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Dave Feldman Profile picture
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5 Mar, 4 tweets, 2 min read
1/ The blood test (assay) I'm especially interested in is called "OxPL-ApoB" and it's ultimately offered through @BostonHeartDX

If looking to get it:

1- You may want to first check with your doctor to see if they can order it directly (sometimes insurance will cover)...
2/
2- There may be online services that you can order it with, but to be honest, I haven't checked which do yet

3- We offer it and other Boston Heart tests through OwnYourLabs, but full disclosure, this program is a beta right now. You can sign up here: ownyourlabs.com/boston-heart-b…
3/ If you do get the test, please consider sharing back the data to us at CholesterolCode.com. I'm especially interested to see if this data further confirms my hypothesis that the majority of hyper-responders will generally have relatively low OxPL in spite of very high ApoB
4/ You can always @Lpa_Doc directly about this assay and the research behind it.

Lastly, you can also check out my own recent data on it here: cholesterolcode.com/preliminary-da…

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More from @DaveKeto

Dave Feldman Profile picture
19 Feb
1/ Interesting thread by @mak_sysepi regarding a recent study on HDL-C with a massive sampling -- 15.8 million! academic.oup.com/ije/advance-ar…

Note this tracks my favorite metric, All Cause Mortality.

(Hat tip @DrNadolsky)
2/ If you already follow my work, you've known for some time that I value looking to HDL-C alongside triglycerides. In particular, I like to compare the "low carb lipid triad" to atherogenic dyslipidemia. HDL-C is a very important part of each...
3/ The paper we're working on for the Lipid Energy Model focuses on studies that demonstrate HDL-C going up and down as it relates to metabolism, both in a health and diseased state.

It's worth noting a very common association of high HDL is alcoholism.
Read 5 tweets
Dave Feldman Profile picture
9 Feb
A paper combining Triglycerides/HDL-C and All Cause Mortality with dialysis patients --

This deserves a #ReadingThread... (at least between my calls and meetings today)...
"In fact, traditional CV [cardiovascular] risk factors, such as hypercholesterolemia and obesity, have not been found to be reliable predictors of mortality risk in these patients..."
"... as previous studies have shown these factors are paradoxically associated with better survival in the hemodialysis population."
Read 15 tweets
Dave Feldman Profile picture
7 Feb
1/ There really has been a sea change I've noticed lately with regard to both the #LipidTriad and the #LMHR phenotype.

I'm not going to call anyone out, but I will say there are many who I had several spirited discussions in the past few years...
2/ ...who considered high LDL = "high" risk, full stop...

... but are now expressing #LMHRs may actually be at "low" risk of cardiovascular disease (in spite of having LDL in the 95% of the pop), just that they "could be better" if they have these LDL levels lower than they are.
3/ It's worth emphasizing their position hasn't changed with regard to higher LDL = *more risk* of CVD. Thus, it would still be better for #LMHRs to have lower LDL, all else being equal.

But the magnitude of difference in the assumed risk has changed for many.
Read 6 tweets
Dave Feldman Profile picture
6 Feb
Ah — looks like I have a podcast for my walk today...

... I may be adding comments to this thread as I listen...

@FoodLiesOrg @TuckerGoodrich @drcateshanahan
Had to run minute 23 Tucker brings up that there are some amount of these PUFAs that are necessary and obligate to the body, but the quantity is typically much higher in the modern diet due to heavy use of seed oils...
... I bring this up because I do think @TuckerGoodrich is often portrayed by others as though he’s against any amount of PUFA in the diet. As always, I’m #ProNuance. ;)
Read 5 tweets
Dave Feldman Profile picture
2 Feb
Really enjoying this latest podcast on @PeterAttiaMD’s #Drive so far.

Now up to the part where Dr Yassine is discussing GLUT1 regulation with the brain... (about 22m in)...
Hmmm...

“... the brain is not efficient at using fat as a source of ATP.”
“... nevertheless when the brain goes through crises and cannot utilize glucose, we have a mechanism to extract ATP from fat. But the result is a price tag, the result is oxidative stress.”

Please visit this around minute 24 to get the full context.
Read 5 tweets
Dave Feldman Profile picture
23 Jan
1/ So I did my 3D body scan this morning (and no, not posting pictures or sharing absolute numbers).

This will have been 5 days since isocaloric carb swapping in low-ish carb for keto.

Waist +0.9"
Stomach +1.2"
Hip +0.4"

Wow... in 5 days? This was pretty unexpected...
2/ Just a reminder-- this is the most controlled experiment between phases I've had to date. Replicated sleep and exercise timing and patterns, even water quantity and consumption window.

Given this shorter time span for the phase, I wasn't expecting much difference to emerge...
3/ Although, to be sure, both my wife and I noticed I was putting on more weight in the midsection even before the scan. I'm holding at around 199.6 for two days in a row, so while I gained around 2.5lbs initially, it seems to have leveled off.
Read 8 tweets

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