I've heard this objection over and over again in response to my argument that lockdowns don't pass a cost-benefit test and it's such an obvious non-sequitur that it just baffles me that people keep raising it. marginalrevolution.com/marginalrevolu…
The idea is that, if lockdowns don't make a big difference because voluntary behavior changes have a similar effect on transmission, they also don't make a big difference on people's well-being because people are going to do the same thing no matter what.
But that's a non-sequitur because the fact that state-enforced lockdowns don't have a large effect on transmission beyond what voluntary behavior changes would achieve in the absence of government interventions doesn't mean that people behave in the same way under a lockdown.
For instance, under current lockdown rules in France, people can't have a drink at the terrace of a café. If this were allowed, it would have no meaningful effect on transmission, but the fact that it's not possible has a meaningful effect on people's well-being. This isn't hard.
The fact that so many people fail to grasp this obvious point has been a constant source of sorrow for me, but apparently they really don't since every time I make this argument about the role of voluntary behavior changes people raise that objection 🤷♂️
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I'm very happy to announce that my paper has been accepted for publication by the European Journal of Epidemiology.
Folks, before you take this seriously (although in a sense it’s very serious), I recommend that you actually read the abstract 😄
The worst part is that I could totally write that paper. The modeling itself would only take 30 minutes, but the really fun part would be the write-up.
Sauf qu'il n'y a aucune raison de penser que E(années de vie restantes | age = x & sexe = y & victime du COVID-19) est égal à E(années de vie restantes | age = x & sexe = y) et qu'il est même parfaitement évident que ce n'est pas le cas 🤷♂️
À n'importe quel âge, l'immense majorité des gens qui sont infectés par SARS-CoV-2 survivent, donc ceux qui en meurent sont vraisemblablement plus fragiles que la moyenne des gens du même age et du même sexe et auraient sans doute vécu moins longtemps.
Ce tableau est donc trompeur dans le contexte du débat sur le nombre d'années de vie perdues par les victimes du COVID-19. Bref, avant de faire le mariole et de donner des leçons de démographie aux autres, mieux vaut réfléchir un peu et s'assurer qu'on ne dit pas de connerie...
Not only did your study show no such thing, but it rests on demonstrably false assumptions, so it's really extraordinary that you continue to peddle those results. Here is a thread in which I explain why this study is worthless and should never have been used to guide policy 🧵
First, the model used in that study assumes that B.1.1.7, the UK variant, is 59% more transmissible than the historical lineage. This estimate is based from Gaymard et al. (2021), which obtained it by fitting a simple exponential growth model to only 2 data points from January.
As I explained at length in this post, even if we just use those 2 data points from January, this estimate is highly sensitive to the assumptions we make about the distribution of the generation time and there is a lot of uncertainty about that. cspicenter.org/blog/waronscie…
Des nouvelles de B.1.1.7, le « variant anglais » qui était censé provoquer un tsunami en raison de sa transmissibilité accrue, à partir des dernières données de Santé publique France 😂
Même chose mais quand on fait la comparaison uniquement avec la souche historique plutôt qu’avec tous les variants non-B.1.1.7. En gros, la première méthode est sans doute un peu biaisée, tandis que celle-ci ne l’est pas mais l’erreur de mesure est plus grande.
Je rappelle que les génies de l’Inserm et de l’Institut Pasteur continuent de faire l’hypothèse qu’il est 50% à 70% plus transmissible dans les modèles qu’ils utilisent pour faire les projections qu’ils présentent au gouvernement 👌
Top 3 things I've been wrong about during the pandemic:
1) That lockdowns were a good policy 2) Relatedly, that most of the uncertainty early on was about the IFR, as opposed to how to model the spread 3) That a vaccine wouldn't be approved until mid-2021 at the earliest
I should actually have put number 2 in first position, because it's the reason I was wrong on lockdowns. I thought there was a chance the IFR was significantly lower than 1% because of Japan, which I assumed was swimming in virus, yet still had very few COVID-19 deaths by March.
That's because I assumed a SIR model with constant contact rate was a good representation of transmission in the absence of strong government interventions, but in fact it's not and the explanation for Japan's low COVID-19 mortality was just that the virus had not spread much.
61% of people tested negative for antibodies, but instead of concluding that most people in that sample who claimed to suffer from "long COVID" are just hypochondriacs who were never even infected, the conclusion is that "long COVID" patients have a weaker antibody response 😂
But the problem is not that medical researchers make that ridiculous inference with a straight face, not at all, it's that a journal asked them to exclude people who tested negative, which apparently is racist because most of them were minorities 🙃
It's amazing, just have a look at how many doctors and scientists liked/retweeted this garbage, medical research is rotten to the core. The mere fact that a prominent researcher can say that with a straight face and apparently no fear of damaging her reputation speaks volumes.