1/ A #tweetorial about simulation in NCC
Today @namorris opened his remarks on Sim in NCC @ #NCS2021 w/ a simple question about the correct first line treatment for SE? Everyone got it.
Then he posed a tougher question.
How often does that happen?
No one voted "always"
2/
Even the most groundbreaking research won’t benefit our patients if we aren’t delivering it correctly.
I so highly encourage you to check out Nick’s talk on-demand if you have access to #NCS2021.
It is 🚨critical🚨 that we teach more effectively!
3/ Convinced?
Some practical, take-aways from this talk about finding right Simulation Solution.
4/
Simulation is an amazing educational tool. But it can be used in other ways, too!
Simulation can be used to:
⭐️to evaluate trainees
⚡️ as a research tool to understand learners and systems
✨to understanding systems integration & improve care delivery.
5/ Defining the simulation’s goal is defining the “Why”.
And if you are interested in approaching this as research, it is crucial to approach it with a hypothesis and have a methodology in mind from the start. Another amazing pearl from @namorris.
6/ From the “why” you also will need to define the purpose of the sim -- the “what”:
“what is the learning object” “What are we trying to improve with this?”
Lots of options:
7/ What you want to accomplish defines “how” you should accomplish this. Simulation can be done through tissue models, task trainers, manikins, apps, virtual reality, standardized patient, and more
8/ It seems straight forward but the fidelity is only “high” or “low” in reference to your goal --
👉Teaching LPs on a task trainer is “high fidelity”
👉Teaching communication and leadership training with a task trainer…
9/ So we’ve got the “Why do this” & “What is the purpose” & “How are we going to do this”
But u also need “How are we going to run the case?”
A tradition model is to run the case & debrief @ the end
But, as @Capt_Ammonia loves, you can also rapid cycle & debrief as you go!
10/ Putting it all together. Each simulation:
👉has an overall goal: the “WHY” do this
👉has a purpose & specific learning objectives: the “WHAT” is it we are trying to accomplish
👉has a modality and method: the ”HOW” we are going to run the case
12/ But in the context of stroke, seizure, brain death, etc if you want to train learners to initiate a diagnostic workup, interpret radiology, correctly dose medications, screen for confounders, communicate findings… The manikin’s exam matters less.
13/ Work Sahar Zafar & I did @ MGH Learning Lab demonstrating that residents gained the same amount of confidence and knowledge in simulations with SPs as they did with a manikin. So glad @DanHarrisonMD and others are carrying this work forward @harvardneuromds!
14/ 2⃣nd challenge: Does a learner’s performance in the sim lab correlate with how they are going to perform in real life?
This study (@namorris) suggests so! Sim-Based Assessment of Graduate Neurology Trainees' Performance Managing Acute Ischemic Stroke pubmed.ncbi.nlm.nih.gov/34706974/
15/ Finally – how are we going to prove that the work we do in a simulation lab translates to what really matters: improving patient outcomes.
It can be done but it's a challenge and requires a lot of accounting for confounders... the opposite of:
1/🧵
In the early days of fellowship, I remember checking our SAH patients’ transcranial dopplers (TCD), scanning the Vmeans & if they were ~<70 cm/sec throughout thinking:
“Great. Perfect. TCDs globally low. Nothing to worry about here!”
🚨Note. This is not a #tweetorial about if large vessel vasospasm is the cause of DCI or just an epiphenomenon OR if treating vasospasm is the way to improve functional outcomes …That is important!... but that is not this tweetorial. pubmed.ncbi.nlm.nih.gov/21285966/
3/ Given #TCDs is a pretty large topic, this @medtweetorial will be told in 3 parts:
Part 1⃣:
⭐️Basic principles of TCDs
⭐️Use of TCDs to detect Vasospasm
Part 2⃣: The Pulsatility Index - why it matters
Part 3⃣: The Utility of TCDs as an ancillary test in BDT
1/ 1st week of NeuroICU fellowship. A #tweetorial summary:
1⃣ Pt in DI. Give anti-diuretic hormone (ADH), call it “pit drip”
2⃣Pt in distributive shock. Give ADH, call it “vaso”
3⃣Pt on ASA needs EVD. Give ADH (sort of), call it “DDAVP”
4⃣ Fellow postcall & confused, give….
2/ Just kidding… everyone knows the drug for that is
3/ All the names and purposes of ADH had me feeling ⬇️
So – a review of all things ADH including:
✅It’s various aliases
✅Receptors and function
✅Clinical utility in NeuroICU (+general ICUs)
A 70 yo W with history of HTN presented with significant IVH from a ruptured AVM.
Admission EKG showed this:
A #brugada pattern. She had no personal or family history of syncope / sudden death. And on admission (time of this EKG) she was not febrile. About 12 hours later we repeated the EKG:
Trops normal and ECHO later in the day demonstrated a normal EF and grade 1 DD, but no wall motion abnormality. No apical ballooning. There was mildly increase LV wall thickness.
2/ With the TREAT-CAD trial, lots of talk about dissection treatment. Whether your team anti-platelets or team anticoagulation (🙋🏻 Must. Give. Heparin (@MGHNeurology) 4 ever. I know you feel this, @namorris!) consideration about the location of dissection is possibly important.
3/ Also, regardless of your team… TREAT-CAD was not able to demonstrate non-inferiority of ASA, just saying.
Aspirin versus anticoagulation in cervical artery dissection (TREAT-CAD): an open-label, randomised, non-inferiorit… pubmed.ncbi.nlm.nih.gov/33765420/