@YoureAllDunces

I am trying to find out why the long-known data of the spike protein being the engine of COVID-19 was not disclosed prior to EUA of vaccines. This is my second attempt to address this since stories keep changing. I'm going back to original articles/videos...
...and creating a kind of index document, complete with links, identifying content that could cause people to be misinformed and lead to rabbit hole research, etc.
In the course of conducting research, the following related information was found:

In this video, we find out what makes a SELF-AMPLIFYING vaccine as opposed to a TRANSMISSIBLE vaccine, a frequent question.

This is one of the most informative of the videos to date and has its own index as it is broken into chapters with separate titles:

Doctor Crotty informs us as follows:

1. It was always known that not all of the vaccine would stay inside the arm muscle. While the Doctor does not specify where the vaccine developers wanted this part of the vaccine to go, he does say he wanted the lipid nanoparticle...
packages to get into some immune cells. "Rare" immune cells are the ones that the developers wanted to receive the mRNA packages. I infer from this, because of its location under the arm, the place they WANT the vaccine to go is the lymph system.
It was always known that not all the spike proteins made through the mRNA vaccines would be expressed on the surface of the cells. Dr. Crotty does not elaborate about where the spike proteins that do not express on cell surfaces exactly go.
Amounts of wandering spike proteins are not provided, nor are there estimates of how much vaccine does not remain in the arm. I can't see how any of the vaccine developers could know that.
In all of the articles and videos I have examined to date, not one has disclosed:
1. The description of the claim that the vaccine developers for all the vaccine providers engineered mRNA code that would prevent the binding of the vaccine spike protein to cell ACE2 receptor.
2. That the spike protein is the engine of the collection of symptoms, or the disease, that is called COVID-19.

This means that the claim to the existence of mRNA code that creates a safety feature to prevent spike protein from binding to the ACE2 receptors post dates the...
...release of the vaccines to the public under the Emergency Use Authorization.

The fact that the spike protein is the engine of COVID-19 was also withheld from pubic disclosure in all articles and videos reviewed thus far.

Investigation continuing.

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More from @Farrier105

19 Oct
@YoureAllDunces Something else has to be broached as we confront the addition of this claimed safety feature for the vaccine spike protein that is supposed to prevent binding to ACE2 receptors.
The creation of the safety feature would further change the RNA genome of the vaccine's spike protein from the genome of the virus' spike protein. I don't think this fact is remotely debatable, but desperation can result in just about anything.
These differences in the appearances, and the genetic structures between the spike protein of the vaccine and the spike protein of the virus inspire questions about the nature of what constitutes a VARIANT of a virus.
Read 7 tweets
23 Sep
@LL4DJT The Spike Protein is the real problem with SARS-COV-2 and COVID-19, the disease caused by SARS-COV-2.

studyfinds.org/covid-alters-g…
@LL4DJT The vaccines cause your cells to make the Spike Protein, which is the dangerous aspect of the virus, which is just a type of CARRIER of the spike protein and basically does little to impact human health. An analogy of the relationship of the spike protein to the virus is here:
Read 12 tweets
17 Sep
@YoureAllDunces Comparison of "Spreading Speed" between SARS-COV-1 (Original SARS 2003-2004) and SARS-COV-2:

frontiersin.org/articles/10.33…
"Compared to SARS-CoV and MERS-CoV, SARS-CoV-2 is causing an even more severe pandemic due to its spreading speed and the population affected. Deep studies comparing the different disease-causing coronaviruses will shed light on the fundamental mechanisms of coronavirus
related diseases.

"Even though S proteins of SARS-CoV and SARS-CoV-2 share very similar structures, the binding affinities of S protein and ACE2 of SARS-CoV-2 are much higher than SARS-CoV (Koehl, 2018). This might be the key reason for SARS-CoV-2's faster-spreading speed,
Read 12 tweets
17 Sep
@MichaelWeddle15 @with_integrity @stranahan Why does Trump enact Neocon policies like the coup in Venezuela and putting troops in Syria in the first place? It needs to be explained why CrowdStrike told the story they told to WAPO on June 13, 2016--indirectly--that Fancy Bear did NOT steal the emails.
@MichaelWeddle15 @with_integrity @stranahan That makes them look stupid when Guccifer 2.0 shows up and claims he stole the emails. When you look at Shawn Henry's declassified testimony to Congress, it APPPEARS that CrowdStrike thought Cozy Bear could have stolen the emails. CrowdStrike admitted they did not witness...
@MichaelWeddle15 @with_integrity @stranahan ...most of the documents being exfiltrated, and cannot prove the Russians did it, only that they STAGED documents for exfiltration.

That is NOT setting the table for the "Russians stole the emails and gave them to Wikileaks" scenario. So, why were at the DNC?
Read 21 tweets
15 Sep
@RWMaloneMD Doctor: I have been researching the S1 protein of the original SARS virus from early 2000s to see if it performed similar actions against human health as does the SARS-COV-2 S1 protein. The SARS spike was just as dangerous as it turns out.
Read 13 tweets

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