one still hurts so much. When I put any weight on it, it feels like the muscle is torn. So, what kind of testing can I have done to see what causes this ?
Reply: I have just found this thread how interesting! I too had terrible trouble walking,
3/4 so much leg pain that I had to keep stopping. I thought it must be my trainers, so bought new ones along with expensive insoles. I happened to have an immunologists appointment and he ordered, amongst other things, an MRI and found that I have a partially torn hamstring,
4/4 was very worrying.
MY MUSCLE HAS COME AWAY FROM THE BONE right at the top of my leg. Was told to stop taking Statins immediately. Now be seeing a cardio to get something different, though would prefer nothing and take my chances. Feel much better now, 8 weeks & no statins.
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1/5 Love it love it ! - Statins Forum today - Makes my heart sing: - "Hi there! I’d like to share my story. I have known of my high cholesterol since I was 30 (now just shy of 49). High being over 425 regularly. I tried every statin there is only to have horrible side effects
2/5 eventually resulting in Rhabdomylosis. My cardiologist has been trying for over a year to put me on Repatha. I kept declining. Besides seeing the numbers on a lab report, I felt like a healthy female for my age. It didn’t feel like the right choice.
3/5 2 weeks ago my doctor suggested the CAC scan after seeing my cholesterol numbers (275 LDL/80 HDL. 386 total) He told me that if it comes back as zero he will drop the statin/injection discussion.
1/4 What a great question (thanks @TMackeown ). "Given that most of the deaths/serious effects of Covid-19 are in the elderly, who are also the most likely to have been on statins for some time, do you think there is a connection between the lowering of LDL/ApoB100 and the
2/4 susceptibility to infection - not just from MRSA ?"
If you google Statins/Covid you will find that statins are most efficacious in every way 😂 (We would expect that wouldn't we ?). However if we turn to independent researchers we find the truth.
3/4 See the link below
bmj.com/content/368/bm…
It gets worse because Statins act through the reversible block of (HMG-CoAR), thereby reducing cholesterol synthesis, 7-dehydrocholesterol and vitamin D production.
1/4 Gelam honey extract has been found to protect pancreatic hamster cells from hyperglycaemic conditions. Significantly, this honey decreased ROS production, glucose-induced lipid peroxidation, increased insulin content and the cell viability under hyperglycaemic conditions.
2/4 Significantly, honey has been shown to enhance endothelial function through quenching lipophilic cumoxyl and cumoperoxyl radicals, suppressing cell damage, inhibiting cell membrane oxidation and decreasing reactive oxygen species (ROS) generation and GSH
3/4 recovery in endothelial cells. Honey exhibits a broad-spectrum of antibacterial activity against both Gram-positive bacteria and Gram-negative bacteria. Since Infectious agents have been linked to atherosclerotic disease,
1/6 In 1984 Merck discovered a way to accelerate mechanism based cell alterations with a Statin by giving rats a second cholesterol lowering drug called a “bile resin”. The combination lowered cholesterol better than Lovastatin alone (Grrrreat !).
2/6 It elevated Reductase more than Lovastatin alone, and it made more abnormal-appearing ER than Lovastatin. In case you are wondering ER = endoplasmic reticulum. This drug combination is what reportedly killed the majority of Baycol users.
3/6 The liver has a very high demand for cholesterol to make bile that is made in the liver and then stored in the gall bladder for digestion of fat. The bile is then recycled back to the liver from the intestines for reuse.
1/4 Oh how we have been played ! Brown and Goldstein studied the defective genetics of the LDL receptor in people with FH who have extremely high cholesterol. It was for these patients that the FDA gave Dallas an IND (Investigational New Drug) permission to test Lovastatin,
2/4 Brown and Goldstein studied the defective genetics of the LDL receptor in people with FH who have extremely high cholesterol. It was for these patients that the FDA gave Dallas an IND (Investigational New Drug) permission to test Lovastatin, and not the general public.
3/ 4 A compilation of FH is that painful deposits of oxidised cholesterol would improve this condition. Notably, two of these six FH patients with cholesterol levels over 500 didn’t even have CVD !!
1/4 What infuriates me about this paper is there is NO overwhelming evidence to support lowering LDL-C to reduce cardiovascular morbidity and mortality. What prompted this paper is the fact that the Pharmaceutical industry had new drugs to sell i.e. PCSK9 inhibitors.
2/4 Far more profitable than Statins, and the reason why they now confess that Statins are toxic, to promote their new and more profitable PCSK9’s. The problem is the new PCSK9’s are no better than Statins. The pharma Industry has known for the past 40+ years the damage
3/4 that Statins do, but have been in denial. Here they are stupid enough to make the admission about Statin Toxicity and its mechanisms. If you look at the Fourier trial for PCSK9’s you will see that more people died in the treatment arm of the trial than in the placebo group.